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Blood, ISSN 0006-4971, 2009, Volume 114, Issue 13, pp. 2667 - 2677
Journal Article
2008, Advances in experimental medicine and biology, ISBN 0387097880, Volume 640., xxvii, 357
This book shows how the structural similarity of MIRRs determines the general principles underlying MIRR-mediated transmembrane signaling mechanisms. In so... 
Killer cells | Cell receptors | Models, Immunological | Receptors | Signal Transduction | Immune recognition | Receptors, Immunologic | Cellular signal transduction | T cells | Oncology | Biomedicine general | Biomedicine
Book
Journal of Allergy and Clinical Immunology, The, ISSN 0091-6749, 2017, Volume 140, Issue 3, pp. 809 - 821.e3
Background Hypertension is considered an immunologic disorder. However, the role of the IL-17 family in genetic hypertension in the spontaneously hypertensive... 
Allergy and Immunology | Hypertension | CD161 | IL-17 | spontaneously hypertensive rat | digoxin | T cells | retinoic acid receptor–related orphan receptor γt | TH17 | Toll-like receptor | gene expression | innate immune system | INTERLEUKIN 17 | ACTIVATION | ENDOTHELIAL DYSFUNCTION | ORGAN DAMAGE | IMMUNOLOGY | NICOTINIC ACETYLCHOLINE-RECEPTORS | RESPONSES | ALLERGY | INFLAMMATION | T(H)17 | retinoic acid receptor-related orphan receptor gamma t | DIFFERENTIATION | TH17 CELLS | LYMPHOCYTES | Animals, Newborn | Rats, Inbred SHR | Nuclear Receptor Subfamily 1, Group F, Member 3 - immunology | Nuclear Receptor Subfamily 1, Group F, Member 3 - antagonists & inhibitors | Poly I-C - pharmacology | Cells, Cultured | Interleukin-17 - immunology | Interleukin-17 - physiology | Interleukin-17 - genetics | Rats, Inbred WKY | Male | Hypertension - immunology | Spleen - cytology | Toll-Like Receptor 3 - agonists | NK Cell Lectin-Like Receptor Subfamily B - immunology | Digoxin - pharmacology | Hypertension - physiopathology | Vasodilation | Aging - immunology | Animals | Aorta, Thoracic - physiology | Blood Pressure - drug effects | Nuclear Receptor Subfamily 1, Group F, Member 3 - genetics | RNA - metabolism | Flow cytometry | Transcription factors | Digoxin | Disease | Helper cells | Nervous system | Lymphocytes T | Birth | Kinases | Signal transduction | Splenocytes | Lymphocytes | Rodents | Measurement methods | Blood pressure | Immune system | Spleen | Immune response | Cytokines | Rats | Pharmacology | Inflammation | T cell receptors | Interleukin 17 | Endothelium | Acids | Prostate | Surface markers | Retinoic acid | Nicotine
Journal Article
PLoS ONE, ISSN 1932-6203, 06/2013, Volume 8, Issue 6, p. e65860
Dopamine (DA), a neurotransmitter in the nervous system, has been shown to modulate immune function. We have previously reported that five subtypes of DA... 
PROTEIN-KINASE-A | ACTIVATED T-CELLS | NUCLEAR-FACTOR | MULTIDISCIPLINARY SCIENCES | FACTOR-KAPPA-B | PROLIFERATION | SECRETION | MEDIATED MECHANISM | EXPRESSION | LYMPHOCYTES | Enzyme Activators - pharmacology | Phosphorylation | Receptors, Dopamine D3 - metabolism | Receptors, Dopamine D4 - genetics | Receptors, Dopamine D3 - genetics | Isoquinolines - pharmacology | Killer Cells, Natural - immunology | Cyclic AMP-Dependent Protein Kinases - antagonists & inhibitors | Cyclic AMP - metabolism | Second Messenger Systems | Cyclic AMP-Dependent Protein Kinases - metabolism | Dopamine Agonists - pharmacology | Gene Expression | Colforsin - pharmacology | Adenylyl Cyclases - metabolism | Benzazepines - pharmacology | Sulfonamides - pharmacology | Mice, Inbred ICR | Animals | Dopamine Antagonists - pharmacology | Cyclic AMP Response Element-Binding Protein - metabolism | 2,3,4,5-Tetrahydro-7,8-dihydroxy-1-phenyl-1H-3-benzazepine - pharmacology | Cell Line, Tumor | Receptors, Dopamine D5 - metabolism | Receptors, Dopamine D4 - metabolism | Protein Processing, Post-Translational | Quinpirole - pharmacology | Receptors, Dopamine D5 - genetics | Killer Cells, Natural - metabolism | Cytotoxicity, Immunologic | Phenols | Cyclic adenylic acid | Dopamine | Lymphocytes | Dopamine receptors | Drugs | Flow cytometry | Toxicity | Cytotoxicity | Nervous system | Lymphocytes T | mRNA | Activation | Haloperidol | Kinases | Dopamine D4 receptors | Immunosuppressive agents | Proteins | Signal transduction | Receptors | Cell activation | Modulation | Clonal selection | Physiology | Natural killer cells | Dopamine D2 receptors | Spleen | Dopamine D5 receptors | Adenosine | Immune response | Cytokines | Dopamine D3 receptors | T cell receptors | Cyclic AMP response element-binding protein | Gene expression | Lymphoma | Medicine | Cytometry | Signaling | Dopamine D1 receptors | Forskolin | Receptor mechanisms | Laboratory animals
Journal Article
Nature Immunology, ISSN 1529-2908, 05/2012, Volume 13, Issue 5, pp. 439 - 447
Lymphocyte activation must be tightly regulated to ensure sufficient immunity to pathogens and prevent autoimmunity. Protein tyrosine phosphatases (PTPs) serve... 
SIGNAL-TRANSDUCTION | IMMUNE CELLS | T-CELL-RECEPTOR | PTPN22 ALLELIC VARIANT | IMMUNOLOGY | IMMATURE B-CELLS | SRC-FAMILY KINASES | ANTIGEN RECEPTOR | NEGATIVE REGULATORY TYROSINE | CUTTING EDGE | PHOSPHOTYROSINE PHOSPHATASE | Antigens, CD - immunology | Protein Tyrosine Phosphatase, Non-Receptor Type 6 - immunology | Protein Tyrosine Phosphatase, Non-Receptor Type 2 - immunology | Humans | Autoimmunity - genetics | NK Cell Lectin-Like Receptor Subfamily K - immunology | Protein Tyrosine Phosphatases - metabolism | Leukocyte Common Antigens - immunology | Signal Transduction - immunology | Protein Tyrosine Phosphatases - genetics | Protein Tyrosine Phosphatase, Non-Receptor Type 22 - immunology | Signaling Lymphocytic Activation Molecule Family Member 1 | Autoimmunity - immunology | Receptors, Antigen, T-Cell - immunology | Lymphocyte Activation | Protein Tyrosine Phosphatase, Non-Receptor Type 22 - genetics | Receptor-Like Protein Tyrosine Phosphatases, Class 3 - immunology | Protein Tyrosine Phosphatase, Non-Receptor Type 2 - genetics | Receptors, Cell Surface - immunology | Receptors, Antigen, B-Cell - immunology | Animals | Lymphocytes - enzymology | Mice | Protein Tyrosine Phosphatase, Non-Receptor Type 6 - deficiency | Protein Tyrosine Phosphatase, Non-Receptor Type 12 - immunology | Tyrosine | Autoimmunity | Phosphatases | Immune response | Lymphocytes | Physiological aspects | Research | Properties
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 8/2010, Volume 107, Issue 33, pp. 14751 - 14756
In several murine models of autoimmune arthritis, Th17 cells are the dominant initiators of inflammation. In human arthritis the majority of IL-17–secreting... 
T lymphocytes | Synovial fluid | Phenotypes | Cytokines | Juvenile rheumatoid arthritis | Cultured cells | Arthritis | Joint diseases | Joints | T cell antigen receptors | Juvenile | CD161 | RORC2 | RHEUMATOID-ARTHRITIS | GAMMA | MULTIDISCIPLINARY SCIENCES | JUVENILE IDIOPATHIC ARTHRITIS | CD4(+) T-CELLS | INDUCTION | MEMORY CELLS | IL-17 | juvenile | INFLAMED JOINTS | T-H-17 CELLS | LINEAGE | Receptors, CCR6 - immunology | Humans | Interleukin-17 - immunology | Molecular Sequence Data | T-Box Domain Proteins - immunology | Interferon-gamma - metabolism | Th1 Cells - immunology | Th1 Cells - metabolism | Flow Cytometry | T-Lymphocytes, Helper-Inducer - immunology | Base Sequence | Nuclear Receptor Subfamily 1, Group F, Member 3 - metabolism | Receptors, CCR6 - metabolism | Interferon-gamma - genetics | Child | Cell Lineage - genetics | Nuclear Receptor Subfamily 1, Group F, Member 3 - genetics | Transforming Growth Factor beta - immunology | Amino Acid Sequence | Nuclear Receptor Subfamily 1, Group F, Member 3 - immunology | Gene Expression | T-Lymphocytes, Helper-Inducer - metabolism | NK Cell Lectin-Like Receptor Subfamily B - metabolism | Interleukin-17 - genetics | Interleukin-12 - metabolism | Reverse Transcriptase Polymerase Chain Reaction | Arthritis, Juvenile - genetics | NK Cell Lectin-Like Receptor Subfamily B - immunology | T-Box Domain Proteins - genetics | T-Box Domain Proteins - metabolism | Arthritis, Juvenile - metabolism | Interleukin-17 - metabolism | NK Cell Lectin-Like Receptor Subfamily B - genetics | Arthritis, Juvenile - immunology | Interferon-gamma - immunology | Interleukin-12 - immunology | Receptors, CCR6 - genetics | Transforming Growth Factor beta - metabolism | Cell Lineage - immunology | Autoimmunity | Research | Adaptation (Physiology) | Animal models | Transcription factors | Helper cells | Interleukin 12 | Interleukin 23 | Inflammation | Lymphocytes T | Transforming growth factor- beta | double prime T-cell receptor | CCR6 protein | Interleukin 17 | Plasticity | Children | Biological Sciences
Journal Article
PLoS ONE, ISSN 1932-6203, 10/2016, Volume 11, Issue 10, p. e0164517
Journal Article
Current Opinion in Immunology, ISSN 0952-7915, 2004, Volume 16, Issue 5, pp. 626 - 633
Journal Article
European Journal of Immunology, ISSN 0014-2980, 05/2010, Volume 40, Issue 5, pp. 1418 - 1427
Human cytomegalovirus (hCMV) infection is usually asymptomatic but may cause disease in immunocompromised hosts. It has been reported that hCMV infection may... 
C‐type lectins | Killer immunoglobulin‐like receptor(KIR) | T cell | NK cell | Human cytomegalovirus | Killer immunoglobulin-like receptor (KIR) | C-type lectins | NATURAL-KILLER-CELLS | CD94/NKG2A | IMMUNOLOGY | HLA-E | FAMILY | COMPLEX CLASS-I | INHIBITORY RECEPTOR | LIGAND | CLASS-I MOLECULES | EXPRESSION | T-CELLS | Immunoglobulin G - blood | NK Cell Lectin-Like Receptor Subfamily C - biosynthesis | Antigens, CD - biosynthesis | Humans | NK Cell Lectin-Like Receptor Subfamily C - genetics | Child, Preschool | Cytomegalovirus Infections - urine | Infant | Male | Receptors, Natural Killer Cell - biosynthesis | Cytomegalovirus Infections - immunology | Antibodies, Viral - blood | Antigens, CD - genetics | NK Cell Lectin-Like Receptor Subfamily D - biosynthesis | Receptors, Immunologic - biosynthesis | Immunoglobulin G - immunology | NK Cell Lectin-Like Receptor Subfamily D - genetics | Killer Cells, Natural - immunology | Female | Child | Cytomegalovirus - isolation & purification | Leukocyte Immunoglobulin-like Receptor B1 | Urine - virology | Killer Cells, Natural - classification | Saliva - virology | Cytomegalovirus - immunology | Cytomegalovirus Infections - blood | NK Cell Lectin-Like Receptor Subfamily B - genetics | Gene Expression Regulation, Viral | NK Cell Lectin-Like Receptor Subfamily B - biosynthesis | Killer Cells, Natural - metabolism | Receptors, Immunologic - genetics | Receptors, Natural Killer Cell - genetics | Cytomegalovirus | T cell receptors | Infections
Journal Article