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Nature, ISSN 0028-0836, 10/2017, Volume 550, Issue 7674, pp. 67 - 73
Despite their fundamental biological and clinical importance, the molecular mechanisms that regulate the first cell fate decisions in the human embryo are not... 
ANALYZER | HUMAN EMBRYOS | INNER CELL MASS | FGF | MULTIDISCIPLINARY SCIENCES | GENES | HUMAN ZYGOTES | GENERATION | CAS9 | NORMALIZATION | POU DOMAIN | Human Embryonic Stem Cells - cytology | Humans | Zygote - metabolism | Substrate Specificity | Male | Embryo, Mammalian - metabolism | Octamer Transcription Factor-3 - genetics | Ectoderm - metabolism | Gene Expression Regulation, Developmental | Female | Nanog Homeobox Protein - genetics | Human Embryonic Stem Cells - metabolism | Germ Layers - metabolism | Blastocyst - metabolism | Embryonic Development - genetics | CRISPR-Cas Systems - genetics | Gene Editing | Cell Lineage | Animals | Embryo, Mammalian - embryology | Embryo, Mammalian - cytology | Octamer Transcription Factor-3 - metabolism | Mice | Octamer Transcription Factor-3 - deficiency | Nanog Homeobox Protein - metabolism | Embryonic development | Genetic aspects | Nucleotide sequencing | Methods | DNA sequencing | Genes | Oct-4 protein | Embryo cells | Genomes | CDX2 protein | Kinases | Trophectoderm | Proteins | Embryogenesis | Embryology | Cell fate | Null cells | Deoxyribonucleic acid--DNA | CRISPR | Gene expression | Ribonucleic acid--RNA | Embryos | Embryonic growth stage | Studies | Zygotes | Molecular modelling | Microinjection | Stem cells | Protein expression | Genetic engineering | Mutation | Pluripotency
Journal Article
Nature Cell Biology, ISSN 1465-7392, 07/2017, Volume 19, Issue 7, pp. 820 - 832
Understanding the roles of splicing factors and splicing events during tumorigenesis would open new avenues for targeted therapies. Here we identify an... 
NONCODING RNA | PAXILLIN | COLORECTAL-CANCER | GENE-EXPRESSION | SELF-RENEWAL | PROLIFERATION | LIVER DEVELOPMENT | PROGRESSION | TUMOR-INITIATING CELLS | CANCER STEM-CELLS | CELL BIOLOGY | Paxillin - metabolism | RNA-Binding Proteins - genetics | Up-Regulation | Paxillin - genetics | Cell Proliferation | Alternative Splicing | Exons | Humans | Gene Expression Regulation, Neoplastic | Male | MicroRNAs - metabolism | SOXB1 Transcription Factors - metabolism | Octamer Transcription Factor-3 - genetics | Transfection | RNA Interference | SOXB1 Transcription Factors - genetics | Time Factors | Carcinoma, Hepatocellular - genetics | 3' Untranslated Regions | Binding Sites | Nanog Homeobox Protein - genetics | Argonaute Proteins - metabolism | Argonaute Proteins - genetics | Liver Neoplasms - genetics | Mice, Inbred C57BL | Gene Expression Profiling - methods | RNA, Long Noncoding - genetics | Tumor Burden | Hep G2 Cells | Animals | Carrier Proteins - metabolism | Mice, Nude | Octamer Transcription Factor-3 - metabolism | Liver Neoplasms - metabolism | Protein Binding | High-Throughput Nucleotide Sequencing | Mice | MicroRNAs - genetics | RNA, Long Noncoding - metabolism | Nanog Homeobox Protein - metabolism | RNA-Binding Proteins - metabolism | Carcinoma, Hepatocellular - metabolism | Alternative splicing | Translation | Transcription | Splicing | MiRNA | Hepatocellular carcinoma | Gene expression | Argonaute 2 protein | Gene sequencing | Degradation | Liver cancer | 3' Untranslated regions | Ribonucleic acids | Translation elongation | Tumorigenesis | Splicing factors | Elongation
Journal Article
Journal Article
Journal Article
NATURE CELL BIOLOGY, ISSN 1465-7392, 05/2018, Volume 20, Issue 5, pp. 565 - 565
A robust network of transcription factors and an open chromatin landscape are hallmarks of the naive pluripotent state. Recently, the acetyllysine reader Brd4... 
GROUND-STATE PLURIPOTENCY | NAIVE PLURIPOTENCY | PROTEIN BRD4 | BET BROMODOMAIN INHIBITION | CANCER | EXPRESSION | MEDIATOR COMPLEX | GENOME | DNA DEMETHYLATION | P-TEFB | CELL BIOLOGY | Chromatin - metabolism | Male | Mice, 129 Strain | Mouse Embryonic Stem Cells - metabolism | DNA-Binding Proteins - metabolism | Cell Self Renewal | Gene Expression Regulation, Developmental | Kruppel-Like Transcription Factors - metabolism | Female | Cell Differentiation | Acetylation | Nuclear Proteins - genetics | Binding Sites | STAT3 Transcription Factor - genetics | STAT3 Transcription Factor - metabolism | Nanog Homeobox Protein - genetics | Proto-Oncogene Proteins - metabolism | Cell Line | Signal Transduction | Mice, Inbred C57BL | Chromatin Assembly and Disassembly | Nuclear Proteins - metabolism | Proto-Oncogene Proteins - genetics | Transcription Factors - genetics | DNA-Binding Proteins - genetics | Mice, Inbred ICR | Transcription Factors - metabolism | Cell Lineage | Phenotype | Animals | Histones - genetics | Protein Binding | Mice | Protein Processing, Post-Translational | Histones - metabolism | Chromatin - genetics | Kruppel-Like Transcription Factors - genetics | Nanog Homeobox Protein - metabolism | Transcription factors | Cellular signal transduction | Research | Stem cells | Chromatin | KLF4 protein | Embryo cells | Stat3 protein | Gene expression | Pluripotency | Deoxyribonucleic acid--DNA | Acetyllysine
Journal Article
Journal Article
Molecular Cell, ISSN 1097-2765, 11/2016, Volume 64, Issue 3, pp. 624 - 635
Journal Article
Journal Article
Journal Article
Brain, ISSN 0006-8950, 04/2018, Volume 141, Issue 4, pp. 1000 - 1016
See Lerche (doi:10.1093/brain/awy073) for a scientific commentary on this article. PRRT2 mutations cause heterogeneous paroxysmal neurological disorders. Using... 
Voltage-dependent sodium channels | Paroxysmal disorders | Proline-rich transmembrane protein 2 | Induced pluripotent stem cells | Neuronal excitability | HEMIPLEGIC MIGRAINE | SYNAPTIC-TRANSMISSION | DISORDERS | proline-rich transmembrane protein 2 | CORTICAL NETWORKS | NEUROSCIENCES | CLINICAL NEUROLOGY | paroxysmal disorders | NEONATAL-INFANTILE SEIZURES | GENE | AXON INITIAL SEGMENT | MUTATION | voltage-dependent sodium channels | induced pluripotent stem cells | MICE | PAROXYSMAL KINESIGENIC DYSKINESIA | neuronal excitability | Membrane Potentials - genetics | Humans | Neurons - cytology | Nervous System Diseases - genetics | Cerebral Cortex - cytology | NAV1.2 Voltage-Gated Sodium Channel - metabolism | PAX6 Transcription Factor - genetics | SOXB1 Transcription Factors - metabolism | SOXB1 Transcription Factors - genetics | HEK293 Cells | Neurons - physiology | Cell Differentiation | Membrane Proteins - metabolism | Nervous System Diseases - pathology | Siblings | Nanog Homeobox Protein - genetics | PAX6 Transcription Factor - metabolism | Axon Initial Segment - physiology | Gene Expression Regulation - genetics | Membrane Proteins - genetics | Mice, Inbred C57BL | Fibroblasts - pathology | Mutation - genetics | Nerve Tissue Proteins - genetics | Mice, Knockout | Nerve Tissue Proteins - metabolism | Animals | NAV1.6 Voltage-Gated Sodium Channel - metabolism | Consanguinity | Mice | NAV1.6 Voltage-Gated Sodium Channel - genetics | Induced Pluripotent Stem Cells | Nanog Homeobox Protein - metabolism | Original
Journal Article
Journal Article