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Free Radical Biology and Medicine, ISSN 0891-5849, 11/2015, Volume 88, Issue Pt B, pp. 417 - 426
In light of the emerging interplay between redox and metabolic signaling pathways we investigated the potential cross talk between nuclear factor E2-related... 
Xanthohumol | Nrf2 | ER stress | AMPK | HO-1 | PERK | LKB1 | MEF | PHOSPHORYLATION | BIOCHEMISTRY & MOLECULAR BIOLOGY | KINASE | MECHANISMS | ENERGY SENSOR | CELL DEFENSE PATHWAY | ENDOTHELIAL-CELLS | ENDOCRINOLOGY & METABOLISM | NRF2-MEDIATED INDUCTION | HEME OXYGENASE-1 | STRESS | AMP-Activated Protein Kinases - metabolism | Heme Oxygenase-1 - metabolism | Reactive Oxygen Species - metabolism | Oxidation-Reduction | Oxidative Stress - physiology | Propiophenones - pharmacology | Mitochondria - metabolism | Mitochondria - drug effects | Receptor Cross-Talk - physiology | Blotting, Western | Animals | Flow Cytometry | Fibroblasts - drug effects | NF-E2-Related Factor 2 - metabolism | Signal Transduction - physiology | Flavonoids - pharmacology | Membrane Proteins - metabolism | Mice | Real-Time Polymerase Chain Reaction | Unfolded Protein Response - physiology | Fibroblasts - metabolism | RNA | Glycogen | Synthesis | ACC, acetyl-CoA carboxylase | CHO, Chinese hamster ovary | GSH, glutathione (reduced) | OCR, oxygen consumption rate | HO-1, heme oxygenase 1 | CaMKK, calcium calmodulin-dependent kinase kinase | Maf, small musculoaponeurotic fibrosarcoma | PERK, protein kinase RNA-like endoplasmic reticulum kinase | Xn, xanthohumol | Hrd1, synoviolin | TAK, transforming growth factor β-activated kinase | UPR, unfolded protein response | Hrd1 (HMG-CoA reductase degradation)-ubiquitin ligase | MEF, mouse embryonic fibroblasts | βTrcP1, β-transducin-repeat containing protein 1 | mTOR, mammalian target of rapamycin | ROS, reactive oxygen species | LKB1, liver kinase B1 | Keap1, Kelch-like ECH-associated protein | GSSG, oxidized glutathione | ARE, antioxidant response element | WT, wild type | GSK3β, glycogen synthase kinase 3β | Nrf2, nuclear factor E2-related factor 2 | AMPK, AMP-activated kinase | DCF, dichlorofluorescein | NADPH, nicotinamide adenine dinucleotide phosphate | DMSO, dimethyl sulfoxide | ER, endoplasmic reticulum | FCS, fetal calf serum
Journal Article
by Seo, JY and Lim, SS and Kim, J and Lee, KW and Kim, JS
PHYTOTHERAPY RESEARCH, ISSN 0951-418X, 05/2017, Volume 31, Issue 5, pp. 801 - 811
Given the evidence for detoxifying/antioxidant enzyme-inducing activities by alantolactone (AL) and isoalantolactone (IAL), the purpose of this study was to... 
cognitive function | OXIDATIVE STRESS | CHEMISTRY, MEDICINAL | DETOXIFYING ENZYMES | ALZHEIMERS-DISEASE | MEMORY DEFICITS | ROS GENERATION | MODEL | Alzheimer | INULA-HELENIUM | alantolactone | Nrf2 signaling pathway | DEHYDROGLYASPERIN C | neuroprotection | PHARMACOLOGY & PHARMACY | isoalantolactone | NRF2-MEDIATED INDUCTION | NF-KAPPA-B
Journal Article
Molecular and Cellular Biochemistry, ISSN 0300-8177, 5/2015, Volume 403, Issue 1, pp. 287 - 299
Journal Article
PLoS ONE, ISSN 1932-6203, 04/2015, Volume 10, Issue 4, pp. e0124000 - e0124000
Inhibition of carcinogenesis may be a consequence of attenuation of oxidative stress via activation of antioxidant defence system, restoration and... 
NRF2-MEDIATED ANTIOXIDANT | GROWTH-FACTOR-BETA | NAD(P)H-QUINONE OXIDOREDUCTASE-1 | TGF-BETA | NITRIC-OXIDE SYNTHASE | MULTIDISCIPLINARY SCIENCES | IN-VIVO | SULFHYDRYL-GROUPS | GLUTATHIONE-S-TRANSFERASE | INDUCTION | EXPRESSION | Consensus Sequence | Glutathione Reductase - metabolism | Antioxidants - metabolism | Male | NAD(P)H Dehydrogenase (Quinone) - genetics | RNA, Messenger - metabolism | Lymphoma, T-Cell - prevention & control | Glutathione Reductase - genetics | Lymphoma, T-Cell - metabolism | Glutathione Transferase - genetics | Liver - drug effects | Cyclooxygenase 2 - genetics | Protein Binding - drug effects | Inflammation Mediators - metabolism | NF-E2-Related Factor 2 - genetics | Anticarcinogenic Agents - pharmacology | Tumor Microenvironment - drug effects | Liver - metabolism | RNA, Messenger - genetics | Curcumin - pharmacology | Tumor Suppressor Protein p53 - metabolism | Glutathione Transferase - metabolism | Transforming Growth Factor beta1 - genetics | Lymphoma, T-Cell - drug therapy | Animals | Nitric Oxide Synthase Type II - genetics | Signal Transduction - drug effects | NF-E2-Related Factor 2 - metabolism | Cyclooxygenase 2 - metabolism | NAD(P)H Dehydrogenase (Quinone) - metabolism | Mice | Mice, Inbred AKR | Nitric Oxide Synthase Type II - metabolism | Prevention | Antioxidants | Patient outcomes | Development and progression | Genetic aspects | Research | Non-Hodgkin's lymphomas | Transforming growth factors | Tumor proteins | Risk factors | Oxidative stress | Regulations | Laboratories | Zoology | Liver | p53 Protein | Smooth muscle | Biochemistry | Lymphocytes T | Activation | Metastasis | Carcinogenesis | Bearing | Proteins | Signal transduction | Angiogenesis | Carcinogens | Metabolites | Restoration | Rodents | Modulation | Cell cycle | Attenuation | Curcumin | Inhibition | Growth factors | Deoxyribonucleic acid--DNA | Enzymes | Inflammation | Gene expression | Lymphoma | Nitric-oxide synthase | Signaling | Phytochemicals | Hepatocytes | Nitric oxide | Glycolysis | Lymphomas | Cyclooxygenase-2 | Molecular biology | T-cell lymphoma | Cancer | Apoptosis | Tumors | Index Medicus | Deoxyribonucleic acid | DNA
Journal Article
Journal of Agricultural and Food Chemistry, ISSN 0021-8561, 04/2016, Volume 64, Issue 14, pp. 2855 - 2864
Journal Article
Polymers, ISSN 2073-4360, 12/2018, Volume 10, Issue 12, p. 1384
Osteoclastic bone resorption enables orthodontic tooth movement (OTM) in orthodontic treatment. Previously, we demonstrated that local epigallocatechin gallate... 
Epigallocatechin gallate | RANKL | Gelatin | Nrf2 | ROS | Osteoclast | Antioxidant enzyme | PERIODONTAL TISSUE | POLYMER SCIENCE | epigallocatechin gallate | OSTEOPROTEGERIN | INDUCTION | antioxidant enzyme | OSTEOCLASTOGENESIS | NRF2-MEDIATED ANTIOXIDANT | (-)-EPIGALLOCATECHIN GALLATE | GENE-TRANSFER | DIFFERENTIATION | osteoclast | gelatin
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