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The Journal of cell biology, ISSN 1540-8140, 2014, Volume 204, Issue 2, pp. 231 - 245
Journal Article
Cell death and differentiation, ISSN 1476-5403, 2014, Volume 21, Issue 10, pp. 1511 - 1521
Necroptosis is a form of programmed cell death that depends on the activation of receptor interacting protein kinase-1 (RIPK1) and RIPK3 by receptors such as... 
APOPTOSIS | RIPOPTOSOME | MLKL | MACROPHAGES | DOWNSTREAM | BIOCHEMISTRY & MOLECULAR BIOLOGY | MIXED LINEAGE KINASE | PROGRAMMED NECROSIS | DOMAIN-LIKE | CLEAVAGE | CELL-DEATH | CELL BIOLOGY | Protein Kinases - metabolism | Protein Structure, Tertiary | Receptor-Interacting Protein Serine-Threonine Kinases - metabolism | Tumor Necrosis Factor-alpha - metabolism | Cell Line | Phosphorylation | Protein Kinases - genetics | Cell Survival | Humans | Protein Multimerization | Receptors, Tumor Necrosis Factor, Type I | Caspase 8 - metabolism | Imidazoles - pharmacology | Receptor-Interacting Protein Serine-Threonine Kinases - genetics | Necrosis - physiopathology | RNA Interference | Indoles - pharmacology | RNA, Small Interfering | Apoptosis - physiology | Enzyme Activation | Receptor-Interacting Protein Serine-Threonine Kinases - antagonists & inhibitors | Tumor Necrosis Factor-alpha - antagonists & inhibitors | Receptor-Interacting Protein Serine-Threonine Kinases/genetics | Protein Kinases/genetics | Receptor-Interacting Protein Serine-Threonine Kinases/antagonists & inhibitors | Caspase 8/metabolism | Receptor-Interacting Protein Serine-Threonine Kinases/metabolism | Tumor Necrosis Factor-alpha/antagonists & inhibitors | Indoles/pharmacology | Life Sciences | Tumor Necrosis Factor-alpha/metabolism | Imidazoles/pharmacology | Immunology | Apoptosis/physiology | Protein Kinases/metabolism | Necrosis/physiopathology | Original Paper
Journal Article
FEBS Letters, ISSN 0014-5793, 2008, Volume 582, Issue 1, pp. 117 - 131
Dyslipidemia and insulin resistance are commonly associated with catabolic or lipodystrophic conditions (such as cancer and sepsis) and with pathological... 
Type 2 diabetes | Obesity | Dyslipidemia | Insulin resistance | Lipid metabolism | Antiadipogenesis | Metabolic syndrome | TNF signalling | C/EBPα | extracellular signal-regulated kinase | white adipose tissue | JNK | inhibitor of NFκB | LPL | WAT | IKKβ | IκB kinase beta | AMP-activated protein kinase | hormone sensitive lipase | IκB | sTNF-α | c-jun N-terminal kinase | tumour necrosis factor-alpha | HSL | NFκB | mTNF-α | transmembrane tumour necrosis factor-alpha | T2D | TNFR2 | nuclear factor-kappa B | soluble tumour necrosis factor-alpha | tumour necrosis factor-alpha receptor 1 | PPARγ | TNFR1 | tumour necrosis factor-alpha receptor 2 | CCAAT/enhancer binding protein | ERK1/2 | type 2 diabetes | TNF-α | brown adipose tissue | BAT | lipoprotein lipase | AMPK | peroxisome proliferator-activated receptor gamma | ACTIVATED RECEPTOR-GAMMA | BIOCHEMISTRY & MOLECULAR BIOLOGY | lipid metabolism | BROWN ADIPOCYTES | MESSENGER-RNA LEVELS | INDUCED INSULIN-RESISTANCE | CELL BIOLOGY | metabolic syndrome | TUMOR-NECROSIS-FACTOR | HUMAN ADIPOSE-TISSUE | BIOPHYSICS | PLECKSTRIN HOMOLOGY DOMAIN | ADIPONECTIN GENE-EXPRESSION | dyslipidemia | insulin resistance | NF-KAPPA-B | obesity | SIGNAL-RELATED KINASE | antiadipogenesis | Animals | Body Temperature Regulation - physiology | Adipocytes - metabolism | Humans | Tumor Necrosis Factor-alpha - physiology | Adipocytes - cytology | Lipid Metabolism | Cell Differentiation - physiology
Journal Article
Neuropsychopharmacology (New York, N.Y.), ISSN 1740-634X, 2011, Volume 36, Issue 5, pp. 979 - 992
Patients with chronic pain usually suffer from working memory deficits, which may decrease their intellectual ability significantly. Despite intensive clinical... 
working memory | spared nerve injury | tumor necrosis factor-α | chronic neuropathic pain | long-term potentiation | presynaptic boutons | ATTENTION | NEUTRALIZING ANTIBODIES | PSYCHIATRY | WALLERIAN DEGENERATION | SYNAPTIC-TRANSMISSION | NECROSIS-FACTOR-ALPHA | NEUROSCIENCES | DORSAL-HORN | tumor necrosis factor-alpha | NEUROPATHIC PAIN | INHIBITION | PHARMACOLOGY & PHARMACY | EXPRESSION | Tumor Necrosis Factor-alpha - metabolism | Electric Stimulation | Tumor Necrosis Factor-alpha - genetics | Male | Memory, Short-Term - physiology | Hippocampus - drug effects | Thalidomide - pharmacology | RNA, Messenger - metabolism | Excitatory Postsynaptic Potentials - physiology | Receptors, Tumor Necrosis Factor, Type I - deficiency | Time Factors | Up-Regulation - physiology | Statistics, Nonparametric | Peripheral Nervous System Diseases - pathology | Peripheral Nervous System Diseases - complications | Immunosuppressive Agents - pharmacology | Disease Models, Animal | Maze Learning - physiology | Rats | Memory, Short-Term - drug effects | Up-Regulation - genetics | Maze Learning - drug effects | Mice, Knockout | Peripheral Nervous System Diseases - metabolism | Up-Regulation - drug effects | Tumor Necrosis Factor-alpha - pharmacology | Animals | Memory Disorders - etiology | Long-Term Potentiation | Hyperalgesia - etiology | Presynaptic Terminals - metabolism | Mice | Hippocampus - physiopathology | learning & memory | pain | plasticity | analgesics | tumor necrosis factor alpha | neuropharmacology | peripheral nerve injury | Original
Journal Article
The Journal of clinical investigation, ISSN 0021-9738, 2009, Volume 119, Issue 10, pp. 3011 - 3023
Cytokines orchestrate the tumor-promoting interplay between malignant cells and the immune system. In many experimental and human cancers, the cytokine... 
RHEUMATOID-ARTHRITIS | MEDICINE, RESEARCH & EXPERIMENTAL | TGF-BETA | INTERLEUKIN-17 FAMILY | IN-VIVO | GROWTH | IL-17-PRODUCING T-CELLS | FACTOR RECEPTOR P55 | PHASE-II | NECROSIS-FACTOR-ALPHA | NF-KAPPA-B | Humans | Tumor Necrosis Factor-alpha - genetics | Interleukin-17 - immunology | Antibodies, Monoclonal - therapeutic use | Gene Expression Profiling | Chimera - genetics | CD4-Positive T-Lymphocytes - immunology | Interleukin-23 - genetics | Ovarian Neoplasms - genetics | Chimera - immunology | Interleukin-2 Receptor alpha Subunit - genetics | Infliximab | Microarray Analysis | Tumor Necrosis Factor-alpha - immunology | Anti-Inflammatory Agents - therapeutic use | Female | Ovarian Neoplasms - drug therapy | Disease Models, Animal | Interleukin-2 Receptor alpha Subunit - immunology | Receptors, Interleukin - immunology | Mice, Inbred C57BL | Receptors, Tumor Necrosis Factor, Type I | Interleukin-17 - genetics | Clinical Trials as Topic | Receptors, Interleukin - genetics | Interleukin-23 - immunology | Mice, Knockout | Animals | Signal Transduction - physiology | Mice | Ovarian Neoplasms - immunology | Interleukin-2 Receptor alpha Subunit | Signal Transduction | Anti-Inflammatory Agents | Interleukin-23 | Receptors, Interleukin | Antibodies, Monoclonal | Tumor Necrosis Factor-alpha | Life Sciences | Immunology | CD4-Positive T-Lymphocytes | Interleukin-17 | Ovarian Neoplasms | Chimera
Journal Article
Cell metabolism, ISSN 1550-4131, 2014, Volume 20, Issue 1, pp. 103 - 118
Chronic inflammation constitutes an important link between obesity and its pathophysiological sequelae. In contrast to the belief that inflammatory signals... 
OBESITY | HOMEOSTASIS | INSULIN-RESISTANCE | ENDOCRINOLOGY & METABOLISM | FAT | NECROSIS-FACTOR-ALPHA | DIFFERENTIATION | IDENTIFICATION | ADIPOGENESIS | EXPRESSION | PROGRESSION | CELL BIOLOGY | Tumor Necrosis Factor-alpha - metabolism | Inflammation - pathology | Adipogenesis - drug effects | Tumor Necrosis Factor-alpha - genetics | Adipose Tissue, White - metabolism | Body Weight - drug effects | Adrenergic Agonists - pharmacology | Male | Serum Amyloid A Protein - genetics | Fatty Acid-Binding Proteins - metabolism | Recombinant Proteins - biosynthesis | Inflammation - metabolism | Leptin - blood | Diet, High-Fat | Adiponectin - metabolism | Female | Models, Animal | Glucose Tolerance Test | Lipopolysaccharides - toxicity | Serum Amyloid A Protein - metabolism | Fatty Liver - physiopathology | Mice, Inbred C57BL | Mice, Transgenic | Recombinant Proteins - genetics | Recombinant Proteins - pharmacology | Intra-Abdominal Fat - metabolism | Fatty Acid-Binding Proteins - genetics | Tumor Necrosis Factor-alpha - pharmacology | Animals | Mice | Adipose Tissue, White - drug effects | Adipose tissues | Glucose metabolism | Analysis | Physiological aspects | Inflammation | Glucose | Cells | Dextrose | Medicinska grundvetenskaper | Basic Medicine | Physiology | Immunologi inom det medicinska området | Immunology in the medical area | Fysiologi
Journal Article