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Journal Article
NATURE COMMUNICATIONS, ISSN 2041-1723, 01/2017, Volume 8, Issue 1, p. 14121
Journal Article
Nature Communications, ISSN 2041-1723, 07/2016, Volume 7, Issue 1, pp. 12057 - 12057
Journal Article
Journal Article
Nature Communications, ISSN 2041-1723, 12/2018, Volume 9, Issue 1, pp. 1423 - 12
Beta-transducin repeat containing E3 ubiquitin protein ligase (BTRC) is crucial for the degradation of I kappa B alpha. Our previous transcriptome sequencing... 
UBIQUITINATION | HEPATOCELLULAR-CARCINOMA | INVASION | GENE | MIR-339-5P | MULTIDISCIPLINARY SCIENCES | KINASE | ALPHA | TETRASPANIN FUNCTIONS | CANCER | EXPRESSION | Tumor Necrosis Factor-alpha - metabolism | Neoplasm Transplantation | RNA, Small Interfering - genetics | Carcinoma, Squamous Cell - genetics | Carcinoma, Squamous Cell - metabolism | Carcinoma, Squamous Cell - pathology | Humans | Middle Aged | Tumor Necrosis Factor-alpha - genetics | Gene Expression Regulation, Neoplastic | Male | MicroRNAs - metabolism | NF-KappaB Inhibitor alpha - genetics | NF-kappa B - metabolism | Esophageal Neoplasms - pathology | Matrix Metalloproteinase 9 - metabolism | beta-Transducin Repeat-Containing Proteins - metabolism | Neoplasm Grading | Matrix Metalloproteinase 9 - genetics | Esophageal Neoplasms - metabolism | Female | Vascular Cell Adhesion Molecule-1 - genetics | Chemokine CCL2 - metabolism | Tetraspanins - genetics | Tetraspanins - metabolism | beta-Transducin Repeat-Containing Proteins - genetics | Esophageal Squamous Cell Carcinoma | Signal Transduction | Chemokine CCL2 - genetics | Lymphatic Metastasis | Urokinase-Type Plasminogen Activator - genetics | Tetraspanins - antagonists & inhibitors | Intercellular Adhesion Molecule-1 - metabolism | Animals | Esophageal Neoplasms - genetics | NF-kappa B - genetics | Urokinase-Type Plasminogen Activator - metabolism | Mice, Nude | Intercellular Adhesion Molecule-1 - genetics | NF-KappaB Inhibitor alpha - metabolism | Cell Line, Tumor | Aged | Mice | MicroRNAs - genetics | Neoplasm Staging | Vascular Cell Adhesion Molecule-1 - metabolism | RNA, Small Interfering - metabolism | Ubiquitin | Transducin | Metastasis | Tissues | Lymph nodes | Metastases | Degradation | Ubiquitination | Transcription activation | Bioindicators | Ubiquitin-protein ligase | Translocation | NF-κB protein | Squamous cell carcinoma | Intercellular adhesion molecule 1 | Therapeutic applications | Tumor necrosis factor-α | Gene expression | Nuclear transport | Gelatinase B | Esophagus | Signaling | Proteasomes | Biomarkers | Monocyte chemoattractant protein 1
Journal Article
STEM CELLS, ISSN 1066-5099, 11/2016, Volume 34, Issue 11, pp. 2707 - 2720
Osteogenic differentiation and bone formation is suppressed under condition of inflammation induced by proinflammation cytokines. A number of studies indicate... 
lncRNA | NF‐κB | Human adipose‐derived stem cells | Osteogenic differentiation | MIR31HG | Human adipose-derived stem cells | NF-κB | lncRNA MIR31HG | TRANSCRIPTION FACTORS | REGENERATION | CELL & TISSUE ENGINEERING | CELL BIOLOGY | BREAST-CANCER | NF-kappa B | LONG NONCODING RNA | ONCOLOGY | BONE-FORMATION | NUCLEAR-FACTOR | BIOTECHNOLOGY & APPLIED MICROBIOLOGY | INFLAMMATION | OSTEOBLASTS | GENE-EXPRESSION | HEMATOLOGY | NF-KAPPA-B | RNA, Small Interfering - genetics | Humans | Adipose Tissue - cytology | NF-KappaB Inhibitor alpha - genetics | Transplantation, Heterologous | Stem Cells - cytology | NF-kappa B p50 Subunit - genetics | Stem Cells - metabolism | Transcription Factor RelA - genetics | Stem Cell Transplantation | Adipose Tissue - metabolism | Cell Differentiation | Osteoblasts - cytology | Osteogenesis - genetics | Tissue Engineering | Promoter Regions, Genetic | NF-kappa B p50 Subunit - metabolism | Signal Transduction | Osteoblasts - drug effects | Osteogenesis - drug effects | Gene Expression Regulation | RNA, Long Noncoding - genetics | Feedback, Physiological | Animals | Transcription Factor RelA - metabolism | Mice, Nude | NF-KappaB Inhibitor alpha - metabolism | Lipopolysaccharides - pharmacology | Protein Binding | Stem Cells - drug effects | Mice, Inbred BALB C | Primary Cell Culture | Osteoblasts - metabolism | RNA, Long Noncoding - antagonists & inhibitors | Adipose Tissue - drug effects | RNA, Long Noncoding - metabolism | RNA, Small Interfering - metabolism | Tissue engineering | Stem cells | Inflammation | NF-κB protein | Cytokines | Circuits | Engineering | Bone growth | Biomedical materials | Biocompatibility | Bone (long) | Bone | Inhibition | Differentiation | In vitro methods and tests | Osteogenesis
Journal Article
Oncotarget, ISSN 1949-2553, 2016, Volume 7, Issue 48, pp. 78883 - 78895
The nuclear export protein, exportin-1 (XPO1/CRM1), is overexpressed in many cancers and correlates with poor prognosis. Selinexor, a first-in-class Selective... 
NF-κB | XPO1 | SINE | Proteasome inhibitors | Selinexor | SIGNAL | ACTIVATION | PHOSPHORYLATION | proteasome inhibitors | NECROSIS-FACTOR-ALPHA | CRM1 | CANCER | CELL BIOLOGY | NF-kappa B | selinexor | SERUM-LEVELS | PROTEINS | CORRELATE | KINASES | Osteosarcoma - drug therapy | Phosphorylation | Humans | Drug Resistance, Neoplasm | NF-KappaB Inhibitor alpha - genetics | NF-kappa B - metabolism | Bortezomib - pharmacology | Bone Neoplasms - pathology | Transcription Factor RelA - genetics | Antineoplastic Combined Chemotherapy Protocols - pharmacology | Dose-Response Relationship, Drug | Cell Nucleus - metabolism | Fibrosarcoma - pathology | Transfection | RNA Interference | Time Factors | Cell Nucleus - pathology | Proteolysis | Female | Bone Neoplasms - drug therapy | Cell Death - drug effects | Hydrazines - pharmacology | Bone Neoplasms - enzymology | Osteosarcoma - enzymology | Proteasome Inhibitors - pharmacology | Mice, SCID | Mice, Inbred ICR | Drug Synergism | Triazoles - pharmacology | Fibrosarcoma - genetics | Animals | Karyopherins - metabolism | NF-kappa B - genetics | Signal Transduction - drug effects | Transcription Factor RelA - metabolism | Active Transport, Cell Nucleus - drug effects | NF-KappaB Inhibitor alpha - metabolism | Receptors, Cytoplasmic and Nuclear - antagonists & inhibitors | Cell Line, Tumor | Proteasome Endopeptidase Complex - metabolism | Cell Nucleus - drug effects | Fibrosarcoma - enzymology | Karyopherins - antagonists & inhibitors | Fibrosarcoma - drug therapy | Osteosarcoma - pathology | Receptors, Cytoplasmic and Nuclear - metabolism
Journal Article
Scientific Reports, ISSN 2045-2322, 06/2016, Volume 6, Issue 1, p. 27026
The present study was designed to determine whether sulfur dioxide (SO2) could be endogenously produced in adipocyte and served as a novel adipocyte-derived... 
3T3-L1 ADIPOCYTES | SYSTEM | METABOLISM | INSULIN-RESISTANCE | MULTIDISCIPLINARY SCIENCES | GENE-EXPRESSION | SENSITIVITY | GENERATION | SECRETION | TISSUES | SULFIDE | Aspartate Aminotransferases - genetics | Interleukin-8 - genetics | RNA, Small Interfering - genetics | Nitriles - pharmacology | Transcription Factor RelA - antagonists & inhibitors | Adipose Tissue, White - metabolism | Adipocytes - cytology | Male | NF-KappaB Inhibitor alpha - genetics | Sulfones - pharmacology | Anti-Inflammatory Agents - metabolism | Transcription Factor RelA - genetics | Aspartate Aminotransferases - antagonists & inhibitors | Adipose Tissue, White - cytology | Thiocarbamates - pharmacology | Isoenzymes - metabolism | Proline - pharmacology | Chemokine CCL2 - metabolism | Interleukin-8 - metabolism | Phosphorylation - drug effects | Proline - analogs & derivatives | Signal Transduction | Isoenzymes - genetics | Gene Expression Regulation | Sulfur Dioxide - metabolism | Rats | Chemokine CCL2 - genetics | 3T3-L1 Cells | Rats, Sprague-Dawley | Aspartate Aminotransferases - metabolism | Tumor Necrosis Factor-alpha - pharmacology | Adipose Tissue, Brown - cytology | Animals | Transcription Factor RelA - metabolism | Adipocytes - metabolism | NF-KappaB Inhibitor alpha - metabolism | Adipose Tissue, Brown - metabolism | Mice | Isoenzymes - antagonists & inhibitors | RNA, Small Interfering - metabolism
Journal Article