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1. Full Text SHIP1 intrinsically regulates NK cell signaling and education, resulting in tolerance of an MHC class I-mismatched bone marrow graft in mice
by Gumbleton, Matthew and Vivier, Eric and Kerr, William G
The Journal of immunology (1950), ISSN 0022-1767, 03/2015, Volume 194, Issue 6, pp. 2847 - 2854
Life Sciences & Biomedicine | Immunology | Science & Technology | Self Tolerance - immunology | Inositol Polyphosphate 5-Phosphatases | Phosphoric Monoester Hydrolases - deficiency | NK Cell Lectin-Like Receptor Subfamily K - immunology | Interferon-gamma - metabolism | Mice, 129 Strain | Bone Marrow Transplantation - methods | Haptens - immunology | NK Cell Lectin-Like Receptor Subfamily D - immunology | Dermatitis, Contact - immunology | Flow Cytometry | Phosphoric Monoester Hydrolases - immunology | NK Cell Lectin-Like Receptor Subfamily K - metabolism | Natural Cytotoxicity Triggering Receptor 1 - genetics | Self Tolerance - genetics | Killer Cells, Natural - immunology | Natural Cytotoxicity Triggering Receptor 1 - immunology | Cytokines - immunology | Dermatitis, Contact - genetics | NK Cell Lectin-Like Receptor Subfamily A - metabolism | Phosphoric Monoester Hydrolases - genetics | Cytokines - metabolism | Mice, Inbred C57BL | Histocompatibility Antigens Class I - immunology | Homeostasis - immunology | Mice, Transgenic | NK Cell Lectin-Like Receptor Subfamily A - immunology | Histocompatibility - immunology | Mice, Knockout | Animals | Interferon-gamma - immunology | Natural Cytotoxicity Triggering Receptor 1 - metabolism | Graft Rejection - immunology | Killer Cells, Natural - metabolism | Graft Rejection - genetics | Phosphatidylinositol-3,4,5-Trisphosphate 5-Phosphatases | NK Cell Lectin-Like Receptor Subfamily D - metabolism | Homeostasis - genetics | Index Medicus | Abridged Index Medicus
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2. Full Text The Egyptian Rousette Genome Reveals Unexpected Features of Bat Antiviral Immunity
by Pavlovich, Stephanie S and Lovett, Sean P and Koroleva, Galina and Guito, Jonathan C and Arnold, Catherine E and Nagle, Elyse R and Kulcsar, Kirsten and Lee, Albert and Thibaud-Nissen, Françoise and Hume, Adam J and Mühlberger, Elke and Uebelhoer, Luke S and Towner, Jonathan S and Rabadan, Raul and Sanchez-Lockhart, Mariano and Kepler, Thomas B and Palacios, Gustavo
Cell (Cambridge), ISSN 0092-8674, 05/2018, Volume 173, Issue 5, pp. 1098 - 1110.e18
type I interferon | genome | antiviral immunity | innate immunity | natural killer cell receptors | filovirus | Chiroptera | Biochemistry & Molecular Biology | Life Sciences & Biomedicine | Science & Technology | Cell Biology | Chiroptera - immunology | Marburg Virus Disease - immunology | Disease Reservoirs - virology | Humans | Egypt | Immunity, Innate - genetics | NK Cell Lectin-Like Receptor Subfamily C - genetics | Phylogeny | Genetic Variation | NK Cell Lectin-Like Receptor Subfamily C - classification | NK Cell Lectin-Like Receptor Subfamily D - genetics | Amino Acid Sequence | Cell Line | Chiroptera - classification | Marburgvirus - physiology | Marburg Virus Disease - pathology | Interferon Type I - classification | Chromosome Mapping | Histocompatibility Antigens Class I - genetics | Sequence Alignment | Animals | NK Cell Lectin-Like Receptor Subfamily C - chemistry | Histocompatibility Antigens Class I - classification | NK Cell Lectin-Like Receptor Subfamily D - chemistry | Interferon Type I - genetics | NK Cell Lectin-Like Receptor Subfamily D - classification | Genome | Chiroptera - genetics | Evolution, Molecular | Bats | Medical colleges | Medical research | Microbiology | Genomics | Genomes | Biological response modifiers | Marburg virus disease | Virus diseases | Killer cells | Anopheles | Analysis | Medicine, Experimental | Interferon | Index Medicus
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3. Full Text Impaired natural killer cell self-education and "missing-self" responses in Ly49-deficient mice
by Beĺanger, Simon and Tu, Megan M and Rahim, Mir Munir Ahmed and Mahmoud, Ahmad B and Patel, Rajen and Tai, Lee-Hwa and Troke, Angela D and Wilhelm, Brian T and Landry, Josette-Renée and Zhu, Qinzhang and Tung, Kenneth S and Raulet, David H and Makrigiannis, Andrew P
Blood, ISSN 0006-4971, 07/2012, Volume 120, Issue 3, pp. 592 - 602
Life Sciences & Biomedicine | Hematology | Science & Technology | Hematologic and hematopoietic diseases | Biological and medical sciences | Medical sciences | Antigens, Ly - genetics | NK Cell Lectin-Like Receptor Subfamily K - immunology | Lectins, C-Type - genetics | Cell Differentiation - genetics | Lectins, C-Type - metabolism | Neoplasms - genetics | Mice, Mutant Strains | NK Cell Lectin-Like Receptor Subfamily D - genetics | Killer Cells, Natural - immunology | NK Cell Lectin-Like Receptor Subfamily A - genetics | Receptors, Immunologic - immunology | Mice, Inbred C57BL | Histocompatibility Antigens Class I - immunology | Gene Silencing - immunology | Histocompatibility Antigens Class I - genetics | Cell Degranulation - immunology | NK Cell Lectin-Like Receptor Subfamily A - immunology | NK Cell Lectin-Like Receptor Subfamily K - genetics | Cell Differentiation - immunology | Killer Cells, Natural - cytology | Animals | Antigens, Ly - immunology | Neoplasms - immunology | Cell Line, Tumor | Transsexualism - genetics | Mice | Receptors, Immunologic - genetics | NK Cell Lectin-Like Receptor Subfamily D - metabolism | Receptors, Immunologic - metabolism | Index Medicus | Abridged Index Medicus | Immunobiology
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4. Full Text Cytomegalovirus Infection Drives Adaptive Epigenetic Diversification of NK Cells with Altered Signaling and Effector Function
by Schlums, Heinrich and Cichocki, Frank and Tesi, Bianca and Theorell, Jakob and Beziat, Vivien and Holmes, Tim D and Han, Hongya and Chiang, Samuel C.C and Foley, Bree and Mattsson, Kristin and Larsson, Stella and Schaffer, Marie and Malmberg, Karl-Johan and Ljunggren, Hans-Gustaf and Miller, Jeffrey S and Bryceson, Yenan T
Immunity (Cambridge, Mass.), ISSN 1074-7613, 03/2015, Volume 42, Issue 3, pp. 443 - 456
Life Sciences & Biomedicine | Immunology | Science & Technology | Cell Proliferation | Humans | Transcription Factors - deficiency | NK Cell Lectin-Like Receptor Subfamily C - genetics | Receptors, IgG - deficiency | Intracellular Signaling Peptides and Proteins - immunology | Cytomegalovirus Infections - immunology | Gene Expression Profiling | Killer Cells, Natural - pathology | Cytomegalovirus Infections - genetics | Protein-Tyrosine Kinases - deficiency | Protein-Tyrosine Kinases - immunology | T-Lymphocytes, Cytotoxic - pathology | Intracellular Signaling Peptides and Proteins - deficiency | NK Cell Lectin-Like Receptor Subfamily C - immunology | DNA Methylation | Protein-Tyrosine Kinases - genetics | Receptors, IgG - genetics | Microarray Analysis | Antibodies - immunology | Cytomegalovirus Infections - virology | Killer Cells, Natural - immunology | Cytomegalovirus Infections - pathology | Kruppel-Like Transcription Factors - deficiency | Transcription Factors - immunology | Intracellular Signaling Peptides and Proteins - genetics | Syk Kinase | Adaptive Immunity | T-Lymphocytes, Cytotoxic - immunology | Promoter Regions, Genetic | GPI-Linked Proteins - immunology | Signal Transduction | Killer Cells, Natural - virology | T-Lymphocytes, Cytotoxic - virology | NK Cell Lectin-Like Receptor Subfamily C - deficiency | Immunophenotyping | Receptors, IgG - immunology | Transcription Factors - genetics | Killer Cells, Natural - classification | Promyelocytic Leukemia Zinc Finger Protein | Cytomegalovirus - immunology | Epigenesis, Genetic - immunology | Kruppel-Like Transcription Factors - genetics | Kruppel-Like Transcription Factors - immunology | GPI-Linked Proteins - genetics | Virus diseases | Proteins | Epigenetic inheritance | Killer cells | Immunotherapy | Genomics | Medical genetics | Cytomegalovirus infections | T cells | Methylation | Health aspects | Antigens | Flow cytometry | Cytomegalovirus | Data analysis | Biomedical research | Transplants & implants | Cytokines | Leukemia | Cytotoxicity | Infections | Kinases | Diversification | Gene expression | Experiments | Scholarships & fellowships | Lymphocytes | DNA methylation | Epigenetics | Viral infections | Immune system | Cancer | Index Medicus | Medicin och hälsovetenskap
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5. Full Text Cutting edge: NKG2D signaling enhances nk cell responses but alone is insufficient to drive expansion during mouse cytomegalovirus infection
by Nabekura, Tsukasa and Gotthardt, Dagmar and Niizuma, Kouta and Trsan, Tihana and Jenus, Tina and Jonjic, Stipan and Lanier, Lewis L
The Journal of immunology (1950), ISSN 0022-1767, 09/2017, Volume 199, Issue 5, pp. 1567 - 1571
Life Sciences & Biomedicine | Immunology | Science & Technology | NK Cell Lectin-Like Receptor Subfamily A - metabolism | Cell Proliferation | Signal Transduction | Lymphocyte Activation | Mice, Inbred C57BL | Cells, Cultured | Immunity, Innate | NK Cell Lectin-Like Receptor Subfamily K - genetics | Mice, Knockout | Muromegalovirus - immunology | Animals | NK Cell Lectin-Like Receptor Subfamily K - metabolism | Adaptor Proteins, Signal Transducing - genetics | Killer Cells, Natural - immunology | Protein Binding | NK Cell Lectin-Like Receptor Subfamily A - genetics | Mice | Adaptor Proteins, Signal Transducing - metabolism | Receptors, Immunologic - genetics | Receptors, Immunologic - metabolism | Herpesviridae Infections - immunology | DAP12 protein | Lymphocyte receptors | Cell proliferation | NKG2 antigen | Cytomegalovirus | Signaling | Viruses | Infections | DAP10 protein | Natural killer cells | Expansion | Index Medicus | Abridged Index Medicus
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6. Full Text Cutting edge: CD8+ T cell priming in the absence of NK cells leads to enhanced memory responses
by Soderquest, Katrina and Walzer, Thierry and Zafirova, Biljana and Klavinskis, Linda S and Polić, Bojan and Vivier, Eric and Lord, Graham M and Martín-Fontecha, Alfonso
The Journal of immunology (1950), ISSN 0022-1767, 03/2011, Volume 186, Issue 6, pp. 3304 - 3308
Life Sciences & Biomedicine | Immunology | Science & Technology | CD8-Positive T-Lymphocytes - cytology | Pore Forming Cytotoxic Proteins - genetics | Pore Forming Cytotoxic Proteins - biosynthesis | Coculture Techniques | Humans | Melanoma, Experimental - metabolism | Killer Cells, Natural - pathology | Lymphocyte Activation - genetics | Lymphocyte Activation - immunology | Melanoma, Experimental - immunology | NK Cell Lectin-Like Receptor Subfamily K - metabolism | Pore Forming Cytotoxic Proteins - physiology | CD8-Positive T-Lymphocytes - metabolism | Killer Cells, Natural - immunology | Female | Perforin | Immunologic Memory - genetics | NK Cell Lectin-Like Receptor Subfamily K - physiology | NK Cell Lectin-Like Receptor Subfamily K - deficiency | Mice, Inbred C57BL | Cells, Cultured | Melanoma, Experimental - pathology | Mice, Transgenic | Up-Regulation - genetics | Mice, Knockout | Animals | Up-Regulation - immunology | Mice | Killer Cells, Natural - metabolism | CD8-Positive T-Lymphocytes - immunology | Lymphocyte Depletion - methods | Index Medicus | Abridged Index Medicus | Up-Regulation | NK Cell Lectin-Like Receptor Subfamily K | Killer Cells, Natural | Lymphocyte Activation | Lymphocyte Depletion | CD8-Positive T-Lymphocytes | Life Sciences | Melanoma, Experimental | Immunologic Memory | Pore Forming Cytotoxic Proteins
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7. Full Text Balance between activating NKG2D, DNAM‐1, NKp44 and NKp46 and inhibitory CD94/NKG2A receptors determine natural killer degranulation towards rheumatoid arthritis synovial fibroblasts
by Nielsen, Natasja and Pascal, Veronique and Fasth, Andreas E. R and Sundström, Yvonne and Galsgaard, Elisabeth D and Ahern, David and Andersen, Martin and Baslund, Bo and Bartels, Else M and Bliddal, Henning and Feldmann, Marc and Malmström, Vivianne and Berg, Louise and Spee, Pieter and Söderström, Kalle
Immunology, ISSN 0019-2805, 08/2014, Volume 142, Issue 4, pp. 581 - 593
natural killer cytotoxicity | CD94/NKG2A | NKG2D | fibroblast‐like synoviocytes | Fibroblast-like synoviocytes | Natural killer cytotoxicity | Life Sciences & Biomedicine | Immunology | Science & Technology | Humans | NK Cell Lectin-Like Receptor Subfamily K - immunology | Synovial Membrane - immunology | Synovial Membrane - pathology | Male | Killer Cells, Natural - pathology | NK Cell Lectin-Like Receptor Subfamily D - immunology | Arthritis, Rheumatoid - metabolism | NK Cell Lectin-Like Receptor Subfamily C - immunology | NK Cell Lectin-Like Receptor Subfamily K - metabolism | Killer Cells, Natural - immunology | Female | Histocompatibility Antigens Class I - biosynthesis | Natural Cytotoxicity Triggering Receptor 1 - immunology | Fibroblasts - metabolism | Natural Cytotoxicity Triggering Receptor 2 - immunology | Cell Line | NK Cell Lectin-Like Receptor Subfamily C - metabolism | Antigens, Differentiation, T-Lymphocyte - immunology | Histocompatibility Antigens Class I - immunology | Antigens, Differentiation, T-Lymphocyte - metabolism | Cell Degranulation - immunology | Fibroblasts - pathology | Synovial Membrane - metabolism | Arthritis, Rheumatoid - pathology | Up-Regulation - immunology | Natural Cytotoxicity Triggering Receptor 2 - metabolism | Fibroblasts - immunology | Natural Cytotoxicity Triggering Receptor 1 - metabolism | Killer Cells, Natural - metabolism | Arthritis, Rheumatoid - immunology | NK Cell Lectin-Like Receptor Subfamily D - metabolism | Arthritis | Histocompatibility antigens | Immunity | Analysis | HLA histocompatibility antigens | Autoimmune diseases | Antigens | Immune system | Index Medicus | fibroblast-like synoviocytes | CD94 | Original | NKG2A | Medicin och hälsovetenskap
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8. Full Text NKG2D receptor and its ligands in host defense
by Lanier, Lewis L
Cancer immunology research, ISSN 2326-6066, 06/2015, Volume 3, Issue 6, pp. 575 - 582
Oncology | Immunology | Life Sciences & Biomedicine | Science & Technology | Neoplasms - metabolism | Communicable Diseases - therapy | Humans | Communicable Diseases - genetics | NK Cell Lectin-Like Receptor Subfamily K - genetics | Communicable Diseases - metabolism | Lymphocyte Subsets - immunology | Lymphocyte Subsets - metabolism | Lymphocyte Activation - immunology | Neoplasms - therapy | Animals | Disease Resistance - physiology | NK Cell Lectin-Like Receptor Subfamily K - metabolism | Neoplasms - genetics | Neoplasms - immunology | Communicable Diseases - immunology | Killer Cells, Natural - immunology | Ligands | Killer Cells, Natural - metabolism | Index Medicus
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9. Full Text Desiccating stress-induced chemokine expression in the epithelium is dependent on upregulation of NKG2D/RAE-1 and release of IFN-γ in experimental dry eye
by Coursey, Terry G and Bohat, Ritu and Barbosa, Flavia L and Pflugfelder, Stephen C and De Paiva, Cintia S
The Journal of immunology (1950), ISSN 0022-1767, 11/2014, Volume 193, Issue 10, pp. 5264 - 5272
Life Sciences & Biomedicine | Immunology | Science & Technology | Chemokine CXCL11 - genetics | Antigens, Ly - genetics | NK Cell Lectin-Like Receptor Subfamily K - immunology | Homeodomain Proteins - immunology | Adoptive Transfer | Epithelium, Corneal - pathology | Xerophthalmia - genetics | T-Lymphocytes - transplantation | Conjunctiva - pathology | Gene Deletion | Chemokine CXCL10 - immunology | Female | Chemokine CXCL11 - immunology | Interferon-gamma - genetics | T-Lymphocytes - pathology | Epithelium, Corneal - immunology | Disease Models, Animal | Xerophthalmia - pathology | Conjunctiva - immunology | Membrane Proteins - genetics | Desiccation | Mice, Inbred C57BL | Gene Expression Regulation | Membrane Proteins - immunology | Chemokine CXCL9 - immunology | Immunity, Innate | NK Cell Lectin-Like Receptor Subfamily K - genetics | Chemokine CXCL9 - genetics | NK Cell Lectin-Like Receptor Subfamily B - immunology | Homeodomain Proteins - genetics | Mice, Knockout | Antibodies - pharmacology | Animals | NK Cell Lectin-Like Receptor Subfamily B - antagonists & inhibitors | Antigens, Ly - immunology | NK Cell Lectin-Like Receptor Subfamily B - genetics | Chemokine CXCL10 - genetics | Interferon-gamma - immunology | T-Lymphocytes - immunology | Mice | Xerophthalmia - immunology | Index Medicus | Abridged Index Medicus |