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Nature Communications, ISSN 2041-1723, 12/2017, Volume 8, Issue 1, pp. 2247 - 15
Journal Article
British Journal of Pharmacology, ISSN 0007-1188, 06/2017, Volume 174, Issue 12, pp. 1647 - 1669
Journal Article
Advances in experimental medicine and biology, ISSN 0065-2598, 2019, Volume 1127, p. 85
Arachidonic acid (AA) is a polyunsaturated fatty acid that participates in the inflammatory response mainly through bioactive-lipids formation in macrophages... 
Arachidonic Acid - metabolism | Inflammation - metabolism | Macrophages - metabolism | Phosphorylation | Reactive Oxygen Species - metabolism | NADPH Oxidase 2 - metabolism | Reactive Nitrogen Species - metabolism | Respiratory Burst | Humans | Superoxides - metabolism | Phosphoproteins - metabolism
Journal Article
Nature Cell Biology, ISSN 1465-7392, 03/2018, Volume 20, Issue 3, pp. 307 - 319
Reactive oxygen species (ROS) contribute to tissue damage and remodelling mediated by the inflammatory response after injury. Here we show that ROS, which... 
STEM-CELLS | PHAGOCYTOSIS | ACTIVATION | SUPEROXIDE-PRODUCTION | ROS | KINASE | PTEN | NEURONS | MECHANISMS | OUTGROWTH | CELL BIOLOGY | Ganglia, Spinal - injuries | Sciatic Nerve - injuries | Axons - enzymology | Reactive Oxygen Species - metabolism | Exosomes - pathology | Sciatic Nerve - physiopathology | Spinal Cord Injuries - genetics | Spinal Cord Injuries - pathology | Endocytosis | CX3C Chemokine Receptor 1 - metabolism | Ganglia, Spinal - enzymology | Peripheral Nerve Injuries - physiopathology | Spinal Cord Injuries - enzymology | Proto-Oncogene Proteins c-akt - metabolism | Phosphatidylinositol 3-Kinase - metabolism | Disease Models, Animal | Dyneins - metabolism | Cell Line | NADPH Oxidase 2 - deficiency | Macrophages - pathology | Signal Transduction | Sciatic Nerve - pathology | Mice, Inbred C57BL | Peripheral Nerve Injuries - pathology | PTEN Phosphohydrolase - metabolism | Nuclear Proteins - metabolism | Endosomes - pathology | Nerve Regeneration | Macrophages - enzymology | Mice, Knockout | Animals | Peripheral Nerve Injuries - enzymology | NADPH Oxidase 2 - metabolism | Axons - pathology | Ganglia, Spinal - pathology | Exosomes - enzymology | Endosomes - enzymology | Spinal Cord Injuries - physiopathology | NADPH Oxidase 2 - genetics | Nerve Degeneration | Peripheral Nerve Injuries - genetics | Sciatic Nerve - enzymology | Axons | Regeneration | Reactive oxygen species | NADP (Coenzyme) | Nervous system | Cellular signal transduction | Research | Gene expression | Oxidase | Recovery of function | Spinal injuries | CX3CR1 protein | Inflammatory response | AKT protein | Exosomes | Macrophages | Inactivation | NAD(P)H oxidase | Signal transduction | Dorsal root ganglia | Neurodegeneration | Dieback | CYBB protein | Degeneration | Cell body | Oxygen | Deactivation | Inflammation | Ganglia | 1-Phosphatidylinositol 3-kinase | Signaling | Dynein | PTEN protein | Sciatic nerve | Endosomes
Journal Article
Journal Article
Journal Article
Journal Article
Journal Article
Free Radical Biology and Medicine, ISSN 0891-5849, 10/2016, Volume 99, pp. 167 - 178
Excessive levels of reactive oxygen species (ROS) and increased expression of NADPH oxidases (Nox) have been proposed to contribute to pulmonary artery... 
Epigenetics | Histone deacetylases | NADPH oxidase | Pulmonary artery hypertension | OXYGEN SPECIES PRODUCTION | ACTIVATION | BIOCHEMISTRY & MOLECULAR BIOLOGY | HDAC INHIBITION | CELL-PROLIFERATION | 5 NOX5 | NAD(P)H OXIDASE | THERAPEUTIC TARGET | DISEASE | ENDOCRINOLOGY & METABOLISM | EXPRESSION | RIGHT-VENTRICULAR HYPERTROPHY | Transcription, Genetic - drug effects | Antihypertensive Agents - pharmacology | Endothelial Cells | Luciferases - metabolism | Reactive Oxygen Species - metabolism | Epigenesis, Genetic | Humans | Monocrotaline | Male | RNA Polymerase II - metabolism | Hypertension, Pulmonary - chemically induced | Luciferases - genetics | p300-CBP Transcription Factors - genetics | Isoenzymes - metabolism | HEK293 Cells | Fibroblasts | Hypertension, Pulmonary - drug therapy | Genes, Reporter | Cell Line | Promoter Regions, Genetic | Histone Deacetylases - genetics | Signal Transduction | Isoenzymes - genetics | Rats | Histone Deacetylases - metabolism | Hypertension, Pulmonary - genetics | Rats, Sprague-Dawley | p300-CBP Transcription Factors - metabolism | Reactive Oxygen Species - antagonists & inhibitors | Animals | NADPH Oxidase 2 - metabolism | Histone Deacetylase Inhibitors - pharmacology | RNA Polymerase II - genetics | Mice | NADPH Oxidase 2 - antagonists & inhibitors | NADPH Oxidase 2 - genetics | Hypertension, Pulmonary - pathology | Isoenzymes - antagonists & inhibitors | Oxidases | Epigenetic inheritance | Chromatin | RNA | Analysis | Genes | Histones | Superoxide | DNA binding proteins | Genetic transcription | Pulmonary hypertension
Journal Article