Articles

Search Articles

Advanced search
Help

Catalogue

Articles

Databases

X
Search Filters
Format Format
Format Format
X
Sort by Item Count (A-Z)
Filter by Count
Journal Article (3762) 3762
Publication (206) 206
Dissertation (27) 27
Book Review (21) 21
Book Chapter (12) 12
Newsletter (12) 12
Conference Proceeding (9) 9
Book / eBook (5) 5
Newspaper Article (3) 3
Data Set (1) 1
Magazine Article (1) 1
Paper (1) 1
more...
Subjects Subjects
Subjects Subjects
X
Sort by Item Count (A-Z)
Filter by Count
humans (2582) 2582
nasopharyngeal carcinoma (2281) 2281
index medicus (1450) 1450
nasopharyngeal neoplasms - pathology (1246) 1246
cell line, tumor (1158) 1158
oncology (1147) 1147
cancer (1077) 1077
carcinoma (897) 897
apoptosis (880) 880
female (834) 834
male (786) 786
nasopharyngeal neoplasms - metabolism (783) 783
expression (758) 758
nasopharyngeal neoplasms - genetics (702) 702
animals (688) 688
metastasis (624) 624
middle aged (616) 616
proteins (544) 544
adult (529) 529
gene expression (514) 514
mice (512) 512
research (454) 454
nasopharyngeal cancer (444) 444
cell proliferation (442) 442
aged (420) 420
gene expression regulation, neoplastic (415) 415
tumors (412) 412
genetic aspects (401) 401
health aspects (384) 384
cell cycle (372) 372
cell biology (371) 371
stomatognathic diseases (363) 363
otorhinolaryngologic diseases (352) 352
prognosis (344) 344
epstein-barr virus (343) 343
biochemistry & molecular biology (331) 331
nasopharyngeal neoplasms - drug therapy (320) 320
mice, nude (317) 317
kinases (311) 311
proliferation (307) 307
development and progression (303) 303
cancer therapies (300) 300
signal transduction (299) 299
care and treatment (295) 295
chemotherapy (295) 295
cell growth (291) 291
apoptosis - drug effects (287) 287
nasopharyngeal carcinoma-cells (287) 287
research article (275) 275
medicine, research & experimental (274) 274
tumor cells, cultured (274) 274
studies (267) 267
activation (265) 265
epstein-barr-virus (265) 265
radiation therapy (259) 259
transfection (254) 254
nasopharyngeal neoplasms - virology (253) 253
carcinoma - pathology (252) 252
cell proliferation - drug effects (248) 248
invasion (246) 246
breast-cancer (242) 242
growth (240) 240
analysis (238) 238
down-regulation (236) 236
herpesvirus 4, human - genetics (235) 235
medicine (233) 233
medical prognosis (232) 232
immunohistochemistry (221) 221
phosphorylation (214) 214
nasopharyngeal neoplasms - radiotherapy (213) 213
radiotherapy (207) 207
antineoplastic agents - pharmacology (200) 200
flow cytometry (194) 194
carcinoma - metabolism (191) 191
carcinoma, squamous cell - pathology (187) 187
mice, inbred balb c (187) 187
blotting, western (186) 186
cells (184) 184
microrna (183) 183
micrornas - genetics (182) 182
multidisciplinary sciences (175) 175
genes (174) 174
physiological aspects (174) 174
migration (173) 173
neoplasms. tumors. oncology. including cancer and carcinogens (173) 173
neoplasm invasiveness (172) 172
inhibition (170) 170
polymerase chain reaction (170) 170
carcinoma - genetics (169) 169
gene (167) 167
rna interference (167) 167
pathology (165) 165
in-vitro (163) 163
adolescent (162) 162
deoxyribonucleic acid--dna (160) 160
gene-expression (160) 160
metastases (160) 160
reverse transcriptase polymerase chain reaction (159) 159
protein (158) 158
biology (155) 155
more...
Library Location Library Location
Language Language
Language Language
X
Sort by Item Count (A-Z)
Filter by Count
English (2971) 2971
Chinese (765) 765
Japanese (84) 84
French (2) 2
German (1) 1
Italian (1) 1
Korean (1) 1
Polish (1) 1
more...
Publication Date Publication Date
Click on a bar to filter by decade
Slide to change publication date range


1. Full Text EBV-miR-BART7-3p promotes the EMT and metastasis of nasopharyngeal carcinoma cells by suppressing the tumor suppressor PTEN
by Cai, L.-M and Cai, L-M and Lyu, X.-M and Lyu, X-M and Luo, W.-R and Luo, W-R and Cui, X-F and Cui, X.-F and Ye, Y.-F and Ye, Y-F and Yuan, C-C and Yuan, C.-C and Peng, Q.-X and Peng, Q-X and Wu, D-H and Wu, D.-H and Liu, T-F and Liu, T.-F and Wang, E and Wang, E and Marincola, F-M and Marincola, F.-M and Yao, K-T and Yao, K.-T and Fang, W-Y and Fang, W.-Y and Cai, H-B and Cai, H.-B and Li, X and Li, X
Oncogene, ISSN 0950-9232, 04/2015, Volume 34, Issue 17, pp. 2156 - 2166
The epithelial-mesenchymal transition (EMT) is crucial to cancer progression and metastasis. Although multiple cellular miRNAs have been identified to regulate... 
PROTEIN | BIOCHEMISTRY & MOLECULAR BIOLOGY | E-CADHERIN | CANCER STEM-CELLS | ENCODED MICRORNAS | CELL BIOLOGY | EPITHELIAL-MESENCHYMAL TRANSITION | LUNG-CANCER | INVASION | ONCOLOGY | TARGETING PTEN | GENETICS & HEREDITY | EXPRESSION | EPSTEIN-BARR-VIRUS | Nasopharyngeal Neoplasms - genetics | Nasopharyngeal Neoplasms - metabolism | Carcinoma | Herpesvirus 4, Human - genetics | Humans | Nasopharyngeal Neoplasms - virology | Glycogen Synthase Kinase 3 beta | Phosphatidylinositol 3-Kinases - metabolism | RNA, Neoplasm - metabolism | Proto-Oncogene Proteins c-akt - genetics | Nasopharyngeal Neoplasms - pathology | Neoplasm Metastasis | Cell Nucleus - metabolism | RNA, Viral - genetics | Cell Nucleus - pathology | Epithelial-Mesenchymal Transition | Epstein-Barr Virus Infections - genetics | Female | RNA, Viral - metabolism | Proto-Oncogene Proteins c-akt - metabolism | Cell Line | PTEN Phosphohydrolase - genetics | Nasopharyngeal Carcinoma | Neoplasm Invasiveness | PTEN Phosphohydrolase - biosynthesis | Signal Transduction - genetics | Glycogen Synthase Kinase 3 - metabolism | Epstein-Barr Virus Infections - pathology | Phosphatidylinositol 3-Kinases - genetics | Cell Nucleus - genetics | Glycogen Synthase Kinase 3 - genetics | RNA, Neoplasm - genetics | Herpesvirus 4, Human - metabolism | Epstein-Barr Virus Infections - metabolism | MicroRNA | Biological products | Innovations | Development and progression | Epstein-Barr virus | Genetic aspects | Nasopharyngeal cancer | Gene expression | Health aspects | Cellular biology | Metastasis | Cancer
Journal Article
Details down arrow
Digital rights down arrow
loading Loading Rights
2. Full Text MYC regulation of CHK1 and CHK2 promotes radioresistance in a stem cell-like population of nasopharyngeal carcinoma cells
by Wang, Wen-Jun and Wu, Si-Pei and Liu, Jia-Bin and Shi, Yong-Sheng and Huang, Xue and Zhang, Qian-Bing and Yao, Kai-Tai
Cancer Research, ISSN 0008-5472, 02/2013, Volume 73, Issue 3, pp. 1219 - 1231
Radiotherapy is the most successful nonsurgical treatment for nasopharyngeal carcinoma (NPC). Despite this, the prognosis remains poor. Although NPCs initially... 
CANCER-CELLS | ONCOLOGY | GROWTH ARREST | DNA-DAMAGE RESPONSE | GLIOMA | LINE | C-MYC | IDENTIFICATION | EXPRESSION | CHECKPOINTS | CYCLE | Nasopharyngeal Carcinoma | Carcinoma | Protein-Serine-Threonine Kinases - physiology | Humans | Radiation Tolerance | Organic Chemicals - analysis | Nasopharyngeal Neoplasms - pathology | Nasopharyngeal Neoplasms - radiotherapy | Proto-Oncogene Proteins c-myc - physiology | Animals | DNA Repair | Protein Kinases - physiology | Cell Line, Tumor | Checkpoint Kinase 2 | Checkpoint Kinase 1 | Female | Mice | DNA Damage | Neoplastic Stem Cells - radiation effects
Journal Article
Details down arrow
Digital rights down arrow
loading Loading Rights
3. Full Text Effects of CKMT1 on radiosensitivity of nasopharyngeal carcinoma cells
by Lan, Ruilong and Huang, Fei and Zhong, Guangxian and Chen, Ruiqing and Wang, Zeng and Chen, Junying and Fu, Lengxi and Hong, Jinsheng and Zhang, Lurong
International Journal of Radiation Biology, ISSN 0955-3002, 05/2019, Volume 95, Issue 5, pp. 597 - 606
Purpose: Radioresistance is an important factor for unsatisfactory prognosis in Nasopharyngeal carcinoma (NPC) patients. Ubiquitous mitochondrial creatine... 
CKMT1 | nasopharyngeal carcinoma | radiosensitivity | STAT3 | REPAIR | MITOCHONDRIAL CREATINE-KINASE | NUCLEAR SCIENCE & TECHNOLOGY | POOR-PROGNOSIS | RADIORESISTANCE | GROWTH | BIOLOGY | EXPRESSION | RADIOLOGY, NUCLEAR MEDICINE & MEDICAL IMAGING
Journal Article
Details down arrow
Digital rights down arrow
loading Loading Rights
4. Full Text Targeting Rad50 sensitizes human nasopharyngeal carcinoma cells to radiotherapy
by Chang, Lihong and Huang, Jiancong and Wang, Kai and Li, Jingjia and Yan, Ruicheng and Zhu, Ling and Ye, Jin and Wu, Xifu and Zhuang, Shimin and Li, Daqing and Zhang, Gehua
BMC Cancer, ISSN 1471-2407, 03/2016, Volume 16, Issue 1, p. 190
Background: The Mre11-Rad50-Nbs1 (MRN) complex is well known for its crucial role in initiating DNA double strand breaks (DSBs) repair pathways to resistant... 
Nasopharyngeal carcinoma | Rad50 | Radiosensitization | MRN complex | STRAND BREAK REPAIR | MRE11/RAD50/NBS1 COMPLEX | MRE11-RAD50-NBS1 | DEPLETION | DISRUPTION | ONCOLOGY | NBS1 | DNA-REPAIR | HUMAN TUMOR-CELLS | Nasopharyngeal Neoplasms - genetics | Carcinoma | Humans | Multiprotein Complexes | DNA Repair Enzymes - genetics | DNA Breaks, Double-Stranded - radiation effects | DNA-Binding Proteins - metabolism | G2 Phase Cell Cycle Checkpoints - radiation effects | MRE11 Homologue Protein | Nasopharyngeal Neoplasms - radiotherapy | Disease Models, Animal | Radiation, Ionizing | Gene Expression | Nasopharyngeal Carcinoma | Cell Cycle Proteins - metabolism | Radiation Tolerance - genetics | Nuclear Proteins - metabolism | DNA-Binding Proteins - genetics | Xenograft Model Antitumor Assays | Animals | Cell Line, Tumor | Protein Binding | Gene Expression Regulation, Neoplastic - radiation effects | Mice | Mutation | Apoptosis | Complications and side effects | Care and treatment | Adenoviruses | DNA damage | Physiological aspects | Development and progression | Research | Radiotherapy | Health aspects | Cancer
Journal Article
Details down arrow
Digital rights down arrow
loading Loading Rights
5. Full Text RIPK4 promoted the tumorigenicity of nasopharyngeal carcinoma cells
by Gong, Yongqian and Luo, Xinggu and Yang, Jing and Jiang, Qingshan and Liu, Zhifeng
Biomedicine & Pharmacotherapy, ISSN 0753-3322, 12/2018, Volume 108, pp. 1 - 6
RIPK4 (receptor interacting serine/threonine kinase 4) has been reported to be aberrantly expressed in several cancer types. However, its expression pattern... 
NPC | NF-kB signaling | VEGF | RIPK4 | MEDICINE, RESEARCH & EXPERIMENTAL | IMMUNITY | EFFICACY | AXITINIB | SAFETY | PROTEIN-KINASE | CANCER | PKK | PHARMACOLOGY & PHARMACY | NF-kappa B signaling | NF-KAPPA-B | Human Umbilical Vein Endothelial Cells - metabolism | Humans | Middle Aged | Gene Expression Regulation, Neoplastic | Nasopharyngeal Carcinoma - blood supply | Male | NF-kappa B - metabolism | Vascular Endothelial Growth Factor A - metabolism | RNA, Messenger - metabolism | Carcinogenesis - metabolism | Nasopharyngeal Carcinoma - enzymology | I-kappa B Kinase - metabolism | Female | Protein-Serine-Threonine Kinases - metabolism | Tumor Stem Cell Assay | Cell Proliferation - genetics | Signal Transduction | RNA, Messenger - genetics | Protein-Serine-Threonine Kinases - genetics | Up-Regulation - genetics | Carcinogenesis - pathology | Animals | Nasopharyngeal Carcinoma - genetics | Mice, Nude | Nasopharyngeal Carcinoma - pathology | Cell Line, Tumor | Aged | Endothelial growth factors | Carcinoma | Cancer
Journal Article
Details down arrow
Digital rights down arrow
loading Loading Rights
6. Full Text MALAT1/miR-124/Capn4 axis regulates proliferation, invasion and EMT in nasopharyngeal carcinoma cells
by Shi, Baoyuan and Wang, Yandan and Yin, Fengfang
Cancer Biology & Therapy, ISSN 1538-4047, 10/2017, Volume 18, Issue 10, pp. 792 - 800
Background: Long non-coding RNA MALAT1 (Metastasis-associated lung Adenocarcinoma transcript-1) has been demonstrated to play a critical role in the regulation... 
miR-124 | NPC | MALAT1 | proliferation | invasion | Capn4 | METASTASIS | LONG NONCODING RNAS | MICRORNAS | IN-VITRO | CANCER DEVELOPMENT | ONCOLOGY | GASTRIC-CANCER | EXPRESSION | MOLECULAR-MECHANISMS | Nasopharyngeal Neoplasms - genetics | Up-Regulation | Calpain - metabolism | Humans | Gene Expression Regulation, Neoplastic | MicroRNAs - metabolism | Calpain - genetics | Epithelial-Mesenchymal Transition - genetics | RNA, Messenger - metabolism | Cell Movement - genetics | Nasopharyngeal Neoplasms - pathology | Transfection | RNA Interference | Neoplasm Invasiveness - pathology | Carcinoma - pathology | Cell Proliferation - genetics | Nasopharyngeal Carcinoma | Down-Regulation | RNA, Long Noncoding - genetics | Signal Transduction - genetics | Disease Progression | Cell Line, Tumor | Carcinoma - genetics | MicroRNAs - genetics | RNA, Long Noncoding - metabolism | RNA, Small Interfering - metabolism
Journal Article
Details down arrow
Digital rights down arrow
loading Loading Rights
7. Full Text Epstein-Barr Virus_Encoded LMP1 Upregulates MicroRNA-21 to Promote the Resistance of Nasopharyngeal Carcinoma Cells to Cisplatin-Induced Apoptosis by Suppressing PDCD4 and Fas-L
by Yang, Guang-Da and Huang, Tie-Jun and Peng, Li-Xia and Yang, Chang-Fu and Liu, Ran-Yi and Huang, Hong-Bing and Chu, Qiao-Qiao and Yang, Hong-Jie and Huang, Jia-Ling and Zhu, Zhen-Yu and Qian, Chao-Nan and Huang, Bi-Jun
PLoS ONE, ISSN 1932-6203, 10/2013, Volume 8, Issue 10, p. e78355
Approximately 30% of patients with Epstein-Barr virus (EBV)-positive advanced nasopharyngeal carcinoma (NPC) display chemoresistance to cisplatin-based... 
CANCER-CELLS | B-CELLS | SURVIVAL | MEMBRANE-PROTEIN 1 | MULTIDISCIPLINARY SCIENCES | EBV | MIR-21 | LATENCY | LIGAND | EXPRESSION | LINES | RNA-Binding Proteins - genetics | Carcinoma | Apoptosis - drug effects | Humans | Nasopharyngeal Neoplasms - virology | MicroRNAs - metabolism | Viral Matrix Proteins - pharmacology | Tetrazolium Salts | Forkhead Transcription Factors - metabolism | Luciferases | Thiazoles | Apoptosis Regulatory Proteins - genetics | Inhibitory Concentration 50 | Viral Matrix Proteins - metabolism | Colony-Forming Units Assay | Gene Expression Regulation, Neoplastic - drug effects | Nasopharyngeal Neoplasms - drug therapy | Real-Time Polymerase Chain Reaction | RNA-Binding Proteins - antagonists & inhibitors | Cell Line | Nasopharyngeal Carcinoma | Cisplatin - pharmacology | Reverse Transcriptase Polymerase Chain Reaction | Blotting, Western | Drug Resistance, Neoplasm - genetics | Fas Ligand Protein - antagonists & inhibitors | Cisplatin - therapeutic use | Signal Transduction - drug effects | Apoptosis Regulatory Proteins - antagonists & inhibitors | Fluorescent Antibody Technique | MicroRNAs - genetics | Fas Ligand Protein - genetics | Forkhead Box Protein O3 | Infection | Prognosis | MicroRNA | Tumor necrosis factor | Epstein-Barr virus | B cells | Health aspects | Cisplatin | Apoptosis | Cancer | Cell proliferation | Laboratories | Genes | Crosstalk | Chemoresistance | Viruses | Oncology | Homology | AKT protein | Infections | Metastasis | Proteins | Nasopharyngeal carcinoma | Disease resistance | Signal transduction | Coding | Tumor necrosis factor-TNF | Tumorigenesis | Bioindicators | FOXO3 protein | MiRNA | Ribonucleic acid--RNA | 1-Phosphatidylinositol 3-kinase | Membrane proteins | Medicine | Signaling | Chemotherapy | FasL protein | Cell death | MicroRNAs | Medical prognosis | Cell lines | Biomarkers | Ligands | Latent membrane protein 1 | RNA | Ribonucleic acid
Journal Article
Details down arrow
Digital rights down arrow
loading Loading Rights
8. Full Text Chloroquine sensitizes nasopharyngeal carcinoma cells but not nasoepithelial cells to irradiation by blocking autophagy
by Makowska, Anna and Eble, Michael and Prescher, Kirsten and Ho, Mareike and Kontny, Udo
PLoS ONE, ISSN 1932-6203, 11/2016, Volume 11, Issue 11, p. e0166766
Background Treatment of nasopharyngeal carcinoma requires the application of high dosages of radiation, leading to severe long-term complications in the... 
HYDROXYCHLOROQUINE | APOPTOSIS | 5-FLUOROURACIL | IN-VITRO | INHIBITION | MULTIDISCIPLINARY SCIENCES | INDUCTION | CONTRIBUTES | EPSTEIN-BARR-VIRUS | CHEMOTHERAPY | TEMOZOLOMIDE | RNA, Small Interfering - genetics | Apoptosis - drug effects | Apoptosis - radiation effects | Epithelial Cells - drug effects | Humans | Autophagy-Related Protein 5 - genetics | Autophagy - drug effects | Chloroquine - pharmacology | Nasopharyngeal Neoplasms - radiotherapy | RNA Interference | Autophagy - genetics | Nasopharyngeal Neoplasms - drug therapy | Autophagy-Related Proteins - genetics | Carcinoma - drug therapy | In Situ Nick-End Labeling | Epithelial Cells - radiation effects | Nasopharyngeal Carcinoma | Radiation-Sensitizing Agents - pharmacology | Ubiquitin-Conjugating Enzymes - genetics | Carcinoma - radiotherapy | Autophagy-Related Protein 7 - genetics | Nasal Mucosa - cytology | Beclin-1 - genetics | Cell Line, Tumor | Cell Proliferation - drug effects | Cell Proliferation - radiation effects | Autophagy (Cytology) | Care and treatment | Chemotherapy | Chloroquine | Patient outcomes | Causes of | Development and progression | Nasopharyngeal cancer | Drug resistance | Health aspects | Cancer | Pediatrics | Biotechnology | Xenotransplantation | Colorectal cancer | Radiation | Oncology | Autophagy | Cancer therapies | Experiments | Proteins | Nasopharyngeal carcinoma | Cell growth | Xenografts | Sensitizing | Radiation effects | Hematology | Complications | Tumor cells | Melanoma | siRNA | Tumor cell lines | Electron microscopy | Cell lines | Stem cells | Irradiation | In vivo methods and tests | Immunofluorescence | Phagocytosis | Apoptosis
Journal Article
Details down arrow
Digital rights down arrow
loading Loading Rights
9. Full Text Positive regulation of HIF-1A expression by EBV oncoprotein LMP1 in nasopharyngeal carcinoma cells
by Sung, Wei-Wen and Chu, Yi-Chih and Chen, Peir-Rong and Liao, Ming-Hui and Lee, Jeng-Woei
Cancer Letters, ISSN 0304-3835, 2016, Volume 382, Issue 1, pp. 21 - 31
Highlights • LMP1 up-regulates HIF-1A transcription and post-transcription in NPC cells. • Down-regulation of RNA-destabilizing proteins contributes to... 
Hematology, Oncology and Palliative Medicine | LMP-1 | Nasopharyngeal carcinoma | EBV | HIF-1α | HIF-1 alpha | TRISTETRAPROLIN | TRANSCRIPTION | FACTOR-I | KAPPA-B | MESSENGER-RNA | GENE | ONCOLOGY | HYPOXIA-INDUCIBLE FACTOR-1-ALPHA | HIF-1-ALPHA | BINDING | EPSTEIN-BARR-VIRUS | Nasopharyngeal Neoplasms - genetics | RNA-Binding Proteins - genetics | Up-Regulation | Nasopharyngeal Neoplasms - metabolism | Viral Matrix Proteins - genetics | Herpesvirus 4, Human - genetics | Humans | Carcinoma - virology | Gene Expression Regulation, Neoplastic | Nasopharyngeal Neoplasms - virology | NF-kappa B - metabolism | RNA, Messenger - metabolism | Transfection | Time Factors | Hypoxia-Inducible Factor 1, alpha Subunit - metabolism | Epstein-Barr Virus Infections - genetics | Tristetraprolin - metabolism | Transcription, Genetic | Viral Matrix Proteins - metabolism | 3' Untranslated Regions | Binding Sites | STAT3 Transcription Factor - metabolism | Nasopharyngeal Carcinoma | Epstein-Barr Virus Infections - virology | Signal Transduction | Hypoxia-Inducible Factor 1, alpha Subunit - genetics | RNA, Messenger - genetics | Extracellular Signal-Regulated MAP Kinases | RNA Stability | Host-Pathogen Interactions | Tristetraprolin - genetics | Cell Line, Tumor | Carcinoma - genetics | Carcinoma - metabolism | Herpesvirus 4, Human - metabolism | Epstein-Barr Virus Infections - metabolism | Cell Transformation, Viral | RNA-Binding Proteins - metabolism | RNA | Colleges & universities | Viruses | Infections | Kinases | Gene expression | Proteins | Angiogenesis | Hepatitis | Cell growth | Mutagenesis | Plasmids | Rodents | MicroRNAs | Conflicts of interest | Epigenetics | Hypoxia | Mutation | Index Medicus
Journal Article
Details down arrow
Digital rights down arrow
loading Loading Rights
10. Full Text Inhibitory effects of 3-bromopyruvate in human nasopharyngeal carcinoma cells
by ZOU, XUE and ZHANG, MENGXIAO and SUN, YIMING and ZHAO, SURONG and WEI, YINGMEI and ZHANG, XUDONG and JIANG, CHENCHEN and LIU, HAO
Oncology Reports, ISSN 1021-335X, 10/2015, Volume 34, Issue 4, pp. 1895 - 1904
Tumor cells depend on aerobic glycolysis for adenosine triphosphate (ATP) production, which is therefore targeted by therapeutic agents. The compound... 
3-bromopyruvate | nasopharyngeal carcinoma cells | apoptosis | reactive oxygen species | necroptosis | Reactive oxygen species | Necroptosis | Nasopharyngeal carcinoma cells | Apoptosis | CANCER-CELLS | RIP KINASES | SURVIVAL | MITOCHONDRIA | INVOLVEMENT | DEATH | NECROSIS | THERAPY | ONCOLOGY | Nasopharyngeal Neoplasms - genetics | Nasopharyngeal Carcinoma | Carcinoma | Reactive Oxygen Species - metabolism | Pyruvates - administration & dosage | Apoptosis - drug effects | Humans | Mitochondria - drug effects | Mitochondria - pathology | Nasopharyngeal Neoplasms - pathology | Xenograft Model Antitumor Assays | Animals | Membrane Potential, Mitochondrial | Cell Line, Tumor | Mice | Nasopharyngeal Neoplasms - drug therapy | Studies | Medical research | Immunoglobulins | Cell growth | Tumor necrosis factor-TNF | Radiation therapy | Metabolism | Kinases | Cancer therapies | Tumors
Journal Article
Details down arrow
Digital rights down arrow
loading Loading Rights
11. Full Text Sodium butyrate induces autophagic apoptosis of nasopharyngeal carcinoma cells by inhibiting AKT/mTOR signaling
by Huang, Wei and Zeng, Chong and Liu, Weidong and Liu, Jie and Yuan, Li and Wang, Lei and Zhu, Hecheng and Xu, Yang and Luo, Yi and Xie, Dan and Jiang, Xingjun and Ren, Caiping
Biochemical and Biophysical Research Communications, ISSN 0006-291X, 06/2019, Volume 514, Issue 1, pp. 64 - 70
Previously, we confirmed the anti-tumor effects of sodium butyrate (NaBu) in nasopharyngeal carcinoma (NPC). However, its molecular mechanisms have not be... 
Nasopharyngeal carcinoma | AKT/mTOR | Sodium butyrate | Autophagy | Apoptosis
Journal Article
Details down arrow
Digital rights down arrow
loading Loading Rights
12. Full Text LncRNA UCA1 affects epithelial-mesenchymal transition, invasion, migration and apoptosis of nasopharyngeal carcinoma cells
by Han, Ri and Chen, Shunjin and Wang, Jianqi and Zhao, Yunteng and Li, Gang
Cell Cycle, ISSN 1538-4101, 11/2019, Volume 18, Issue 21, pp. 3044 - 3053
Objective: In this study, long non-coding RNA urothelial carcinoma associated 1 (lncRNA UCA1) in nasopharyngeal carcinoma (NPC) and its effect on the malignant... 
Nasopharyngeal carcinoma | Colony formation | UCA1
Journal Article
Details down arrow
Digital rights down arrow
loading Loading Rights
13. Full Text MiR-223 targeting MAFB suppresses proliferation and migration of nasopharyngeal carcinoma cells
by Yang, Wanyong and Lan, Xi and Li, Dongmin and Li, Tao and Lu, Shemin
BMC Cancer, ISSN 1471-2407, 12/2015, Volume 15, Issue 1, p. 461
Background: Mounting evidence suggests that miRNAs have major functions in tumor pathogenesis, and this study aimed to identify the candidate miRNA and... 
Nasopharyngeal carcinoma | MiR-223 | MAFB | Proliferation | Migration | DOWN-REGULATION | SENSITIVITY | MICRORNA-223 | HEPATOCELLULAR-CARCINOMA | ONCOLOGY | GASTRIC-CANCER | GROWTH | OSTEOSARCOMA | EXPRESSION | TUMORIGENESIS | PROGRESSION | Nasopharyngeal Neoplasms - genetics | Cell Proliferation - genetics | Nasopharyngeal Carcinoma | Carcinoma | MafB Transcription Factor - genetics | Humans | Gene Expression Regulation, Neoplastic | MafB Transcription Factor - biosynthesis | MicroRNAs - biosynthesis | Carcinogenesis | Cell Movement - genetics | Nasopharyngeal Neoplasms - pathology | Xenograft Model Antitumor Assays | RNA, Messenger - biosynthesis | Animals | Nasopharyngeal Neoplasms - blood | Cell Line, Tumor | MafB Transcription Factor - antagonists & inhibitors | Mice | MicroRNAs - blood | MicroRNAs - genetics | Cell proliferation | Care and treatment | MicroRNA | Analysis | Luciferase | Genetic aspects | Nasopharyngeal cancer | Diagnosis | Genetic transcription
Journal Article
Details down arrow
Digital rights down arrow
loading Loading Rights
14. Full Text Downregulation of lncRNA ANRIL inhibits proliferation, induces apoptosis, and enhances radiosensitivity in nasopharyngeal carcinoma cells through regulating miR-125a
by Hu, Xigang and Jiang, Huijuan and Jiang, Xiaojun
Cancer Biology & Therapy, ISSN 1538-4047, 05/2017, Volume 18, Issue 5, pp. 331 - 338
Increasing evidence demonstrated that long non-coding RNA ANRIL serves as a fatal oncogene in many cancers, including nasopharyngeal carcinoma (NPC). However,... 
ANRIL | NPC | miR-125a | apoptosis | proliferation | radiosensitivity | METASTASIS | IDENTIFICATION | LUNG-CANCER | LONG NONCODING RNA | ONCOLOGY | POOR-PROGNOSIS | RADIORESISTANCE | PROMOTES | EXPRESSION | PROGRESSION | AXIS | Nasopharyngeal Neoplasms - genetics | Cell Proliferation - genetics | Nasopharyngeal Carcinoma | Apoptosis - radiation effects | Humans | Gene Expression Regulation, Neoplastic | Cell Survival - genetics | Radiation Tolerance - genetics | Apoptosis - genetics | RNA, Long Noncoding - genetics | Carcinoma - radiotherapy | Cell Survival - radiation effects | Nasopharyngeal Neoplasms - pathology | Nasopharyngeal Neoplasms - radiotherapy | Flow Cytometry | Cell Line, Tumor | Carcinoma - genetics | MicroRNAs - genetics | Carcinoma - pathology | Cell Proliferation - radiation effects
Journal Article
Details down arrow
Digital rights down arrow
loading Loading Rights