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Plant Physiology and Biochemistry, ISSN 0981-9428, 01/2017, Volume 110, pp. 167 - 177
Understanding the adverse impact of nanoparticles in crop plants has emerged as one of the most interesting fields of plant research. Therefore, this study has... 
Pea seedlings | Oxidative stress | Anatomical structures | Nitric oxide | Silver nanoparticles | ARABIDOPSIS-THALIANA | RICE SEEDLINGS | CHLOROPLASTS | TOXICITY | CROP PLANTS | PLANT SCIENCES | RESPONSES | ANTIOXIDANT SYSTEMS | PROTECTIVE ROLE | GROWTH | Nitroprusside - metabolism | Reactive Oxygen Species - metabolism | Glutathione - metabolism | Glutathione Reductase - metabolism | Fluorescence | Nitroprusside - pharmacology | Ascorbic Acid - metabolism | Plant Roots - drug effects | Ascorbate Peroxidases - metabolism | X-Ray Diffraction | Plant Shoots - drug effects | Silver - toxicity | Plant Leaves - drug effects | Plant Proteins - metabolism | Nitric Oxide Donors - pharmacology | Peas - metabolism | Superoxide Dismutase - metabolism | Metal Nanoparticles - ultrastructure | Malondialdehyde - metabolism | Microscopy, Electron, Transmission | Plant Roots - metabolism | Metal Nanoparticles - chemistry | Nitric Oxide Donors - metabolism | Silver - chemistry | Peas - drug effects | Chlorophyll - metabolism | Seedlings - drug effects | Hydrogen Peroxide - metabolism | Chlorophyll - chemistry | Metal Nanoparticles - toxicity | Plant Leaves - metabolism | Photosynthesis - drug effects | Plant Shoots - metabolism | Oxidative Stress - drug effects | Nitric Oxide - metabolism | Seedlings - metabolism | Nanoparticles | Chlorophyll | Silver | Peas | Phytochemistry | Superoxide | Photosynthesis | Nanotechnology | Index Medicus
Journal Article
Cell and Tissue Research, ISSN 0302-766X, 6/2004, Volume 316, Issue 3, pp. 315 - 323
The purpose of this study was to determine the cellular distribution and degradation in rat liver following intravenous injection of superparamagnetic iron... 
Medicine | Contrast agents | Iron oxide particles | Magnetic resonance imaging | Rat (Wister) | Liver | Metabolism | magnetic resonance imaging | LIVER ENDOTHELIAL-CELLS | liver | TRANSVERSE RELAXATION | iron oxide particles | contrast agents | ENDOCYTOSIS | CELL BIOLOGY | PHARMACOKINETICS | ENHANCEMENT | rat (wister) | KUPFFER CELLS | metabolism | PARTICLE-SIZE | FERRITIN | MR CONTRAST AGENTS | Injections, Intravenous | Rats, Wistar | Contrast Media - pharmacokinetics | Magnetic Resonance Imaging - methods | Male | Hepatocytes - metabolism | Ferrosoferric Oxide | Dextrans | Ferritins - metabolism | Liver - drug effects | Time Factors | Iron - pharmacokinetics | Kupffer Cells - metabolism | Hepatocytes - drug effects | Magnetite Nanoparticles | Oxides - metabolism | Hemosiderin - metabolism | Endothelial Cells - metabolism | Liver - metabolism | Rats | Iron - metabolism | Metabolic Clearance Rate - physiology | Kupffer Cells - drug effects | Oxides - pharmacokinetics | Cell Compartmentation - physiology | Animals | Cell Compartmentation - drug effects | Contrast Media - metabolism | Liver - cytology | Endothelial Cells - drug effects | Index Medicus | Cell Compartmentation/drug effects/physiology | Medical and Health Sciences | Endothelial Cells/drug effects/metabolism | Rats; Wistar | Medicin och hälsovetenskap | Injections; Intravenous | Magnetic Resonance Imaging/methods | Iron/metabolism/pharmacokinetics | Hemosiderin/metabolism | Liver/cytology/drug effects/metabolism | Oxides/metabolism/pharmacokinetics | Contrast Media/metabolism/pharmacokinetics | Ferritin/metabolism | Kupffer Cells/drug effects/metabolism | Metabolic Clearance Rate/physiology | Hepatocytes/drug effects/metabolism
Journal Article
Nature Cell Biology, ISSN 1465-7392, 11/2017, Volume 19, Issue 11, pp. 1358 - 1370
Metabolic reprogramming is a hallmark of cancer. Herein we discover that the key glycolytic enzyme pyruvate kinase M2 isoform (PKM2), but not the related... 
BREAST-CANCER | OXIDATIVE-PHOSPHORYLATION | PROTEIN | TUMOR PROGRESSION | PYRUVATE-KINASE M2 | CA2+ TRANSFER | MITOCHONDRIA | METABOLIC REQUIREMENTS | CELL-PROLIFERATION | ISOFORM | CELL BIOLOGY | Calcium - metabolism | Humans | Endoplasmic Reticulum - metabolism | Carcinogenesis - metabolism | Cell Movement - physiology | Glycolysis - physiology | CARD Signaling Adaptor Proteins - metabolism | MCF-7 Cells | Inositol 1,4,5-Trisphosphate Receptors - metabolism | HEK293 Cells | Female | Membrane Proteins - metabolism | Cell Line | Cell Proliferation - physiology | Oxidative Phosphorylation | Guanylate Cyclase - metabolism | Mitochondria - metabolism | Carcinogenesis - pathology | Animals | Carrier Proteins - metabolism | Mice, Nude | Neoplasm Metastasis - pathology | Thyroid Hormones - metabolism | Cell Line, Tumor | Mice | Mice, Inbred BALB C | Methylation | Cell proliferation | Energy metabolism | Phosphorylation | Calcium | Leukocyte migration | Oxidative metabolism | Pyruvic acid | Kinases | Calcium influx | Metastases | Nanoparticles | Mitochondria | Receptors | Arginine | Tumorigenesis | Inhibition | Inositol 1,4,5-trisphosphate | Inositol 1,4,5-trisphosphate receptors | Cell survival | Pyruvate kinase | Protein arginine methyltransferase | Breast cancer | Metabolism | Calcium (mitochondrial) | Energy balance | Oxidative phosphorylation | Glycolysis | Endoplasmic reticulum | Cell migration | Calcium (reticular) | Cancer | Index Medicus
Journal Article
Particle and Fibre Toxicology, ISSN 1743-8977, 08/2013, Volume 10, Issue 1, pp. 35 - 35
Background: Nanotechnology, particularly the use of multi-walled carbon nanotubes (MWCNT), is a rapidly growing discipline with implications for advancement in... 
Pulmonary inflammation | MWCNT | Pulmonary fibrosis | Airway epithelium | Co-culture | Endothelium | LUNG | PROTEIN | PARTICLES | TOXICITY | IN-VITRO MODELS | NANOPARTICLES | REACTIVE OXYGEN | INFLAMMATION | TOXICOLOGY | NF-KAPPA-B | EXPRESSION | Phosphorylation | Reactive Oxygen Species - metabolism | Respiratory Mucosa - drug effects | Cadherins - metabolism | Capillaries - pathology | Coculture Techniques | Humans | Actins - metabolism | Neovascularization, Pathologic | Capillary Permeability - drug effects | Capillaries - drug effects | NF-kappa B - metabolism | Vascular Endothelial Growth Factor A - metabolism | Antigens, CD - metabolism | Respiratory Mucosa - pathology | Blood-Air Barrier - drug effects | Nanotubes, Carbon - toxicity | Alveolar Epithelial Cells - pathology | Time Factors | Blood-Air Barrier - pathology | Inflammation Mediators - metabolism | p38 Mitogen-Activated Protein Kinases - metabolism | Capillaries - metabolism | STAT3 Transcription Factor - metabolism | Endothelial Cells - metabolism | Alveolar Epithelial Cells - metabolism | Alveolar Epithelial Cells - drug effects | Intercellular Adhesion Molecule-1 - metabolism | Blood-Air Barrier - metabolism | Respiratory Mucosa - metabolism | Endothelial Cells - pathology | Vascular Cell Adhesion Molecule-1 - metabolism | Endothelial Cells - drug effects | Muscle proteins | Epithelial cells | Analysis | Nanotubes | Nanoparticles | Angiogenesis | Occupational safety | Transmission electron microscopy | Lungs | Rodents | Nanomaterials | Carbon | Cell adhesion & migration | Index Medicus
Journal Article
Journal of Molecular and Cellular Cardiology, ISSN 0022-2828, 2016, Volume 102, pp. 10 - 21
Abstract We recently reported that increased NADPH oxidase 4 (NOX4) expression and activity during aging results in enhanced cellular and mitochondrial... 
Cardiovascular | Nanoparticles | Reactive oxygen species | Inflammation | Cytokines | Atherosclerosis | MITOCHONDRIAL OXIDATIVE STRESS | CARDIAC & CARDIOVASCULAR SYSTEMS | MELITTIN-DERIVED PEPTIDES | TGF-BETA | MONOCYTE CHEMOATTRACTANT PROTEIN-1 | CELL BIOLOGY | GROWTH-FACTOR-BETA | SIRNA TRANSFECTION | SMOOTH-MUSCLE-CELLS | NF-KAPPA-B | TRANSCRIPTIONAL REGULATION | CORONARY-ARTERY-DISEASE | Carotid Arteries - metabolism | RNA, Small Interfering - genetics | Vasculitis - metabolism | Reactive Oxygen Species - metabolism | Muscle, Smooth, Vascular - metabolism | Atherosclerosis - genetics | Humans | Vasculitis - etiology | Vasculitis - pathology | NADPH Oxidases - metabolism | Cell Survival - genetics | Male | NF-kappa B - metabolism | Gene Knockdown Techniques | Aging - genetics | RNA Interference | Inflammation Mediators - metabolism | NADPH Oxidases - genetics | Cytokines - genetics | Myocytes, Smooth Muscle - metabolism | Disease Models, Animal | Atherosclerosis - pathology | Cytokines - metabolism | MAP Kinase Kinase Kinases - metabolism | Mitochondria - metabolism | NADPH Oxidase 4 | Atherosclerosis - metabolism | Hydrogen Peroxide - metabolism | Mice, Knockout | Animals | Biomarkers | Carotid Arteries - pathology | Mice | Aging - metabolism | Oxidases | RNA | Genes | Transforming growth factors | Gene expression | Medical colleges | Anopheles | Peptides | Peroxides | Index Medicus
Journal Article
Nature Communications, ISSN 2041-1723, 12/2017, Volume 8, Issue 1, pp. 2270 - 2270
Drugs that mirror the cellular effects of starvation mimics are considered promising therapeutics for common metabolic disorders, such as obesity, liver... 
LOOP-HELIX PROTEIN | REGULATOR | ACTIVATED PROTEIN-KINASE | LIPID-METABOLISM | MULTIDISCIPLINARY SCIENCES | CALCIUM | CALCINEURIN | RECEPTOR | AUTOPHAGY | ENERGY-METABOLISM | LYSOSOMAL BIOGENESIS | Biguanides - pharmacology | Calcium - metabolism | Caloric Restriction | Longevity - drug effects | Humans | Metabolic Syndrome - metabolism | Autophagy - drug effects | Basic Helix-Loop-Helix Leucine Zipper Transcription Factors - agonists | Autophagosomes - drug effects | Lysosomes - metabolism | Liver - drug effects | Diet, High-Fat | Lactams - pharmacology | Lysosomes - drug effects | Starvation | Caenorhabditis elegans - metabolism | Fatty Liver - metabolism | Liver - metabolism | Enzyme Inhibitors - pharmacology | Mitochondria - metabolism | Autophagosomes - metabolism | Mitochondria - drug effects | Digoxin - pharmacology | Animals | Caenorhabditis elegans - drug effects | High-Throughput Screening Assays | Lipid Metabolism - drug effects | Mice | HeLa Cells | Drugs | Animal models | Digoxin | Calcium | Biodegradability | Liver | Disorders | Autophagy | Nanoparticles | Dietary restrictions | Mitochondria | Fatty liver | Biocompatibility | Lipid metabolism | Biodegradation | Metabolic diseases | Lead compounds | Metabolism | Steatosis | Mode of action | Life span | Nematodes | Aging (natural) | Metabolic disorders | Nanotechnology | Index Medicus
Journal Article
Fertility and Sterility, ISSN 0015-0282, 2015, Volume 104, Issue 6, pp. 1552 - 1560.e3
Objective To investigate the engraftment and proliferation of superparamagnetic iron oxide nanoparticles (SPIOs)-labeled human CD133+ bone marrow-derived stem... 
Internal Medicine | Obstetrics and Gynecology | Asherman syndrome | bone marrow-derived stem cells (BMDSCs) | CD133+cells | superparamagnetic iron oxide nanoparticles (SPIOs) | cells | CD133 | POPULATION | MOBILIZATION | REGENERATION | PHENOTYPE | MOUSE ENDOMETRIUM | CD133(+) cells | IDENTIFICATION | RAT MODEL | OBSTETRICS & GYNECOLOGY | REPRODUCTIVE BIOLOGY | THIN ENDOMETRIUM | DISEASE | DIFFERENTIATION | Cell Proliferation | Prospective Studies | Humans | Middle Aged | Cell Tracking - methods | Glycoproteins - metabolism | Ki-67 Antigen - metabolism | Insulin-Like Growth Factor I - genetics | Gynatresia - physiopathology | Stem Cells - metabolism | Gynatresia - genetics | Antigens, CD - metabolism | Atrophy | Stem Cell Transplantation | Peptides - metabolism | Thrombospondin 1 - genetics | Bone Marrow Transplantation | Adult | Female | Disease Models, Animal | Endometrium - metabolism | Biomarkers - metabolism | Gynatresia - surgery | Paracrine Communication | Cell Survival | Glycosaminoglycans - metabolism | Gene Expression Regulation | Graft Survival | Clinical Trials as Topic | Mice, SCID | AC133 Antigen | Collagen - metabolism | Animals | Gynatresia - metabolism | Endometrium - physiopathology | Mice, Inbred NOD | Mice | Thrombospondin 1 - metabolism | Endometrium - pathology | Gynatresia - pathology | Bone Marrow Cells - metabolism | Insulin-Like Growth Factor I - metabolism | Index Medicus
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 12/2014, Volume 111, Issue 48, pp. 17104 - 17109
Metastasis portends a poor prognosis for cancer patients. Primary tumor cells disseminate through the bloodstream before the appearance of detectable... 
Vimentin | Epithelial cells | Blood cells | Cell lines | Stem cells | Fluorescence | Breast cancer | Blood | Tumors | Cancer | Diagnostic | Cancer metastasis | MRNA | Nanotechnology | NanoFlares | nanotechnology | mRNA | diagnostic | cancer metastasis | SURVIVAL | MECHANISM | MULTIDISCIPLINARY SCIENCES | PERIPHERAL-BLOOD | EPITHELIAL-MESENCHYMAL TRANSITION | REGULATOR | PREDICT | METASTATIC BREAST-CANCER | PROGRESSION | MESSENGER-RNA DETECTION | Carbocyanines - chemistry | Cadherins - metabolism | Vimentin - metabolism | Humans | Neoplastic Cells, Circulating - metabolism | Carbocyanines - metabolism | Transplantation, Heterologous | Green Fluorescent Proteins - genetics | RNA, Messenger - metabolism | DNA, Antisense - genetics | Breast Neoplasms - metabolism | Vimentin - genetics | Base Sequence | Biomarkers, Tumor - metabolism | Female | Cadherins - genetics | Gold - chemistry | Green Fluorescent Proteins - metabolism | Nanotechnology - methods | Metal Nanoparticles - chemistry | RNA, Messenger - genetics | Neoplastic Cells, Circulating - chemistry | DNA, Antisense - metabolism | Fibronectins - metabolism | Breast Neoplasms - genetics | Breast Neoplasms - blood | Cell Line, Tumor | Luminescent Proteins - genetics | Biomarkers, Tumor - genetics | Fibronectins - genetics | DNA, Antisense - chemistry | Microscopy, Fluorescence | Luminescent Proteins - metabolism | Health aspects | Identification and classification | Cancer cells | Flow cytometry | Metastasis | Ribonucleic acid--RNA | Risk assessment | Index Medicus | Biological Sciences
Journal Article
Plant Physiology and Biochemistry, ISSN 0981-9428, 01/2017, Volume 110, pp. 59 - 69
The present study describes the role of zinc oxide nanoparticles (ZnONPs) in reversing oxidative stress symptoms induced by heavy metal (Cd and Pb) exposure in... 
Lead | Cadmium | Genetic variability | Phytotoxicity | Antioxidative enzymes | Photosynthetic pigments | CATALASE | PLANT | TOLERANCE | GRAPHENE | CHROMIUM VI PHYTOTOXICITY | PLANT SCIENCES | PEROXIDASE | GROWTH | STRESS | MACROALGAE | Metals, Heavy - metabolism | Antioxidants - chemistry | Cadmium - metabolism | Seedlings - growth & development | Zinc Oxide - chemistry | Plant Leaves - drug effects | Plant Proteins - metabolism | Lead - toxicity | DNA, Plant - genetics | Superoxide Dismutase - metabolism | Malondialdehyde - metabolism | Cadmium - toxicity | Lead - metabolism | Metal Nanoparticles - chemistry | Fabaceae - growth & development | Antioxidants - pharmacology | Chlorophyll - metabolism | Seedlings - drug effects | DNA, Plant - metabolism | Catalase - metabolism | Metal Nanoparticles - administration & dosage | Metals, Heavy - toxicity | Plant Leaves - metabolism | Photosynthesis - drug effects | Plant Leaves - growth & development | Antioxidants - administration & dosage | Carotenoids - metabolism | Fabaceae - metabolism | Random Amplified Polymorphic DNA Technique | Fabaceae - drug effects | Oxidative Stress - drug effects | Peroxidase - metabolism | Seedlings - metabolism | Nanoparticles | Enzymes | Plant physiology | Analysis | Genetically modified organisms | Green technology | Zinc oxide | Genetic engineering | Superoxide | Molecular biology | Photosynthesis | Index Medicus
Journal Article
Molecular Therapy, ISSN 1525-0016, 07/2017, Volume 25, Issue 7, pp. 1718 - 1729
Inhibition of Notch signaling via systemic drug administration triggers conversion of white adipocytes into beige adipocytes (browning) and reduces adiposity.... 
PLGA | Notch signaling | adipocyte | browning | adipose tissue | nanoparticle | Notch inhibitor | drug delivery | obesity | dibenzazepine | MEDICINE, RESEARCH & EXPERIMENTAL | STEM-CELLS | ACTIVATION | ADIPOCYTES | NOTCH | HUMANS | GLUCOSE-HOMEOSTASIS | INHIBITION | FAT-CELLS | BIOTECHNOLOGY & APPLIED MICROBIOLOGY | ADAPTIVE THERMOGENESIS | GENETICS & HEREDITY | BONE | Peroxisome Proliferator-Activated Receptor Gamma Coactivator 1-alpha - agonists | Peroxisome Proliferator-Activated Receptor Gamma Coactivator 1-alpha - genetics | Nanoparticles - chemistry | Obesity - drug therapy | Adipose Tissue, White - metabolism | Male | Obesity - genetics | Drug Carriers | Cell Cycle Proteins - antagonists & inhibitors | Dibenzazepines - pharmacology | Basic Helix-Loop-Helix Transcription Factors - metabolism | Nanoparticles - metabolism | Iodide Peroxidase - metabolism | Apoptosis Regulatory Proteins - genetics | Cell Cycle Proteins - genetics | Peroxisome Proliferator-Activated Receptor Gamma Coactivator 1-alpha - metabolism | Polyglycolic Acid - metabolism | Energy Metabolism - genetics | Adipose Tissue, White - pathology | Basic Helix-Loop-Helix Transcription Factors - genetics | Lactic Acid - chemistry | Signal Transduction | Lactic Acid - metabolism | Mice, Inbred C57BL | Transcription Factor HES-1 - metabolism | Cell Cycle Proteins - metabolism | Gene Expression Regulation | Basic Helix-Loop-Helix Transcription Factors - antagonists & inhibitors | Adipose Tissue, Brown - pathology | Apoptosis Regulatory Proteins - metabolism | Obesity - metabolism | Obesity - pathology | Anti-Obesity Agents - chemistry | Animals | Transcription Factor HES-1 - antagonists & inhibitors | Transcription Factor HES-1 - genetics | Polyglycolic Acid - chemistry | Apoptosis Regulatory Proteins - agonists | Dibenzazepines - chemistry | Mice, Obese | Adipose Tissue, Brown - drug effects | Adipose Tissue, Brown - metabolism | Mice | Iodide Peroxidase - genetics | Anti-Obesity Agents - pharmacology | Adipose Tissue, White - drug effects | Energy Metabolism - drug effects | Obesity | Immunoglobulins | Adipose tissue | Homeostasis | Adipocytes | Gene expression | Metabolism | Nanoparticles | Proteins | Microscopy | Internalization | Poly(lactide-co-glycolide) | Biotechnology industry | Metabolic disorders | Index Medicus | Original
Journal Article