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The Plant cell, ISSN 1532-298X, 2012, Volume 24, Issue 1, pp. 336 - 352
..., and identified the peroxisomal enzyme glycolate oxidase (GOX) as an essential component of nonhost resistance. GOX-silenced N... 
Oxidases | Statistical significance | Pathogens | Disease resistance | Leaves | Inoculation | Plant growth | Genes | Plants | Plant cells | NADPH OXIDASE | DEFENSE RESPONSES | BIOCHEMISTRY & MOLECULAR BIOLOGY | OXIDATIVE BURST | BACTERIAL VIRULENCE | PLANT-DISEASE RESISTANCE | TOBACCO MOSAIC-VIRUS | CELL-DEATH | PLANT SCIENCES | CELL BIOLOGY | HYDROGEN-PEROXIDE | PHYTOPHTHORA-INFESTANS | HYPERSENSITIVE RESPONSE | Arabidopsis - enzymology | Reactive Oxygen Species - metabolism | Plant Diseases - immunology | Arabidopsis - immunology | Molecular Sequence Data | Plant Diseases - microbiology | Pseudomonas syringae - pathogenicity | Tobacco - immunology | Alcohol Oxidoreductases - genetics | Plant Diseases - genetics | Plant Immunity - genetics | Plants, Genetically Modified - genetics | Plants, Genetically Modified - immunology | Plants, Genetically Modified - enzymology | Gene Expression Regulation, Plant - genetics | Alcohol Oxidoreductases - metabolism | Signal Transduction - genetics | Plant Immunity - physiology | Arabidopsis - genetics | Arabidopsis - microbiology | Plants, Genetically Modified - microbiology | Tobacco - genetics | Gene Expression Regulation, Plant - physiology | Signal Transduction - physiology | Tobacco - microbiology | Tobacco - enzymology | Arabidopsis thaliana | Reactive oxygen species | Plant immunology | Plant genetics | Physiological aspects | Genetic aspects | Research | Tobacco (Plant) | Gene expression | Health aspects
Journal Article
Journal Article
Arteriosclerosis, Thrombosis, and Vascular Biology, ISSN 1079-5642, 12/2006, Volume 26, Issue 12, pp. 2688 - 2695
Journal Article
Journal of Biological Chemistry, ISSN 0021-9258, 09/2016, Volume 291, Issue 38, pp. 20030 - 20041
Journal Article
Nature immunology, ISSN 1529-2916, 2010, Volume 11, Issue 5, pp. 411 - 418
Sensors of pathogens, such as Toll-like receptors (TLRs), detect microbes to activate transcriptional programs that orchestrate adaptive responses to specific... 
UNFOLDED PROTEIN RESPONSE | PATHWAY | ER STRESS | HOST-DEFENSE | ENDOPLASMIC-RETICULUM STRESS | FRANCISELLA-TULARENSIS INFECTION | IMMUNOLOGY | LIVE VACCINE STRAIN | INFLAMMATORY RESPONSE | NEGATIVE REGULATOR | SYSTEMS BIOLOGY | RNA, Small Interfering - genetics | Endoribonucleases - genetics | Tularemia - metabolism | Toll-Like Receptor 2 - genetics | Membrane Glycoproteins - metabolism | Lipopeptides - pharmacology | Transcriptional Activation - drug effects | NADPH Oxidases - metabolism | Tularemia - genetics | Protein Splicing - genetics | Transcriptional Activation - immunology | X-Box Binding Protein 1 | Protein Splicing - drug effects | NADPH Oxidases - genetics | Stress, Physiological - drug effects | TNF Receptor-Associated Factor 6 - genetics | Transcription Factor CHOP - biosynthesis | Transcription Factors - immunology | Cytokines - genetics | Protein-Serine-Threonine Kinases - metabolism | DNA-Binding Proteins - immunology | Endoribonucleases - metabolism | Macrophages - pathology | Stress, Physiological - genetics | Myeloid Differentiation Factor 88 - genetics | Toll-Like Receptor 4 - genetics | Signal Transduction - genetics | Toll-Like Receptor 4 - immunology | Toll-Like Receptor 2 - metabolism | Toll-Like Receptor 4 - metabolism | Mice, Knockout | Macrophages - metabolism | Signal Transduction - drug effects | Tunicamycin - pharmacology | Lipopolysaccharides - pharmacology | Toll-Like Receptor 2 - immunology | Mice | Stress, Physiological - immunology | Endoribonucleases - immunology | Transcription Factor CHOP - genetics | NADPH Oxidases - immunology | DNA-Binding Proteins - metabolism | Signal Transduction - immunology | Macrophages - virology | Mice, Mutant Strains | Tularemia - immunology | Membrane Glycoproteins - immunology | Macrophages - immunology | Cytokines - immunology | Cell Line | Francisella tularensis - immunology | Protein Splicing - immunology | Protein-Serine-Threonine Kinases - genetics | Transcription Factors - genetics | DNA-Binding Proteins - genetics | Immunity, Innate | NADPH Oxidase 2 | Regulatory Factor X Transcription Factors | Mice, Inbred C3H | Membrane Glycoproteins - genetics | Transcription Factors - metabolism | Animals | TNF Receptor-Associated Factor 6 - metabolism | Macrophages - drug effects | Protein-Serine-Threonine Kinases - immunology | Francisella tularensis - pathogenicity | Myeloid Differentiation Factor 88 - metabolism | Cytokines - biosynthesis | Cell receptors | Transcription factors | Immune response | Physiological aspects | Genetic aspects | Research | Health aspects | Francisella tularensis
Journal Article
The Plant cell, ISSN 1532-298X, 2017, Volume 29, Issue 4, pp. 775 - 790
Journal Article
PloS one, ISSN 1932-6203, 2015, Volume 10, Issue 7, p. e0129743
Reactive oxygen species (ROS) produced by nicotinamide adenine dinucleotide phosphate oxidase (NOX... 
TRANSFORMATION | NADPH OXIDASE | HEPATOCYTE APOPTOSIS | OXIDATIVE STRESS | SIGNALING PATHWAYS | HEDGEHOG PATHWAY | MULTIDISCIPLINARY SCIENCES | INVOLVEMENT | NAD(P)H OXIDASES | PROLIFERATION | EXPRESSION | NADPH Oxidases - chemistry | Reactive Oxygen Species - metabolism | Humans | NADPH Oxidases - metabolism | Hedgehog Proteins - metabolism | Hepatic Stellate Cells - metabolism | Platelet-Derived Growth Factor - genetics | Myofibroblasts - metabolism | Carbon Tetrachloride Poisoning - metabolism | Hedgehog Proteins - genetics | Hepatitis - genetics | Hepatitis - metabolism | Liver Cirrhosis - metabolism | NADPH Oxidases - genetics | NADH, NADPH Oxidoreductases - metabolism | Liver Cirrhosis - genetics | Hepatic Stellate Cells - pathology | Myofibroblasts - pathology | NADH, NADPH Oxidoreductases - genetics | Signal Transduction | NADPH Oxidases - antagonists & inhibitors | Hepatitis - pathology | NADPH Oxidase 4 | Pyrazoles - metabolism | Carbon Tetrachloride Poisoning - pathology | NADPH Oxidase 1 | Mice, Knockout | Platelet-Derived Growth Factor - metabolism | Animals | Pyridines - metabolism | Liver Cirrhosis - pathology | Carbon Tetrachloride Poisoning - genetics | Mice | NADH, NADPH Oxidoreductases - antagonists & inhibitors | Niacinamide | Platelet-derived growth factor | Carbon tetrachloride | Fibrosis | Inflammation | Comparative analysis | Liver cirrhosis | Phosphates | Oxidative stress | Pathogenesis | Liver | Genes | Lipid peroxidation | Activation | NAD(P)H oxidase | Lipopolysaccharides | Veterinarians | Hepatitis | Cell activation | NOX4 protein | Rodents | Cell cycle | Peroxidation | Medical research | Stellate cells | Enzymes | Oxygen | NADPH-diaphorase | Adenine | CCL4 protein | Medicine | Cirrhosis | Signaling | Injury prevention | Inhibitors | Hedgehog protein | Nicotinamide adenine dinucleotide | Nicotinamide | Laboratory animals | Plastics | Apoptosis | Bile
Journal Article
Trends in Endocrinology & Metabolism, ISSN 1043-2760, 2014, Volume 25, Issue 9, pp. 452 - 463
Highlights • Endothelial cells express four NADPH oxidase isoforms: NOX1, 2, 4, and 5. • NOX1, 2, and 5 make superoxide and cause endothelial dysfunction and vascular disease... 
Endocrinology & Metabolism | hyperlipidemia | NADPH oxidases | vascular disease | selective NOX1/2 inhibitors | inflammation | reactive oxygen species | hypertension | diabetes | endothelial dysfunction | Hypertension | Vascular disease | Selective NOX1/2 inhibitors | Reactive oxygen species | Hyperlipidemia | Endothelial dysfunction | Inflammation | Diabetes | OXYGEN SPECIES PRODUCTION | OXIDATIVE STRESS | SPONTANEOUSLY HYPERTENSIVE-RATS | NITRIC-OXIDE SYNTHASE | LOW-DENSITY-LIPOPROTEIN | ANGIOTENSIN-II | REDOX SIGNALING PLATFORMS | INDUCED UP-REGULATION | ENDOCRINOLOGY & METABOLISM | HYDROGEN-PEROXIDE PRODUCTION | CORONARY-ARTERY-DISEASE | NADPH Oxidases - chemistry | Membrane Glycoproteins - metabolism | Humans | NADPH Oxidases - metabolism | Endothelium, Vascular - drug effects | Endothelium, Vascular - enzymology | Molecular Targeted Therapy | Membrane Glycoproteins - antagonists & inhibitors | NADPH Oxidases - genetics | Membrane Proteins - metabolism | Vascular Diseases - drug therapy | Membrane Proteins - genetics | NADPH Oxidases - antagonists & inhibitors | Enzyme Inhibitors - pharmacology | Mice, Transgenic | NADPH Oxidase 5 | NADPH Oxidase 4 | Vascular Diseases - enzymology | Enzyme Inhibitors - therapeutic use | NADPH Oxidase 2 | NADPH Oxidase 1 | Cardiovascular Agents - pharmacology | Membrane Glycoproteins - genetics | Mice, Knockout | Cardiovascular Agents - therapeutic use | Animals | Membrane Proteins - antagonists & inhibitors | Models, Biological | Oxidases | Blood circulation disorders | Superoxide | Peroxides | Endothelium
Journal Article
Journal Article
Free radical biology & medicine, ISSN 0891-5849, 2016, Volume 99, pp. 167 - 178
Excessive levels of reactive oxygen species (ROS) and increased expression of NADPH oxidases (Nox... 
Epigenetics | Histone deacetylases | NADPH oxidase | Pulmonary artery hypertension | OXYGEN SPECIES PRODUCTION | ACTIVATION | BIOCHEMISTRY & MOLECULAR BIOLOGY | HDAC INHIBITION | CELL-PROLIFERATION | 5 NOX5 | NAD(P)H OXIDASE | THERAPEUTIC TARGET | DISEASE | ENDOCRINOLOGY & METABOLISM | EXPRESSION | RIGHT-VENTRICULAR HYPERTROPHY | Transcription, Genetic - drug effects | Antihypertensive Agents - pharmacology | Endothelial Cells | Luciferases - metabolism | Reactive Oxygen Species - metabolism | Epigenesis, Genetic | Humans | Monocrotaline | Male | RNA Polymerase II - metabolism | Hypertension, Pulmonary - chemically induced | Luciferases - genetics | p300-CBP Transcription Factors - genetics | Isoenzymes - metabolism | HEK293 Cells | Fibroblasts | Hypertension, Pulmonary - drug therapy | Genes, Reporter | Cell Line | Promoter Regions, Genetic | Histone Deacetylases - genetics | Signal Transduction | Isoenzymes - genetics | Rats | Histone Deacetylases - metabolism | Hypertension, Pulmonary - genetics | Rats, Sprague-Dawley | p300-CBP Transcription Factors - metabolism | Reactive Oxygen Species - antagonists & inhibitors | Animals | NADPH Oxidase 2 - metabolism | Histone Deacetylase Inhibitors - pharmacology | RNA Polymerase II - genetics | Mice | NADPH Oxidase 2 - antagonists & inhibitors | NADPH Oxidase 2 - genetics | Hypertension, Pulmonary - pathology | Isoenzymes - antagonists & inhibitors | Oxidases | Epigenetic inheritance | Chromatin | RNA | Analysis | Genes | Histones | Superoxide | DNA binding proteins | Genetic transcription | Pulmonary hypertension
Journal Article