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1. Full Text Andrographolide Suppresses Proliferation of Nasopharyngeal Carcinoma Cells via Attenuating NF- κ B Pathway
by Peng, Tao and Hu, Min and Wu, Ting-Ting and Zhang, Cen and Chen, Zhe and Huang, Shuo and Zhou, Xu-Hong
BioMed research international, ISSN 2314-6133, 3/2015, Volume 2015, pp. 735056 - 7
Life Sciences & Biomedicine | Biotechnology & Applied Microbiology | Medicine, Research & Experimental | Science & Technology | Research & Experimental Medicine | Diterpenes - pharmacology | Nasopharyngeal Carcinoma | Nasopharyngeal Neoplasms - metabolism | Carcinoma | Apoptosis - drug effects | Humans | NF-kappa B - metabolism | Down-Regulation - drug effects | Signal Transduction - drug effects | Cell Cycle Checkpoints - drug effects | Cell Line, Tumor | Antineoplastic Agents - pharmacology | Cell Proliferation - drug effects | Nasopharyngeal Neoplasms - drug therapy | Index Medicus
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2. miR-495 enhances the efficacy of radiotherapy by targeting GRP78 to regulate EMT in nasopharyngeal carcinoma cells
by Feng, Xueping and Lv, Wuwu and Wang, Shuanglian and He, Qian
Oncology reports, ISSN 1021-335X, 09/2018, Volume 40, Issue 3, pp. 1223 - 1232
Nasopharyngeal carcinoma | Epithelial-mesenchymal transition | Radioresistance | Glucose-regulated protein 78 | MiR-495 | Life Sciences & Biomedicine | Oncology | Science & Technology | Nasopharyngeal Neoplasms - genetics | Prognosis | Follow-Up Studies | Apoptosis - radiation effects | Humans | Middle Aged | Gene Expression Regulation, Neoplastic | Male | Case-Control Studies | Nasopharyngeal Neoplasms - pathology | Young Adult | Heat-Shock Proteins - genetics | Nasopharyngeal Neoplasms - radiotherapy | Epithelial-Mesenchymal Transition | Aged, 80 and over | Biomarkers, Tumor - metabolism | Adult | Female | Tumor Cells, Cultured | Carcinoma - pathology | Child | Nasopharyngeal Carcinoma | Heat-Shock Proteins - metabolism | Computational Biology | Radiation Tolerance - genetics | Carcinoma - radiotherapy | Adolescent | Carcinoma - genetics | Aged | Biomarkers, Tumor - genetics | MicroRNAs - genetics | Cell Proliferation - radiation effects
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3. Full Text Blood diffusion and Th1-suppressive effects of galectin-9-containing exosomes released by Epstein-Barr virus-infected nasopharyngeal carcinoma cells
by Klibi, Jihène and Niki, Toshiro and Riedel, Alexander and Pioche-Durieu, Catherine and Souquere, Sylvie and Rubinstein, Eric and Moulec, Sylvestre L. E and Guigay, Joël and Hirashima, Mitsuomi and Guemira, Fethi and Adhikary, Dinesh and Mautner, Josef and Busson, Pierre
Blood, ISSN 0006-4971, 02/2009, Volume 113, Issue 9, pp. 1957 - 1966
Life Sciences & Biomedicine | Hematology | Science & Technology | Hematologic and hematopoietic diseases | Biological and medical sciences | Infectious diseases | Medical sciences | Viral diseases | Exosomes - metabolism | Nasopharyngeal Neoplasms - metabolism | Apoptosis - drug effects | Receptors, Virus - metabolism | Blood Vessels - metabolism | Humans | Exosomes - pathology | Transplantation, Heterologous | CD4-Positive T-Lymphocytes - pathology | CD4-Positive T-Lymphocytes - immunology | Carcinoma - complications | Nasopharyngeal Neoplasms - blood | T-Lymphocytes, Helper-Inducer - immunology | Nasopharyngeal Neoplasms - complications | Protein Binding - drug effects | Galectins - immunology | Nasopharyngeal Neoplasms - immunology | Epstein-Barr Virus Infections - blood | T-Lymphocytes, Helper-Inducer - pathology | Diffusion | Tumor Escape - immunology | Carcinoma - blood | Carcinoma - immunology | Epstein-Barr Virus Infections - immunology | CD4-Positive T-Lymphocytes - metabolism | Galectins - antagonists & inhibitors | Hepatitis A Virus Cellular Receptor 2 | Antibodies - pharmacology | Animals | Apoptosis - immunology | Cell Line, Tumor | Receptors, Virus - immunology | Epstein-Barr Virus Infections - complications | Galectins - blood | Mice | Carcinoma - metabolism | HeLa Cells | Receptors, Virus - antagonists & inhibitors | Epstein-Barr Virus Infections - metabolism | Galectins - metabolism | Index Medicus | Abridged Index Medicus
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4. Full Text Epstein‐Barr virus nuclear antigen 1 (EBNA1) protein induction of epithelial‐mesenchymal transition in nasopharyngeal carcinoma cells
by Wang, Lu and Tian, Wen‐Dong and Xu, Xia and Nie, Biao and Lu, Juan and Liu, Xiong and Zhang, Bao and Dong, Qi and Sunwoo, John B and Li, Gang and Li, Xiang‐Ping
Cancer, ISSN 0008-543X, 02/2014, Volume 120, Issue 3, pp. 363 - 372
microRNA | nasopharyngeal carcinoma | metastasis | epithelial‐mesenchymal transition | Epstein‐Barr virus | epithelial-mesenchymal transition | Epstein-Barr virus | Life Sciences & Biomedicine | Oncology | Science & Technology | Biological and medical sciences | Multiple tumors. Solid tumors. Tumors in childhood (general aspects) | Medical sciences | Otorhinolaryngology. Stomatology | Upper respiratory tract, upper alimentary tract, paranasal sinuses, salivary glands: diseases, semeiology | Tumors | Nasopharyngeal Carcinoma | Transcription Factors - physiology | Carcinoma | Signal Transduction | Neoplasm Invasiveness | Humans | Epstein-Barr Virus Nuclear Antigens - physiology | Transforming Growth Factor beta1 - physiology | Nasopharyngeal Neoplasms - pathology | Epithelial-Mesenchymal Transition | Cell Line, Tumor | MicroRNAs - physiology | Homeodomain Proteins - physiology | Cell Movement | Zinc Finger E-box-Binding Homeobox 1 | Physiological aspects | Development and progression | Nasopharyngeal cancer | Research | Metastasis | Index Medicus | Abridged Index Medicus
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5. Full Text A positive feedback loop between Wnt/β‐catenin signaling and hTERT regulates the cancer stem cell‐like traits in radioresistant nasopharyngeal carcinoma cells
by Chen, Kaihua and Chen, Li and Li, Ling and Qu, Song and Yu, Binbin and Sun, Yongchu and Wan, Fangzhu and Chen, Xishan and Liang, Renba and Zhu, Xiaodong
Journal of cellular biochemistry, ISSN 0730-2312, 11/2020, Volume 121, Issue 11, pp. 4612 - 4622
Wnt/β‐catenin signaling | nasopharyngeal carcinoma | cancer stem cells | hTERT | radioresistance | Wnt/β-catenin signaling | Biochemistry & Molecular Biology | Life Sciences & Biomedicine | Science & Technology | Cell Biology | Phenotypes | Telomerase reverse transcriptase | Radiosensitivity | RNA-directed DNA polymerase | Wnt protein | Transcription | Gene regulation | Radioresistance | Feedback loops | Positive feedback | Proteins | Nasopharyngeal carcinoma | Signaling | Signal transduction | Feedback | Stem cells | Telomerase | Catenin | Cytoplasm | Cancer | Index Medicus
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6. Full Text Andrographolide suppresses proliferation of nasopharyngeal carcinoma cells via attenuating NF- B pathway
by Peng, Tao and Hu, Min and Wu, Ting-Ting and Zhang, Cen and Chen, Zhe and Huang, Shuo and Zhou, Xu-Hong
BioMed research international, ISSN 2314-6133, 2015, Volume 2015
Cell proliferation | Prevention | Care and treatment | Usage | Transcription factors | Cancer cells | Physiological aspects | Diterpenes | Nasopharyngeal cancer | Health aspects | Studies | Cell culture | Cell cycle | Cancer | Apoptosis
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7. Full Text Downregulation of miR-144 by triptolide enhanced p85α−PTEN complex formation causing S phase arrest of human nasopharyngeal carcinoma cells
by Wu, Chien-Wei and Wang, Shyang-Guang and Lin, Meng-Liang and Chen, Shih-Shun
European journal of pharmacology, ISSN 0014-2999, 07/2019, Volume 855, pp. 137 - 148
PTEN | Akt | S phase arrest | Triptolide | p85α | Life Sciences & Biomedicine | Pharmacology & Pharmacy | Science & Technology | Diterpenes - pharmacology | Cyclin-Dependent Kinase 2 - metabolism | Class Ia Phosphatidylinositol 3-Kinase - metabolism | Humans | Caspase 3 - metabolism | PTEN Phosphohydrolase - metabolism | Cyclin-Dependent Kinase 2 - chemistry | Down-Regulation - drug effects | Threonine - metabolism | p21-Activated Kinases - metabolism | Nasopharyngeal Neoplasms - pathology | Phenanthrenes - pharmacology | Proteolysis - drug effects | Epoxy Compounds - pharmacology | Cell Line, Tumor | MicroRNAs - genetics | Phosphorylation - drug effects | S Phase Cell Cycle Checkpoints - drug effects | Index Medicus
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8. Full Text The FOXM1-ABCC5 axis contributes to paclitaxel resistance in nasopharyngeal carcinoma cells
by Hou, Youxiang and Zhu, Qianling and Li, Zheng and Peng, Yongbo and Yu, Xiaohui and Yuan, Bowen and Liu, Yijun and Liu, Youhong and Yin, Linglong and Peng, Yuchong and Jiang, Zhenghua and Li, Jinping and Xie, Bowen and Duan, Yumei and Tan, Guolin and Gulina, Kurban and Gong, Zhicheng and Sun, Lunquan and Fan, Xuegong and Li, Xiong
Cell death & disease, ISSN 2041-4889, 2017, Volume 8, Issue 3, pp. e2659 - e2659
Life Sciences & Biomedicine | Science & Technology | Cell Biology | Carcinoma - drug therapy | Nasopharyngeal Neoplasms - genetics | Cell Proliferation - genetics | Nasopharyngeal Carcinoma | Drug Resistance, Multiple - genetics | Signal Transduction | Humans | Gene Expression Regulation, Neoplastic | Neoplasm Recurrence, Local - drug therapy | Epithelial-Mesenchymal Transition - genetics | Cell Movement - genetics | Nasopharyngeal Neoplasms - pathology | Drug Resistance, Neoplasm - genetics | Neoplastic Stem Cells - metabolism | Cell Line, Tumor | Neoplastic Stem Cells - pathology | Carcinoma - genetics | Forkhead Box Protein M1 - genetics | Multidrug Resistance-Associated Proteins - genetics | Neoplasm Recurrence, Local - genetics | Nasopharyngeal Neoplasms - drug therapy | Carcinoma - pathology | Paclitaxel - administration & dosage | Chemotherapy | Drug resistance | Cells | Cancer | Index Medicus | Original
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9. Full Text Down-regulation of miRNA-204 by LMP-1 enhances CDC42 activity and facilitates invasion of EBV-associated nasopharyngeal carcinoma cells
by Ma, Lei and Deng, Xubin and Wu, Minhua and Zhang, Gong and Huang, Jianqing
FEBS letters, ISSN 0014-5793, 05/2014, Volume 588, Issue 9, pp. 1562 - 1570
Nasopharyngeal carcinoma | Stat-3 | miR-204 | Epstein–Barr virus | Latent membrane protein 1 | Cdc42 | Epstein-Barr virus | Biochemistry & Molecular Biology | Biophysics | Life Sciences & Biomedicine | Science & Technology | Cell Biology | Neoplasm Transplantation | Nasopharyngeal Neoplasms - metabolism | Carcinoma | Nasopharyngeal Neoplasms - mortality | Prognosis | Humans | Middle Aged | Gene Expression Regulation, Neoplastic | Nasopharyngeal Neoplasms - virology | Male | MicroRNAs - metabolism | cdc42 GTP-Binding Protein - metabolism | Viral Matrix Proteins - physiology | Nasopharyngeal Neoplasms - pathology | RNA Interference | Base Sequence | Female | Binding Sites | STAT3 Transcription Factor - metabolism | Nasopharyngeal Carcinoma | Signal Transduction | Neoplasm Invasiveness | Down-Regulation | Kaplan-Meier Estimate | Tumor Burden | Disease Progression | Herpesvirus 4, Human | Epstein-Barr Virus Infections - pathology | Animals | Mice, Nude | Cell Line, Tumor | Epstein-Barr Virus Infections - mortality | Mice | Mice, Inbred BALB C | MicroRNAs - genetics | Epstein-Barr Virus Infections - metabolism | MicroRNA | Index Medicus
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10. Full Text Epigallocatechin‐3‐gallate inhibits migration of human nasopharyngeal carcinoma cells by repressing MMP‐2 expression
by Ho, Hsu‐Chueh and Huang, Cheng‐Chen and Lu, Yen‐Ting and Yeh, Chia‐Ming and Ho, Yu‐Ting and Yang, Shun‐Fa and Hsin, Chung‐Han and Lin, Chiao‐Wen
Journal of cellular physiology, ISSN 0021-9541, 11/2019, Volume 234, Issue 11, pp. 20915 - 20924
nasopharyngeal carcinoma | metastasis | epigallocatechin‐3‐gallate | MMP‐2 | epigallocatechin-3-gallate | MMP-2 | Physiology | Life Sciences & Biomedicine | Science & Technology | Cell Biology | Catechin | Carcinoma | Green tea | Cancer | Repressing | Phosphorylation | Matrix metalloproteinases | Epigallocatechin-3-gallate | Metastasis | Matrix metalloproteinase | Src protein | Kinases | Lymph nodes | Anticancer properties | Metastases | Antioxidants | Nasopharyngeal carcinoma | Tea | Signaling | Signal transduction | Chemotherapy | Inhibition | Cell migration | Index Medicus
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11. Sodium selenite (Na 2 SeO 3 ) induces apoptosis through the mitochondrial pathway in CNE-2 nasopharyngeal carcinoma cells
by Cui, Zhongyi and Li, Caihong and Li, Xiangyong and Zhang, Qunzhou and Zhang, Yuefei and Shao, Jingjing and Zhou, Keyuan
International journal of oncology, ISSN 1019-6439, 06/2015, Volume 46, Issue 6, pp. 2506 - 2514
Cell proliferation | Sodium selenite | CNE-2 | Cell cycle | Apoptosis | Life Sciences & Biomedicine | Oncology | Science & Technology | Nasopharyngeal Neoplasms - genetics | Nasopharyngeal Carcinoma | Nasopharyngeal Neoplasms - metabolism | Carcinoma | Reactive