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Journal Article
JOURNAL OF NEURAL TRANSMISSION, ISSN 0300-9564, 08/2014, Volume 121, Issue 8, pp. 1029 - 1075
Journal Article
Neuroscience, ISSN 0306-4522, 2007, Volume 146, Issue 3, pp. 1169 - 1181
Abstract The in vitro differentiation of P19 murine embryonal carcinoma cells to neurons resembles developmental stages which are encountered during neuronal... 
Neurology | purinergic receptors | P19 embryonal carcinoma cells | ATP | neuronal differentiation | maturation of synapses | intrinsic modulation of cholinergic and glutamate–NMDA receptor activity | intrinsic modulation of cholinergic and glutamate-NMDA receptor activity | STEM-CELLS | INTRACELLULAR CALCIUM | NEUROSCIENCES | P2Y RECEPTORS | SELECTIVE EXPRESSION | GENE-EXPRESSION | CENTRAL-NERVOUS-SYSTEM | NEURAL PROGENITOR CELLS | LONG-TERM POTENTIATION | EXTRACELLULAR NUCLEOTIDES | GROWTH-FACTOR | Immunohistochemistry | Purinergic P1 Receptor Antagonists | Receptors, N-Methyl-D-Aspartate - metabolism | Receptors, Muscarinic - biosynthesis | Neoplastic Stem Cells - metabolism | Receptors, Purinergic P2 - biosynthesis | Receptors, Purinergic P2 - genetics | Neurons - metabolism | Purinergic Antagonists | Pyridoxal Phosphate - analogs & derivatives | Cell Differentiation - physiology | Suramin - pharmacology | Receptors, Muscarinic - genetics | Cell Line | Triazines - pharmacology | Pyridoxal Phosphate - pharmacology | Receptors, Cholinergic - metabolism | Receptors, Purinergic P1 - genetics | Receptors, Purinergic P1 - biosynthesis | Embryonal Carcinoma Stem Cells | Purinergic P2 Receptor Antagonists | Reverse Transcriptase Polymerase Chain Reaction | Blotting, Western | Receptors, Purinergic - genetics | Microscopy, Confocal | Animals | Fluorescent Antibody Technique | Receptors, Nicotinic - biosynthesis | Mice | Mice, Inbred BALB C | Receptors, Nicotinic - genetics | Receptors, Purinergic - biosynthesis | Cell research | Carcinoma | Neurons | Cancer
Journal Article
Journal Article
Behavioural Brain Research, ISSN 0166-4328, 2009, Volume 205, Issue 1, pp. 96 - 101
There is increasing evidence that a dysfunction of the N-methyl- d-aspartate (NMDA) receptor system plays a key role in the pathophysiology of schizophrenia.... 
Development | NMDA | Schizophrenia | CNS | Behaviour | NEUROSCIENCES | HYPOFUNCTION MODEL | MK-801 | CREB | MEMORY | PSYCHOSIS | OBJECT RECOGNITION | BEHAVIORAL SCIENCES | BLOCKADE | TEMPORAL CORTEX | METHYL-D-ASPARTATE | COGNITIVE DEFICITS | Methyl aspartate | Lymphocytes | Health aspects
Journal Article
Brain, Behavior, and Immunity, ISSN 0889-1591, 2016, Volume 57, pp. 360 - 370
Journal Article
Neuron, ISSN 0896-6273, 03/2012, Volume 73, Issue 5, pp. 962 - 977
Chronic stress could trigger maladaptive changes associated with stress-related mental disorders; however, the underlying mechanisms remain elusive. In this... 
UBIQUITIN-PROTEASOME SYSTEM | DENDRITIC MORPHOLOGY | RAT | HIPPOCAMPAL | NEURONS | SCHIZOPHRENIA | C-ELEGANS | SYNAPTIC PLASTICITY | NEUROSCIENCES | BRAIN | WORKING-MEMORY | Receptors, Glutamate - metabolism | RNA, Small Interfering - genetics | Immunoprecipitation | Prefrontal Cortex - physiopathology | 2-Amino-5-phosphonovalerate - pharmacology | Male | Excitatory Postsynaptic Potentials - drug effects | Prefrontal Cortex - pathology | Cognition Disorders - etiology | 6-Cyano-7-nitroquinoxaline-2,3-dione - pharmacology | Restraint, Physical - adverse effects | Nedd4 Ubiquitin Protein Ligases | Pyramidal Cells - drug effects | Disease Models, Animal | GABA-A Receptor Antagonists | Pyramidal Cells - physiopathology | Receptors, Glutamate - genetics | F-Box Proteins - metabolism | Cognition Disorders - pathology | Endosomal Sorting Complexes Required for Transport - metabolism | Ubiquitin-Protein Ligases - metabolism | Rats | Excitatory Amino Acid Antagonists - pharmacology | Bicuculline - pharmacology | Rats, Sprague-Dawley | Neuropsychological Tests | Stress, Psychological - pathology | Animals | Analysis of Variance | Prefrontal Cortex - metabolism | In Vitro Techniques | Recognition (Psychology) | Stress, Psychological - complications | RNA, Small Interfering - metabolism | Ubiquitin | Corticosteroids | Disease transmission | Neurons | Ligases | Physiological aspects | Amino acids | Stress (Psychology) | Glutamate | Mental illness | Mental disorders | Rodents | Memory | Behavior | Experiments | Variance analysis | Stress | stress | glucocorticoid receptor | AMPA receptor | ubiquitination | degradation | corticosterone | NMDA receptor | recognition memory
Journal Article
Cancer Science, ISSN 1347-9032, 12/2018, Volume 109, Issue 12, pp. 3874 - 3882
Autocrine and paracrine factors, including glutamate and epidermal growth factor (EGF), are potent inducers of brain tumor cell invasion, a pathological... 
xCT | glioma | epidermal growth factor receptor | GluN2B | glutamate | MIGRATION | CELLS | TYROSINE PHOSPHORYLATION | NMDA RECEPTORS | EGFR | INVASION | ONCOLOGY | BIOLOGY | DEFICIENT | EXPRESSION | MODULATION | Neoplasm Transplantation | Phosphorylation | Humans | Receptors, N-Methyl-D-Aspartate - metabolism | Sulfasalazine - pharmacology | Brain Neoplasms - metabolism | Glioma - metabolism | Amino Acid Transport System y+ - metabolism | Receptors, N-Methyl-D-Aspartate - chemistry | Dizocilpine Maleate - administration & dosage | Protein Domains | Cell Survival - drug effects | ErbB Receptors - metabolism | Brain Neoplasms - drug therapy | Disease Progression | Drug Synergism | Sulfasalazine - administration & dosage | Cell Movement - drug effects | Animals | Signal Transduction - drug effects | Cell Line, Tumor | Cell Proliferation - drug effects | Glutamic Acid - metabolism | Mice | Epidermal Growth Factor - pharmacology | Dizocilpine Maleate - pharmacology | Glioma - drug therapy | Methyl aspartate | Epidermal growth factor | Gliomas | Brain tumors | Development and progression | Aspartate | Glutamate | Immunoglobulins | Cell survival | Epidermal growth factor receptors | Paracrine signalling | N-Methyl-D-aspartic acid receptors | Glutamic acid receptors | Kinases | Cell adhesion & migration | Glioma cells | Sulfasalazine | Autocrine signalling | Cell migration | Tumors | Original
Journal Article