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Annual Review of Immunology, ISSN 0732-0582, 2009, Volume 27, Issue 1, pp. 229 - 265
Journal Article
Journal of Autoimmunity, ISSN 0896-8411, 2015, Volume 66, pp. 76 - 88
Journal Article
2016, Methods in molecular biology, ISBN 1493935666, Volume 1417
Web Resource
Journal Article
American Journal of Obstetrics and Gynecology, ISSN 0002-9378, 03/2018, Volume 218, Issue 3, pp. 294 - 314.e2
Evidence from family and twin-based studies provide strong support for a significant contribution of maternal and fetal genetics to the timing of parturition... 
rare variants | single nucleotide polymorphism | nucleotide oligomerization domain (NOD) protein | inflammatory bowel disease | periodontal disease | sequencing | NOD-like receptor (NLR) | mutation | genetics | innate immunity | inflammation | inflammasome | genome-wide association | missing heritability | toll-like receptor (TLR) | interleukin | DNA variant | preterm premature rupture of membranes (PPROM) | INTERLEUKIN-1 RECEPTOR ANTAGONIST | ADVERSE PREGNANCY OUTCOMES | ACTIVATING PEPTIDE-1 INTERLEUKIN-8 | OBSTETRICS & GYNECOLOGY | AMNIOTIC-FLUID INTERLEUKIN-6 | TUMOR-NECROSIS-FACTOR | NOD-LIKE RECEPTORS | FACTOR-ALPHA GENE | INFLAMMATORY-BOWEL-DISEASE | SINGLE-NUCLEOTIDE POLYMORPHISMS | Genetic Predisposition to Disease | Genome-Wide Association Study | Premature Birth - genetics | Humans | Immunity, Innate - genetics | Premature Birth - etiology | Whole Exome Sequencing | Pregnancy | Inflammation - complications | Inflammatory Bowel Diseases - genetics | Inflammation - genetics | Female | Periodontal Diseases - genetics | Mutations | high penetrance | DNA Variants | NOD-like receptors (NLR) | Nucleotide-binding Oligomerization Domain (NOD) proteins | Innate immunity | Inflammasome | Inflammation | Preterm premature rupture of membranes (PPROM) | Inflammatory bowel disease | Exomes | Periodontal disease | Toll-like receptors (TLR) | Defensins | Interleukins | Transcriptomics | Genetics | single nucleotide polymorphisms | Sequencing | genome wide association studies
Journal Article
DIABETES CARE, ISSN 0149-5992, 09/2012, Volume 35, Issue 9, pp. 1817 - 1820
OBJECTIVE-Type 1 diabetes is a condition in which pancreatic islets are destroyed by self-reactive T cells. The process is facilitated by deficits in the... 
NOD MICE | ENDOCRINOLOGY & METABOLISM | MELLITUS
Journal Article