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MOLECULAR CANCER RESEARCH, ISSN 1541-7786, 06/2018, Volume 16, Issue 6, pp. 986 - 999
Journal Article
Circulation, ISSN 0009-7322, 02/2017, Volume 135, Issue 9, pp. 881 - 897
BACKGROUND:Chronic heart failure (HF) is associated with altered signal transduction via β-adrenoceptors and G proteins and with reduced cAMP formation.... 
heart failure | receptors, adrenergic, beta | myocardial contraction | signal transduction | ACTIVATION | CARDIAC & CARDIOVASCULAR SYSTEMS | receptors | HETEROTRIMERIC G-PROTEINS | PHOSPHORYLATION | CARDIAC MYOCYTES | BETA-ADRENERGIC-STIMULATION | CYCLIC-AMP | adrenergic | RECOMBINANT ADENOVIRUSES | PERIPHERAL VASCULAR DISEASE | NDPK-B | ENERGY PHOSPHATE TRANSFER | beta | GAMMA DIMERS | Rats, Wistar | Humans | Embryo, Nonmammalian - metabolism | Male | NM23 Nucleoside Diphosphate Kinases - genetics | NM23 Nucleoside Diphosphate Kinases - metabolism | GTP-Binding Protein alpha Subunits, G12-G13 - metabolism | NM23 Nucleoside Diphosphate Kinases - analysis | Protein Isoforms - metabolism | RNA Interference | Protein Isoforms - chemistry | Cell Membrane - metabolism | Cyclic AMP - metabolism | Disease Models, Animal | Cyclic AMP - analysis | Cell Line | Myocytes, Cardiac - cytology | Mice, Inbred C57BL | NM23 Nucleoside Diphosphate Kinases - antagonists & inhibitors | Rats | Heart Failure - metabolism | Heart Failure - pathology | Zebrafish - growth & development | Mice, Knockout | Animals | Myocytes, Cardiac - drug effects | Isoproterenol - pharmacology | Myocytes, Cardiac - metabolism | Protein Binding | Mice | Protein Isoforms - genetics | RNA, Small Interfering - metabolism | Heart failure | Polymerase chain reaction | Care and treatment | Usage | Cellular signal transduction | Research | Index Medicus | Abridged Index Medicus
Journal Article
Arteriosclerosis, Thrombosis, and Vascular Biology, ISSN 1079-5642, 08/2015, Volume 35, Issue 8, pp. 1852 - 1861
OBJECTIVE—Vascular smooth muscle cells (VSMC) proliferation is a hallmark of atherosclerosis and vascular restenosis. The intermediate conductance Ca-activated... 
atherosclerosis | neointima formation | SK4 protein | NM23B nucleoside diphosphate kinase | KCa3.1 protein | Nme2 protein | KCNN4 protein | CD4 T-CELLS | ENDOTHELIAL DYSFUNCTION | REVERSIBLE HISTIDINE PHOSPHORYLATION | PHENOTYPIC MODULATION | 1 protein | KCa3 | CA2+-ACTIVATED K+ CHANNEL | VASCULAR-DISEASE | BLOOD-PRESSURE | BETA-GAMMA DIMERS | HUMAN ERYTHROCYTES | PERIPHERAL VASCULAR DISEASE | HEMATOLOGY | CORONARY SMOOTH-MUSCLE | Cell Proliferation | Intermediate-Conductance Calcium-Activated Potassium Channels - genetics | Myocytes, Smooth Muscle - pathology | Mice, 129 Strain | Intermediate-Conductance Calcium-Activated Potassium Channels - metabolism | NM23 Nucleoside Diphosphate Kinases - genetics | NM23 Nucleoside Diphosphate Kinases - metabolism | Neointima | Carotid Arteries - enzymology | Carotid Artery Injuries - genetics | NM23 Nucleoside Diphosphate Kinases - deficiency | Carotid Artery Injuries - pathology | Disease Models, Animal | Signal Transduction | Myocytes, Smooth Muscle - enzymology | Mice, Inbred C57BL | Cells, Cultured | Mice, Knockout | Muscle, Smooth, Vascular - pathology | Animals | Carotid Artery Injuries - enzymology | Membrane Potentials | Intermediate-Conductance Calcium-Activated Potassium Channels - deficiency | Carotid Arteries - pathology | Muscle, Smooth, Vascular - enzymology | Index Medicus
Journal Article
Journal of Cerebral Blood Flow & Metabolism, ISSN 0271-678X, 7/2017, Volume 37, Issue 7, pp. 2471 - 2484
Journal Article
FASEB Journal, ISSN 0892-6638, 04/2017, Volume 31, Issue 4, pp. 1531 - 1546
Journal Article
Journal of Biological Chemistry, ISSN 0021-9258, 2017, Volume 292, Issue 37, pp. 15205 - 15215
Transcriptional activation of the human telomerase reverse transcriptase (hTERT) gene, which remains repressed in adult somatic cells, is critical during... 
CELLS | ACTIVATION | NM23-H2 | DNA | BIOCHEMISTRY & MOLECULAR BIOLOGY | KINASE | TRANSCRIPTION | GENE-EXPRESSION | HUMAN CANCERS | PROMOTER | GENOME | Epigenetic Repression | Humans | Protein Multimerization | Neoplasm Proteins - antagonists & inhibitors | Fibrosarcoma - metabolism | Neoplasm Proteins - metabolism | NM23 Nucleoside Diphosphate Kinases - genetics | NM23 Nucleoside Diphosphate Kinases - metabolism | Repressor Proteins - antagonists & inhibitors | Fibrosarcoma - pathology | Telomerase - genetics | Chromatin Immunoprecipitation | RNA Interference | Carcinoma - enzymology | G-Quadruplexes | Telomerase - metabolism | Carcinoma - pathology | Neoplasm Proteins - genetics | Genes, Reporter | Repressor Proteins - metabolism | Recombinant Proteins - metabolism | NM23 Nucleoside Diphosphate Kinases - chemistry | Promoter Regions, Genetic | Repressor Proteins - chemistry | Mutagenesis, Site-Directed | Histone Demethylases - chemistry | NM23 Nucleoside Diphosphate Kinases - antagonists & inhibitors | Cells, Cultured | Recombinant Proteins - chemistry | Repressor Proteins - genetics | Neoplasm Proteins - chemistry | Histone Demethylases - metabolism | Point Mutation | Cell Line, Tumor | Carcinoma - metabolism | Telomerase - antagonists & inhibitors | Fibrosarcoma - enzymology | Amino Acid Substitution | Index Medicus | Gene Regulation | transcription regulation | chromatin modification | G-quadruplex | telomerase reverse transcriptase (TERT) | NME2 | REST repressor complex | epigenetics
Journal Article
Arteriosclerosis, Thrombosis, and Vascular Biology, ISSN 1079-5642, 10/2014, Volume 34, Issue 10, pp. 2292 - 2300
OBJECTIVE—Nucleoside diphosphate kinase B (NDPKB) participates in the activation of heterotrimeric and monomeric G proteins, which are pivotal mediators in... 
VEGFR2 | Angiogenesis | Nme2 | NDPKB | Endothelial permeability | Nucleoside diphosphate kinase | CELLS | HETEROTRIMERIC G-PROTEIN | ACTIVATION | ZEBRAFISH | angiogenesis | nucleoside diphosphate kinase | endothelial permeability | BETA-GAMMA DIMERS | IN-VITRO | INHIBITION | VE-CADHERIN | PERIPHERAL VASCULAR DISEASE | MICE | DIFFERENTIATION | HEMATOLOGY | Hindlimb | Cadherins - metabolism | Humans | Ischemia - enzymology | Ischemia - genetics | Zebrafish - embryology | NM23 Nucleoside Diphosphate Kinases - genetics | NM23 Nucleoside Diphosphate Kinases - metabolism | Recovery of Function | Antigens, CD - metabolism | Gene Knockdown Techniques | Transfection | RNA Interference | Time Factors | Gene Expression Regulation, Developmental | Retinal Neovascularization - enzymology | Retinal Neovascularization - genetics | NM23 Nucleoside Diphosphate Kinases - deficiency | Disease Models, Animal | Muscle, Skeletal - blood supply | Signal Transduction | Zebrafish Proteins - metabolism | Mice, Inbred C57BL | Cells, Cultured | Vascular Endothelial Growth Factor Receptor-2 - metabolism | Ischemia - physiopathology | Regional Blood Flow | Zebrafish - genetics | Mice, Knockout | Zebrafish Proteins - deficiency | Animals | Human Umbilical Vein Endothelial Cells - enzymology | Zebrafish - metabolism | Mice | Zebrafish Proteins - genetics | Retinal Neovascularization - physiopathology | Endothelial Cells - enzymology | Neovascularization, Physiologic | Index Medicus
Journal Article
Planta, ISSN 0032-0935, 1/2015, Volume 241, Issue 1, pp. 95 - 107
Main conclusion The study is the first to reveal the proteomic response in plants to a single PAH stress, and indicates that NDPK3 is a positive regulator in... 
Life Sciences | Plant | Phenanthrene | Abiotic stress | Forestry | Positive regulator | Agriculture | Ecology | Plant Sciences | Nucleoside diphosphate kinase-3 | ENHANCED TOLERANCE | IDENTIFICATION | PISUM-SATIVUM | PLANT SCIENCES | METABOLISM | INDUCED OXIDATIVE STRESS | PHYTOREMEDIATION | FUNCTIONAL-ANALYSIS | PLANTS | EXPRESSION | POLYACRYLAMIDE-GEL | Proteome - genetics | Reactive Oxygen Species - metabolism | Glutathione Reductase - metabolism | Peroxiredoxins - metabolism | Electrophoresis, Gel, Two-Dimensional | NM23 Nucleoside Diphosphate Kinases - genetics | NM23 Nucleoside Diphosphate Kinases - metabolism | Glutathione Reductase - genetics | Arabidopsis Proteins - metabolism | Ascorbate Peroxidases - metabolism | Glutathione Transferase - genetics | Peroxiredoxins - genetics | Ascorbate Peroxidases - genetics | Stress, Physiological - drug effects | Proteomics - methods | Gene Ontology | Arabidopsis Proteins - genetics | Polycyclic Aromatic Hydrocarbons - pharmacology | Arabidopsis - drug effects | Catalase - genetics | Glutathione Transferase - metabolism | Transcriptome - drug effects | Reverse Transcriptase Polymerase Chain Reaction | Arabidopsis - metabolism | Arabidopsis - genetics | Catalase - metabolism | Gene Expression Regulation, Plant - drug effects | Signal Transduction - drug effects | Mutation | Proteome - metabolism | Arabidopsis thaliana | Polycyclic aromatic hydrocarbons | Glutathione transferase | Analysis | Nucleosides | Physiological aspects | Forests and forestry | Superoxide | Plants | Index Medicus
Journal Article