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1. Full Text Naftopidil for the treatment of lower urinary tract symptoms compatible with benign prostatic hyperplasia
by Hwang, Eu Chang and Gandhi, Shreyas and Jung, Jae Hung and Imamura, Mari and Kim, Myung Ha and Pang, Ran and Dahm, Philipp
The Cochrane database of systematic reviews, ISSN 1469-493X, 10/2018, Volume 10, Issue 10, pp. CD007360 - CD007360
Background Benign prostatic hyperplasia (BPH) is a common condition in ageing men that may cause lower urinary tract symptoms (LUTS). Treatment aims are to... 
Naphthalenes [adverse effects; therapeutic use] | 75 MG | EFFICACY | Prostatic Hyperplasia [complications] | STORAGE SYMPTOM | SEXUAL FUNCTION | CLINICAL PROGRESSION | Randomized Controlled Trials as Topic | MEDICINE, GENERAL & INTERNAL | OVERACTIVE BLADDER | Sulfonamides [adverse effects; therapeutic use] | Piperazines [adverse effects; therapeutic use] | ALPHA-ADRENOCEPTOR ANTAGONISTS | Adrenergic alpha-Antagonists [adverse effects; therapeutic use] | Prostatism [drug therapy; etiology] | TAMSULOSIN HYDROCHLORIDE | ALPHA-1 BLOCKERS | HEALTH-STATUS | Benzilates - adverse effects | Naphthalenes - adverse effects | Benzilates - therapeutic use | Humans | Adrenergic alpha-Antagonists - therapeutic use | Male | Prostatic Hyperplasia - complications | Prostatism - drug therapy | Urological Agents - adverse effects | Adrenergic alpha-Antagonists - adverse effects | Ethamsylate - therapeutic use | Lower Urinary Tract Symptoms - drug therapy | Lower Urinary Tract Symptoms - etiology | Ethamsylate - adverse effects | Urological Agents - therapeutic use | Prostatism - etiology | Tamsulosin - adverse effects | Tamsulosin - therapeutic use | Piperazines - therapeutic use | Piperazines - adverse effects | Indoles - adverse effects | Sulfonamides - therapeutic use | Quality of Life | Sulfonamides - adverse effects | Indoles - therapeutic use | Plant Extracts - therapeutic use | Drug Combinations | Naphthalenes - therapeutic use | Plant Extracts - adverse effects | Index Medicus
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2. Full Text A case of X-linked hypophosphatemic rickets: Complications and the therapeutic use of cinacalcet
by Ræder, Helge and Shaw, Nick and Netelenbos, Coen and Bjerknes, Robert
European Journal of Endocrinology, ISSN 0804-4643, 2008, Volume 159, Issue 1, pp. S101 - S105
In hypophosphatemic rickets, there are both inherited and acquired forms, where X-linked dominant hypophosphatemic rickets (XI,H) is the most prevalent genetic... 
SECONDARY HYPERPARATHYROIDISM | NEPHROCALCINOSIS | CALCIMIMETICS | ENDOCRINOLOGY & METABOLISM | CHILDREN | Phosphates - administration & dosage | Familial Hypophosphatemic Rickets - drug therapy | Humans | Infant | Phosphates - therapeutic use | Mutation, Missense | Hyperparathyroidism - chemically induced | Familial Hypophosphatemic Rickets - genetics | Renal Insufficiency - chemically induced | Hypertension - chemically induced | PHEX Phosphate Regulating Neutral Endopeptidase - genetics | Hyperparathyroidism - drug therapy | Female | Hypercalcemia - drug therapy | Genetic Diseases, X-Linked | Bone Density Conservation Agents - adverse effects | Administration, Oral | Bone Density Conservation Agents - therapeutic use | Hypercalcemia - chemically induced | Nephrocalcinosis - chemically induced | Hydroxycholecalciferols - adverse effects | Hydroxycholecalciferols - therapeutic use | Cinacalcet Hydrochloride | Phosphates - adverse effects | Naphthalenes - therapeutic use | Index Medicus
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3. Full Text Recent changes in therapeutic approaches and association with outcomes among patients with secondary hyperparathyroidism on chronic hemodialysis: The DOPPS study
by Tentori, Francesca and Wang, Mia and Bieber, Brian A and Karaboyas, Angelo and Li, Yun and Jacobson, Stefan H and Andreucci, Vittorio E and Fukagawa, Masafumi and Frimat, Luc and Mendelssohn, David C and Port, Friedrich K and Pisoni, Ronald L and Robinson, Bruce M
Clinical Journal of the American Society of Nephrology, ISSN 1555-9041, 2015, Volume 10, Issue 1, pp. 98 - 109
Background and objectivesElevated parathyroid hormone levels may be associated with adverse clinical outcomes in patients on dialysis. After the introduction... 
ESRD | dialysis | parathyroid hormone | PERITONEAL-DIALYSIS | hyperparathyroidism | DIALYSIS PATIENTS | CLINICAL-PRACTICE GUIDELINE | CARDIOVASCULAR-DISEASE | MAINTENANCE HEMODIALYSIS | UROLOGY & NEPHROLOGY | PARATHYROID-HORMONE | MINERAL METABOLISM | RENAL REPLACEMENT | CHRONIC KIDNEY-DISEASE | CKD | PRACTICE PATTERNS | Hyperparathyroidism, Secondary - therapy | Up-Regulation | Naphthalenes - adverse effects | Prospective Studies | Parathyroidectomy - adverse effects | Humans | Middle Aged | Male | Parathyroid Hormone - blood | Renal Insufficiency, Chronic - complications | Renal Insufficiency, Chronic - therapy | Renal Dialysis - mortality | Time Factors | Renal Insufficiency, Chronic - mortality | Adult | Female | Parathyroidectomy - mortality | Renal Insufficiency, Chronic - diagnosis | Dietary Supplements - adverse effects | Parathyroidectomy - trends | Risk Assessment | Risk Factors | Treatment Outcome | Biomarkers - blood | Calcimimetic Agents - adverse effects | Calcimimetic Agents - therapeutic use | Renal Insufficiency, Chronic - blood | Vitamin D - adverse effects | Hyperparathyroidism, Secondary - blood | Cinacalcet Hydrochloride | Hyperparathyroidism, Secondary - diagnosis | Hyperparathyroidism, Secondary - mortality | Renal Dialysis - adverse effects | Aged | Vitamin D - therapeutic use | Hyperparathyroidism, Secondary - etiology | Naphthalenes - therapeutic use | Index Medicus | Original
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4. Full Text Actions of the dual FAAH/MAGL inhibitor JZL195 in a murine neuropathic pain model
by Adamson Barnes, Nicholas S and Mitchell, Vanessa A and Kazantzis, Nicholas P and Vaughan, Christopher W
British Journal of Pharmacology, ISSN 0007-1188, 01/2016, Volume 173, Issue 1, pp. 77 - 87
Background and PurposeWhile cannabinoids have been proposed as a potential treatment for neuropathic pain, they have limitations. Cannabinoid receptor agonists... 
FAAH | CONCISE GUIDE | ACID AMIDE HYDROLASE | MONOACYLGLYCEROL LIPASE | PHARMACOLOGY | PHARMACOLOGY & PHARMACY | INFLAMMATORY PAIN | BLOCKADE | RECEPTORS | OMEGA-CONOTOXINS | RAT MODEL | Naphthalenes - adverse effects | Monoacylglycerol Lipases - antagonists & inhibitors | Male | Dose-Response Relationship, Drug | Benzamides - therapeutic use | Piperidines - pharmacology | Benzamides - pharmacology | Benzodioxoles - therapeutic use | Morpholines - adverse effects | Morpholines - therapeutic use | Carbamates - pharmacology | Disease Models, Animal | Morpholines - pharmacology | Amidohydrolases - antagonists & inhibitors | Benzoxazines - adverse effects | Piperazines - therapeutic use | Piperazines - adverse effects | Piperazines - pharmacology | Neuralgia - drug therapy | Benzoxazines - therapeutic use | Naphthalenes - pharmacology | Animals | Hyperalgesia - drug therapy | Piperidines - therapeutic use | Benzoxazines - pharmacology | Carbamates - adverse effects | Mice | Carbamates - therapeutic use | Benzodioxoles - pharmacology | Naphthalenes - therapeutic use | Care and treatment | Pain | Lipase | Fatty acids | Analysis | Pain management | Index Medicus | Research Papers | Research Paper
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5. Full Text Prevention of gut leakiness by a probiotic treatment leads to attenuated HPA response to an acute psychological stress in rats
by Ait-Belgnaoui, Afifa and Durand, Henri and Cartier, Christel and Chaumaz, Gilles and Eutamene, Hélène and Ferrier, Laurent and Houdeau, Eric and Fioramonti, Jean and Bueno, Lionel and Theodorou, Vassilia
Psychoneuroendocrinology, ISSN 0306-4530, 2012, Volume 37, Issue 11, pp. 1885 - 1895
Summary Background and aims Intestinal barrier impairment is incriminated in the pathophysiology of intestinal gut disorders associated with psychiatric... 
Endocrinology & Metabolism | Psychiatry | Lipopolysaccharide | HPA axis | Brain-gut interaction | Intestinal permeability | LIGHT-CHAIN KINASE | INDUCED VISCERAL HYPERSENSITIVITY | PSYCHIATRY | COLONIC RESPONSES | FECAL MICROBIOTA | BARRIER FUNCTION | ANXIETY-LIKE BEHAVIOR | NEUROSCIENCES | ENDOCRINOLOGY & METABOLISM | LACTOBACILLUS-FARCIMINIS TREATMENT | IRRITABLE-BOWEL-SYNDROME | VAGAL-AFFERENTS | Rats, Wistar | Colon - drug effects | Myosin-Light-Chain Kinase - antagonists & inhibitors | Corticotropin-Releasing Hormone - analysis | Inflammation - blood | Anti-Bacterial Agents - therapeutic use | Lipopolysaccharides - blood | Inflammation - metabolism | Inflammation - drug therapy | Stress, Psychological - metabolism | Female | Corticosterone - blood | Hypothalamo-Hypophyseal System - physiology | Adrenocorticotropic Hormone - blood | Pituitary-Adrenal System - physiology | Lactobacillus | Hypothalamo-Hypophyseal System - drug effects | Acute Disease | Permeability - drug effects | Restraint, Physical - psychology | Pituitary-Adrenal System - drug effects | Stress, Psychological - drug therapy | Rats | Colon - metabolism | Azepines - therapeutic use | Animals | Probiotics - therapeutic use | Cytokines - biosynthesis | Naphthalenes - therapeutic use | Prevention | Corticotropin releasing hormone | Gastrointestinal agents | Myosin | ACTH | Permeability | Stress (Psychology) | Muscle proteins | Index Medicus
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6. Full Text Nitric oxide releasing hydrogel enhances the therapeutic efficacy of mesenchymal stem cells for myocardial infarction
by Yao, Xinpeng and Liu, Yi and Gao, Jie and Yang, Liang and Mao, Duo and Stefanitsch, Christina and Li, Yang and Zhang, Jun and Ou, Lailiang and Kong, Deling and Zhao, Qiang and Li, Zongjin
Biomaterials, ISSN 0142-9612, 2015, Volume 60, pp. 130 - 140
Abstract Stem cell therapy has been proved to be an effective approach to ameliorate the heart remodeling post myocardial infarction (MI). However, poor cell... 
Advanced Basic Science | Dentistry | Myocardial infarction | Molecular imaging | Adipose-derived mesenchymal stem cells (AD-MSCs) | Angiogenesis | Hydrogel | Nitric oxide | SURVIVAL | MATERIALS SCIENCE, BIOMATERIALS | ENGINEERING, BIOMEDICAL | REGENERATION | CARDIOMYOCYTES | CARDIAC REPAIR | DELIVERY | TRANSPLANTATION | S-NITROSYLATION | ENGRAFTMENT | CHITOSAN HYDROGEL | GROWTH-FACTOR | NIH 3T3 Cells | Optical Imaging | Galactose - metabolism | Hydrogel, Polyethylene Glycol Dimethacrylate - chemistry | Adipose Tissue - cytology | Delayed-Action Preparations - chemistry | Myocardial Infarction - therapy | Mesenchymal Stromal Cells - cytology | Ventricular Remodeling | Myocardial Infarction - pathology | beta-Galactosidase - metabolism | Nitric Oxide Donors - therapeutic use | Naphthalenes - metabolism | Oligopeptides - chemistry | Hydrogel, Polyethylene Glycol Dimethacrylate - metabolism | Naphthalenes - chemistry | Mice, Inbred C57BL | Cells, Cultured | Myocardium - pathology | Oligopeptides - metabolism | Delayed-Action Preparations - metabolism | Nitric Oxide Donors - administration & dosage | Galactose - chemistry | Animals | Heart - drug effects | Mice | Mesenchymal Stem Cell Transplantation | Neovascularization, Physiologic | Heart | Luciferase | Stem cells | Genetic engineering | Transplantation | Galactose | Vascular endothelial growth factor | Health aspects | Heart attack | Index Medicus | Biomedical materials | Effectiveness | Hydrogels | Migration | Mathematical models | Remodeling
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7. Full Text A peripherally restricted cannabinoid receptor agonist produces robust anti-nociceptive effects in rodent models of inflammatory and neuropathic pain
by Yu, Xiao Hong and Cao, Chang Qing and Martino, Giovanni and Puma, Carole and Morinville, Anne and St-Onge, Stéphane and Lessard, Étienne and Perkins, Martin N and Laird, Jennifer M.A
Pain, ISSN 0304-3959, 11/2010, Volume 151, Issue 2, pp. 337 - 344
Cannabinoids are analgesic in man, but their use is limited by their psychoactive properties. One way to avoid cannabinoid receptor subtype 1 (CB1R)-mediated... 
Nociceptive flexor reflex | Analgesia | Cannabinoid receptor subtype 1 agonist | Rat | AZ11713908 | Peripheral site of action | EFFICACY | CUTANEOUS HYPERALGESIA | ALLODYNIA | RANDOMIZED CONTROLLED-TRIAL | NEUROSCIENCES | RAT MODEL | CLINICAL NEUROLOGY | STRATEGIES | ANTIHYPERALGESIC PROPERTIES | THERMAL NOCICEPTION | CB1 | ANESTHESIOLOGY | Inflammation - chemically induced | Humans | Calcium Channel Blockers - therapeutic use | Male | Naphthalenes - blood | Morpholines - blood | Dose-Response Relationship, Drug | Time Factors | Inflammation - drug therapy | Freund's Adjuvant - adverse effects | Neuralgia - chemically induced | Benzoxazines - blood | Carrageenan - adverse effects | Morpholines - therapeutic use | Benzimidazoles - therapeutic use | Disease Models, Animal | Mice, Inbred C57BL | Rats | Rats, Sprague-Dawley | Neuralgia - drug therapy | Benzoxazines - therapeutic use | Mice, Knockout | Receptor, Cannabinoid, CB1 - metabolism | Cannabinoids - therapeutic use | Pain Measurement - methods | Animals | Sulfonamides - therapeutic use | Anti-Inflammatory Agents, Non-Steroidal - therapeutic use | Mice | Receptor, Cannabinoid, CB1 - deficiency | Calcium Channel Blockers - blood | Naphthalenes - therapeutic use | Receptor, Cannabinoid, CB2 - deficiency | Models | Analgesics | Analysis | Chronic pain | Index Medicus
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8. Full Text Cannabinoids Elicit Antidepressant-Like Behavior and Activate Serotonergic Neurons through the Medial Prefrontal Cortex
by Bambico, Francis Rodriguez and Katz, Noam and Debonnel, Guy and Gobbi, Gabriella
Journal of Neuroscience, ISSN 0270-6474, 10/2007, Volume 27, Issue 43, pp. 11700 - 11711
Preclinical and clinical studies show that cannabis modulates mood and possesses antidepressant-like properties, mediated by the agonistic activity of... 
Medial prefrontal cortex | receptor | Forced swim test | Serotonin | Dorsal raphe nucleus | Depression | Cannabinoid | medial prefrontal cortex | RAT CEREBRAL-CORTEX | cannabinoid | FORCED SWIMMING TEST | NEUROSCIENCES | EXTRACELLULAR GLUTAMATE | VENTRAL TEGMENTAL AREA | dorsal raphe nucleus | serotonin | GLUTAMATERGIC TRANSMISSION | CB1 receptor | ACID AMIDE HYDROLASE | ADULT-RAT | IN-VIVO | depression | forced swim test | CB1 RECEPTORS | Morpholines - pharmacology | Rats | Male | Rats, Sprague-Dawley | Benzoxazines - therapeutic use | Cannabinoids - pharmacology | Naphthalenes - pharmacology | Antidepressive Agents - therapeutic use | Depression - drug therapy | Receptor, Cannabinoid, CB1 - metabolism | Cannabinoids - therapeutic use | Depression - metabolism | Animals | Prefrontal Cortex - drug effects | Receptor, Cannabinoid, CB1 - agonists | Serotonin - metabolism | Benzoxazines - pharmacology | Prefrontal Cortex - metabolism | Antidepressive Agents - pharmacology | Neurons - metabolism | Neurons - drug effects | Morpholines - therapeutic use | Naphthalenes - therapeutic use | Index Medicus
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9. Full Text Effects of WIN 55,212-2 mesylate on the anticonvulsant action of lamotrigine, oxcarbazepine, pregabalin and topiramate against maximal electroshock-induced seizures in mice
by Luszczki, Jarogniew J and Wlaz, Aleksandra and Karwan, Slawomir and Florek-Luszczki, Magdalena and Czuczwar, Stanislaw J
European Journal of Pharmacology, ISSN 0014-2999, 11/2013, Volume 720, Issue 1-3, pp. 247 - 254
The aim of this study was to determine the effect of WIN 55,212-2 mesylate (WIN – a non-selective cannabinoid CB and CB receptor agonist) on the protective... 
Antiepileptic drug | Maximal electroshock-induced seizure | Cannabinoid | WIN 55,212-2 mesylate | CB1 RECEPTOR AGONIST | CARBONIC-ANHYDRASE | GLUTAMATERGIC SYNAPTIC-TRANSMISSION | ACTIVATED CALCIUM-CHANNELS | RAT HIPPOCAMPAL-NEURONS | ADENOSINE RECEPTORS | CANNABINOID RECEPTOR | ANTI-CONVULSANT CARBAMAZEPINE | PHARMACOLOGY & PHARMACY | CORTICOSTRIATAL SYSTEM | ANTIEPILEPTIC DRUGS | Receptor, Cannabinoid, CB2 - agonists | Triazines - therapeutic use | Seizures - drug therapy | Male | Seizures - metabolism | Pregabalin | Brain - metabolism | Seizures - physiopathology | Drug Interactions | Receptor, Cannabinoid, CB1 - agonists | Cannabinoid Receptor Agonists - therapeutic use | gamma-Aminobutyric Acid - therapeutic use | Drug Therapy, Combination | Hand Strength | Morpholines - therapeutic use | gamma-Aminobutyric Acid - pharmacokinetics | Anticonvulsants - pharmacokinetics | Fructose - pharmacokinetics | Carbamazepine - analogs & derivatives | Anticonvulsants - therapeutic use | Carbamazepine - therapeutic use | Memory, Long-Term - drug effects | Fructose - therapeutic use | Benzoxazines - therapeutic use | Fructose - analogs & derivatives | Animals | Avoidance Learning - drug effects | Mice | Muscle Strength - drug effects | Carbamazepine - pharmacokinetics | gamma-Aminobutyric Acid - analogs & derivatives | Electroshock | Naphthalenes - therapeutic use | Seizures - psychology | Triazines - pharmacokinetics | Topiramate | Liquid chromatography | Seizures (Medicine) | Analysis | Index Medicus
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10. Full Text Terutroban versus aspirin in patients with cerebral ischaemic events (PERFORM): a randomised, double-blind, parallel-group trial
by Bousser, Marie-Germaine, Prof and Amarenco, Pierre, Prof and Chamorro, Angel, Prof and Fisher, Marc, Prof and Ford, Ian, MD and Fox, Kim M, Prof and Hennerici, Michael G, Prof and Mattle, Heinrich P, Prof and Rothwell, Peter M, Prof and de Cordoüe, Agnès, MD and Fratacci, Marie-Dominique, MD and PERFORM Study Investigators
Lancet, The, ISSN 0140-6736, 2011, Volume 377, Issue 9782, pp. 2013 - 2022
Summary Background Patients with ischaemic stroke or transient ischaemic attack (TIA) are at high risk of recurrent stroke or other cardiovascular events. We... 
Internal Medicine | STROKE | MEDICINE, GENERAL & INTERNAL | ATTACK PERFORM | CLOPIDOGREL | CARDIOVASCULAR EVENTS | PREVENTION | ATHEROSCLEROSIS | PLACEBO-CONTROLLED TRIAL | THROMBOXANE A | RECEPTOR ANTAGONIST | CORONARY ANGIOPLASTY | Stroke - prevention & control | Ischemic Attack, Transient - drug therapy | Double-Blind Method | Humans | Middle Aged | Propionates - therapeutic use | Male | Treatment Outcome | Secondary Prevention | Stroke - drug therapy | Ischemic Attack, Transient - prevention & control | Receptors, Thromboxane - antagonists & inhibitors | Female | Aged | Aspirin - therapeutic use | Platelet Aggregation Inhibitors - therapeutic use | Naphthalenes - therapeutic use | Stroke (Disease) | Aspirin | Research | Drug therapy | Comparative analysis | Health aspects | Studies | Stroke | Heart attacks | Mortality | Blood pressure | Acute coronary syndromes | Blood clots | Dementia | Drugs | Myocardial infarction | Statistical analysis | Transient ischemic attack | Motivation | Clinical trials | Data processing | Bleeding | Risk factors | Index Medicus | Abridged Index Medicus | Ischemic Attack, Transient | Naphthalenes | Life Sciences | Human health and pathology | Receptors, Thromboxane | Propionates | Platelet Aggregation Inhibitors
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