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1. Full Text The genomic complexity of primary human prostate cancer
by Berger, Michael F and Lawrence, Michael S and Demichelis, Francesca and Drier, Yotam and Cibulskis, Kristian and Sivachenko, Andrey Y and Sboner, Andrea and Esgueva, Raquel and Pflueger, Dorothee and Sougnez, Carrie and Onofrio, Robert and Carter, Scott L and Park, Kyung and Habegger, Lukas and Ambrogio, Lauren and Fennell, Timothy and Parkin, Melissa and Saksena, Gordon and Voet, Douglas and Ramos, Alex H and Pugh, Trevor J and Wilkinson, Jane and Fisher, Sheila and Winckler, Wendy and Mahan, Scott and Ardlie, Kristin and Baldwin, Jennifer and Simons, Jonathan W and Kitabayashi, Naoki and MacDonald, Theresa Y and Kantoff, Philip W and Chin, Lynda and Gabriel, Stacey B and Gerstein, Mark B and Golub, Todd R and Meyerson, Matthew and Tewari, Ashutosh and Lander, Eric S and Getz, Gad and Rubin, Mark A and Garraway, Levi A
Nature, ISSN 0028-0836, 02/2011, Volume 470, Issue 7333, pp. 214 - 220
Prostate cancer is the second most common cause of male cancer deaths in the United States. However, the full range of prostate cancer genomic alterations is... 
MYELOID-LEUKEMIA GENOME | GENE FUSIONS | ANDROGEN RECEPTOR | HUMAN BREAST | PATHWAY | MULTIDISCIPLINARY SCIENCES | MUTATION | PTEN | TUMOR-SUPPRESSOR | REARRANGEMENTS | PROGRESSION | Chromatin - metabolism | PTEN Phosphohydrolase - genetics | Cell Adhesion Molecules - genetics | Chromosome Breakpoints | Humans | Gene Expression Regulation, Neoplastic | PTEN Phosphohydrolase - metabolism | Male | Signal Transduction - genetics | Case-Control Studies | Genome, Human - genetics | Carrier Proteins - genetics | Prostatic Neoplasms - genetics | Epigenesis, Genetic - genetics | Chromosome Aberrations | Recombination, Genetic - genetics | Transcription, Genetic | Chromatin - genetics | Care and treatment | Genetic aspects | Prostate cancer | Health aspects | Genomics | Stem cells | Epigenetics | Gene loci | Genetic engineering | Mutation | Stress response | Chromosomes | Deoxyribonucleic acid--DNA
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2. Full Text Widespread deregulation of microRNA expression in human prostate cancer
by Ozen, M and Creighton, C.J and Ozdemir, M and Ittmann, M
Oncogene, ISSN 0950-9232, 03/2008, Volume 27, Issue 12, pp. 1788 - 1793
MicroRNAs (miRNAs) are small regulatory RNAs that can regulate gene expression by binding to mRNA sequences and repressing target-gene expression... 
EIF4EBP1 | miRNA | Microarray | Prostate cancer | BREAST-CANCER | prostate cancer | microarray | ONCOLOGY | BIOCHEMISTRY & MOLECULAR BIOLOGY | SEL1L | GENETICS & HEREDITY | TARGETS | SIGNATURE | CELL BIOLOGY | Prostatic Neoplasms - metabolism | MicroRNAs - antagonists & inhibitors | Oligonucleotide Array Sequence Analysis | Humans | Male | MicroRNAs - biosynthesis | MicroRNAs - metabolism | Up-Regulation - genetics | Gene Expression Profiling | Down-Regulation - genetics | Microspheres | Prostatic Neoplasms - genetics | Gene Expression Regulation, Neoplastic - physiology | Usage | DNA microarrays | Genetic aspects | Research | Gene expression | Risk factors | Oncology | Ribonucleic acid--RNA
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3. Full Text N-Myc Drives Neuroendocrine Prostate Cancer Initiated from Human Prostate Epithelial Cells
by Lee, John K and Phillips, John W and Smith, Bryan A and Park, Jung Wook and Stoyanova, Tanya and McCaffrey, Erin F and Baertsch, Robert and Sokolov, Artem and Meyerowitz, Justin G and Mathis, Colleen and Cheng, Donghui and Stuart, Joshua M and Shokat, Kevan M and Gustafson, W. Clay and Huang, Jiaoti and Witte, Owen N
Cancer Cell, ISSN 1535-6108, 04/2016, Volume 29, Issue 4, pp. 536 - 547
amplification and overexpression are common in neuroendocrine prostate cancer (NEPC). However, the impact of aberrant N-Myc expression in prostate... 
INACTIVATION | ORIGIN | ONCOLOGY | ADENOCARCINOMA | C-MYC | STEM-CELL | DIFFERENTIATION | IDENTIFICATION | CARCINOMA | EXPRESSION | BASAL-CELLS | CELL BIOLOGY | Adenocarcinoma - pathology | Epithelial Cells - metabolism | Humans | Neoplasm Proteins - physiology | Gene Expression Regulation, Neoplastic | Laser Capture Microdissection | Male | Antineoplastic Agents - therapeutic use | Genes, myc | Aurora Kinase A - antagonists & inhibitors | Molecular Targeted Therapy | Recombinant Fusion Proteins - metabolism | Exome | Neoplasm Metastasis | Proto-Oncogene Proteins c-myc - physiology | Prostatic Neoplasms - genetics | Aurora Kinase A - physiology | Neoplastic Stem Cells - metabolism | Cell Transformation, Neoplastic - genetics | Neoplastic Stem Cells - pathology | Adenocarcinoma - genetics | Neoplasm Proteins - genetics | Neuroendocrine Tumors - pathology | Prostatic Neoplasms - pathology | Transduction, Genetic | Neoplasm Invasiveness | Phenylurea Compounds - therapeutic use | Epithelial Cells - pathology | Proto-Oncogene Proteins c-akt - physiology | Mice, SCID | Azepines - therapeutic use | Neuroendocrine Tumors - genetics | Orchiectomy | Xenograft Model Antitumor Assays | Animals | Protein Kinase Inhibitors - therapeutic use | Pyrimidines - therapeutic use | Cell Line, Tumor | Mice, Inbred NOD | Enzyme Activation | Molecular biology | Health aspects | Prostate cancer | Stem cells | Medical genetics | Cell death
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4. Full Text Identification of a Cell of Origin for Human Prostate Cancer
by Andrew S. Goldstein and Jiaoti Huang and Changyong Guo and Isla P. Garraway and Owen N. Witte
Science, ISSN 0036-8075, 7/2010, Volume 329, Issue 5991, pp. 568 - 571
Luminal cells are believed to be the cells of origin for human prostate cancer, because the disease is characterized by luminal cell expansion and the absence... 
Epithelial cells | Neurons | Stromal cells | REPORTS | Stem cells | Basal cell neoplasms | Prostate cancer | Prostate | Cells | Endothelial cells | Cell transplantation | PROGENITORS | STEM-CELLS | EPITHELIAL-CELLS | SUBPOPULATION | MULTIDISCIPLINARY SCIENCES | PTEN | LEADS | INTRAEPITHELIAL NEOPLASIA | EXPRESSION | CARCINOMA | BASAL | Transcriptional Regulator ERG | Prostatic Neoplasms - metabolism | Prostatic Intraepithelial Neoplasia - pathology | Epithelial Cells - metabolism | Humans | Receptors, Androgen - metabolism | Male | Prostate - metabolism | Prostate - pathology | Keratins - analysis | Flow Cytometry | Biomarkers, Tumor - metabolism | Proto-Oncogene Proteins c-akt - metabolism | Prostatic Intraepithelial Neoplasia - metabolism | Prostatic Neoplasms - pathology | Epithelium - pathology | Transduction, Genetic | Cell Separation | Epithelial Cells - pathology | Mice, SCID | Animals | Cell Transformation, Neoplastic | Prostate - cytology | Mice, Inbred NOD | Trans-Activators - metabolism | Mice | Usage | Cancer cells | Physiological aspects | Causes of | Genetic aspects | Histology | Gene expression | Cellular biology | Pathogenesis | Human | Antigens | Origins | Biomedical materials | In vivo testing | In vivo tests | Cancer
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5. Full Text Insulin receptor expression by human prostate cancers
by Cox, Michael E and Gleave, Martin E and Zakikhani, Mahvash and Bell, Robert H and Piura, Esther and Vickers, Elaine and Cunningham, Matthew and Larsson, Ola and Fazli, Ladan and Pollak, Michael
The Prostate, ISSN 0270-4137, 01/2009, Volume 69, Issue 1, pp. 33 - 40
BACKGROUND Although recent laboratory and population studies suggest that prostate cancer may be responsive to insulin, there is a gap in knowledge concerning... 
metabolic syndrome | Gleason grade | prostate cancer | insulin receptor | obesity | insulin‐like growth factor receptor | Insulin-like growth factor receptor | Obesity | Insulin receptor | Metabolic syndrome | Prostate cancer | CELLS | IGF-I | HYBRID RECEPTORS | FACTOR AXIS | RISK | HYPERINSULINEMIA | insulin-like growth factor receptor | ENDOCRINOLOGY & METABOLISM | RESISTANCE | UROLOGY & NEPHROLOGY | PROGRESSION | GROWTH-FACTOR-I | Immunohistochemistry | Antibody Specificity | Prostatic Neoplasms - metabolism | Receptor, IGF Type 1 - metabolism | Oligonucleotide Array Sequence Analysis | Humans | Gene Expression Regulation, Neoplastic | Male | Metabolic Syndrome - metabolism | Prostatic Neoplasms - physiopathology | Liver Neoplasms | RNA, Messenger - metabolism | Receptor, Insulin - genetics | Metabolic Syndrome - physiopathology | Receptor, Insulin - immunology | Hyperinsulinism - physiopathology | Hyperinsulinism - metabolism | Prostatic Neoplasms - pathology | Obesity - physiopathology | Receptor, IGF Type 1 - genetics | Obesity - metabolism | Obesity - pathology | Hyperinsulinism - pathology | Receptor, IGF Type 1 - immunology | Cell Line, Tumor | Receptor, Insulin - metabolism | Signal Transduction - physiology | Metabolic Syndrome - pathology
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6. Full Text SIRT1 is significantly elevated in mouse and human prostate cancer
by Huffman, Derek M and Grizzle, William E and Bamman, Marcas M and Kim, Jeong-Su and Eltoum, Isam A and Elgavish, Ada and Nagy, Tim R
Cancer Research, ISSN 0008-5472, 07/2007, Volume 67, Issue 14, pp. 6612 - 6618
Evidence suggests that the histone deacetylase, SIRT1, is a mediator of life span extension by calorie restriction; however, SIRT1 may paradoxically increase... 
LONGEVITY | ONCOLOGY | DEACETYLASE | ADENOCARCINOMA | TUMOR-SUPPRESSOR | MECHANISMS | BODY-COMPOSITION | HIC-1 GENE | CELL-SURVIVAL | EXPRESSION | CALORIE RESTRICTION | Immunohistochemistry | Prostatic Neoplasms - metabolism | Humans | Mice, Inbred C57BL | Gene Expression Regulation, Neoplastic | Male | Mice, Transgenic | Prostate - metabolism | Sirtuins - biosynthesis | Animals | Prostatic Neoplasms - genetics | Sirtuin 1 | Cell Line, Tumor | Cell Differentiation | Mice | Sirtuins - genetics
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7. Full Text The androgen receptor cistrome is extensively reprogrammed in human prostate tumorigenesis
by Pomerantz, Mark M and Li, Fugen and Takeda, David Y and Lenci, Romina and Chonkar, Apurva and Chabot, Matthew and Cejas, Paloma and Vazquez, Francisca and Cook, Jennifer and Shivdasani, Ramesh A and Bowden, Michaela and Lis, Rosina and Hahn, William C and Kantoff, Philip W and Brown, Myles and Loda, Massimo and Long, Henry W and Freedman, Matthew L
Nature Genetics, ISSN 1061-4036, 11/2015, Volume 47, Issue 11, pp. 1346 - 1351
Master transcription factors interact with DNA to establish cell type identity and to regulate gene expression in mammalian cells(1,2). The genome-wide map of... 
FOXA1 | ELEMENTS | GENE | GENETICS & HEREDITY | TRANSCRIPTIONAL PROGRAM | DIFFERENTIATION | MUTATIONS | HOXB13 | ENHANCERS | CANCER | EXPRESSION | Prostatic Neoplasms - metabolism | Homeodomain Proteins - metabolism | Humans | Receptors, Androgen - metabolism | Immunoblotting | Male | Carcinogenesis - metabolism | Prostate - metabolism | Prostate - pathology | Prostatic Neoplasms - genetics | Chromatin Immunoprecipitation | RNA Interference | Cell Transformation, Neoplastic - genetics | Carcinogenesis - genetics | Kaplan-Meier Estimate | Hepatocyte Nuclear Factor 3-alpha - genetics | Transcriptome - genetics | Binding Sites - genetics | Cell Transformation, Neoplastic - metabolism | Sequence Analysis, DNA | Homeodomain Proteins - genetics | Hepatocyte Nuclear Factor 3-alpha - metabolism | Receptors, Androgen - genetics | Cell Line, Tumor | Protein Binding | Genome-Wide Association Study - methods | Cluster Analysis | Hormone receptors | Epigenetic inheritance | Gene mutations | Oncology, Experimental | Genetic research | Development and progression | Genetic aspects | Research | Prostate cancer | Health aspects | Cancer | Genomes | Tumorigenesis | Gene expression | Experiments | Binding sites | Tumors
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8. Full Text PRUNE2 is a human prostate cancer suppressor regulated by the intronic long noncoding RNA PCA3
by Ahmad Salameh and Alessandro K. Lee and Marina Cardó-Vila and Diana N. Nunes and Eleni Efstathiou and Fernanda I. Staquicini and Andrey S. Dobroff and Serena Marchiò and Nora M. Navone and Hitomi Hosoya and Richard C. Lauer and Sijin Wen and Carolina C. Salmeron and Anh Hoang and Irene Newsham and Leandro A. Lima and Dirce M. Carraro and Salvatore Oliviero and Mikhail G. Kolonin and Richard L. Sidman and Kim-Anh Do and Patricia Troncoso and Christopher J. Logothetis and Ricardo R. Brentani and George A. Calin and Webster K. Cavenee and Emmanuel Dias-Neto and Renata Pasqualini and Wadih Arap
Proceedings of the National Academy of Sciences, ISSN 0027-8424, 07/2015, Volume 112, Issue 27, pp. 8403 - 8408
Prostate cancer antigen 3 (PCA3) is the most specific prostate cancer biomarker but its function remains unknown. Here we identify PRUNE2, a target... 
PCA3 | ADAR | Long noncoding RNA | Prostate cancer | PRUNE2 | BMCC1 | prostate cancer | RETINOBLASTOMA | ACT | GENE | HOMOLOGY BCH DOMAIN | MULTIDISCIPLINARY SCIENCES | long noncoding RNA | EXPRESSION | Prostatic Neoplasms - metabolism | Adenosine Deaminase - metabolism | Humans | Gene Expression Regulation, Neoplastic | Molecular Sequence Data | Immunoblotting | Male | Neoplasm Proteins - metabolism | Prostatic Neoplasms - genetics | MCF-7 Cells | RNA Interference | Tumor Suppressor Proteins - genetics | HEK293 Cells | Antigens, Neoplasm - metabolism | RNA Precursors - metabolism | Neoplasm Proteins - genetics | Antigens, Neoplasm - genetics | Prostatic Neoplasms - pathology | Tumor Suppressor Proteins - metabolism | Introns - genetics | Adenosine Deaminase - genetics | RNA Precursors - genetics | RNA, Long Noncoding - genetics | Mice, SCID | Reverse Transcriptase Polymerase Chain Reaction | Xenograft Model Antitumor Assays - methods | Animals | Cell Line, Tumor | Protein Binding | HeLa Cells | RNA, Long Noncoding - metabolism | RNA sequencing | Genetic aspects | Methods | Biological Sciences
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9. Full Text High aldehyde dehydrogenase activity identifies tumor-initiating and metastasis-initiating cells in human prostate cancer
by Van Den Hoogen, Christel and Van Der Horst, Geertje and Cheung, Henry and Buijs, Jeroen T and Lippitt, Jenny M and Guzmán-Ramírez, Natalia and Hamdy, Freddie C and Eaton, Colby L and Thalmann, George N and Cecchini, Marco G and Pelger, Rob C. M and Van Der Pluijm, Gabri
Cancer Research, ISSN 0008-5472, 06/2010, Volume 70, Issue 12, pp. 5163 - 5173
Metastatic progression of advanced prostate cancer is a major clinical problem. Identifying the cell(s) of origin in prostate cancer and its distant metastases... 
BREAST-CANCER | PROGENITOR CELLS | STEM-CELLS | ONCOLOGY | FUNCTIONAL MARKER | BONE METASTASIS | ACUTE MYELOID-LEUKEMIA | MODEL | EXPRESSION | CD44 | REVEALS | Prostatic Neoplasms - pathology | Bone Neoplasms - enzymology | Cell Proliferation | Humans | Bone Neoplasms - secondary | Male | Blotting, Western | Disease Progression | Immunoenzyme Techniques | Prostate - pathology | Animals | Flow Cytometry | Cell Transformation, Neoplastic | Mice, Nude | Biomarkers, Tumor - metabolism | Cell Line, Tumor | Prostatic Neoplasms - enzymology | Mice | Mice, Inbred BALB C | Prostate - enzymology | Aldehyde Dehydrogenase - metabolism
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10. Full Text Animal models of human prostate cancer: The consensus report of the new york meeting of the mouse models of human cancers consortium prostate pathology committee
by Ittmann, Michael and Huang, Jiaoti and Radaelli, Enrico and Martin, Philip and Signoretti, Sabina and Sullivan, Ruth and Simons, Brian W and Ward, Jerrold M and Robinson, Brian D and Chu, Gerald C and Loda, Massimo and Thomas, George and Borowsky, Alexander and Cardiff, Robert D
Cancer Research, ISSN 0008-5472, 05/2013, Volume 73, Issue 9, pp. 2718 - 2736
Animal models, particularly mouse models, play a central role in the study of the etiology, prevention, and treatment of human prostate cancer. While tissue... 
ANDROGEN RECEPTOR | XENOGRAFT MODELS | TO-MESENCHYMAL TRANSITION | PTEN LOSS | ONCOLOGY | GENETICALLY-ENGINEERED MICE | REACTIVE STROMA | IN-VIVO | GROWTH | INTRAEPITHELIAL NEOPLASIA | EXPRESSION | Genetic Engineering - methods | Neoplasm Transplantation | New York | Prostatic Neoplasms - pathology | Humans | Gene Expression Regulation, Neoplastic | Rats | Male | Prostatic Neoplasms - diagnosis | Consensus | Disease Progression | Neoplasm Metastasis | Adenocarcinoma - metabolism | Animals | Societies, Medical | Mice | Oncogenes | Disease Models, Animal | prostate cancer | animal models | transgenic | pathology | xenograft | genetically engineered mice
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11. Full Text Castration resistance in human prostate cancer is conferred by a frequently occurring androgen receptor splice variant
by Sun, Shihua and Sprenger, Cynthia C. T and Vessella, Robert L and Haugk, Kathleen and Soriano, Kathryn and Mostaghel, Elahe A and Page, Stephanie T and Coleman, Ilsa M and Nguyen, Holly M and Sun, Huiying and Nelson, Peter S and Plymate, Stephen R
Journal of Clinical Investigation, ISSN 0021-9738, 08/2010, Volume 120, Issue 8, pp. 2715 - 2730
Progression of prostate cancer following castration is associated with increased androgen receptor (AR) expression and signaling despite AR blockade. Recent... 
TUMOR-CELL LINES | MEDICINE, RESEARCH & EXPERIMENTAL | ACTIVATION | EPITHELIAL-CELLS | IGF-IR | GENE-EXPRESSION | MUTATIONS | MEDICAL CASTRATION | PROGRESSION | GROWTH-FACTOR-I | T-ANTIGEN | Neoplasm Transplantation | Prostatic Neoplasms - pathology | Humans | Gene Expression Regulation, Neoplastic | Male | Transplantation, Heterologous | Orchiectomy | Prostatic Neoplasms - therapy | Neoplasm Metastasis | RNA Splicing | Animals | Prostatic Neoplasms - genetics | Receptors, Androgen - genetics | Androgen Antagonists - therapeutic use | Cell Line, Tumor | Mice | Hydroxamic Acids - pharmacology | Receptors, Androgen - physiology | Complications and side effects | Hormone receptors | Castration | Development and progression | Properties | Prostate cancer | Risk factors
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12. Full Text Diversity of TMPRSS2-ERG fusion transcripts in the human prostate
by Clark, J and Merson, S and Jhavar, S and Flohr, P and Edwards, S and Foster, C.S and Eeles, R and Martin, F.L and