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1. Full Text Epigenetic Modification and Antibody-Dependent Expansion of Memory-like NK Cells in Human Cytomegalovirus-Infected Individuals
by Lee, Jaewon and Zhang, Tianxiang and Hwang, Ilwoong and Kim, Ahrom and Nitschke, Larissa and Kim, MinJae and Scott, Jeannine M and Kamimura, Yosuke and Lanier, Lewis L and Kim, Sungjin
Immunity (Cambridge, Mass.), ISSN 1074-7613, 03/2015, Volume 42, Issue 3, pp. 431 - 442
Life Sciences & Biomedicine | Immunology | Science & Technology | Cell Proliferation | Humans | NK Cell Lectin-Like Receptor Subfamily C - genetics | Receptors, IgG - deficiency | Intracellular Signaling Peptides and Proteins - immunology | Cytomegalovirus Infections - immunology | Gene Expression Profiling | Killer Cells, Natural - pathology | Cytomegalovirus Infections - genetics | Protein-Tyrosine Kinases - deficiency | Protein-Tyrosine Kinases - immunology | Intracellular Signaling Peptides and Proteins - deficiency | NK Cell Lectin-Like Receptor Subfamily C - immunology | DNA Methylation | Protein-Tyrosine Kinases - genetics | Receptors, IgG - genetics | Microarray Analysis | Antibodies - immunology | Cytomegalovirus Infections - virology | Killer Cells, Natural - immunology | Cytomegalovirus Infections - pathology | Intracellular Signaling Peptides and Proteins - genetics | Syk Kinase | Promoter Regions, Genetic | GPI-Linked Proteins - immunology | Signal Transduction | Killer Cells, Natural - virology | NK Cell Lectin-Like Receptor Subfamily C - deficiency | Immunophenotyping | Receptors, IgG - immunology | Killer Cells, Natural - classification | Cytomegalovirus - immunology | Epigenesis, Genetic - immunology | Immunologic Memory | GPI-Linked Proteins - genetics | Proteins | Viral antibodies | Epigenetic inheritance | Killer cells | Molecular genetics | Antibodies | Gene expression | Antigens | Cytomegalovirus | Cloning | Colleges & universities | Infections | Experiments | Long term | DNA methylation | Epigenetics | Expansion | Viral infections | Deoxyribonucleic acid--DNA | Immune system | Index Medicus
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2. Full Text Cytokine activation induces human memory-like NK cells
by Romee, Rizwan and Schneider, Stephanie E and Leong, Jeffrey W and Chase, Julie M and Keppel, Catherine R and Sullivan, Ryan P and Cooper, Megan A and Fehniger, Todd A
Blood, ISSN 0006-4971, 12/2012, Volume 120, Issue 24, pp. 4751 - 4760
Life Sciences & Biomedicine | Hematology | Science & Technology | Hematologic and hematopoietic diseases | Biological and medical sciences | Medical sciences | Receptors, Interleukin-12 - immunology | Antigens, CD - immunology | Interleukin-18 - immunology | Humans | Lectins, C-Type - immunology | Interferon-gamma - metabolism | Interleukin-15 - immunology | Antigens, CD - metabolism | NK Cell Lectin-Like Receptor Subfamily D - immunology | Lectins, C-Type - metabolism | NK Cell Lectin-Like Receptor Subfamily C - immunology | Lymphocyte Activation - immunology | Flow Cytometry | Time Factors | Killer Cells, Natural - immunology | Receptors, Interleukin-12 - genetics | Interferon-gamma - genetics | CD56 Antigen - immunology | Immunologic Memory - immunology | Cytokines - immunology | Interleukin-12 - pharmacology | Receptors, Interleukin-18 - metabolism | CD56 Antigen - metabolism | NK Cell Lectin-Like Receptor Subfamily C - metabolism | Antigens, Differentiation, T-Lymphocyte - immunology | Antigens, Differentiation, T-Lymphocyte - metabolism | Reverse Transcriptase Polymerase Chain Reaction | Interleukin-15 - pharmacology | Receptors, Interleukin-12 - metabolism | Lymphocyte Activation - drug effects | Interferon-gamma - immunology | Interleukin-12 - immunology | K562 Cells | Receptors, Interleukin-18 - genetics | Receptors, Interleukin-18 - immunology | Cell Proliferation - drug effects | Interleukin-18 - pharmacology | Killer Cells, Natural - drug effects | Killer Cells, Natural - metabolism | NK Cell Lectin-Like Receptor Subfamily D - metabolism | Cytokines - pharmacology | Index Medicus | Abridged Index Medicus | Immunobiology
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3. Full Text The Egyptian Rousette Genome Reveals Unexpected Features of Bat Antiviral Immunity
by Pavlovich, Stephanie S and Lovett, Sean P and Koroleva, Galina and Guito, Jonathan C and Arnold, Catherine E and Nagle, Elyse R and Kulcsar, Kirsten and Lee, Albert and Thibaud-Nissen, Françoise and Hume, Adam J and Mühlberger, Elke and Uebelhoer, Luke S and Towner, Jonathan S and Rabadan, Raul and Sanchez-Lockhart, Mariano and Kepler, Thomas B and Palacios, Gustavo
Cell (Cambridge), ISSN 0092-8674, 05/2018, Volume 173, Issue 5, pp. 1098 - 1110.e18
type I interferon | genome | antiviral immunity | innate immunity | natural killer cell receptors | filovirus | Chiroptera | Biochemistry & Molecular Biology | Life Sciences & Biomedicine | Science & Technology | Cell Biology | Chiroptera - immunology | Marburg Virus Disease - immunology | Disease Reservoirs - virology | Humans | Egypt | Immunity, Innate - genetics | NK Cell Lectin-Like Receptor Subfamily C - genetics | Phylogeny | Genetic Variation | NK Cell Lectin-Like Receptor Subfamily C - classification | NK Cell Lectin-Like Receptor Subfamily D - genetics | Amino Acid Sequence | Cell Line | Chiroptera - classification | Marburgvirus - physiology | Marburg Virus Disease - pathology | Interferon Type I - classification | Chromosome Mapping | Histocompatibility Antigens Class I - genetics | Sequence Alignment | Animals | NK Cell Lectin-Like Receptor Subfamily C - chemistry | Histocompatibility Antigens Class I - classification | NK Cell Lectin-Like Receptor Subfamily D - chemistry | Interferon Type I - genetics | NK Cell Lectin-Like Receptor Subfamily D - classification | Genome | Chiroptera - genetics | Evolution, Molecular | Bats | Medical colleges | Medical research | Microbiology | Genomics | Genomes | Biological response modifiers | Marburg virus disease | Virus diseases | Killer cells | Anopheles | Analysis | Medicine, Experimental | Interferon | Index Medicus
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4. Full Text Cytomegalovirus Infection Drives Adaptive Epigenetic Diversification of NK Cells with Altered Signaling and Effector Function
by Schlums, Heinrich and Cichocki, Frank and Tesi, Bianca and Theorell, Jakob and Beziat, Vivien and Holmes, Tim D and Han, Hongya and Chiang, Samuel C.C and Foley, Bree and Mattsson, Kristin and Larsson, Stella and Schaffer, Marie and Malmberg, Karl-Johan and Ljunggren, Hans-Gustaf and Miller, Jeffrey S and Bryceson, Yenan T
Immunity (Cambridge, Mass.), ISSN 1074-7613, 03/2015, Volume 42, Issue 3, pp. 443 - 456
Life Sciences & Biomedicine | Immunology | Science & Technology | Cell Proliferation | Humans | Transcription Factors - deficiency | NK Cell Lectin-Like Receptor Subfamily C - genetics | Receptors, IgG - deficiency | Intracellular Signaling Peptides and Proteins - immunology | Cytomegalovirus Infections - immunology | Gene Expression Profiling | Killer Cells, Natural - pathology | Cytomegalovirus Infections - genetics | Protein-Tyrosine Kinases - deficiency | Protein-Tyrosine Kinases - immunology | T-Lymphocytes, Cytotoxic - pathology | Intracellular Signaling Peptides and Proteins - deficiency | NK Cell Lectin-Like Receptor Subfamily C - immunology | DNA Methylation | Protein-Tyrosine Kinases - genetics | Receptors, IgG - genetics | Microarray Analysis | Antibodies - immunology | Cytomegalovirus Infections - virology | Killer Cells, Natural - immunology | Cytomegalovirus Infections - pathology | Kruppel-Like Transcription Factors - deficiency | Transcription Factors - immunology | Intracellular Signaling Peptides and Proteins - genetics | Syk Kinase | Adaptive Immunity | T-Lymphocytes, Cytotoxic - immunology | Promoter Regions, Genetic | GPI-Linked Proteins - immunology | Signal Transduction | Killer Cells, Natural - virology | T-Lymphocytes, Cytotoxic - virology | NK Cell Lectin-Like Receptor Subfamily C - deficiency | Immunophenotyping | Receptors, IgG - immunology | Transcription Factors - genetics | Killer Cells, Natural - classification | Promyelocytic Leukemia Zinc Finger Protein | Cytomegalovirus - immunology | Epigenesis, Genetic - immunology | Kruppel-Like Transcription Factors - genetics | Kruppel-Like Transcription Factors - immunology | GPI-Linked Proteins - genetics | Virus diseases | Proteins | Epigenetic inheritance | Killer cells | Immunotherapy | Genomics | Medical genetics | Cytomegalovirus infections | T cells | Methylation | Health aspects | Antigens | Flow cytometry | Cytomegalovirus | Data analysis | Biomedical research | Transplants & implants | Cytokines | Leukemia | Cytotoxicity | Infections | Kinases | Diversification | Gene expression | Experiments | Scholarships & fellowships | Lymphocytes | DNA methylation | Epigenetics | Viral infections | Immune system | Cancer | Index Medicus | Medicin och hälsovetenskap
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5. Full Text Impaired natural killer cell self-education and "missing-self" responses in Ly49-deficient mice
by Beĺanger, Simon and Tu, Megan M and Rahim, Mir Munir Ahmed and Mahmoud, Ahmad B and Patel, Rajen and Tai, Lee-Hwa and Troke, Angela D and Wilhelm, Brian T and Landry, Josette-Renée and Zhu, Qinzhang and Tung, Kenneth S and Raulet, David H and Makrigiannis, Andrew P
Blood, ISSN 0006-4971, 07/2012, Volume 120, Issue 3, pp. 592 - 602
Life Sciences & Biomedicine | Hematology | Science & Technology | Hematologic and hematopoietic diseases | Biological and medical sciences | Medical sciences | Antigens, Ly - genetics | NK Cell Lectin-Like Receptor Subfamily K - immunology | Lectins, C-Type - genetics | Cell Differentiation - genetics | Lectins, C-Type - metabolism | Neoplasms - genetics | Mice, Mutant Strains | NK Cell Lectin-Like Receptor Subfamily D - genetics | Killer Cells, Natural - immunology | NK Cell Lectin-Like Receptor Subfamily A - genetics | Receptors, Immunologic - immunology | Mice, Inbred C57BL | Histocompatibility Antigens Class I - immunology | Gene Silencing - immunology | Histocompatibility Antigens Class I - genetics | Cell Degranulation - immunology | NK Cell Lectin-Like Receptor Subfamily A - immunology | NK Cell Lectin-Like Receptor Subfamily K - genetics | Cell Differentiation - immunology | Killer Cells, Natural - cytology | Animals | Antigens, Ly - immunology | Neoplasms - immunology | Cell Line, Tumor | Transsexualism - genetics | Mice | Receptors, Immunologic - genetics | NK Cell Lectin-Like Receptor Subfamily D - metabolism | Receptors, Immunologic - metabolism | Index Medicus | Abridged Index Medicus | Immunobiology
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6. Full Text The killer cell lectin‐like receptor B1 (KLRB1) 503T>C polymorphism (rs1135816) and acute rejection after liver transplantation
by Thude, H and Rother, S and Sterneck, M and Klempnauer, J and Nashan, B and Schwinzer, R and Koch, M
HLA : immune response genetics, ISSN 2059-2302, 01/2018, Volume 91, Issue 1, pp. 52 - 55
killer cell lectin‐like receptor B1 | acute rejection | CD161 | liver transplantation | killer cell lectin-like receptor B1 | Pathology | Immunology | Life Sciences & Biomedicine | Science & Technology | Cell Biology | Acute Disease | Genetic Predisposition to Disease | Liver Transplantation | Humans | Middle Aged | Male | NK Cell Lectin-Like Receptor Subfamily B - immunology | NK Cell Lectin-Like Receptor Subfamily B - genetics | Adult | Female | Aged | Polymorphism, Single Nucleotide | Graft Rejection - immunology | Graft Rejection - genetics
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7. Full Text SHIP1 intrinsically regulates NK cell signaling and education, resulting in tolerance of an MHC class I-mismatched bone marrow graft in mice
by Gumbleton, Matthew and Vivier, Eric and Kerr, William G
The Journal of immunology (1950), ISSN 0022-1767, 03/2015, Volume 194, Issue 6, pp. 2847 - 2854
Life Sciences & Biomedicine | Immunology | Science & Technology | Self Tolerance - immunology | Inositol Polyphosphate 5-Phosphatases | Phosphoric Monoester Hydrolases - deficiency | NK Cell Lectin-Like Receptor Subfamily K - immunology | Interferon-gamma - metabolism | Mice, 129 Strain | Bone Marrow Transplantation - methods | Haptens - immunology | NK Cell Lectin-Like Receptor Subfamily D - immunology | Dermatitis, Contact - immunology | Flow Cytometry | Phosphoric Monoester Hydrolases - immunology | NK Cell Lectin-Like Receptor Subfamily K - metabolism | Natural Cytotoxicity Triggering Receptor 1 - genetics | Self Tolerance - genetics | Killer Cells, Natural - immunology | Natural Cytotoxicity Triggering Receptor 1 - immunology | Cytokines - immunology | Dermatitis, Contact - genetics | NK Cell Lectin-Like Receptor Subfamily A - metabolism | Phosphoric Monoester Hydrolases - genetics | Cytokines - metabolism | Mice, Inbred C57BL | Histocompatibility Antigens Class I - immunology | Homeostasis - immunology | Mice, Transgenic | NK Cell Lectin-Like Receptor Subfamily A - immunology | Histocompatibility - immunology | Mice, Knockout | Animals | Interferon-gamma - immunology | Natural Cytotoxicity Triggering Receptor 1 - metabolism | Graft Rejection - immunology | Killer Cells, Natural - metabolism | Graft Rejection - genetics | Phosphatidylinositol-3,4,5-Trisphosphate 5-Phosphatases | NK Cell Lectin-Like Receptor Subfamily D - metabolism | Homeostasis - genetics | Index Medicus | Abridged Index Medicus
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8. Full Text Synergized regulation of NK cell education by NKG2A and specific Ly49 family members
by Zhang, Xiaoqian and Feng, Jin and Chen, Shasha and Yang, Haoyan and Dong, Zhongjun
Nature communications, ISSN 2041-1723, 11/2019, Volume 10, Issue 1, pp. 5010 - 12
Science & Technology - Other Topics | Multidisciplinary Sciences | Science & Technology | NK Cell Lectin-Like Receptor Subfamily A - metabolism | Multigene Family | NK Cell Lectin-Like Receptor Subfamily C - metabolism | Mice, Inbred C57BL | Histocompatibility Antigens Class I - immunology | NK Cell Lectin-Like Receptor Subfamily C - genetics | Histocompatibility Antigens Class I - genetics | Mice, SCID | NK Cell Lectin-Like Receptor Subfamily A - immunology | Mice, Knockout | Histocompatibility Antigens Class I - metabolism | NK Cell Lectin-Like Receptor Subfamily C - immunology | Animals | CRISPR-Cas Systems | Killer Cells, Natural - immunology | Mice, Inbred NOD | NK Cell Lectin-Like Receptor Subfamily A - genetics | Gene Editing - methods | Killer Cells, Natural - metabolism | Ly-49 antigen | NKG2 antigen | CRISPR | Receptors | Phenotypes | Major histocompatibility complex | Clonal deletion | Mushrooms | Education | Natural killer cells | Gene deletion | Synergism | Index Medicus
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9. Full Text Induction of lectin-like transcript 1 (LLT1) protein cell surface expression by pathogens and interferon-γ contributes to modulate immune responses
by Germain, Claire and Meier, Anders and Jensen, Teis and Knapnougel, Perrine and Poupon, Gwenola and Lazzari, Anne and Neisig, Anne and Håkansson, Katarina and Dong, Tao and Wagtmann, Nicolai and Galsgaard, Elizabeth D and Spee, Pieter and Braud, Veronique M
The Journal of biological chemistry, ISSN 0021-9258, 11/2011, Volume 286, Issue 44, pp. 37964 - 37975
Life Sciences & Biomedicine | Biochemistry & Molecular Biology | Science & Technology | Cell Line | Humans | Gene Expression Regulation | Lectins, C-Type - biosynthesis | Interferon-gamma - metabolism | Animals | Immune System | T-Lymphocytes - metabolism | Models, Biological | Hematopoietic Stem Cells - cytology | NK Cell Lectin-Like Receptor Subfamily B - biosynthesis | Interferons - metabolism | Leukocytes, Mononuclear - cytology | Cell Membrane - metabolism | Mice | Receptors, Cell Surface - biosynthesis | Toll-Like Receptors - metabolism | Index Medicus | Cell Membrane | Interferon-gamma | T-Lymphocytes | Interferons | Lectins, C-Type | Receptors, Cell Surface | Leukocytes, Mononuclear | Life Sciences | Toll-Like Receptors | Immunology | NK Cell Lectin-Like Receptor Subfamily B | Hematopoietic Stem Cells | T Cell | CD161 | Natural Killer (NK) Cell | Pathogen-associated Molecular Pattern (PAMP) | Interferon | Toll-like Receptor (TLR) | LLT1
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