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Journal Article
Cell, ISSN 0092-8674, 2006, Volume 126, Issue 2, pp. 375 - 387
Journal Article
Science, ISSN 0036-8075, 6/2011, Volume 332, Issue 6037, pp. 1565 - 1568
Synthetic biology has advanced the design of genetic devices that can be used to reprogram metabolic activities in mammalian cells. By functionally linking the... 
Cell culture techniques | Diabetes complications | HEK293 cells | Bioreactors | Medical treatment | REPORTS | Cell lines | Mice | Diabetes | Gene expression | Transgenes | RETINAL GANGLION-CELLS | RESPONSES | VISUAL FUNCTION | MELANOPSIN | ROD | MULTIDISCIPLINARY SCIENCES | LIGHT | ECTOPIC EXPRESSION | PHOTOPIGMENT | MAMMALIAN-CELLS | DEGENERATION | Diabetes Mellitus, Type 2 - genetics | Humans | Alkaline Phosphatase - metabolism | Homeostasis | Diabetes Mellitus, Type 2 - metabolism | Insulin - blood | Glucagon-Like Peptide 1 - genetics | Rod Opsins - genetics | Light Signal Transduction | Transfection | Isoenzymes - metabolism | Light | Rod Opsins - metabolism | Transcription, Genetic | Genes, Reporter | Genetic Engineering - methods | Cell Line | Alkaline Phosphatase - genetics | Glucagon-Like Peptide 1 - metabolism | Signal Transduction | Isoenzymes - genetics | NFATC Transcription Factors - metabolism | Gene Expression Regulation | GPI-Linked Proteins - metabolism | Animals | Synthetic Biology - methods | Cell Line, Tumor | Optical Fibers | Blood Glucose - metabolism | GPI-Linked Proteins - genetics | NFATC Transcription Factors - genetics | Blood sugar | Physiological aspects | Cellular signal transduction | Genetic aspects | Synthetic biology | Research | Genetic transcription | Glucose | Chemical synthesis | Rodents | Blood
Journal Article
Developmental Cell, ISSN 1534-5807, 2002, Volume 3, Issue 6, pp. 889 - 901
Signaling by RANKL is essential for terminal differentiation of monocytes/mcrophages into osteoclasts. The TRAF6 and c-Fos signaling pathways both play... 
LYMPH-NODE ORGANOGENESIS | CELLS | CYCLOSPORINE-A | OSTEOPROTEGERIN-LIGAND | KEY REGULATOR | C-FOS | RECEPTOR ACTIVATOR | GENE-EXPRESSION | DEVELOPMENTAL BIOLOGY | OSTEOPETROSIS | NF-KAPPA-B | CELL BIOLOGY | Transcription, Genetic - drug effects | Genetic Testing | Membrane Glycoproteins - metabolism | Oligonucleotide Array Sequence Analysis | RANK Ligand | Osteoclasts - cytology | Promoter Regions, Genetic - drug effects | RNA, Messenger - metabolism | Calcineurin - genetics | Cell Culture Techniques | Active Transport, Cell Nucleus - genetics | Signal Transduction - genetics | Nuclear Proteins | Signal Transduction - drug effects | Calcium Signaling - drug effects | Active Transport, Cell Nucleus - drug effects | Hematopoietic Stem Cells - cytology | Proto-Oncogene Proteins c-fos - genetics | Mice | Transcription, Genetic - genetics | Calcium Signaling - genetics | Receptor Activator of Nuclear Factor-kappa B | Transcription Factors - deficiency | Promoter Regions, Genetic - genetics | DNA-Binding Proteins - deficiency | Cell Differentiation - genetics | Carrier Proteins - pharmacology | Hematopoietic Stem Cells - drug effects | Gene Expression Regulation - genetics | Membrane Glycoproteins - pharmacology | Mice, Inbred C57BL | Cells, Cultured | Proto-Oncogene Proteins c-fos - metabolism | Hematopoietic Stem Cells - metabolism | NFATC Transcription Factors | Transcription Factors - genetics | DNA-Binding Proteins - genetics | Osteoclasts - metabolism | Membrane Glycoproteins - genetics | Gene Expression Regulation - drug effects | Proteins - genetics | Carrier Proteins - genetics | TNF Receptor-Associated Factor 6 | Animals | Carrier Proteins - metabolism | Proteins - metabolism | Cell Differentiation - drug effects | Calcineurin - metabolism | Osteoclasts - drug effects
Journal Article
Immunity, ISSN 1074-7613, 02/2013, Volume 38, Issue 2, pp. 225 - 236
It is widely appreciated that T cells increase glycolytic flux during activation, but the role of mitochondrial flux is unclear. Here, we have shown that... 
COMPLEX-III | SUPEROXIDE | PROTEIN | GLUCOSE-METABOLISM | LYMPHOCYTE ACTIVATION | IMMUNE-RESPONSES | AEROBIC GLYCOLYSIS | TRANSDUCTION | PROLIFERATION | IMMUNOLOGY | GLUTAMINE UPTAKE | Electron Transport Complex III - metabolism | Cell Proliferation | Reactive Oxygen Species - metabolism | Iron-Sulfur Proteins - genetics | Homeodomain Proteins - immunology | Mitochondria - immunology | Interleukin-2 - immunology | NFATC Transcription Factors - immunology | Mitochondria - genetics | T-Lymphocytes - metabolism | Lymphopenia - metabolism | Female | Tumor Necrosis Factor Receptor Superfamily, Member 7 - immunology | Gene Expression | Signal Transduction | Lymphocyte Activation | Mitochondria - metabolism | Tumor Necrosis Factor Receptor Superfamily, Member 7 - genetics | Homeodomain Proteins - genetics | Mice, Knockout | Animals | Iron-Sulfur Proteins - deficiency | T-Lymphocytes - immunology | Lymphopenia - immunology | Mice | Interleukin-2 - biosynthesis | NFATC Transcription Factors - genetics | Antigens | Medical colleges | Glucose metabolism | Cytochrome b | Mitochondrial DNA | Glucose | T cells | Dextrose | Flow cytometry | Oxygen | Lymphatic system | Transcription factors | Laboratories | T cell receptors | Infections | Biosynthesis | Metabolism | Cell growth | Mitochondria | Metabolites | Immune system | Apoptosis
Journal Article
Nature, ISSN 0028-0836, 06/2006, Volume 441, Issue 7093, pp. 595 - 600
Journal Article
Circulation, ISSN 0009-7322, 09/2006, Volume 114, Issue 11, pp. 1159 - 1168
Background - Cellular hypertrophy requires coordinated regulation of progrowth and antigrowth mechanisms. In cultured neonatal cardiomyocytes, Foxo... 
Calcineurin | Angiotensin | Hypertrophy | SKELETAL-MUSCLE ATROPHY | ACTIVATION | CARDIAC & CARDIOVASCULAR SYSTEMS | PROTEIN | hypertrophy | UBIQUITIN LIGASE | PROTEASOMAL DEGRADATION | AKT | calcineurin | PATHWAY | DOWNSTREAM | IN-VIVO | angiotensin | PERIPHERAL VASCULAR DISEASE | MICE | HEMATOLOGY | Cardiomegaly - pathology | Male | Intracellular Signaling Peptides and Proteins - metabolism | Phosphatidylinositol 3-Kinases - metabolism | Proto-Oncogene Proteins c-akt - genetics | Forkhead Transcription Factors - metabolism | Muscle Proteins - metabolism | Phosphoric Monoester Hydrolases - drug effects | Proto-Oncogene Proteins c-akt - metabolism | Intracellular Signaling Peptides and Proteins - genetics | Angiotensin II - pharmacology | Myocytes, Cardiac - cytology | Mice, Inbred C57BL | NFATC Transcription Factors - metabolism | Cells, Cultured | Cardiomegaly - physiopathology | Gene Expression Regulation - physiology | Rats | Transcription Factors - genetics | Forkhead Transcription Factors - genetics | Nerve Tissue Proteins - genetics | Rats, Sprague-Dawley | Calcineurin - physiology | Muscle Proteins - genetics | Nerve Tissue Proteins - metabolism | Phosphatidylinositol 3-Kinases - genetics | Transcription Factors - metabolism | Animals | Myocytes, Cardiac - drug effects | Myocytes, Cardiac - metabolism | Signal Transduction - physiology | Forkhead Box Protein O1 | Mice | Cardiomegaly - genetics | Forkhead Box Protein O3 | Phosphoric Monoester Hydrolases - metabolism | NFATC Transcription Factors - genetics
Journal Article
Nature, ISSN 0028-0836, 06/2010, Volume 465, Issue 7299, pp. 808 - 812
Journal Article
Journal Article