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Journal Article
NATURE COMMUNICATIONS, ISSN 2041-1723, 01/2017, Volume 8, Issue 1, p. 14121
Journal Article
Journal of Immunotherapy, ISSN 1524-9557, 11/2010, Volume 33, Issue 9, pp. 983 - 990
Journal Article
NeuroReport, ISSN 0959-4965, 08/2017, Volume 28, Issue 12, pp. 705 - 711
Journal Article
Journal Article
PLoS Pathogens, ISSN 1553-7366, 07/2017, Volume 13, Issue 7, p. e1006503
Myeloid-derived suppressor cells (MDSCs) are expanded in tumor microenvironments, including that of Epstein-Barr virus (EBV)-associated nasopharyngeal... 
AIM2 INFLAMMASOME | CANCER CELLS | INFLAMMASOME ACTIVATION | MICROBIOLOGY | NLRP3 INFLAMMASOME | VIROLOGY | EPITHELIAL-CELLS | GLUCOSE-METABOLISM | AEROBIC GLYCOLYSIS | NF-KAPPA-B | GLUT1 EXPRESSION | EPSTEIN-BARR-VIRUS | PARASITOLOGY | Nasopharyngeal Neoplasms - genetics | Nasopharyngeal Neoplasms - metabolism | Viral Matrix Proteins - genetics | Cell Proliferation | Herpesvirus 4, Human - genetics | Granulocyte-Macrophage Colony-Stimulating Factor - metabolism | Humans | Carcinoma - virology | Gene Expression Regulation, Neoplastic | Nasopharyngeal Neoplasms - virology | Nasopharyngeal Neoplasms - physiopathology | Myeloid-Derived Suppressor Cells - cytology | Epstein-Barr Virus Infections - genetics | Viral Matrix Proteins - metabolism | Interleukin-6 - metabolism | Myeloid-Derived Suppressor Cells - metabolism | Epstein-Barr Virus Infections - physiopathology | Nasopharyngeal Carcinoma | Epstein-Barr Virus Infections - virology | Interleukin-6 - genetics | Signal Transduction | Carcinoma - physiopathology | Granulocyte-Macrophage Colony-Stimulating Factor - genetics | Host-Pathogen Interactions | Cell Line, Tumor | Glycolysis | Carcinoma - genetics | Carcinoma - metabolism | Herpesvirus 4, Human - metabolism | Epstein-Barr Virus Infections - metabolism | Genetic aspects | Epstein-Barr virus | Research | Nasopharyngeal cancer | Membrane proteins | Laboratories | Viruses | Oncology | Infections | In vitro testing | Suppressor cells | Degradation | Interleukin 6 | Proteins | Nasopharyngeal carcinoma | Mitochondria | Ubiquitination | Interleukin 1 | Life sciences | Expansion | Glucose transporter | Biodegradation | GLUT1 protein | Granulocyte-macrophage colony-stimulating factor | Roles | Gene expression | Medicine | Immunosuppression | Collaboration | Microenvironments | Cyclooxygenase-2 | Transporter | Cancer | Latent membrane protein 1 | Tumors
Journal Article
Journal Article
Human Brain Mapping, ISSN 1065-9471, 01/2018, Volume 39, Issue 1, pp. 407 - 427
Radiation therapy, a major method of treatment for brain cancer, may cause severe brain injuries after many years. We used a rare and unique cohort of... 
amplitude of low‐frequency fluctuations | resting state | nasopharyngeal carcinoma | functional connectivity | radiation therapy | structural connectivity | diffusion tensor imaging | irradiation injury | prognosis | functional magnetic resonance imaging | amplitude of low-frequency fluctuations | RESTING-STATE FMRI | INTENSITY-MODULATED RADIOTHERAPY | NASOPHARYNGEAL CARCINOMA PATIENTS | NEUROSCIENCES | NEUROIMAGING | TEMPORAL-LOBE NECROSIS | COGNITIVE FUNCTION | APPEARING WHITE-MATTER | IN-VIVO | CENTRAL-NERVOUS-SYSTEM | CRANIAL IRRADIATION | METABOLIC ALTERATIONS | RADIOLOGY, NUCLEAR MEDICINE & MEDICAL IMAGING | Brain - diagnostic imaging | Prognosis | Follow-Up Studies | Humans | Middle Aged | Radiation Injuries - diagnostic imaging | Male | Brain Injuries - physiopathology | Carcinoma - diagnostic imaging | Young Adult | Multimodal Imaging | Nasopharyngeal Neoplasms - radiotherapy | Rest | Nasopharyngeal Neoplasms - physiopathology | Adult | Female | Retrospective Studies | Radiotherapy Dosage | Nasopharyngeal Neoplasms - diagnostic imaging | Nasopharyngeal Carcinoma | Cross-Sectional Studies | Brain - physiopathology | Brain - radiation effects | Carcinoma - radiotherapy | Carcinoma - physiopathology | Diffusion Tensor Imaging | Radiation Injuries - etiology | Magnetic Resonance Imaging | Brain Injuries - diagnostic imaging | Models, Biological | Aged | Radiation Injuries - physiopathology | Brain Injuries - etiology | Dose-Response Relationship, Radiation | Brain | Injuries | Radiation | Intervention | Neuroimaging | Temporal lobe | Radiation effects | Abnormalities | Cognitive ability | Radiation therapy | Substantia alba | Necrosis | Nasopharyngeal carcinoma | Injury prevention | Head injuries | Neural networks | Predictions | Gangrene | Radiation injuries | Biomarkers | Irradiation | Mathematical models | Brain injury | Cancer | Structure-function relationships | Index Medicus
Journal Article
Journal Article
The Anatomical Record, ISSN 1932-8486, 02/2014, Volume 297, Issue 2, pp. 215 - 221
ABSTRACT Nasopharyngeal carcinoma (NPC) is one of the most common cancers originating in the nasopharynx, and chemoresistance is an essential aspect of NPC... 
sorcin | multidrug resistance | nasopharyngeal carcinoma | Nasopharyngeal carcinoma | Sorcin | Multidrug resistance | GASTRIC-CANCER CELLS | MULTIDRUG-RESISTANCE | ANATOMY & MORPHOLOGY | Nasopharyngeal Neoplasms - metabolism | Carcinoma | Gene Silencing - drug effects | Humans | Calcium-Binding Proteins - antagonists & inhibitors | NF-kappa B - metabolism | Antineoplastic Agents - therapeutic use | Pharyngeal Neoplasms - drug therapy | Pharyngeal Neoplasms - physiopathology | Nasopharyngeal Neoplasms - physiopathology | Gene Expression Regulation, Neoplastic - drug effects | Nasopharyngeal Neoplasms - drug therapy | Nose Neoplasms - metabolism | Gene Expression Regulation, Neoplastic - physiology | Calcium-Binding Proteins - metabolism | Nasopharyngeal Carcinoma | RNA, Small Interfering - pharmacology | Nose Neoplasms - physiopathology | PTEN Phosphohydrolase - metabolism | Pharyngeal Neoplasms - metabolism | ATP Binding Cassette Transporter, Subfamily B | ATP Binding Cassette Transporter, Subfamily B, Member 1 - metabolism | Cisplatin - therapeutic use | Nose Neoplasms - drug therapy | Cell Line, Tumor | Drug Resistance, Neoplasm - physiology | Gene Silencing - physiology | Multidrug Resistance-Associated Proteins - metabolism | Calcium-Binding Proteins - genetics | Chemotherapy | RNA | Glutathione transferase | Drug resistance | Drug therapy | Cancer | Genes | Rodents
Journal Article