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index medicus (534) 534
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Journal of Experimental Medicine, ISSN 0022-1007, 08/2012, Volume 209, Issue 9, pp. 1537 - 1551
Journal Article
Cancer Research, ISSN 0008-5472, 10/2009, Volume 69, Issue 19, pp. 7569 - 7576
Journal Article
Cancer Research, ISSN 0008-5472, 03/2009, Volume 69, Issue 6, pp. 2400 - 2407
Despite rapid advances in many fronts, pancreatic cancer (PC) remains one of the most difficult human malignancies to treat due, in part, to de novo and... 
TARGET | STEM-CELLS | GROWTH INHIBITION | INVASION | TUMOR PROGRESSION | ONCOLOGY | PROSTATE-CANCER | TAMOXIFEN RESISTANCE | DOWN-REGULATION | E-CADHERIN | NF-KAPPA-B | RNA, Small Interfering - genetics | Pancreatic Neoplasms - metabolism | Receptors, Notch - metabolism | Humans | Deoxycytidine - pharmacology | Drug Resistance, Neoplasm | Intercellular Signaling Peptides and Proteins - biosynthesis | NF-kappa B - metabolism | Receptor, Notch2 - genetics | Receptors, Notch - genetics | Proto-Oncogene Proteins - biosynthesis | Cell Movement - physiology | Pancreatic Neoplasms - drug therapy | Intercellular Signaling Peptides and Proteins - metabolism | Transfection | Receptors, Notch - biosynthesis | Serrate-Jagged Proteins | Antimetabolites, Antineoplastic - pharmacology | Membrane Proteins - metabolism | Jagged-1 Protein | Calcium-Binding Proteins - metabolism | Proto-Oncogene Proteins - metabolism | Calcium-Binding Proteins - biosynthesis | Signal Transduction | Membrane Proteins - genetics | Down-Regulation | Pancreatic Neoplasms - pathology | Intercellular Signaling Peptides and Proteins - genetics | Receptor, Notch2 - metabolism | Epithelial Cells - pathology | Pancreatic Neoplasms - genetics | Proto-Oncogene Proteins - genetics | Membrane Proteins - biosynthesis | Phenotype | Receptor, Notch2 - biosynthesis | Mesoderm - pathology | RNA, Messenger | Deoxycytidine - analogs & derivatives | Receptor, Notch4 | Calcium-Binding Proteins - genetics | Index Medicus
Journal Article
Cell Death and Differentiation, ISSN 1350-9047, 11/2018, Volume 25, Issue 10, pp. 1837 - 1854
Zika virus (ZV) infects neural stem cells (NSCs) and causes quiescence in NSCs, reducing the pool of brain cells, leading to microcephaly. Despite... 
PROGENITOR CELLS | NOTCH2 | APOPTOSIS | ACTIVATION | BIOCHEMISTRY & MOLECULAR BIOLOGY | NEUROGENESIS | PROLIFERATION | INFECTION | DIFFERENTIATION | PRECURSOR CELLS | BRAIN | CELL BIOLOGY | Up-Regulation | MicroRNAs - antagonists & inhibitors | Humans | MicroRNAs - metabolism | Receptor, Notch2 - genetics | Gene Regulatory Networks | Neural Stem Cells - cytology | Viral Envelope Proteins - metabolism | G1 Phase Cell Cycle Checkpoints | Cell Differentiation | 3' Untranslated Regions | Zika Virus - metabolism | Viral Envelope Proteins - genetics | Signal Transduction | Cell Survival | Down-Regulation | Neural Stem Cells - virology | Receptor, Notch2 - metabolism | Genetic Vectors - metabolism | Genetic Vectors - genetics | Fetus - cytology | Viral Nonstructural Proteins - metabolism | Receptor, Notch2 - chemistry | Antagomirs - metabolism | MicroRNAs - chemistry | Neural Stem Cells - metabolism | Apoptosis | Platelet-derived growth factor | Wnt protein | p53 Protein | Stem cell transplantation | Zika virus | Subventricular zone | Proteins | Epidermal growth factor | Cell cycle | Neurospheres | Pax3 protein | G1 phase | Structural proteins | Fetuses | MiRNA | Envelope protein | Electroporation | Microencephaly | Molecular modelling | 3' Untranslated regions | MicroRNAs | Stem cells | Neural stem cells | Ventricle | Cell migration | Cell biology | Neuroscience
Journal Article
PLoS ONE, ISSN 1932-6203, 02/2010, Volume 5, Issue 2, p. e9258
Background: The Notch pathway is essential for proper epidermal differentiation during embryonic skin development. Moreover, skin specific loss of Notch... 
B-CELL DEVELOPMENT | HUMAN EPITHELIAL-CELLS | STEM-CELLS | IN-VITRO | MOUSE KERATINOCYTES | BIOLOGY | MICE | T-CELL | GROWTH-FACTOR | EXPRESSION | THYMIC STROMAL LYMPHOPOIETIN | Dermatitis, Atopic - genetics | Skin - metabolism | Humans | Receptor, Notch2 - genetics | Receptors, Notch - genetics | Myeloproliferative Disorders - genetics | Granulocyte Colony-Stimulating Factor - genetics | Dermatitis, Atopic - mortality | Flow Cytometry | Receptors, Cytokine - metabolism | Receptors, Cytokine - genetics | Granulocyte Colony-Stimulating Factor - metabolism | Skin - pathology | Immunoglobulins | Cytokines - metabolism | Mice, Inbred C57BL | Receptors, Notch - physiology | Mice, Transgenic | Survival Rate | Signal Transduction - genetics | Dermatitis, Atopic - physiopathology | Reverse Transcriptase Polymerase Chain Reaction | Mice, Knockout | Receptor, Notch2 - physiology | Animals | Mice, Nude | Models, Biological | Survival Analysis | Myeloproliferative Disorders - physiopathology | Signal Transduction - physiology | Mice | Skin - physiopathology | Receptor, Notch1 - genetics | Myeloproliferative Disorders - mortality | Receptor, Notch1 - physiology | Embryonic development | Psoriasis | Atopic dermatitis | Bone marrow | Transplantation | Skin | B cells | Syngeneic grafts | Pathogenesis | Bone marrow transplantation | Homeostasis | Lichen planus | Inactivation | Dermatitis | Thymus | Signal transduction | Embryogenesis | Biomedical materials | Immunology | Granulocytes | Granulocyte colony-stimulating factor | Lymphocytes | Rodents | Biocompatibility | Drug dosages | Osteopenia | Neutropenia | Spleen | Repressing | Deactivation | Cytokines | Developmental biology | Dermatology | Leukocytes (eosinophilic) | Neutrophils | Keratinocytes | Breast cancer | Gene expression | Myeloproliferative diseases | Hemopoiesis | Signaling | Thymic stromal lymphopoietin | Stem cells | Mast cells | Infiltration | Notch protein | Bone | Hematopoietic system | Mutation | Eosinophils
Journal Article
The Journal of Clinical Endocrinology & Metabolism, ISSN 0021-972X, 11/2017, Volume 102, Issue 11, pp. 4163 - 4172
CONTEXT:Hajdu-Cheney syndrome (HJCYS) is a rare, multisystem bone disease caused by heterozygous mutations in the NOTCH2 gene. Histomorphometric and bone... 
ACROOSTEOLYSIS | PROXIMAL RADIUS | MINERALIZATION DENSITY DISTRIBUTION | ADOLESCENTS | ENDOCRINOLOGY & METABOLISM | SEVERE OSTEOPOROSIS | NOTCH2 CAUSE | DISORDER | FOLLOW-UP | NORMATIVE DATA | TRUNCATING MUTATIONS | Bone and Bones - physiology | Calcification, Physiologic - drug effects | Humans | Bone Density Conservation Agents - therapeutic use | Male | Receptor, Notch2 - genetics | Hajdu-Cheney Syndrome - metabolism | Hajdu-Cheney Syndrome - physiopathology | Hajdu-Cheney Syndrome - drug therapy | Case-Control Studies | Alendronate - therapeutic use | Bone and Bones - drug effects | Hajdu-Cheney Syndrome - genetics | Bone Density - drug effects | Adolescent | Diphosphonates - therapeutic use | Female | Imidazoles - therapeutic use | Mutation | Osteoclasts - physiology | Child | Osteoclasts - drug effects | Therapy | Radius | Energy use | Spine | Electron imaging | Spine (lumbar) | Backscattering | Medical services | Males | Bone (trabecular) | Density | Bisphosphonates | Bone resorption | Alendronic acid | Bone histomorphometry | Biomedical materials | Bone growth | Computed tomography | Mineralization | Zoledronic acid | Biocompatibility | Bone density | Children | Pretreatment | Bone (cortical) | Bone matrix | Dual energy X-ray absorptiometry | Patients | Pamidronic acid | Osteoclastogenesis | Computation | Biopsy | Bone mineral density | Bone imaging | Notch2 protein | Osteogenesis
Journal Article
Cellular Physiology and Biochemistry, ISSN 1015-8987, 10/2018, Volume 49, Issue 4, pp. 1564 - 1576
Journal Article
Journal Article