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The Journal of Pathology, ISSN 0022-3417, 05/2017, Volume 242, Issue 1, pp. 113 - 125
Journal Article
Journal Article
Cancer Letters, ISSN 0304-3835, 2011, Volume 307, Issue 1, pp. 26 - 36
Abstract Activation of Notch-1 is known to be associated with the development and progression of human malignancies including pancreatic cancer. Emerging... 
Hematology, Oncology and Palliative Medicine | CSC-self renewal | MiRNAs | EMT phenotype | Notch-1 | Genistein | miRNAs | TARGETING NOTCH | DOWN-REGULATION | DRUG-RESISTANCE | BREAST-CANCER | MIR-200 | INHIBITION | ONCOLOGY | SIGNALING PATHWAY | PROSTATE-CANCER | DIFFERENTIATION | EXPRESSION | Cell Adhesion Molecules - genetics | Pancreatic Neoplasms - metabolism | Neoplastic Stem Cells - drug effects | Humans | Wound Healing | Epithelial Cell Adhesion Molecule | Neoplastic Stem Cells - metabolism | Epithelial-Mesenchymal Transition | Hyaluronan Receptors - metabolism | Antigens, Neoplasm - metabolism | Neoplastic Stem Cells - pathology | Tumor Cells, Cultured | Spheroids, Cellular | Anticarcinogenic Agents - pharmacology | Antigens, Neoplasm - genetics | Pancreatic Neoplasms - pathology | RNA, Messenger - genetics | Pancreatic Neoplasms - genetics | Receptor, Notch1 - metabolism | Cell Adhesion - drug effects | Reverse Transcriptase Polymerase Chain Reaction | Cell Adhesion Molecules - metabolism | Hyaluronan Receptors - genetics | Blotting, Western | Cell Movement - drug effects | Genistein - pharmacology | Cell Proliferation - drug effects | MicroRNAs - physiology | Receptor, Notch1 - antagonists & inhibitors | Receptor, Notch1 - genetics | Care and treatment | Pancreatic cancer | Stem cells | Development and progression | Genetic aspects | Drug resistance | Intermediate filament proteins | Cancer | Isoflavones | Metastasis | Cancer therapies | Cell adhesion & migration | Studies | Genotype & phenotype | Cell growth | Medical prognosis | Technological change | Ligands | Pancreas | Acquisitions & mergers | Tumors | Index Medicus | genistein
Journal Article
Oncogene, ISSN 0950-9232, 12/2009, Volume 28, Issue 49, pp. 4326 - 4343
Mammographically dense breast tissue is one of the greatest risk factors for developing breast carcinoma, yet the associated molecular mechanisms remain... 
3D matrix/focal adhesion | Breast cancer | Mechanotransduction | Epithelial morphogenesis | Cell-extracellular matrix interaction | Breast tissue density | CANCER RISK | ACTIVATED PROTEIN-KINASE | mechanotransduction | BIOCHEMISTRY & MOLECULAR BIOLOGY | breast cancer | MALIGNANT PHENOTYPE | CARCINOMA IN-SITU | CELL BIOLOGY | MAMMOGRAPHIC DENSITY | STRESS FIBERS | ONCOLOGY | epithelial morphogenesis | EPITHELIAL-SPECIFIC DISRUPTION | GROWTH-FACTORS | GENETICS & HEREDITY | EXTRACELLULAR-MATRIX | FOCAL ADHESION KINASE | breast tissue density | cell-extracellular matrix interaction | Cell Proliferation | Oligonucleotide Array Sequence Analysis | Cell Count | Humans | Breast Neoplasms - etiology | Gene Expression Profiling | Extracellular Signal-Regulated MAP Kinases - metabolism | Mammary Glands, Human - metabolism | Mechanotransduction, Cellular - physiology | Carcinoma - etiology | Extracellular Matrix - chemistry | Breast Neoplasms - metabolism | Cell Transformation, Neoplastic - genetics | Extracellular Matrix - physiology | Gene Expression - physiology | Female | Mammary Glands, Human - physiology | Extracellular Signal-Regulated MAP Kinases - physiology | Focal Adhesion Kinase 1 - metabolism | Mechanotransduction, Cellular - genetics | Mammary Glands, Human - cytology | Risk Factors | Cells, Cultured | Focal Adhesion Kinase 1 - physiology | Cell Transformation, Neoplastic - metabolism | Phenotype | Breast Neoplasms - genetics | Carcinoma - genetics | Carcinoma - metabolism | Physiological aspects | Development and progression | Genetic aspects | Research | Gene expression | Protein kinases | Risk factors | Genotype & phenotype | Cellular biology | Index Medicus | 3D-Matrix | Focal Adhesion | Cell-Extracellular Matrix Interaction | Epithelial Morphogenesis | Breast Cancer | Breast Tissue Density | Tumor-Stroma Interaction
Journal Article
Journal Article
Journal of Cellular Biochemistry, ISSN 0730-2312, 09/2011, Volume 112, Issue 9, pp. 2296 - 2306
Journal Article
Nature Cell Biology, ISSN 1465-7392, 08/2015, Volume 17, Issue 8, pp. 1049 - 1061
The TOR (target of rapamycin) kinase limits longevity by poorly understood mechanisms. Rapamycin suppresses the mammalian TORC1 complex, which regulates... 
EPITHELIAL-CELL LINE | GROWTH ARREST | DNA-DAMAGE | IN-VIVO | INFLAMMATORY CYTOKINE SECRETION | HUMAN FIBROBLASTS | TUMOR-CELLS | CANCER-THERAPY | REPLICATIVE SENESCENCE | P53 | CELL BIOLOGY | Up-Regulation | Fibroblasts - enzymology | Cell Proliferation | TOR Serine-Threonine Kinases - metabolism | Humans | Gene Expression Regulation, Neoplastic | Interleukin-1alpha - metabolism | Male | NF-kappa B - metabolism | RNA, Messenger - metabolism | Prostatic Neoplasms - secretion | Prostatic Neoplasms - immunology | Dose-Response Relationship, Drug | TOR Serine-Threonine Kinases - antagonists & inhibitors | Prostatic Neoplasms - genetics | TOR Serine-Threonine Kinases - genetics | Transfection | RNA Interference | Time Factors | Cellular Senescence | Inflammation Mediators - metabolism | Transcription, Genetic | Antineoplastic Agents - pharmacology | Interleukin-1alpha - genetics | Interleukin-6 - metabolism | Prostatic Neoplasms - drug therapy | Prostatic Neoplasms - pathology | Anti-Inflammatory Agents - pharmacology | Interleukin-1alpha - secretion | Sirolimus - metabolism | Tumor Burden | Mice, SCID | Sirolimus - pharmacology | Xenograft Model Antitumor Assays | Phenotype | Animals | Fibroblasts - drug effects | Cell Line, Tumor | Prostatic Neoplasms - enzymology | Mitoxantrone - pharmacology | Interleukins | Aging | Genetic aspects | Properties | Phosphotransferases | Carcinogenesis | Health aspects | Cells | Index Medicus
Journal Article
Cell Metabolism, ISSN 1550-4131, 10/2015, Volume 22, Issue 4, pp. 590 - 605
Journal Article
Oncogene, ISSN 0950-9232, 08/2009, Volume 28, Issue 33, pp. 2940 - 2947
Journal Article