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Proceedings of the National Academy of Sciences - PNAS, ISSN 1091-6490, 2016, Volume 113, Issue 51, pp. 14745 - 14750
Journal Article
by Park, SE and Sapkota, K and Kim, S and Kim, H and Kim, SJ
British Journal of Pharmacology, ISSN 0007-1188, 10/2011, Volume 164, Issue 3, pp. 1008 - 1025
...), MAPK cascades, PKB (AKT) and NF‐κB. Accumulation of reaction oxygen species (ROS) was measured by nitroblue tetrazolium and 2′7... 
mitogen‐activated protein kinases | NF‐κB | kaempferol | neuroinflammation | AKT | lipopolysaccharide | microglia | NF-κB | mitogen-activated protein kinases | NITRIC-OXIDE SYNTHASE | ALZHEIMERS-DISEASE | NEURODEGENERATIVE DISEASES | MATRIX METALLOPROTEINASES | NF-kappa B | TOLL-LIKE RECEPTORS | GENE-EXPRESSION | PHARMACOLOGY & PHARMACY | RAT-BRAIN | NF-KAPPA-B | INNATE IMMUNITY | PARKINSONS-DISEASE | Microglia - metabolism | Reactive Oxygen Species - metabolism | Dinoprostone - biosynthesis | NF-kappa B - metabolism | Mitogen-Activated Protein Kinase Kinases - metabolism | Cyclooxygenase 2 - genetics | Nitric Oxide Synthase Type II - antagonists & inhibitors | Protein Binding - drug effects | p38 Mitogen-Activated Protein Kinases - metabolism | Phosphorylation - drug effects | Toll-Like Receptor 4 - antagonists & inhibitors | Proto-Oncogene Proteins c-akt - metabolism | Interleukin-1beta - antagonists & inhibitors | Cell Survival - drug effects | Nitric Oxide - biosynthesis | NF-kappa B - antagonists & inhibitors | Phagocytosis - drug effects | Microglia - drug effects | Signal Transduction | Anti-Inflammatory Agents - pharmacology | Nitric Oxide - antagonists & inhibitors | Cells, Cultured | Microglia - enzymology | Matrix Metalloproteinase 3 - metabolism | Toll-Like Receptor 4 - metabolism | Reactive Oxygen Species - antagonists & inhibitors | Animals | Nitric Oxide Synthase Type II - genetics | p38 Mitogen-Activated Protein Kinases - antagonists & inhibitors | Cyclooxygenase 2 - metabolism | Lipopolysaccharides - pharmacology | Matrix Metalloproteinase Inhibitors | Kaempferols - pharmacology | Mice | Dinoprostone - antagonists & inhibitors | Proto-Oncogene Proteins c-akt - antagonists & inhibitors | Tumor Necrosis Factor-alpha - antagonists & inhibitors | Kinases | Research Papers
Journal Article
The New England journal of medicine, ISSN 1533-4406, 2011, Volume 365, Issue 7, pp. 620 - 628
Journal Article
PLoS ONE, ISSN 1932-6203, 12/2013, Volume 8, Issue 12, p. e80873
...) and RAW264.7 macrophages stimulated by the receptor activator of nuclear factor-kappaB (NF-kappa B) ligand (RANKL). Acteoside pretreatment also prevented bone resorption by mature osteoclasts in a dose-dependent manner... 
REHMANNIA-GLUTINOSA | ACTIVATION | MACROPHAGES | MULTIDISCIPLINARY SCIENCES | RECEPTOR | DIFFERENTIATION | MOLECULAR-BASIS | CELL | MICE LACKING | OSTEOPOROSIS | BONE-RESORPTION | Interleukin-6 - antagonists & inhibitors | RANK Ligand - metabolism | Reactive Oxygen Species - metabolism | NF-kappa B - metabolism | Osteoclasts - cytology | Bone Marrow Cells - drug effects | Female | Cell Differentiation | Interleukin-1beta - biosynthesis | p38 Mitogen-Activated Protein Kinases - metabolism | Interleukin-1beta - antagonists & inhibitors | Phenols - pharmacology | Proto-Oncogene Proteins c-fos - antagonists & inhibitors | Cell Survival - drug effects | Glucosides - pharmacology | NF-kappa B - antagonists & inhibitors | Ovariectomy | Signal Transduction | Bone Marrow Cells - cytology | NFATC Transcription Factors - metabolism | Gene Expression Regulation | Proto-Oncogene Proteins c-fos - metabolism | p38 Mitogen-Activated Protein Kinases - genetics | Antioxidants - pharmacology | Macrophages - cytology | Osteoclasts - metabolism | Mice, Inbred ICR | Bone Resorption - prevention & control | RANK Ligand - genetics | Macrophages - metabolism | Reactive Oxygen Species - antagonists & inhibitors | Animals | NF-kappa B - genetics | RANK Ligand - antagonists & inhibitors | p38 Mitogen-Activated Protein Kinases - antagonists & inhibitors | Interleukin-6 - biosynthesis | Macrophages - drug effects | Proto-Oncogene Proteins c-fos - genetics | Mice | NFATC Transcription Factors - antagonists & inhibitors | Tumor Necrosis Factor-alpha - biosynthesis | Bone Marrow Cells - metabolism | Osteoclasts - drug effects | Tumor Necrosis Factor-alpha - antagonists & inhibitors | NFATC Transcription Factors - genetics | Osteoprogenitor cells | Transcription factors | Phosphorylation | Interleukin | Arthritis | Activation | Kinases | Macrophages | Interleukin 6 | Antioxidants | Bone resorption | Osteoporosis | Bone growth | Bone marrow | Tumor necrosis factor-TNF | Biocompatibility | Inhibition | Pretreatment | Bone loss | TRANCE protein | c-Fos protein | NF-κB protein | Cytokines | c-Jun protein | Pharmacology | Inflammation | Dentistry | Tumor necrosis factor-α | Orthodontics | Medicine | Osteoclastogenesis | Stem cells | Ligands | Osteoclasts | Laboratory animals | Differentiation
Journal Article
Cancer Biology & Therapy, ISSN 1538-4047, 07/2010, Volume 10, Issue 1, pp. 19 - 29
Journal Article
Scientific reports, ISSN 2045-2322, 2018, Volume 8, Issue 1, pp. 998 - 10
Radiation-induced lung injury has restricted radiotherapy for thoracic cancer. The purpose of this study was to investigate the radioprotective effects of... 
CANCER PATIENTS | BROMODOMAINS | TGF-BETA | THERAPY | INFLAMMATION | MULTIDISCIPLINARY SCIENCES | BRD4 INHIBITION | PNEUMONITIS | MECHANISMS | PULMONARY-FIBROSIS | NORMAL TISSUE-INJURY | Humans | NF-kappa B - metabolism | Smad2 Protein - antagonists & inhibitors | Collagen Type I - genetics | MCF-7 Cells | Smad2 Protein - genetics | Lung - radiation effects | Pulmonary Fibrosis - etiology | Fibroblasts - metabolism | NF-kappa B - antagonists & inhibitors | Smad2 Protein - metabolism | Rats | Pulmonary Fibrosis - pathology | Rats, Sprague-Dawley | Smad3 Protein - antagonists & inhibitors | Fibroblasts - drug effects | Proto-Oncogene Proteins c-myc - antagonists & inhibitors | Pulmonary Fibrosis - prevention & control | Cell Line, Tumor | Gene Expression Regulation - radiation effects | Fibroblasts - cytology | Proto-Oncogene Proteins c-myc - genetics | Hydroxyproline - antagonists & inhibitors | Proteins - antagonists & inhibitors | Gamma Rays - adverse effects | Hydroxyproline - biosynthesis | Pulmonary Fibrosis - genetics | Smad3 Protein - metabolism | Molecular Targeted Therapy | Smad3 Protein - genetics | Transforming Growth Factor beta - antagonists & inhibitors | Female | Lung - metabolism | Nuclear Proteins - genetics | Lung - pathology | Collagen Type I - metabolism | Collagen Type I - antagonists & inhibitors | Nuclear Proteins - metabolism | Transcription Factors - antagonists & inhibitors | Transcription Factors - genetics | Proto-Oncogene Proteins c-myc - metabolism | Azepines - pharmacology | Gene Expression Regulation - drug effects | Proteins - genetics | Transcription Factors - metabolism | Triazoles - pharmacology | Animals | Proteins - metabolism | Transforming Growth Factor beta - genetics | Fibroblasts - radiation effects | NF-kappa B - genetics | Nuclear Proteins - antagonists & inhibitors | Cell Proliferation - drug effects | Transforming Growth Factor beta - metabolism | NF-κB protein | Collagen (type I) | Animal models | Cell survival | Lung diseases | Hydroxyproline | Radiation | c-Myc protein | Smad3 protein | Thorax | Breast cancer | Inflammation | Radiation therapy | Myc protein | Esophagus | Computed tomography | Smad2 protein | Rodents | Fibrosis | Fibroblasts | Cancer
Journal Article
Nature (London), ISSN 1476-4687, 2016, Volume 538, Issue 7623, pp. 114 - 117
... Villalobos [4]; Jasper Edgar Neggers [5]; Saurabh Mendiratta [1]; Shuguang Wei [6]; Yosef Landesman [7]; William Senapedis [7]; Erkan Baloglu [7]; Chi-Wan B. Chow [4... 
TARGET | CRM1/XPO1 | ACTIVATION | MODELS | MULTIDISCIPLINARY SCIENCES | KINASE | YAP | MUTATIONS | INHIBITORS | IDENTIFICATION | Lung Neoplasms - drug therapy | Proto-Oncogene Proteins p21(ras) - genetics | Humans | Lung Neoplasms - metabolism | Cell Survival - genetics | Lung Neoplasms - pathology | NF-kappa B - metabolism | Phosphoproteins - antagonists & inhibitors | Phosphoproteins - metabolism | DNA-Binding Proteins - metabolism | Cell Nucleus - metabolism | RNA Interference | Adaptor Proteins, Signal Transducing - antagonists & inhibitors | Female | Verteporfin | Protein-Serine-Threonine Kinases - metabolism | Carcinoma, Non-Small-Cell Lung - pathology | Follistatin-Related Proteins - genetics | Lung Neoplasms - genetics | Cell Survival - drug effects | NF-kappa B - antagonists & inhibitors | DNA-Binding Proteins - antagonists & inhibitors | Signal Transduction | Carcinoma, Non-Small-Cell Lung - genetics | Carcinoma, Non-Small-Cell Lung - metabolism | Nuclear Proteins - metabolism | Transcription Factors - antagonists & inhibitors | Porphyrins - pharmacology | Transcription Factors - metabolism | Xenograft Model Antitumor Assays | Animals | Karyopherins - metabolism | Active Transport, Cell Nucleus - drug effects | Genes, Lethal - genetics | NF-KappaB Inhibitor alpha - metabolism | Nuclear Proteins - antagonists & inhibitors | Receptors, Cytoplasmic and Nuclear - antagonists & inhibitors | Cell Line, Tumor | Cell Proliferation - drug effects | Mice | RNA, Small Interfering | Carcinoma, Non-Small-Cell Lung - drug therapy | Mutation | Adaptor Proteins, Signal Transducing - metabolism | Cell Nucleus - drug effects | Karyopherins - antagonists & inhibitors | Receptors, Cytoplasmic and Nuclear - metabolism | Care and treatment | Genetic aspects | Gene mutations | Health aspects | Guanosine triphosphatase | Lung cancer | Genomics | Genomes | Kinases | Drug resistance | Gene expression | Drug dosages
Journal Article
Journal Article
Nature medicine, ISSN 1546-170X, 2016, Volume 22, Issue 12, pp. 1411 - 1420
Journal Article
Journal of Molecular and Cellular Cardiology, ISSN 0022-2828, 2014, Volume 72, pp. 85 - 94
...) across endothelial cells and how NF-κB and PPAR-γ were involved in this process. TNF-α... 
Cardiovascular | NF-κB | TNF-α | LDL | PPAR-γ | Atherosclerosis | APOPTOSIS | CARDIAC & CARDIOVASCULAR SYSTEMS | ANTAGONIST | TNF-alpha | LOW-DENSITY-LIPOPROTEIN | PPAR-gamma | CELL BIOLOGY | TUMOR-NECROSIS-FACTOR | TRANSPORT | NF-kappa B | INHIBITION | INFLAMMATION | ACTIVATED-RECEPTOR-GAMMA | EXPRESSION | CAVEOLIN-1 | Tumor Necrosis Factor-alpha - metabolism | RNA, Small Interfering - genetics | Human Umbilical Vein Endothelial Cells - metabolism | Nitriles - pharmacology | Atherosclerosis - genetics | Humans | NF-kappa B - metabolism | Filipin - pharmacology | PPAR gamma - metabolism | Sulfones - pharmacology | Caveolin 2 - antagonists & inhibitors | Thiocarbamates - pharmacology | Human Umbilical Vein Endothelial Cells - cytology | Proline - pharmacology | Benzamides - pharmacology | Lipoproteins, LDL - metabolism | PPAR gamma - genetics | Caveolin 1 - antagonists & inhibitors | Receptors, LDL - genetics | Atherosclerosis - pathology | NF-kappa B - antagonists & inhibitors | Human Umbilical Vein Endothelial Cells - drug effects | Proline - analogs & derivatives | Signal Transduction | Atherosclerosis - chemically induced | Receptors, LDL - antagonists & inhibitors | Gene Expression Regulation | Caveolin 1 - genetics | Receptors, LDL - metabolism | Mice, Knockout | Caveolin 1 - metabolism | Caveolin 2 - metabolism | Cinchona Alkaloids - pharmacology | Tumor Necrosis Factor-alpha - pharmacology | Animals | PPAR gamma - antagonists & inhibitors | Lipoproteins, LDL - antagonists & inhibitors | NF-kappa B - genetics | Anilides - pharmacology | Mice | Pyridines - pharmacology | Transcytosis - drug effects | Caveolin 2 - genetics | Atherosclerosis - prevention & control | RNA, Small Interfering - metabolism
Journal Article
International immunopharmacology, ISSN 1567-5769, 2012, Volume 12, Issue 2, pp. 368 - 377
Journal Article