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Proceedings of the National Academy of Sciences - PNAS, ISSN 1091-6490, 2011, Volume 108, Issue 4, pp. 1573 - 1578
...) proteins and of calcium oscillations that became apparent 1-3 d after TNF stimulation. These signaling events culminated in the induction and activation of the calcium-dependent transcription factor, nuclear factor of activated T cells (NFAT... 
T lymphocytes | Myeloid cells | Monocytes | Receptors | Osteoclasts | Bones | Gene expression regulation | Inflammation | Macrophages | Cell fusion | Calcium signaling | Polykaryon | TRANSCRIPTION FACTORS | RHEUMATOID-ARTHRITIS | RANKL | DENDRITIC CELLS | MULTIDISCIPLINARY SCIENCES | calcium signaling | BONE HOMEOSTASIS | polykaryon | TUMOR-NECROSIS-FACTOR | NFAT ACTIVATION | CALCINEURIN | OSTEOCLAST DIFFERENTIATION | EXPRESSION | Gene Expression - drug effects | Calcium - metabolism | Monocytes - cytology | Humans | Immunoblotting | Monocytes - metabolism | NF-kappa B - metabolism | Osteoclasts - cytology | Transcription Factor AP-1 - metabolism | RANK Ligand - pharmacology | Time Factors | Tumor Necrosis Factors - pharmacology | Intracellular Space - drug effects | NFATC Transcription Factors - metabolism | Cells, Cultured | Macrophages - cytology | Reverse Transcriptase Polymerase Chain Reaction | Osteoclasts - metabolism | Monocytes - drug effects | Macrophages - metabolism | Animals | Signal Transduction - drug effects | Cell Differentiation - drug effects | Intracellular Space - metabolism | Macrophages - drug effects | Microscopy, Fluorescence | Osteoclasts - drug effects | NFATC Transcription Factors - genetics | Proteins | Calcium, Dietary | Tumor necrosis factor | Research | T cells | Gene expression | Properties | Biological Sciences
Journal Article
The Journal of biological chemistry, ISSN 1083-351X, 2011, Volume 286, Issue 13, pp. 11275 - 11282
.... As lymphocyte activation is involved in the pathogenesis of many of these diseases, T cells are postulated to be a primary target in minocycline therapy... 
RHEUMATOID-ARTHRITIS | SYSTEMIC-LUPUS-ERYTHEMATOSUS | PROTEIN-KINASE-C | BIOCHEMISTRY & MOLECULAR BIOLOGY | TRANSCRIPTION FACTOR NFAT1 | GENE-EXPRESSION | DOUBLE-BLIND | PLACEBO-CONTROLLED TRIAL | HUMAN-IMMUNODEFICIENCY-VIRUS | LYMPHOCYTE-ACTIVATION | DISEASE PROGRESSION | Enzyme Activators - pharmacology | Minocycline - pharmacology | Humans | NF-kappa B - metabolism | Transcription Factor AP-1 - metabolism | Arthritis, Rheumatoid - metabolism | Cell Nucleus - metabolism | Protein Isoforms - metabolism | Arthritis, Rheumatoid - drug therapy | Phosphorylation - drug effects | Huntington Disease - drug therapy | Multiple Sclerosis - metabolism | Anti-Inflammatory Agents - pharmacology | CD4-Positive T-Lymphocytes - cytology | NFATC Transcription Factors - metabolism | CD4-Positive T-Lymphocytes - metabolism | Cells, Cultured | Enzyme Activation - drug effects | Glycogen Synthase Kinase 3 - metabolism | Huntington Disease - metabolism | NF-kappa B - genetics | Calcium Signaling - drug effects | Lymphocyte Activation - drug effects | Active Transport, Cell Nucleus - drug effects | Anti-Bacterial Agents - pharmacology | Multiple Sclerosis - drug therapy | NFAT Transcription Factor | Immunosupressor | Transcription Regulation | Immunology | Signal Transduction | Transcription Factors | Cellular Immune Response | Lymphocyte
Journal Article
Journal Article
Journal Article
Arteriosclerosis, thrombosis, and vascular biology, ISSN 1524-4636, 2010, Volume 30, Issue 2, pp. 218 - 224
.... Recently, we reported that high glucose activates the Ca/calcineurin-dependent transcription factor nuclear factor of activated T cells (NFAT... 
UTP | Hyperglycemia | Inflammation | Diabetes | Vascular complications | NFAT | TRANSCRIPTION FACTORS | CAROTID-ARTERY | ATHEROSCLEROSIS | NEOINTIMA FORMATION | hyperglycemia | vascular complications | PLASMA | inflammation | UPSTREAM STIMULATORY FACTOR | CALCIUM | HIGH GLUCOSE | PERIPHERAL VASCULAR DISEASE | GENERATION | ACCUMULATION | HEMATOLOGY | diabetes | Osteopontin - genetics | Uridine Triphosphate - metabolism | Diabetes Mellitus, Experimental - drug therapy | Osteopontin - deficiency | Muscle, Smooth, Vascular - metabolism | Cyclosporine - pharmacology | Humans | Transcriptional Activation | Diabetic Angiopathies - etiology | Hyperglycemia - complications | Male | Osteopontin - metabolism | RNA, Messenger - metabolism | Hyperglycemia - drug therapy | Arteries - metabolism | Calcineurin Inhibitors | Transfection | Time Factors | Apyrase - pharmacology | Female | Diabetes Mellitus, Experimental - complications | Diabetic Angiopathies - prevention & control | Diabetes Mellitus, Experimental - metabolism | Binding Sites | Disease Models, Animal | Muscle, Smooth, Vascular - drug effects | Pyrazoles - pharmacology | Diabetic Angiopathies - metabolism | Promoter Regions, Genetic | Glucose Tolerance Test | Signal Transduction | Jurkat Cells | Mice, Inbred C57BL | NFATC Transcription Factors - metabolism | Enzyme Inhibitors - pharmacology | Mice, Knockout | Hyperglycemia - metabolism | Animals | Mice | Mice, Inbred BALB C | Blood Glucose - metabolism | NFATC Transcription Factors - deficiency | NFATC Transcription Factors - antagonists & inhibitors | Calcineurin - metabolism | NFATC Transcription Factors - genetics
Journal Article
Journal Article
The Journal of surgical research, ISSN 0022-4804, 06/2018, Volume 226, pp. 40 - 47
Nuclear factor of activated T-cells 5 (NFAT5) is known to be correlated with migration or invasion of tumor cells based on previous in vitro studies... 
Non–small cell lung cancer | Biomarker | Nuclear factor of activated T-cells 5 | MIGRATION | SURGERY | PROLIFERATION | INDUCTION | IDENTIFICATION | NFAT | Non-small cell lung cancer | CALCIUM | CALCINEURIN | GENE FAMILY | PROTEINS | TRANSCRIPTION FACTOR
Journal Article