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Science, ISSN 0036-8075, 7/2013, Volume 341, Issue 6142, pp. 186 - 191
Journal Article
Blood, ISSN 0006-4971, 07/2012, Volume 120, Issue 3, pp. 592 - 602
Journal Article
Cancer Research, ISSN 0008-5472, 04/2014, Volume 74, Issue 8, pp. 2193 - 2203
The immunoreceptor NKG2D originally identified in natural killer (NK) cells recognizes ligands that are upregulated on tumor cells. Expression of NKG2D ligands... 
TRANSCRIPTION FACTORS | B-CELLS | DAMAGE RESPONSE | SIGNALING PATHWAYS | NATURAL-KILLER | INTERFERON | IMMUNE-SYSTEM | ONCOLOGY | IN-VIVO | C-MYC | INNATE | Immunologic Surveillance | Up-Regulation | Phosphorylation | Nucleocytoplasmic Transport Proteins - biosynthesis | DNA, Neoplasm - metabolism | NK Cell Lectin-Like Receptor Subfamily K - immunology | Interferon Regulatory Factor-3 - genetics | Lymphoma - metabolism | Transfection | DNA Damage - physiology | NK Cell Lectin-Like Receptor Subfamily K - metabolism | DNA Damage - genetics | Killer Cells, Natural - immunology | Membrane Proteins - metabolism | Protein-Serine-Threonine Kinases - metabolism | Interferon Regulatory Factor-3 - immunology | Nucleocytoplasmic Transport Proteins - immunology | Membrane Proteins - genetics | Nuclear Matrix-Associated Proteins - immunology | Mice, Inbred C57BL | Lymphoma - immunology | Protein-Serine-Threonine Kinases - genetics | Lymphoma - genetics | Nuclear Matrix-Associated Proteins - metabolism | Membrane Proteins - immunology | Mice, Transgenic | NK Cell Lectin-Like Receptor Subfamily K - genetics | Nucleocytoplasmic Transport Proteins - genetics | Animals | Nucleocytoplasmic Transport Proteins - metabolism | Nuclear Matrix-Associated Proteins - genetics | Cell Line, Tumor | Interferon Regulatory Factor-3 - metabolism | Ligands | Protein-Serine-Threonine Kinases - immunology | Mice | Killer Cells, Natural - metabolism | Nuclear Matrix-Associated Proteins - biosynthesis | DNA Damage - radiation effects | Index Medicus
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 12/2015, Volume 112, Issue 52, pp. 15988 - 15993
Changes of histone modification status at critical lineage-specifying gene loci in multipotent precursors can influence cell fate commitment. The contribution... 
Histone modification | Innate immunity | NKG2D | Epigenetic regulation | Hematopoietic stem and progenitor cells | METHYLATION | HOMEOSTASIS | histone modification | MULTIDISCIPLINARY SCIENCES | INNATE LYMPHOID-CELLS | epigenetic regulation | CANCER | DEPENDENCE | LINEAGE COMMITMENT | POLYCOMB GROUP PROTEINS | innate immunity | NK CELLS | hematopoietic stem and progenitor cells | GENERATION | EXPRESSION | Polycomb Repressive Complex 2 - genetics | Humans | Interleukin-2 Receptor beta Subunit - metabolism | Cell Survival - genetics | Immunoblotting | Gene Expression Profiling | Cell Differentiation - genetics | Flow Cytometry | NK Cell Lectin-Like Receptor Subfamily K - metabolism | Interleukin-2 Receptor beta Subunit - genetics | Cell Lineage - genetics | Cell Proliferation - genetics | Histone-Lysine N-Methyltransferase - genetics | Mice, Inbred C57BL | Cells, Cultured | Mice, Transgenic | Hematopoietic Stem Cells - metabolism | NK Cell Lectin-Like Receptor Subfamily K - genetics | Reverse Transcriptase Polymerase Chain Reaction | Enhancer of Zeste Homolog 2 Protein | Mice, Knockout | Animals | Histone-Lysine N-Methyltransferase - metabolism | K562 Cells | Cell Line, Tumor | Killer Cells, Natural - metabolism | Polycomb Repressive Complex 2 - metabolism | Physiological aspects | Killer cells | Genetic aspects | Research | Methyltransferases | Cell differentiation | Molecules | Rodents | Immunotherapy | Stem cells | Epigenetics | Cytotoxicity | Tumors | Index Medicus | Biological Sciences
Journal Article
The Journal of Immunology, ISSN 0022-1767, 07/2009, Volume 183, Issue 1, pp. 340 - 351
During mammalian pregnancy maternal-fetal tolerance involves a number of immunosuppressive factors produced by placenta. Recently, placenta-derived exosomes... 
MHC CLASS-I | TROPHOBLAST CELLS | NK CYTOTOXICITY | FAS LIGAND | CHAIN-RELATED MOLECULES | MULTIVESICULAR BODIES | GLYCOPROTEIN UL16 | TOLL-LIKE RECEPTOR | MATERNAL-FETAL TOLERANCE | IMMUNOLOGY | TUMOR-DERIVED EXOSOMES | Endosomes - immunology | Pregnancy Proteins - antagonists & inhibitors | Humans | Pregnancy Proteins - genetics | GPI-Linked Proteins | Exosomes - genetics | Intercellular Signaling Peptides and Proteins - biosynthesis | Endosomes - metabolism | Exosomes - immunology | Female | Endosomes - secretion | Intercellular Signaling Peptides and Proteins - secretion | Exosomes - secretion | Organ Culture Techniques | T-Lymphocytes, Cytotoxic - immunology | Endosomes - genetics | Placenta - immunology | Intercellular Signaling Peptides and Proteins - genetics | NK Cell Lectin-Like Receptor Subfamily K - genetics | Down-Regulation - genetics | Pregnancy Proteins - biosynthesis | NK Cell Lectin-Like Receptor Subfamily K - biosynthesis | Placenta - metabolism | Pregnancy | Immunosuppression | NK Cell Lectin-Like Receptor Subfamily K - antagonists & inhibitors | T-Lymphocytes, Cytotoxic - metabolism | Down-Regulation - immunology | Pregnancy Proteins - secretion | Ligands | Placenta - secretion | Placenta - ultrastructure | Index Medicus | Abridged Index Medicus | Basic Medicine | Clinical Medicine | Obstetrics, Gynecology and Reproductive Medicine | Medical and Health Sciences | Medicin och hälsovetenskap | Medicinska och farmaceutiska grundvetenskaper | Reproduktionsmedicin och gynekologi | Immunologi inom det medicinska området | Klinisk medicin | Immunology in the medical area
Journal Article
Cytotherapy, ISSN 1465-3249, 2009, Volume 11, Issue 3, pp. 341 - 355
Background aims Cancer immunotherapy involving natural killer (NK) cell infusions and administration of therapeutic agents modulating the susceptibility of... 
Advanced Basic Science | Other | natural killer cells | Bortezomib | immunotherapy | expansion | Natural killer cells | Expansion | Immunotherapy | MEDICINE, RESEARCH & EXPERIMENTAL | EFFECTOR FUNCTIONS | NK-CELL | PROLIFERATION | TRANSPLANTATION | CELL & TISSUE ENGINEERING | CELL BIOLOGY | APOPTOSIS-INDUCING LIGAND | BIOTECHNOLOGY & APPLIED MICROBIOLOGY | GENERATION | HEMATOLOGY | CYTOTOXICITY | LYMPHOCYTES | Up-Regulation | Fas Ligand Protein - metabolism | Immunotherapy - methods | Cell Proliferation | Humans | NK Cell Lectin-Like Receptor Subfamily K - immunology | Killer Cells, Natural - pathology | Carcinoma, Renal Cell - immunology | Fas Ligand Protein - immunology | NK Cell Lectin-Like Receptor Subfamily K - metabolism | Cryopreservation | Killer Cells, Natural - immunology | Cell Culture Techniques | Carcinoma, Renal Cell - drug therapy | Carcinoma, Renal Cell - pathology | Lymphocyte Activation | Cytokines - secretion | Immunophenotyping | TNF-Related Apoptosis-Inducing Ligand - immunology | NK Cell Lectin-Like Receptor Subfamily K - genetics | TNF-Related Apoptosis-Inducing Ligand - metabolism | K562 Cells | Killer Cells, Natural - metabolism | Cytotoxicity, Immunologic - immunology | Fas Ligand Protein - genetics | Pyrazines - pharmacology | TNF-Related Apoptosis-Inducing Ligand - genetics | Boronic Acids - pharmacology | Index Medicus
Journal Article
Journal Article
Journal of Biological Chemistry, ISSN 0021-9258, 11/2014, Volume 289, Issue 48, pp. 33311 - 33319
Journal Article
Immunity, ISSN 1074-7613, 2010, Volume 32, Issue 2, pp. 175 - 186
Natural killer (NK) cell cytotoxicity toward target cells depends on synergistic coactivation by NK cell receptors such as NKG2D and 2B4. How synergy occurs is... 
MOLIMMUNO | CELLIMMUNO | NK-CELL CYTOTOXICITY | PATHWAYS | RECEPTOR SIGNALS | VAV PROTEINS | ACTIVATION | NKG2D | KILLER-CELLS | NEGATIVE REGULATION | IMMUNOLOGY | POLARIZATION | T-CELLS | Antigens, CD - immunology | RNA, Small Interfering - genetics | Phosphorylation | Proto-Oncogene Proteins c-vav - genetics | Calcium - metabolism | Humans | NK Cell Lectin-Like Receptor Subfamily K - immunology | Killer Cells, Natural - pathology | HLA Antigens - genetics | Antigens, CD - genetics | Antigens, CD - metabolism | Histocompatibility Antigens Class I - metabolism | Signal Transduction - immunology | Cell Degranulation - genetics | Transfection | NK Cell Lectin-Like Receptor Subfamily K - metabolism | Killer Cells, Natural - immunology | Receptors, Immunologic - immunology | Proto-Oncogene Proteins c-cbl - genetics | Cytotoxicity, Immunologic - genetics | Phospholipase C gamma - metabolism | Histocompatibility Antigens Class I - immunology | HLA Antigens - immunology | Histocompatibility Antigens Class I - genetics | Signaling Lymphocytic Activation Molecule Family | Cell Degranulation - immunology | HLA Antigens - metabolism | NK Cell Lectin-Like Receptor Subfamily K - genetics | Receptor Cross-Talk - immunology | Animals | Cell Line, Tumor | Mice | Killer Cells, Natural - metabolism | Cytotoxicity, Immunologic - immunology | Receptors, Immunologic - genetics | NK Cell Lectin-Like Receptor Subfamily D - metabolism | Receptors, Immunologic - metabolism | Ubiquitin | Immunogenetics | Histocompatibility antigens | Ligases | HLA histocompatibility antigens | Antigens | Insects | Plasmids | Cytotoxicity | Ligands | Kinases | Immune system | Index Medicus
Journal Article
Journal of Leukocyte Biology, ISSN 0741-5400, 12/2014, Volume 96, Issue 6, pp. 1119 - 1129
NK cells directly kill mycobacteria through a contactdependent mechanism leading to the release of perforin and granulysin. Although the mechanisms underlying... 
antibacterial activity | NKG2D | innate immunity | NCR | tuberculosis | Antibacterial activity | Innate immunity | Tuberculosis | FUNCTIONAL-CHARACTERIZATION | IMMUNOLOGY | CELL BIOLOGY | MEDIATED CYTOLYSIS | MEMBRANE-BINDING | MICROBICIDAL ACTIVITY | HOST-DEFENSE | ACTIVATING RECEPTORS | PORE-FORMING PROTEIN | HEMATOLOGY | CYTOTOXICITY | T-CELLS | Natural Cytotoxicity Triggering Receptor 2 - biosynthesis | Bacteriolysis - drug effects | Transcription, Genetic - drug effects | MAP Kinase Signaling System - physiology | Perforin - genetics | Humans | Nanotubes | Natural Cytotoxicity Triggering Receptor 3 - genetics | Cell Wall - drug effects | RNA Interference | Natural Cytotoxicity Triggering Receptor 3 - biosynthesis | Antigens, Differentiation, T-Lymphocyte - physiology | Killer Cells, Natural - immunology | Natural Cytotoxicity Triggering Receptor 2 - antagonists & inhibitors | Killer Cells, Natural - secretion | RNA, Small Interfering - pharmacology | Mycobacterium kansasii | Natural Cytotoxicity Triggering Receptor 3 - antagonists & inhibitors | NK Cell Lectin-Like Receptor Subfamily K - genetics | Bacteriolysis - physiology | NK Cell Lectin-Like Receptor Subfamily K - biosynthesis | Killer Cells, Natural - ultrastructure | MAP Kinase Signaling System - drug effects | NK Cell Lectin-Like Receptor Subfamily K - antagonists & inhibitors | Mycobacterium tuberculosis | Antigens, Differentiation, T-Lymphocyte - genetics | Cell Line, Tumor | Natural Cytotoxicity Triggering Receptor 2 - genetics | Antigens, Differentiation, T-Lymphocyte - biosynthesis |