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Nature Medicine, ISSN 1078-8956, 03/2018, Volume 24, Issue 3, pp. 292 - 303
Adipocytes possess remarkable adaptive capacity to respond to nutrient excess, fasting or cold exposure, and they are thus an important cell type for the... 
MEDICINE, RESEARCH & EXPERIMENTAL | UNFOLDED-PROTEIN RESPONSE | BIOCHEMISTRY & MOLECULAR BIOLOGY | DIET-INDUCED THERMOGENESIS | ADULT HUMANS | ENDOPLASMIC-RETICULUM STRESS | CELL BIOLOGY | LINKS OBESITY | INSULIN-RESISTANCE | FAT | MITOCHONDRIAL DYSFUNCTION | DEGRADATION | TRANSCRIPTION FACTOR | Thermogenesis - genetics | Cold Temperature | Endoplasmic Reticulum - genetics | Humans | Endoplasmic Reticulum - metabolism | Homeostasis | NF-E2-Related Factor 1 - genetics | Obesity - physiopathology | Mitochondria - metabolism | Endoplasmic Reticulum Stress - genetics | Obesity - genetics | Proteasome Endopeptidase Complex - genetics | Animals | Mitochondria - genetics | Gene Deletion | Insulin Resistance - genetics | Inflammation - genetics | Models, Animal | Adipose Tissue, Brown - metabolism | Acclimatization - genetics | Proteasome Endopeptidase Complex - metabolism | Acclimatization - physiology | Inflammation - physiopathology | Adipose tissues | Obesity | Transcription factors | Care and treatment | Cell metabolism | Development and progression | Genetic aspects | Health aspects | Animal models | Adipose tissue | Proteasome activator | Adipocytes | Metabolism | Insulin | Adaptive control | Mitochondria | Thermogenesis | Diet | Clonal deletion | Rodents | Quality control | Proteasomes | Deletion | Adipose tissue (brown) | Endoplasmic reticulum | Adaptation | proteasome | brown adipose tissue | inflammation | proteostasis | insulin resistance | obesity
Journal Article
The Journal of Physiology, ISSN 0022-3751, 09/2016, Volume 594, Issue 18, pp. 5195 - 5207
Journal Article
Cancer Cell, ISSN 1535-6108, 10/2011, Volume 20, Issue 4, pp. 524 - 537
Journal Article
Journal Article
Journal Article
Archives of Toxicology, ISSN 0340-5761, 1/2013, Volume 87, Issue 1, pp. 49 - 72
The completion of the human genome project and the subsequent advent of DNA microarray and high-throughput sequencing technologies have led to a renaissance in... 
Mitochondria | Biomedicine | Environmental Health | Occupational Medicine/Industrial Medicine | Transcriptomics | Pharmacology/Toxicology | Nuclear receptors | Transcription factor | Differentiation | Biomedicine general | Xenobiotic | UNFOLDED-PROTEIN RESPONSE | PROLIFERATOR-ACTIVATED RECEPTORS | OVALBUMIN UPSTREAM PROMOTER | PHOSPHOENOLPYRUVATE-CARBOXYKINASE GENE | ENDOPLASMIC-RETICULUM STRESS | ORPHAN NUCLEAR RECEPTOR | MEDIATED GENE-EXPRESSION | ARYL-HYDROCARBON RECEPTOR | RETINOIC ACID RECEPTOR | TOXICOLOGY | NF-KAPPA-B | Basic-Leucine Zipper Transcription Factors - metabolism | Humans | Toxicogenetics - methods | Receptors, Aryl Hydrocarbon - genetics | STAT Transcription Factors - metabolism | Receptors, Cytoplasmic and Nuclear - genetics | Basic-Leucine Zipper Transcription Factors - genetics | Organ Specificity | Nuclear Respiratory Factor 1 - genetics | Xenobiotics - pharmacology | Gene Expression Regulation - drug effects | Genes, p53 | STAT Transcription Factors - genetics | NF-E2-Related Factor 2 - metabolism | Nuclear Respiratory Factor 1 - metabolism | Receptors, Aryl Hydrocarbon - metabolism | NF-E2-Related Factor 2 - genetics | Oxidative Stress - drug effects | Receptors, Cytoplasmic and Nuclear - metabolism | DNA microarrays | Genes | Genomics | Genetic research | Heat shock proteins | Genetic aspects | Mitochondrial DNA | Universities and colleges | Tumor proteins | Xenobiotics | Gene expression | Toxicology | Molecular biology | Index Medicus
Journal Article
Free Radical Biology and Medicine, ISSN 0891-5849, 2009, Volume 46, Issue 2, pp. 127 - 137
It is well recognized that oxidized LDL (OxLDL) plays a crucial role in the initiation and progression of atherosclerosis. Many biological effects of OxLDL are... 
Oxidative stress | Transcription factors | Free radicals | Gene expression | Oxidized LDL | CYTOSOLIC PHOSPHOLIPASE A | BIOCHEMISTRY & MOLECULAR BIOLOGY | MONOCYTE CHEMOATTRACTANT PROTEIN-1 | FACTOR-KAPPA-B | NECROSIS-FACTOR-ALPHA | DEPENDENT KINASE INHIBITOR | HUMAN ENDOTHELIAL-CELLS | HUMAN ATHEROSCLEROTIC LESIONS | HEME OXYGENASE-1 INDUCTION | ENDOCRINOLOGY & METABOLISM | SMOOTH-MUSCLE-CELLS | GROWTH-FACTOR EXPRESSION | Oxidative Stress | Transcription Factor AP-1 - genetics | Atherosclerosis - genetics | Humans | NF-kappa B - immunology | Peroxisome Proliferator-Activated Receptors - immunology | Transcription Factor AP-1 - immunology | NF-kappa B - metabolism | Smad3 Protein - immunology | STAT Transcription Factors - immunology | Smad3 Protein - metabolism | Transcriptional Activation - immunology | Lipoproteins, LDL - genetics | Hypoxia-Inducible Factor 1 - metabolism | Transcription Factor AP-1 - metabolism | NF-E2-Related Factor 2 - immunology | Tumor Suppressor Protein p53 - genetics | Hypoxia-Inducible Factor 1 - genetics | Smad3 Protein - genetics | NFATC Transcription Factors - immunology | NF-E2-Related Factor 2 - genetics | Lipoproteins, LDL - metabolism | Peroxisome Proliferator-Activated Receptors - genetics | Atherosclerosis - immunology | Signal Transduction | NFATC Transcription Factors - metabolism | Tumor Suppressor Protein p53 - metabolism | Hypoxia-Inducible Factor 1 - immunology | Inflammation | Tumor Suppressor Protein p53 - immunology | Feedback, Physiological | Animals | Cell Cycle | NF-kappa B - genetics | NF-E2-Related Factor 2 - metabolism | Peroxisome Proliferator-Activated Receptors - metabolism | Lipoproteins, LDL - immunology | NFATC Transcription Factors - genetics | Low density lipoproteins | Development and progression | Superoxide dismutase | Superoxide | DNA binding proteins | Transforming growth factors | T cells | Macrophages | Proteins | Atherosclerosis | Genetic aspects | Macrophage colony stimulating factor | Vascular endothelial growth factor
Journal Article
The FEBS Journal, ISSN 1742-464X, 01/2017, Volume 284, Issue 2, pp. 183 - 195
Mitochondria are semi‐autonomous organelles of prokaryotic origin that are postulated to have been acquired by eukaryotic cells through an early endosymbiotic... 
mitophagy | nuclear respiratory factors | autophagy | mitochondrial biogenesis | Parkin | NUCLEAR RESPIRATORY FACTOR-2 | BIOCHEMISTRY & MOLECULAR BIOLOGY | DAMAGED MITOCHONDRIA | QUALITY-CONTROL | CELL-SURVIVAL | MEDIATES MITOPHAGY | SKELETAL-MUSCLE | EMERGING ROLE | ENDOPLASMIC-RETICULUM | TRANSCRIPTION FACTOR | PARKIN RECRUITMENT | Diabetes Mellitus, Type 2 - genetics | Humans | Homeostasis | NF-E2-Related Factor 1 - genetics | Mitochondrial Proteins - genetics | Diabetes Mellitus, Type 2 - metabolism | Cardiovascular Diseases - pathology | Cardiovascular Diseases - genetics | Organelle Biogenesis | Mitochondria - genetics | Mitochondrial Proteins - metabolism | Argonaute Proteins - metabolism | Argonaute Proteins - genetics | Neurodegenerative Diseases - pathology | Cardiovascular Diseases - metabolism | Gene Expression Regulation | Lipid Metabolism | Neurodegenerative Diseases - genetics | Mitochondria - metabolism | Neurodegenerative Diseases - metabolism | NF-E2-Related Factor 1 - metabolism | Iron - metabolism | Mitochondrial Degradation | Diabetes Mellitus, Type 2 - pathology | Apoptosis | Biosynthesis | Mitochondria | Molecular biology | Rodents | Correlation | Alterations | Energy | Physiology | Lipid metabolism | Damage | Cellular manufacture | Stresses | Cell survival | Neurodegenerative diseases | Health | Diabetes mellitus | Mortality | Maintenance | Metabolism | Organelles | Survival | Diseases | Neurological diseases | Surveys | Molecular modelling | Cell death
Journal Article