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1. Full Text Innate mucosal‐associated invariant T (MAIT) cells are activated in inflammatory bowel diseases
by Serriari, N.‐E and Eoche, M and Lamotte, L and Lion, J and Fumery, M and Marcelo, P and Chatelain, D and Barre, A and Nguyen‐Khac, E and Lantz, O and Dupas, J.‐L and Treiner, E
Clinical & Experimental Immunology, ISSN 0009-9104, 05/2014, Volume 176, Issue 2, pp. 266 - 274
I nflammatory bowel diseases are characterized by a deregulated immune response targeting the gut bacterial flora. M ucosal‐associated invariant T ( MAIT )... 
immune system | MHC | class I‐like | cells | inflammatory bowel diseases | innate | Inflammatory bowel diseases | Innate T cells | MHC class I-like | Immune system | NATURAL-KILLER-CELLS | CYTOKINES | CROHNS-DISEASE | ULCERATIVE-COLITIS | IMMUNOLOGY | SUBSET | NKG2D | innate T cells | INFECTION | DEFINES | EXPRESSION | IMPROVES CYTOTOXICITY | T-Lymphocyte Subsets - immunology | Humans | Inflammatory Bowel Diseases - immunology | Interleukin-17 - immunology | NK Cell Lectin-Like Receptor Subfamily K - immunology | Ki-67 Antigen - metabolism | Male | Colitis, Ulcerative - immunology | Lymphocyte Activation - immunology | Flow Cytometry | Ki-67 Antigen - immunology | NK Cell Lectin-Like Receptor Subfamily K - metabolism | Intestinal Mucosa - immunology | Natural Killer T-Cells - metabolism | Inflammatory Bowel Diseases - pathology | Interleukins - immunology | Natural Killer T-Cells - pathology | Adult | Female | Receptors, Immunologic - immunology | Interleukin-17 - blood | Interleukins - blood | Colitis, Ulcerative - pathology | Inflammatory Bowel Diseases - blood | Colitis, Ulcerative - blood | Crohn Disease - immunology | Immunity, Innate - immunology | Microscopy, Confocal | T-Lymphocyte Subsets - pathology | Crohn Disease - pathology | T-Lymphocyte Subsets - metabolism | Crohn Disease - blood | Interferon-gamma - immunology | Interferon-gamma - blood | Receptors, Immunologic - metabolism | Intestinal Mucosa - pathology | Natural Killer T-Cells - immunology | Biological response modifiers | T cells | Analysis | Ulcerative colitis | Medical research | Bowel disease | Index Medicus | Original
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2. Full Text Human Cytomegalovirus Infection Elicits New Decidual Natural Killer Cell Effector Functions
by Siewiera, Johan and El Costa, Hicham and Tabiasco, Julie and Berrebi, Alain and Cartron, Géraldine and Bouteiller, Philippe and Jabrane-Ferrat, Nabila
PLoS Pathogens, ISSN 1553-7366, 04/2013, Volume 9, Issue 4, pp. e1003257 - e1003257
During the first trimester of pregnancy the uterus is massively infiltrated by decidual natural killer cells (dNK). These cells are not killers, but they... 
ACTIVATION | SURFACE EXPRESSION | MICROBIOLOGY | HUMAN NK CELLS | HLA-E | PREGNANCY | VIROLOGY | INHIBITORY RECEPTOR | NKG2D LIGAND | IMMUNOLOGICAL SYNAPSE | POLARIZATION | T-CELLS | PARASITOLOGY | Fas Ligand Protein - metabolism | Decidua - cytology | Humans | NK Cell Lectin-Like Receptor Subfamily K - immunology | Cytomegalovirus Infections - immunology | Cytomegalovirus Infections - metabolism | Pregnancy Trimester, First | NK Cell Lectin-Like Receptor Subfamily D - immunology | NK Cell Lectin-Like Receptor Subfamily C - immunology | NK Cell Lectin-Like Receptor Subfamily K - metabolism | Uterus | HEK293 Cells | Killer Cells, Natural - immunology | Female | Cytomegalovirus Infections - congenital | CD56 Antigen - metabolism | Cell Line | NK Cell Lectin-Like Receptor Subfamily C - metabolism | Decidua - immunology | Placentation | TNF-Related Apoptosis-Inducing Ligand - metabolism | Pregnancy | Cytomegalovirus - immunology | Decidua - virology | Killer Cells, Natural - metabolism | NK Cell Lectin-Like Receptor Subfamily D - metabolism | Virus diseases | Killer cells | Control | Care and treatment | Cytomegaloviruses | Pregnancy, Complications of | Properties | Health aspects | Mother and infant | Index Medicus | Life Sciences | Immunology | Infections | Viral infections | Immune system
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3. Full Text Dual-functional capability of CD3 +CD56 + CIK cells, a T-cell subset that acquires NK function and retains TCR-mediated specific cytotoxicity
by Pievani, Alice and Borleri, Gianmaria and Pende, Daniela and Moretta, Lorenzo and Rambaldi, Alessandro and Golay, Josée and Introna, Martino
Blood, ISSN 0006-4971, 09/2011, Volume 118, Issue 12, pp. 3301 - 3310
CD3(+)CD56(+) cytokine-induced killer (CIK) cells display a potent cytolytic activity. The adhesion molecule lymphocyte function-associated antigen-1 plays a... 
IMMUNOLOGICAL EFFECTOR-CELLS | SEVERE COMBINED IMMUNODEFICIENCY | NKG2D | DENDRITIC CELLS | IN-VIVO | ANTITUMOR-ACTIVITY | NECTIN-2 CD112 | HEMATOLOGY | PHASE-I | INDUCED KILLER-CELLS | CELIAC-DISEASE | Neoplasms - metabolism | T-Lymphocyte Subsets - immunology | T-Lymphocyte Subsets - cytology | Humans | Natural Cytotoxicity Triggering Receptor 3 - metabolism | NK Cell Lectin-Like Receptor Subfamily K - immunology | Lymphocyte Function-Associated Antigen-1 - metabolism | Cytomegalovirus Infections - immunology | Cytomegalovirus Infections - metabolism | Lymphocyte Function-Associated Antigen-1 - immunology | Signal Transduction - immunology | T-Lymphocytes, Cytotoxic - drug effects | Flow Cytometry | NK Cell Lectin-Like Receptor Subfamily K - metabolism | Neoplasms - genetics | Cytomegalovirus Infections - virology | Killer Cells, Natural - immunology | Cytomegalovirus Infections - pathology | Natural Cytotoxicity Triggering Receptor 3 - immunology | CD56 Antigen - immunology | Cytotoxicity, Immunologic - drug effects | Antibodies, Monoclonal - immunology | T-Lymphocytes, Cytotoxic - immunology | Cytokine-Induced Killer Cells - metabolism | Antigens, Differentiation, T-Lymphocyte - immunology | Antibodies, Monoclonal - pharmacology | Antigens, Differentiation, T-Lymphocyte - metabolism | Cytokine-Induced Killer Cells - immunology | Cytomegalovirus - growth & development | Cytomegalovirus - immunology | Killer Cells, Natural - cytology | Signal Transduction - drug effects | Neoplasms - immunology | T-Lymphocyte Subsets - metabolism | CD3 Complex - immunology | Cell Line, Tumor | Cytokine-Induced Killer Cells - cytology | Killer Cells, Natural - metabolism | Primary Cell Culture | Neoplasms - pathology | Index Medicus | Abridged Index Medicus
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4. Full Text Natural killer cell activation enhances immune pathology and promotes chronic infection by limiting CD8⁺ T-cell immunity
by Philipp A. Lang and Karl S. Lang and Haifeng C. Xu and Melanie Grusdat and Ian A. Parish and Mike Recher and Alisha R. Elford and Salim Dhanji and Namir Shaabani and Charles W. Tran and Dilan Dissanayake and Ramtin Rahbar and Magar Ghazarian and Anne Brüstle and Jason Fine and Peter Chen and Casey T. Weaver and Christoph Klose and Andreas Diefenbach and Dieter Häussinger and James R. Carlyle and Susan M. Kaech and Tak W. Mak and Pamela S. Ohashi
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 1/2012, Volume 109, Issue 4, pp. 1210 - 1215
Infections with HIV, hepatitis B virus, and hepatitis C virus can turn into chronic infections, which currently affect more than 500 million patients... 
T lymphocytes | Hepatitis | Receptors | Liver | Viruses | Cytotoxicity | Infections | Mice | Natural killer cells | Immunity | Effector T cells | Virus persistence | Virus elimination | Regulatory innate immunity | NK cell activation | DENDRITIC CELLS | effector T cells | MULTIDISCIPLINARY SCIENCES | NKG2D RECEPTOR | LYMPHOCYTIC CHORIOMENINGITIS VIRUS | DEPENDENT MANNER | regulatory innate immunity | HEPATITIS-C VIRUS | NK CELLS | virus persistence | IN-VIVO | virus elimination | INHIBITORY RECEPTOR | MICE | VIRAL PERSISTENCE | Interferon-alpha - metabolism | Cytotoxicity Tests, Immunologic | Enzyme-Linked Immunosorbent Assay | NK Cell Lectin-Like Receptor Subfamily K - immunology | Immunity, Cellular - immunology | Lymphocyte Activation - immunology | Animals | Flow Cytometry | Lymphocytic choriomeningitis virus | Analysis of Variance | Histological Techniques | Communicable Diseases - immunology | Killer Cells, Natural - immunology | CD8-Positive T-Lymphocytes - immunology | Arenaviridae Infections - immunology | Chronic Disease | Real-Time Polymerase Chain Reaction | Pathology | T cell receptors | Physiology | Rodents | Immune system | Index Medicus | NKG2 antigen | Cell activation | CD8 antigen | Chronic infection | Lymphocytes T | Perforin | Biological Sciences
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5. Full Text Antibody-mediated inhibition of MICA and MICB shedding promotes NK cell-driven tumor immunity
by De Andrade, Lucas Ferrari and En Tay, Rong and Pan, Deng and Luoma, Adrienne M and Ito, Yoshinaga and Badrinath, Soumya and Tsoucas, Daphne and Franz, Bettina and May, Kenneth F and Harvey, Christopher J and Kobold, Sebastian and Pyrdol, Jason W and Yoon, Charles and Yuan, Guo-Cheng and Hodi, F.Stephen and Dranoff, Glenn and Wucherpfennig, Kai W
Science, ISSN 0036-8075, 03/2018, Volume 359, Issue 6383, pp. 1537 - 1542
MICA and MICB are expressed by many human cancers as a result of cellular stress, and can tag cells for elimination by cytotoxic lymphocytes through natural... 
ACTIVATION | I-RELATED CHAIN | LIGANDS | MULTIDISCIPLINARY SCIENCES | NKG2D RECEPTOR | EXPRESSION | CANCER | CYTOTOXICITY | T-CELLS | CUTTING EDGE | MOLECULES | Humans | Melanoma, Experimental - therapy | Mice, Inbred C57BL | Histocompatibility Antigens Class I - immunology | Melanoma, Experimental - pathology | NK Cell Lectin-Like Receptor Subfamily K - immunology | Antibodies, Monoclonal - therapeutic use | Melanoma - pathology | Receptors, IgG - immunology | Histocompatibility Antigens Class I - chemistry | Neoplasm Metastasis | Animals | Melanoma, Experimental - immunology | Melanoma - immunology | Antibodies, Blocking - immunology | Antibodies, Blocking - therapeutic use | Killer Cells, Natural - immunology | Ligands | Immunocompetence | Mice | Protein Domains - immunology | Melanoma - therapy | Antibodies, Monoclonal - immunology | Health aspects | Killer cells | Genetic aspects | Shedding | Animal models | Lung cancer | Antibodies | Cytotoxicity | Activation | Cell surface | Immunity | Metastases | Proteins | NKG2 antigen | Receptors | Cell activation | Proteolysis | Lymphocytes | Inhibition | Natural killer cells | Immune system | Immunoglobulins | Immunostimulation | Melanoma | Fc receptors | CD16 antigen | Major histocompatibility complex | Lysis | Receptor mechanisms | Cancer | Tumors | Index Medicus
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6. Full Text Impaired natural killer cell self-education and "missing-self" responses in Ly49-deficient mice
by Beĺanger, Simon and Tu, Megan M and Rahim, Mir Munir Ahmed and Mahmoud, Ahmad B and Patel, Rajen and Tai, Lee-Hwa and Troke, Angela D and Wilhelm, Brian T and Landry, Josette-Renée and Zhu, Qinzhang and Tung, Kenneth S and Raulet, David H and Makrigiannis, Andrew P
Blood, ISSN 0006-4971, 07/2012, Volume 120, Issue 3, pp. 592 - 602
Ly49-mediated recognition of MHC-I molecules on host cells is considered vital for natural killer (NK)-cell regulation and education; however, gene-deficient... 
NK CELLS | TOLERANCE | REJECTION | ACTIVATING RECEPTORS | MHC | TUMOR-CELLS | CYTOMEGALOVIRUS-INFECTION | CLASS-I MOLECULES | HEMATOLOGY | EXPRESSION | LY49 GENE-CLUSTER | Antigens, Ly - genetics | NK Cell Lectin-Like Receptor Subfamily K - immunology | Lectins, C-Type - genetics | Cell Differentiation - genetics | Lectins, C-Type - metabolism | Neoplasms - genetics | Mice, Mutant Strains | NK Cell Lectin-Like Receptor Subfamily D - genetics | Killer Cells, Natural - immunology | NK Cell Lectin-Like Receptor Subfamily A - genetics | Receptors, Immunologic - immunology | Mice, Inbred C57BL | Histocompatibility Antigens Class I - immunology | Gene Silencing - immunology | Histocompatibility Antigens Class I - genetics | Cell Degranulation - immunology | NK Cell Lectin-Like Receptor Subfamily A - immunology | NK Cell Lectin-Like Receptor Subfamily K - genetics | Cell Differentiation - immunology | Killer Cells, Natural - cytology | Animals | Antigens, Ly - immunology | Neoplasms - immunology | Cell Line, Tumor | Transsexualism - genetics | Mice | Receptors, Immunologic - genetics | NK Cell Lectin-Like Receptor Subfamily D - metabolism | Receptors, Immunologic - metabolism | Index Medicus | Abridged Index Medicus
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7. Full Text Immune evasion mediated by tumor-derived lactate dehydrogenase induction of NKG2D ligands on myeloid cells in glioblastoma patients
by Courtney A. Crane and Kathryn Austgen and Kristen Haberthur and Carly Hofmann and Kara White Moyes and Lia Avanesyan and Lawrence Fong and Michael J. Campbell and Stewart Cooper and Scott A. Oakes and Andrew T. Parsa and Lewis L. Lanier
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 9/2014, Volume 111, Issue 35, pp. 12823 - 12828
Myeloid cells are key regulators of the tumor microenvironment, governing local immune responses. Here we report that tumor-infiltrating myeloid cells and... 
Lactates | Monocytes | Myeloid cells | Receptors | Messenger RNA | Glioblastoma | Ligands | Natural killer cells | Tumors | Cancer | LDH-5 EXPRESSION | TGF-BETA | PROGNOSIS | LDH5 | MULTIDISCIPLINARY SCIENCES | ACTIVATING RECEPTOR | NECK-CANCER | tumor evasion | NK CELLS | PATHWAY | glioma | cancer | SUPPRESSOR-CELLS | RADIOTHERAPY | ASSOCIATION | Myeloid Cells - cytology | GPI-Linked Proteins - immunology | Monocytes - cytology | Humans | Histocompatibility Antigens Class I - immunology | Glioma - immunology | NK Cell Lectin-Like Receptor Subfamily K - immunology | Intracellular Signaling Peptides and Proteins - immunology | Monocytes - immunology | Isoenzymes - immunology | Immunity, Innate - immunology | Killer Cells, Natural - cytology | Brain Neoplasms - immunology | L-Lactate Dehydrogenase - immunology | Glioblastoma - immunology | Myeloid Cells - immunology | HEK293 Cells | Cell Line, Tumor | Killer Cells, Natural - immunology | Immune Evasion - immunology | Immune response | Ligands (Biochemistry) | Oncology, Experimental | Cancer cells | Physiological aspects | Research | Glioblastoma multiforme | Health aspects | Immunological research | Cells | Index Medicus | Biological Sciences
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8. Full Text RAE1 ligands for the NKG2D receptor are regulated by STING-dependent DNA sensor pathways in lymphoma
by Lam, Adeline R and Bert, Nina Le and Ho, Samantha S. W and Shen, Yu J and Tang, Melissa L. F and Xiong, Gordon M and Croxford, J. Ludovic and Koo, Christine X and Ishii, Ken J and Akira, Shizuo and Raulet, David H and Gasser, Stephan
Cancer Research, ISSN 0008-5472, 04/2014, Volume 74, Issue 8, pp. 2193 - 2203
The immunoreceptor NKG2D originally identified in natural killer (NK) cells recognizes ligands that are upregulated on tumor cells. Expression of NKG2D ligands... 
TRANSCRIPTION FACTORS | B-CELLS | DAMAGE RESPONSE | SIGNALING PATHWAYS | NATURAL-KILLER | INTERFERON | IMMUNE-SYSTEM | ONCOLOGY | IN-VIVO | C-MYC | INNATE | Immunologic Surveillance | Up-Regulation | Phosphorylation | Nucleocytoplasmic Transport Proteins - biosynthesis | DNA, Neoplasm - metabolism | NK Cell Lectin-Like Receptor Subfamily K - immunology | Interferon Regulatory Factor-3 - genetics | Lymphoma - metabolism | Transfection | DNA Damage - physiology | NK Cell Lectin-Like Receptor Subfamily K - metabolism | DNA Damage - genetics | Killer Cells, Natural - immunology | Membrane Proteins - metabolism | Protein-Serine-Threonine Kinases - metabolism | Interferon Regulatory Factor-3 - immunology | Nucleocytoplasmic Transport Proteins - immunology | Membrane Proteins - genetics | Nuclear Matrix-Associated Proteins - immunology | Mice, Inbred C57BL | Lymphoma - immunology | Protein-Serine-Threonine Kinases - genetics | Lymphoma - genetics | Nuclear Matrix-Associated Proteins - metabolism | Membrane Proteins - immunology | Mice, Transgenic | NK Cell Lectin-Like Receptor Subfamily K - genetics | Nucleocytoplasmic Transport Proteins - genetics | Animals | Nucleocytoplasmic Transport Proteins - metabolism | Nuclear Matrix-Associated Proteins - genetics | Cell Line, Tumor | Interferon Regulatory Factor-3 - metabolism | Ligands | Protein-Serine-Threonine Kinases - immunology | Mice | Killer Cells, Natural - metabolism | Nuclear Matrix-Associated Proteins - biosynthesis | DNA Damage - radiation effects | Index Medicus
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9. Full Text SHIP1 intrinsically regulates NK cell signaling and education, resulting in tolerance of an MHC class I-mismatched bone marrow graft in mice
by Gumbleton, Matthew and Vivier, Eric and Kerr, William G
Journal of Immunology, ISSN 0022-1767, 03/2015, Volume 194, Issue 6, pp. 2847 - 2854
NK cells are an important component of host immune defense against malignancy and infection. NK cells are educated by MHC class I ligands to ensure... 
ADAPTIVE IMMUNITY | REPERTOIRE | NATURAL-KILLER-CELLS | ACTIVATION | RESPONSIVENESS | INHIBITORY RECEPTORS | DIFFERENTIATION | IMMUNOLOGY | EXPRESSION | SELF-TOLERANCE | CUTTING EDGE | Self Tolerance - immunology | Inositol Polyphosphate 5-Phosphatases | Phosphoric Monoester Hydrolases - deficiency | NK Cell Lectin-Like Receptor Subfamily K - immunology | Interferon-gamma - metabolism | Mice, 129 Strain | Bone Marrow Transplantation - methods | Haptens - immunology | NK Cell Lectin-Like Receptor Subfamily D - immunology | Dermatitis, Contact - immunology | Flow Cytometry | Phosphoric Monoester Hydrolases - immunology | NK Cell Lectin-Like Receptor Subfamily K - metabolism | Natural Cytotoxicity Triggering Receptor 1 - genetics | Self Tolerance - genetics | Killer Cells, Natural - immunology | Natural Cytotoxicity Triggering Receptor 1 - immunology | Cytokines - immunology | Dermatitis, Contact - genetics | NK Cell Lectin-Like Receptor Subfamily A - metabolism | Phosphoric Monoester Hydrolases - genetics | Cytokines - metabolism | Mice, Inbred C57BL | Histocompatibility Antigens Class I - immunology | Homeostasis - immunology | Mice, Transgenic | NK Cell Lectin-Like Receptor Subfamily A - immunology | Histocompatibility - immunology | Mice, Knockout | Animals | Interferon-gamma - immunology | Natural Cytotoxicity Triggering Receptor 1 - metabolism | Graft Rejection - immunology | Killer Cells, Natural - metabolism | Graft Rejection - genetics | Phosphatidylinositol-3,4,5-Trisphosphate 5-Phosphatases | NK Cell Lectin-Like Receptor Subfamily D - metabolism | Homeostasis - genetics | Index Medicus | Abridged Index Medicus
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10. Full Text Suppressing T cell motility induced by anti-CTLA-4 monotherapy improves antitumor effects
by Ruocco, Maria Grazia and Pilones, Karsten A and Kawashima, Noriko and Cammer, Michael and Huang, Julie and Babb, James S and Liu, Mengling and Formenti, Silvia C and Dustin, Michael L and Demaria, Sandra
Journal of Clinical Investigation, ISSN 0021-9738, 10/2012, Volume 122, Issue 10, pp. 3718 - 3730
A promising strategy for cancer immunotherapy is to disrupt key pathways regulating immune tolerance, such as cytotoxic T lymphocyte-associated protein 4... 
BREAST-CANCER | MEDICINE, RESEARCH & EXPERIMENTAL | CTLA-4 BLOCKADE | LYMPHOCYTE ANTIGEN-4 | PHOSPHATIDYLINOSITOL 3-KINASE | IN-VIVO | COMBINATION IMMUNOTHERAPY | TUMOR-CELLS | NKG2D RECEPTOR | IMMUNOLOGICAL SYNAPSE | STOP-SIGNAL | Mammary Neoplasms, Experimental - immunology | Nucleocytoplasmic Transport Proteins - biosynthesis | NK Cell Lectin-Like Receptor Subfamily K - immunology | Antibodies, Monoclonal - therapeutic use | Cell Line, Tumor - transplantation | CD8-Positive T-Lymphocytes - radiation effects | Immunotherapy | Intercellular Adhesion Molecule-1 - biosynthesis | Female | Mammary Neoplasms, Experimental - secondary | Gene Expression Regulation, Neoplastic - drug effects | Neoplasm Proteins - genetics | Cytotoxicity, Immunologic - drug effects | Neoplasm Proteins - biosynthesis | Receptors, CXCR - genetics | Antibodies, Monoclonal - pharmacology | Mammary Neoplasms, Experimental - radiotherapy | Combined Modality Therapy | Lymphocytes, Tumor-Infiltrating - drug effects | Nucleocytoplasmic Transport Proteins - genetics | Mammary Neoplasms, Experimental - therapy | Tumor Microenvironment - immunology | H-2 Antigens - immunology | Animals | NK Cell Lectin-Like Receptor Subfamily K - antagonists & inhibitors | Nuclear Matrix-Associated Proteins - genetics | CD8-Positive T-Lymphocytes - drug effects | Cell Line, Tumor - immunology | Intercellular Adhesion Molecule-1 - genetics | Gene Expression Regulation, Neoplastic - radiation effects | Mice | Mice, Inbred BALB C | CD8-Positive T-Lymphocytes - immunology | CTLA-4 Antigen - antagonists & inhibitors | Nuclear Matrix-Associated Proteins - biosynthesis | Cytotoxicity, Immunologic - radiation effects | Lymphocytes, Tumor-Infiltrating - radiation effects | Cell Movement | Drug Screening Assays, Antitumor | Lymphocytes, Tumor-Infiltrating - immunology | Receptors, CXCR6 | Care and treatment | Tumor-infiltrating lymphocytes | Physiological aspects | Research | T cells | Health aspects | Cancer | Index Medicus | Abridged Index Medicus
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11. Full Text The Intraepithelial T Cell Response to NKG2D-Ligands Links Lymphoid Stress Surveillance to Atopy
by Jessica Strid and Olga Sobolev and Biljana Zafirova and Bojan Polic and Adrian Hayday
Science, ISSN 0036-8075, 12/2011, Volume 334, Issue 6060, pp. 1293 - 1297
Epithelial cells respond to physicochemical damage with up-regulation of major histocompatibility complex-like ligands that can activate the cytolytic... 
T lymphocytes | Antigens | Up regulation | Messenger RNA | Cytokines | Natural killer T cells | REPORTS | Antibodies | Ova | Lymph nodes | Allergies | NKT CELLS | NKG2D | DISRUPTION | LIGANDS | DERMATITIS | MULTIDISCIPLINARY SCIENCES | RECEPTOR | ASTHMA | MICE | MODEL | EXPRESSION | T-Lymphocyte Subsets - immunology | Up-Regulation | Stress, Physiological | NK Cell Lectin-Like Receptor Subfamily K - immunology | Membrane Proteins - immunology | Mice, Transgenic | Receptors, Antigen, T-Cell, gamma-delta - immunology | Th2 Cells - immunology | Lymphoid Tissue - immunology | Animals | Epidermis - immunology | NK Cell Lectin-Like Receptor Subfamily K - metabolism | Hypersensitivity, Immediate - immunology | Ligands | Membrane Proteins - metabolism | Mice | Physiological aspects | Stress (Physiology) | Research | T cells | Epithelial cells | Ligands (Biochemistry) | Skin | Cellular biology | Lymphocytes | Immune system | Index Medicus
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12. Full Text Clinical-grade ex vivo -expanded human natural killer cells up-regulate activating receptors and death receptor ligands and have enhanced cytolytic activity against tumor cells
by Berg, Maria and Lundqvist, Andreas and McCoy, Philip and Samsel, Leigh and Fan, Yong and Tawab, Abdul and Childs, Richard
Cytotherapy, ISSN 1465-3249, 2009, Volume 11, Issue 3, pp. 341 - 355
Background aims Cancer immunotherapy involving natural killer (NK) cell infusions and administration of therapeutic agents modulating the susceptibility of... 
Advanced Basic Science | Other | natural killer cells | Bortezomib | immunotherapy | expansion | Natural killer cells | Expansion | Immunotherapy | MEDICINE, RESEARCH & EXPERIMENTAL | EFFECTOR FUNCTIONS | NK-CELL | PROLIFERATION | TRANSPLANTATION | CELL & TISSUE ENGINEERING | CELL BIOLOGY | APOPTOSIS-INDUCING LIGAND | BIOTECHNOLOGY & APPLIED MICROBIOLOGY | GENERATION | HEMATOLOGY | CYTOTOXICITY | LYMPHOCYTES | Up-Regulation | Fas Ligand Protein - metabolism | Immunotherapy - methods | Cell Proliferation | Humans | NK Cell Lectin-Like Receptor Subfamily K - immunology | Killer Cells, Natural - pathology | Carcinoma, Renal Cell - immunology | Fas Ligand Protein - immunology | NK Cell Lectin-Like Receptor Subfamily K - metabolism | Cryopreservation | Killer Cells, Natural - immunology | Cell Culture Techniques | Carcinoma, Renal Cell - drug therapy | Carcinoma, Renal Cell - pathology | Lymphocyte Activation | Cytokines - secretion | Immunophenotyping | TNF-Related Apoptosis-Inducing Ligand - immunology | NK Cell Lectin-Like Receptor Subfamily K - genetics | TNF-Related Apoptosis-Inducing Ligand - metabolism | K562 Cells | Killer Cells, Natural - metabolism | Cytotoxicity, Immunologic - immunology | Fas Ligand Protein - genetics | Pyrazines - pharmacology | TNF-Related Apoptosis-Inducing Ligand - genetics | Boronic Acids - pharmacology | Index Medicus
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13. Full Text Tumor suppressive microRNAs miR-34a/c control cancer cell expression of ULBP2, a stress-induced ligand of the natural killer cell receptor NKG2D
by Heinemann, Anja and Zhao, Fang and Pechlivanis, Sonali and Eberle, Jürgen and Steinle, Alexander and Diederichs, Sven and Schadendorf, Dirk and Paschen, Annette
Cancer Research, ISSN 0008-5472, 01/2012, Volume 72, Issue 2, pp. 460 - 471
Malignant cells express ligands for the natural killer cell immunoreceptor NKG2D, which sensitizes to early recognition and elimination by cytotoxic... 
ACTIVATION | MELANOMA | I-RELATED CHAIN | ONCOLOGY | IMMUNE-RESPONSES | DOWN-REGULATION | INDUCTION | UP-REGULATION | PROMOTES APOPTOSIS | CUTTING EDGE | P53 | Humans | GPI-Linked Proteins - biosynthesis | NK Cell Lectin-Like Receptor Subfamily K - immunology | Intercellular Signaling Peptides and Proteins - biosynthesis | MicroRNAs - metabolism | RNA, Messenger - metabolism | Transfection | NK Cell Lectin-Like Receptor Subfamily K - metabolism | Melanoma - genetics | HEK293 Cells | Killer Cells, Natural - immunology | 3' Untranslated Regions | Melanoma - metabolism | GPI-Linked Proteins - immunology | Skin Neoplasms - immunology | HCT116 Cells | RNA, Messenger - genetics | Intercellular Signaling Peptides and Proteins - genetics | Tumor Suppressor Protein p53 - metabolism | Imidazoles - pharmacology | Down-Regulation - drug effects | Piperazines - pharmacology | Skin Neoplasms - metabolism | Melanoma - immunology | Skin Neoplasms - genetics | Cell Line, Tumor | MicroRNAs - genetics | Intercellular Signaling Peptides and Proteins - immunology | GPI-Linked Proteins - genetics | Index Medicus
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