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1. Full Text NOD2-mediated dysbiosis predisposes mice to transmissible colitis and colorectal cancer
by Couturier-Maillard, AurĆ©lie and Secher, Thomas and Rehman, Ateequr and Normand, Sylvain and De Arcangelis, AdĆØle and Haesler, Robert and Huot, Ludovic and Grandjean, Teddy and Bressenot, Aude and Delanoye-Crespin, Anne and Gaillot, Olivier and Schreiber, Stefan and Lemoine, Yves and Ryffel, Bernhard and Hot, David and NĆ¹Ć±ez, Gabriel and Chen, Grace and Rosenstiel, Philip and Chamaillard, Mathias
The Journal of clinical investigation, ISSN 0021-9738, 02/2013, Volume 123, Issue 2, pp. 700 - 711
Life Sciences & Biomedicine | Medicine, Research & Experimental | Science & Technology | Research & Experimental Medicine | Nod2 Signaling Adaptor Protein - deficiency | Humans | Colitis - pathology | Male | Nod2 Signaling Adaptor Protein - genetics | Colorectal Neoplasms - etiology | Female | Colorectal Neoplasms - metabolism | Disease Models, Animal | Digestive System - metabolism | Risk Factors | Colorectal Neoplasms - microbiology | Mice, Knockout | Digestive System - pathology | Pregnancy | Colitis - etiology | Receptor-Interacting Protein Serine-Threonine Kinases - genetics | Animals | Colitis - microbiology | Colitis - metabolism | Mice | Receptor-Interacting Protein Serine-Threonine Kinases - deficiency | Colorectal Neoplasms - pathology | Metagenome | Digestive System - microbiology | Care and treatment | Microbiota (Symbiotic organisms) | Colorectal cancer | Physiological aspects | Genetic aspects | Research | Colitis | Risk factors | Index Medicus | Abridged Index Medicus | Receptor-Interacting Protein Serine-Threonine Kinases | Nod2 Signaling Adaptor Protein | Life Sciences | Immunology | Digestive System | Colorectal Neoplasms
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2. Full Text THP-1 Cells and Pro-Inflammatory Cytokine Production: An In Vitro Tool for Functional Characterization of NOD1/NOD2 Antagonists
by Jakopin, Ziga and Corsini, Emanuela
International journal of molecular sciences, ISSN 1422-0067, 09/2019, Volume 20, Issue 17, p. 4265
Biochemistry & Molecular Biology | Physical Sciences | Chemistry | Life Sciences & Biomedicine | Chemistry, Multidisciplinary | Science & Technology | Nod2 Signaling Adaptor Protein - metabolism | Cell Survival | Humans | Nod1 Signaling Adaptor Protein - agonists | Inflammation Mediators - metabolism | Nod2 Signaling Adaptor Protein - antagonists & inhibitors | THP-1 Cells | Nod2 Signaling Adaptor Protein - agonists | Nod1 Signaling Adaptor Protein - antagonists & inhibitors | Nod1 Signaling Adaptor Protein - metabolism | Toll-Like Receptor 4 - metabolism | Cytokines - biosynthesis | NF-ĪŗB protein | Cytokines | Downstream effects | Interleukin | Cytotoxicity | Leukocytes (mononuclear) | Antagonists | Assaying | Kinases | Blood | Cell density | Monocytes | Cell activation | Reporter gene | Peripheral blood mononuclear cells | Ligands | Nod1 protein | Autoimmune diseases | Interleukin 8 | NOD2 protein | Index Medicus | THP-1 | immunomodulation | NOD1 antagonist | TNF-Ī± | cytokines | NOD2 antagonist | IL-8 | functional characterization
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3. Full Text Innate lymphoid cells support regulatory T cells in the intestine through interleukin-2
by Zhou, Lei and Chu, Coco and Teng, Fei and Bessman, Nicholas J and Goc, Jeremy and Santosa, Endi K and Putzel, Gregory G and Kabata, Hiroki and Kelsen, Judith R and Baldassano, Robert N and Shah, Manish A and Sockolow, Robbyn E and Vivier, Eric and Eberl, Gerard and Smith, Kendall A and Sonnenberg, Gregory F
Nature (London), ISSN 0028-0836, 04/2019, Volume 568, Issue 7752, pp. 405 - 409
Science & Technology - Other Topics | Multidisciplinary Sciences | Science & Technology | Inflammation - pathology | T-Lymphocytes, Regulatory - metabolism | Nod2 Signaling Adaptor Protein - deficiency | T-Lymphocytes, Regulatory - classification | Humans | Crohn Disease - metabolism | Male | Nod2 Signaling Adaptor Protein - genetics | Intestines - immunology | T-Lymphocytes, Regulatory - immunology | Antigens - administration & dosage | Interleukin-2 - immunology | Interleukin-1beta - metabolism | Female | Intestine, Small - immunology | Macrophages - immunology | Interleukin-2 - metabolism | Myeloid Differentiation Factor 88 - genetics | Homeostasis - immunology | Interleukin-1beta - immunology | Inflammation - immunology | Crohn Disease - immunology | Immunity, Innate - immunology | Macrophages - metabolism | Animals | Myeloid Differentiation Factor 88 - deficiency | Intestine, Small - cytology | Nod2 Signaling Adaptor Protein - metabolism | Crohn Disease - pathology | Interleukin-2 - deficiency | Antigens - immunology | Mice | Gastrointestinal Microbiome - immunology | Myeloid Differentiation Factor 88 - metabolism | Intestines - cytology | Care and treatment | Crohn's disease | Interleukin-2 | Diagnosis | Research | Genetic regulation | Flow cytometry | Inflammatory bowel diseases | Interleukin | Science | Homeostasis | Lymphocytes T | Macrophages | Small intestine | Cell adhesion & migration | Microbiota | Cell activation | Cell growth | Immunology | Clonal deletion | Interleukin 2 | Lymphocytes | Population | NOD2 protein | Cellular manufacture | Antigens | Lymphatic system | Lymphoid cells | Cytokines | Dendritic cells | Immunoregulation | Gastrointestinal tract | IL-1Ī² | Disease control | Immunological tolerance | Diet | Antibiotics | Large intestine | Gastrointestinal system | MyD88 protein | Index Medicus
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4. Full Text Trace levels of peptidoglycan in serum underlie the NOD-dependent cytokine response to endoplasmic reticulum stress
by Molinaro, Raphael and Mukherjee, Tapas and Flick, Robert and Philpott, Dana J and Girardin, Stephen E
The Journal of biological chemistry, ISSN 0021-9258, 05/2019, Volume 294, Issue 22, pp. 9007 - 9015
Life Sciences & Biomedicine | Biochemistry & Molecular Biology | Science & Technology | Nod2 Signaling Adaptor Protein - deficiency | Cytokines - metabolism | Endoplasmic Reticulum Stress - drug effects | Calcium - metabolism | HCT116 Cells | Humans | Nod1 Signaling Adaptor Protein - deficiency | Nod2 Signaling Adaptor Protein - genetics | Calcimycin - chemistry | Gene Knockout Techniques | Calcimycin - pharmacology | Calcium - chemistry | Nod1 Signaling Adaptor Protein - metabolism | Peptidoglycan - blood | Nod2 Signaling Adaptor Protein - metabolism | Signal Transduction - drug effects | Thapsigargin - pharmacology | Interleukin-8 - metabolism | Chemokine CXCL1 - metabolism | Nod1 Signaling Adaptor Protein - genetics | Index Medicus | Immunology | cell biology | innate immunity | Nod-like receptor (NLR) | cell signaling | calcium intracellular release | endoplasmic reticulum stress (ER stress) | NOD1 | NOD2
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5. Full Text Cellular and molecular mechanisms underlying NOD2 riskā€associated polymorphisms in Crohn's disease
by Strober, Warren and Asano, Naoki and Fuss, Ivan and Kitani, Atsushi and Watanabe, Tomohiro
Immunological reviews, ISSN 0105-2896, 07/2014, Volume 260, Issue 1, pp. 249 - 260
defensins | Crohn's disease | colitis | NOD2 | microbiome | IRFā€4 | Colitis | Defensins | IRF-4 | Microbiome | Life Sciences & Biomedicine | Immunology | Science & Technology | Crohn Disease - genetics | Intestinal Mucosa - metabolism | Nod2 Signaling Adaptor Protein - deficiency | Defensins - biosynthesis | Humans | Inflammatory Bowel Diseases - immunology | Crohn Disease - metabolism | Nod2 Signaling Adaptor Protein - genetics | Intestines - metabolism | Inflammatory Bowel Diseases - metabolism | Intestines - immunology | Microbiota | Intestinal Mucosa - immunology | Inflammatory Bowel Diseases - genetics | Crohn Disease - microbiology | Paneth Cells - metabolism | Genetic Predisposition to Disease | Genetic Association Studies | Immunomodulation | Intestinal Mucosa - microbiology | Immunity, Innate | Crohn Disease - immunology | Polymorphism, Genetic | Animals | Intestines - microbiology | Nod2 Signaling Adaptor Protein - metabolism | Inflammatory Bowel Diseases - microbiology | Microbiota (Symbiotic organisms) | Genetic aspects | Index Medicus
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6. Full Text Nucleotide-Binding Oligomerization Domain-2 Inhibits Toll-Like Receptor-4 Signaling in the Intestinal Epithelium
by Richardson, Ward M and Sodhi, Chhinder P and Russo, Anthony and Siggers, Richard H and Afrazi, Amin and Gribar, Steven C and Neal, Matthew D and Dai, Shipan and Prindle, Thomas and Branca, Maria and Ma, Congrong and Ozolek, John and Hackam, David J
Gastroenterology (New York, N.Y. 1943), ISSN 0016-5085, 2010, Volume 139, Issue 3, pp. 904 - 917.e6
Gastroenterology and Hepatology | TLR4 | NEC | NOD2 | Gastroenterology & Hepatology | Life Sciences & Biomedicine | Science & Technology | Intestinal Mucosa - metabolism | Nod2 Signaling Adaptor Protein - deficiency | Age Factors | Endotoxemia - genetics | Enterocytes - metabolism | Enterocolitis, Necrotizing - genetics | Enterocolitis, Necrotizing - metabolism | Humans | NF-kappa B - metabolism | Nod2 Signaling Adaptor Protein - genetics | Endotoxemia - pathology | Intestinal Mucosa - drug effects | Mitochondrial Proteins - metabolism | Nod2 Signaling Adaptor Protein - agonists | Enterocolitis, Necrotizing - pathology | Disease Models, Animal | Animals, Newborn | Severity of Illness Index | Cell Line | Transduction, Genetic | Enterocolitis, Necrotizing - prevention & control | Mice, Inbred C57BL | Rats | Toll-Like Receptor 4 - genetics | Toll-Like Receptor 4 - metabolism | Mice, Knockout | Acetylmuramyl-Alanyl-Isoglutamine - pharmacology | Animals | Carrier Proteins - metabolism | Nod2 Signaling Adaptor Protein - metabolism | Signal Transduction - drug effects | Protein Array Analysis | Mice | Endotoxemia - metabolism | Apoptosis | Cell Movement | Intestinal Mucosa - pathology | Oligomers | Fluorescence | Mitogens | Nitric oxide | Index Medicus | Abridged Index Medicus
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7. Full Text Crohnā€™s Disease Variants of Nod2 Are Stabilized by the Critical Contact Region of Hsp70
by Schaefer, Amy K and Wastyk, Hannah C and Mohanan, Vishnu and Hou, Ching-Wen and Lauro, Mackenzie L and Melnyk, James E and Burch, Jason M and Grimes, Catherine L
Biochemistry (Easton), ISSN 0006-2960, 08/2017, Volume 56, Issue 34, pp. 4445 - 4448
Life Sciences & Biomedicine | Biochemistry & Molecular Biology | Science & Technology | Crohn Disease - genetics | HSP70 Heat-Shock Proteins - immunology | Humans | Nod2 Signaling Adaptor Protein - chemistry | Crohn Disease - metabolism | Nod2 Signaling Adaptor Protein - immunology | HSP70 Heat-Shock Proteins - genetics | Nod2 Signaling Adaptor Protein - genetics | Mutation, Missense | Crohn Disease - immunology | HSP70 Heat-Shock Proteins - metabolism | Nod2 Signaling Adaptor Protein - metabolism | Protein Domains | HSP70 Heat-Shock Proteins - chemistry | Protein Stability | Amino Acid Substitution | Index Medicus | Hsp70 | nucleotide cofactors | chaperone | innate immunity | muramyl dipeptide | Nod2 | protein-protein interaction | protein stability
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8. Full Text Genetic deficiency of NOD2 confers resistance to invasive aspergillosis
by Gresnigt, M.S and Cunha, C and Jaeger, M and Goncalves, S.M and Malireddi, R.K.S and Ammerdorffer, A and Lubbers, R and Oosting, M and Rasid, O and Jouvion, G and Fitting, C and Jong, D.J. de and Lacerda, J.F and Campos, A and Melchers, W.J.G and Lagrou, K and Maertens, J and Kanneganti, T.D and Carvalho, A and Ibrahim-Granet, O and Veerdonk, F.L. van de
Nature communications, ISSN 2041-1723, 2018, Volume 9, Issue 1, pp. 2636 - 15
Science & Technology - Other Topics | Multidisciplinary Sciences | Science & Technology | Lung - pathology | Nod2 Signaling Adaptor Protein - deficiency | Disease Resistance | Cytokines - metabolism | Humans | Lectins, C-Type | Mice, Inbred C57BL | Risk Factors | Aspergillosis - genetics | Male | Nod2 Signaling Adaptor Protein - genetics | Microbial Viability | Aspergillus | Animals | Aspergillosis - etiology | Aspergillosis - microbiology | Hematopoietic Stem Cell Transplantation - adverse effects | Polymorphism, Single Nucleotide - genetics | Female | Lung - metabolism | Paranasal Sinuses - pathology | Phagocytosis | Fungicides | Stem cell transplantation | Infections | Transplantation | Genetic diversity | Macrophages | Gene polymorphism | Hemopoiesis | Monocytes | Cell activation | Aspergillosis | Immunocompromised hosts | Polymorphism | NOD2 protein | Index Medicus
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9. Full Text The role of NOD1 and NOD2 in host defense against chlamydial infection
by Zou, Yan and Lei, Wenbo and He, Zhansheng and Li, Zhongyu
FEMS microbiology letters, ISSN 0378-1097, 09/2016, Volume 363, Issue 17, p. fnw170
peptidoglycan | NOD1 | NOD2 | inflammation | chlamydia | host defense | Life Sciences & Biomedicine | Microbiology | Science & Technology | Nod1 Signaling Adaptor Protein - chemistry | Cytokines - metabolism | Signal Transduction | Humans | Nod2 Signaling Adaptor Protein - chemistry | Peptidoglycan - chemistry | Nod2 Signaling Adaptor Protein - genetics | Inflammation | Chlamydia - immunology | Immunity, Innate | Nod1 Signaling Adaptor Protein - metabolism | Toll-Like Receptors | Chlamydia Infections - immunology | Animals | Chlamydia Infections - microbiology | Nod2 Signaling Adaptor Protein - metabolism | Chlamydia - pathogenicity | Mice | Nod1 Signaling Adaptor Protein - genetics | Oligomerization | Cytokines | Cell walls | Peptidoglycans | Infections | Parasites | Nucleotides | Pattern recognition | Proteins | Signal transduction | Receptors | Toll-like receptors | Intracellular | Nod1 protein | NOD2 protein | Index Medicus
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10. Full Text Disease phenotype and genotype are associated with shifts in intestinalā€associated microbiota in inflammatory bowel diseases
by Frank, Daniel N and Robertson, Charles E and Hamm, Christina M and Kpadeh, Zegbeh and Zhang, Tianyi and Chen, Hongyan and Zhu, Wei and Sartor, R. Balfour and Boedeker, Edgar C and Harpaz, Noam and Pace, Norman R and Li, Ellen
Inflammatory bowel diseases, ISSN 1078-0998, 01/2011, Volume 17, Issue 1, pp. 179 - 184
microbiota | NOD2 | inflammatory bowel diseases | ATG16L1 | Gastroenterology & Hepatology | Life Sciences & Biomedicine | Science & Technology | Crohn Disease - genetics | Autophagy-Related Proteins | Intestines - pathology | Prognosis | Humans | Colitis, Ulcerative - genetics | Genotype | Nod2 Signaling Adaptor Protein - genetics | Colitis, Ulcerative - pathology | RNA, Ribosomal - genetics | Case-Control Studies | Carrier Proteins - genetics | Metagenome - genetics | Phenotype | Intestines - microbiology | Colitis, Ulcerative - microbiology | Crohn Disease - pathology | Polymorphism, Single Nucleotide - genetics | Crohn Disease - microbiology | Inflammatory bowel diseases | Statistical analysis | rRNA 16S | Data processing | Crohn's disease | Multivariate analysis | Intestine | Surgery | Mathematical models | Models | Age | Genotypes | NOD2 protein | Ulcerative colitis | Index Medicus
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