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PLoS ONE, ISSN 1932-6203, 06/2014, Volume 9, Issue 6, p. e97957
The omega-3 (omega 3) fatty acid docosahexaenoic acid (DHA) can suppress inflammation, specifically IL-1 beta production through poorly understood molecular... 
SIGNALS | CELLS | GPR120 | RECEPTORS | MULTIDISCIPLINARY SCIENCES | Inflammasomes - metabolism | Calcium - metabolism | Humans | NF-kappa B - metabolism | Arrestins - metabolism | Autophagy - drug effects | Cell Nucleus - metabolism | Interleukin-1beta - biosynthesis | Macrophages - immunology | Interleukin-1beta - secretion | Intracellular Space - drug effects | Bone Marrow Cells - cytology | Mice, Inbred C57BL | Macrophages - cytology | Gene Expression Regulation - drug effects | Docosahexaenoic Acids - pharmacology | Macrophages - metabolism | Animals | beta-Arrestins | Active Transport, Cell Nucleus - drug effects | Intracellular Space - metabolism | Lipopolysaccharides - pharmacology | Macrophages - drug effects | Mice | Receptors, G-Protein-Coupled - genetics | HeLa Cells | Cell Nucleus - drug effects | Macrophage Activation - drug effects | Biotechnology | G protein-coupled receptors | Laboratories | Activation | Macrophages | Docosahexaenoic acid | Autophagy | Cell activation | Immunology | Rodents | Bone marrow | Life sciences | Inhibition | Adenosine triphosphate | Chronic illnesses | Translocation | NF-κB protein | Adapters | Priming | Inflammation | Exposure | IL-1β | Metabolism | Fatty acids | Nuclear transport | Infectious diseases | Molecular modelling | Adapter proteins | Phagocytosis | Cancer | Apoptosis
Journal Article
Archives of Toxicology, ISSN 0340-5761, 05/2016, Volume 90, Issue 5, pp. 1163 - 1179
Drug-induced liver injury (DILI) is an important problem both in the clinic and in the development of new safer medicines. Two pivotal adaptation and survival... 
NF-κB signaling | Oxidative stress | Drug-induced liver injury | Live-cell imaging | Nrf2 activation | INDUCED CELL-DEATH | HEPATOCYTES | INDUCTION | LIVER-INJURY | PATHWAY | EXPLORATION | HEPG2 CELLS | ONTOLOGY | MICE | TOXICOLOGY | NF-kappa B signaling | EXPRESSION | Liver - pathology | Humans | Databases, Genetic | Hepatocytes - pathology | Green Fluorescent Proteins - genetics | Hepatocytes - metabolism | RNA, Messenger - metabolism | Transcription Factor RelA - genetics | Dose-Response Relationship, Drug | Transfection | Liver - drug effects | RNA Interference | Time Factors | NF-E2-Related Factor 2 - genetics | Active Transport, Cell Nucleus | Chemical and Drug Induced Liver Injury - pathology | Hepatocytes - drug effects | Recombinant Fusion Proteins - biosynthesis | Liver - metabolism | RNA, Messenger - genetics | Computational Biology | Gene Expression Profiling - methods | Oxidoreductases Acting on Sulfur Group Donors - biosynthesis | Chemical and Drug Induced Liver Injury - genetics | Tumor Necrosis Factor-alpha - toxicity | Hep G2 Cells | Gene Expression Regulation - drug effects | Signal Transduction - drug effects | Transcription Factor RelA - metabolism | Chemical and Drug Induced Liver Injury - metabolism | Green Fluorescent Proteins - biosynthesis | NF-E2-Related Factor 2 - metabolism | Oxidoreductases Acting on Sulfur Group Donors - genetics | Index Medicus | Organ Toxicity and Mechanisms
Journal Article
Cell Death and Differentiation, ISSN 1350-9047, 01/2010, Volume 17, Issue 1, pp. 25 - 34
  Protein ubiquitination is a reversible reaction, in which the ubiquitin chains are deconjugated by a family of deubiquitinases (DUBs). The presence of a... 
CYLD | NF-κB | tumor suppressor | deubiquitination | deubiquitinase
Journal Article
Journal Article
Biochemical Pharmacology, ISSN 0006-2952, 12/2008, Volume 76, Issue 11, pp. 1485 - 1489
Oxidative stress has been implicated in the etiology of neurodegenerative disease, cancer and aging. Indeed, accumulation of reactive oxygen and nitrogen... 
NFκB | Nrf2 | DSS | AOM | Apoptosis | PHENETHYL ISOTHIOCYANATE | RECOGNITION MODEL | PROTEIN | SULFORAPHANE | KEAP1 | INHIBITION | CUL3-BASED E3 LIGASE | PATHWAY | NRF2 POSSESSES | PROSTATE-CANCER CELLS | PHARMACOLOGY & PHARMACY | NF kappa B | NF-κB | apoptosis
Journal Article