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Biochemical and Biophysical Research Communications, ISSN 0006-291X, 10/2019, Volume 519, Issue 1, pp. 29 - 34
Non-steroidal anti-inflammatory drug activated gene-1 (NAG-1), also known as growth differentiation factor 15 (GDF15), is a TGF-β (transforming growth factor... 
High-content screening | Nuclear localization signal | NAG-1 | GDF15 | Peptide transfection | Mitochondrial membrane potential
Journal Article
Molecular Cancer Therapeutics, ISSN 1535-7163, 01/2007, Volume 6, Issue 1, pp. 362 - 369
Prodigiosin (2-methyl-3-pentyl-6-methoxyprodigiosene) is a bacterial metabolite that has anticancer and antimetastatic properties. However, the molecular... 
GSK-3β | NAG-1 | apoptosis | breast cancer | prodigiosin
Journal Article
Cytokine and Growth Factor Reviews, ISSN 1359-6101, 2013, Volume 24, Issue 4, pp. 373 - 384
Abstract GDF-15 (also MIC-1, NAG-1, PLAB, PTGFB) is a member of the TGF-β superfamily, which is widely distributed in mammalian tissues and has been shown to... 
Advanced Basic Science | Cardiovascular disease | Inflammation | Metabolism | Neurotrophic factors | Cancer | CANCER CELLS | BIOCHEMISTRY & MOLECULAR BIOLOGY | GROWTH-DIFFERENTIATION FACTOR-15 | SUPERFAMILY MEMBER | GDF-15 | CELL BIOLOGY | ACTIVATED GENE NAG-1 | SERUM CONCENTRATIONS | INTEGRIN ACTIVATION | MIC-1 | MATERNAL SERUM | EXPRESSION
Journal Article
International journal of obesity (2005), ISSN 0307-0565, 12/2014, Volume 38, Issue 12, pp. 1555 - 1564
Journal Article
Journal Article
Free Radical Biology and Medicine, ISSN 0891-5849, 11/2017, Volume 112, pp. 49 - 59
We have previously reported that a newly synthesized compound, GL-V9 could induce mitochondria-mediated apoptosis in HepG2 cells. However, the underlying... 
GL-V9 | NAG-1 | MMP | Mitochondria | ROS | Apoptosis | DRUG-ACTIVATED GENE | GROWTH-INHIBITION | INDUCED APOPTOSIS | BIOCHEMISTRY & MOLECULAR BIOLOGY | MACROPHAGE INHIBITORY CYTOKINE-1 | NAG-1/GDF15 | MESSENGER-RNA STABILITY | MIC-1 EXPRESSION | P53 | ENDOCRINOLOGY & METABOLISM | SCUTELLARIA-BAICALENSIS GEORGI | COLORECTAL-CANCER CELLS | Epithelial Cells - metabolism | Reactive Oxygen Species - metabolism | Epithelial Cells - drug effects | Humans | Gene Expression Regulation, Neoplastic | Membrane Potential, Mitochondrial - drug effects | Growth Differentiation Factor 15 - genetics | Reactive Oxygen Species - agonists | Carcinoma, Hepatocellular - drug therapy | Carcinoma, Hepatocellular - genetics | Liver Neoplasms - pathology | Antineoplastic Agents - pharmacology | Flavonoids - pharmacology | p38 Mitogen-Activated Protein Kinases - metabolism | Cell Death - drug effects | Epithelial Cells - cytology | Cell Line | Liver Neoplasms - genetics | Signal Transduction | Liver Neoplasms - drug therapy | p38 Mitogen-Activated Protein Kinases - genetics | Mitochondria - metabolism | Mitochondria - drug effects | Mitochondria - pathology | Hep G2 Cells | Xenograft Model Antitumor Assays | Animals | Mice, Nude | Growth Differentiation Factor 15 - agonists | Carcinoma, Hepatocellular - pathology | Liver Neoplasms - metabolism | Cell Line, Tumor | Mice | Oxidative Stress - drug effects | Growth Differentiation Factor 15 - metabolism | Carcinoma, Hepatocellular - metabolism | Chemotherapy | RNA | Cell death | Analysis | Pharmacy | Drugstores | Biochemistry | Superoxide | Hepatoma | Transforming growth factors | Cancer | Genes | Index Medicus
Journal Article
Cancer Letters, ISSN 0304-3835, 2006, Volume 251, Issue 2, pp. 268 - 277
Journal Article
Journal Article