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2013, Methods in molecular biology, ISBN 9781627033824, Volume 1004.
Cell death is an essential process in development, and a major contributor to a wide range of human diseases. Three major classifications of cell death,... 
Necrosis
Web Resource
2007, Advances in experimental medicine and biology, ISBN 9780387706290, Volume 597., xix, 206
It has been established that TNF receptor associated factors (TRAFs) are critical signaling mediators for not only the TNF receptor superfamily, but also the... 
Tumor necrosis factor | Receptors | Cellular signal transduction | Life Sciences | Developmental Biology | Cell Biology
Book
2009, Advances in experimental medicine and biology, ISBN 0387895191, Volume 647, xvii, 220
For two decades, TNF superfamily has been the most investigated area of basic medical research. This book offers a state-of-the-art account of that research,... 
drug therapy | Receptors, Tumor Necrosis Factor | Tumor necrosis factor | Autoimmune Diseases | Tumor Necrosis Factors | Agonists | Inflammation | Neoplasms | Therapeutic use | Biomedicine general | Biomedicine
Book
Journal of Clinical Investigation, ISSN 0021-9738, 11/2012, Volume 122, Issue 11, pp. 4094 - 4104
TNF, an inflammatory cytokine that is enriched in the tumor microenvironment, promotes tumor growth and subverts innate immune responses to cancer cells. We... 
MEDICINE, RESEARCH & EXPERIMENTAL | TUMOR-NECROSIS-FACTOR | INHIBITION | INFLAMMATION | MACROPHAGES | DEATH | EXPRESSION | CANCER | ANTITUMOR | T-CELLS | FACTOR-ALPHA | Neoplasms - metabolism | Tumor Necrosis Factor-alpha - metabolism | Neoplasm Transplantation | Tumor Escape | Receptors, Tumor Necrosis Factor, Type I - immunology | Tumor Necrosis Factor-alpha - genetics | Caspase 8 - metabolism | Graft Rejection - metabolism | Receptors, Tumor Necrosis Factor, Type I - metabolism | Receptors, Tumor Necrosis Factor, Type II - immunology | Caspase 8 - genetics | Signal Transduction - immunology | Neoplasms - genetics | T-Lymphocytes - metabolism | Myeloid Cells - immunology | Receptors, Tumor Necrosis Factor, Type II - metabolism | Tumor Necrosis Factor-alpha - immunology | T-Lymphocytes - pathology | Graft Rejection - pathology | CASP8 and FADD-Like Apoptosis Regulating Protein - genetics | Signal Transduction - genetics | Receptors, Tumor Necrosis Factor, Type I - genetics | CASP8 and FADD-Like Apoptosis Regulating Protein - biosynthesis | Receptors, Tumor Necrosis Factor, Type II - genetics | Mice, Knockout | Caspase 8 - immunology | Animals | Neoplasms - immunology | Cell Line, Tumor | Myeloid Cells - metabolism | T-Lymphocytes - immunology | Mice | Mice, Inbred BALB C | CASP8 and FADD-Like Apoptosis Regulating Protein - immunology | Graft Rejection - immunology | Myeloid Cells - pathology | Graft Rejection - genetics | Neoplasms - pathology | Tumor necrosis factor | Genetic aspects | Cellular signal transduction | Research | Properties | Gene expression | Apoptosis | Index Medicus | Abridged Index Medicus
Journal Article
Molecular and Cellular Biology, ISSN 0270-7306, 10/2006, Volume 26, Issue 19, pp. 7046 - 7055
Article Usage Stats Services MCB Citing Articles Google Scholar PubMed Related Content Social Bookmarking CiteULike Delicious Digg Facebook Google+ Mendeley... 
CANCER-CELLS | APO2L/TRAIL | DEATH DOMAIN | CASPASE-8 ACTIVATION | APOPTOSIS-INDUCING LIGAND | PROTEIN | BIOCHEMISTRY & MOLECULAR BIOLOGY | RESISTANCE | MONOCLONAL-ANTIBODIES | NF-KAPPA-B | EXPRESSION | CELL BIOLOGY | Receptors, Tumor Necrosis Factor - metabolism | Apoptosis Regulatory Proteins - pharmacology | Apoptosis - drug effects | Jurkat Cells | Humans | Membrane Glycoproteins - pharmacology | Cells, Cultured | GPI-Linked Proteins | Enzyme Activation - drug effects | Receptors, TNF-Related Apoptosis-Inducing Ligand | Death Domain Receptor Signaling Adaptor Proteins | Receptors, Tumor Necrosis Factor, Member 10c | Tumor Necrosis Factor Receptor-Associated Peptides and Proteins - metabolism | Tumor Necrosis Factor Decoy Receptors | Tumor Necrosis Factor-alpha - pharmacology | Caspases - metabolism | Membrane Glycoproteins - antagonists & inhibitors | TNF-Related Apoptosis-Inducing Ligand | Membrane Microdomains - drug effects | Protein Binding - drug effects | Apoptosis Regulatory Proteins - antagonists & inhibitors | HeLa Cells | Tumor Necrosis Factor-alpha - antagonists & inhibitors | Index Medicus | Membrane Microdomains | Tumor Necrosis Factor Receptor-Associated Peptides and Proteins | Biochemistry, Molecular Biology | Tumor Necrosis Factor-alpha | Life Sciences | Apoptosis Regulatory Proteins | Receptors, Tumor Necrosis Factor | Protein Binding | Caspases | Enzyme Activation | Membrane Glycoproteins | Hela Cells | Apoptosis
Journal Article
PLoS ONE, ISSN 1932-6203, 10/2013, Volume 8, Issue 10, pp. e76841 - e76841
Journal Article
Journal of Animal and Veterinary Advances, ISSN 1680-5593, 01/2014, Volume 204, Issue 2, pp. 231 - 245
Nuclear factor kappa B (NF-kappa B) essential modulator (NEMO), a regulatory component of the I kappa B kinase (IKK) complex, controls NF-kappa B activation... 
LYS63-LINKED POLYUBIQUITINATION | RECRUITMENT | ACTIVATION | COMPLEX | RECEPTOR-ASSOCIATED KINASE | CONJUGATING ENZYME | ALPHA | CHAINS | NF-KAPPA-B | DEFICIENCY | CELL BIOLOGY | Receptors, Tumor Necrosis Factor - metabolism | Tumor Necrosis Factor-alpha - metabolism | Interleukin-1 - metabolism | Interleukin-1 Receptor-Associated Kinases - metabolism | Interleukin-1 - analysis | Signal Transduction | Humans | Ubiquitin - metabolism | Receptors, Tumor Necrosis Factor - analysis | NF-kappa B - metabolism | I-kappa B Kinase - analysis | Ubiquitin - genetics | Interleukin-1 Receptor-Associated Kinases - analysis | Ubiquitin - physiology | Receptors, Interleukin-1 - analysis | Receptors, Interleukin-1 - metabolism | Tumor Necrosis Factor-alpha - analysis | Ubiquitination | I-kappa B Kinase - metabolism | Interleukin-1 - physiology | Tumor Necrosis Factor-alpha - physiology | Cell Line, Tumor | HeLa Cells | NF-kappa B - analysis | Ubiquitin | Exhibitions | Interleukins | Tumor necrosis factor | TNF inhibitors | Biochemistry | Cytokines | Molecular biology | Kinases | Index Medicus | Interleukin-1 | Biochemistry, Molecular Biology | Interleukin-1 Receptor-Associated Kinases | I-kappa B Kinase | Tumor Necrosis Factor-alpha | Life Sciences | NF-kappa B | Receptors, Tumor Necrosis Factor | Biomolecules | Receptors, Interleukin-1
Journal Article
CELL, ISSN 0092-8674, 07/2003, Volume 114, Issue 2, pp. 181 - 190
Apoptosis induced by TNF-receptor I (TNFR1) is thought to proceed via recruitment of the adaptor FADD and caspase-8 to the receptor complex. TNFR1 signaling is... 
DEATH RECEPTORS | RECRUITMENT | TUMOR-NECROSIS-FACTOR | ACTIVATION | BIOCHEMISTRY & MOLECULAR BIOLOGY | C-FLIP | FAS | FADD | NF-KAPPA-B | FLICE-INHIBITORY PROTEINS | REQUIREMENT | CELL BIOLOGY | Tumor Necrosis Factor-alpha - metabolism | Caspase 9 | Caspase 8 | Humans | Transcriptional Activation | Cytoplasm - metabolism | NF-kappa B - metabolism | RNA-Binding Proteins | fas Receptor - metabolism | TNF Receptor-Associated Factor 1 | Caspases - metabolism | Cell Membrane - metabolism | Tumor Cells, Cultured | Cell Survival - physiology | Receptors, Tumor Necrosis Factor - genetics | Receptors, Tumor Necrosis Factor - metabolism | Signal Transduction | Jurkat Cells | Nuclear Pore Complex Proteins - metabolism | Intracellular Signaling Peptides and Proteins | Fas-Associated Death Domain Protein | Caspase Inhibitors | Proteins - immunology | Proteins - genetics | Tumor Necrosis Factor-alpha - pharmacology | Adaptor Proteins, Signal Transducing | Carrier Proteins - metabolism | Proteins - metabolism | NF-kappa B - genetics | Models, Biological | CASP8 and FADD-Like Apoptosis Regulating Protein | Apoptosis - physiology | Receptors, Tumor Necrosis Factor - immunology | NF-kappa B - drug effects | Chemical inhibitors | Cell research | Cell death | Tumor necrosis factor | Analysis | Physiological aspects | Genetic aspects | Cytoplasm | Apoptosis | Index Medicus | Cell Membrane | Antigens, CD95 | Proteins | Life Sciences | NF-kappa B | Carrier Proteins | Nuclear Pore Complex Proteins | Cell Survival | Biochemistry, Molecular Biology | Tumor Necrosis Factor-alpha | Receptors, Tumor Necrosis Factor | Caspases
Journal Article
1992, ISBN 0824785541, Volume 56., xv, 600 p. : ill. ; 24 cm.
Book
Neuroscience, ISSN 0306-4522, 2008, Volume 157, Issue 2, pp. 414 - 423
Abstract Recent studies suggest that tumor necrosis factor-alpha (TNF) sensitizes primary afferent neurons, and thus facilitates neuropathic pain. Here, we... 
Neurology | DRG | neuropathic pain | ectopic activity | cytokines | TNF receptor | TNF | RHEUMATOID-ARTHRITIS | PRIMARY AFFERENTS | RAT | ALPHA TNF-ALPHA | TACTILE ALLODYNIA | DORSAL-ROOT GANGLION | NEUROSCIENCES | CELL-DEATH | SCIATIC-NERVE | SPINAL NERVE LIGATION | UP-REGULATION | Hyperalgesia - chemically induced | Receptors, Tumor Necrosis Factor - classification | Receptors, Tumor Necrosis Factor, Type II - chemistry | Humans | Male | Ganglia, Spinal - cytology | Ligation - methods | Dose-Response Relationship, Drug | Nerve Fibers - physiology | Receptors, Tumor Necrosis Factor, Type I - chemistry | Time Factors | Reaction Time - drug effects | Receptors, Tumor Necrosis Factor, Type I - pharmacology | Pain Threshold - drug effects | Action Potentials - drug effects | Receptors, Tumor Necrosis Factor - genetics | Physical Stimulation | Receptors, Tumor Necrosis Factor - chemistry | Rats | Nerve Fibers - drug effects | Receptors, Tumor Necrosis Factor - administration & dosage | Mutation - genetics | Receptors, Tumor Necrosis Factor, Type I - genetics | Rats, Sprague-Dawley | Receptors, Tumor Necrosis Factor, Type II - genetics | Spinal Cord - surgery | Hyperalgesia - physiopathology | Animals | Receptors, Tumor Necrosis Factor, Type II - pharmacology | Sensory Receptor Cells - drug effects | Analysis of Variance | In Vitro Techniques | Pain Measurement | Neurosciences | Serum albumin | Tumor necrosis factor | Neurons | Tumors | Necrosis | Index Medicus
Journal Article
Nature, ISSN 0028-0836, 08/2012, Volume 488, Issue 7412, pp. 508 - 511
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 11/2001, Volume 98, Issue 24, pp. 13960 - 13965
In vivo studies have shown T cells to be central to the mechanism by which estrogen deficiency induces bone loss, but the mechanism involved remains, in part,... 
T lymphocytes | Tumor necrosis factor receptors | Biological Sciences | Spleen cells | Estrogens | Bone marrow | Bones | Osteoclasts | Bone density | Mice | Bone marrow cells | Osteoporosis | Mouse | Ovariectomy | PQCT | FACTOR-RECEPTOR | NUDE-MICE | pQCT | FORMATION IN-VITRO | MULTIDISCIPLINARY SCIENCES | osteoporosis | mouse | TUMOR-NECROSIS-FACTOR | POSTMENOPAUSAL OSTEOPOROSIS | HISTOMORPHOMETRY | OSTEOPROTEGERIN LIGAND | GENE-EXPRESSION | OSTEOCLAST DIFFERENTIATION | ovariectomy | LYMPHOCYTES | Tumor Necrosis Factor-alpha - metabolism | Up-Regulation | Tumor Necrosis Factor-alpha - genetics | Antigens, CD - genetics | Receptors, Tumor Necrosis Factor - physiology | Antigens, CD - metabolism | Osteoporosis - metabolism | T-Lymphocytes - metabolism | Receptors, Tumor Necrosis Factor, Type II | Female | Estrogens - metabolism | Receptors, Tumor Necrosis Factor - genetics | Receptors, Tumor Necrosis Factor - metabolism | Estrogens - physiology | Bone Marrow Cells - cytology | Mice, Inbred C57BL | Receptors, Tumor Necrosis Factor, Type I | Cells, Cultured | Mice, Knockout | Animals | T-Lymphocytes - cytology | Mice, Nude | Tumor Necrosis Factor-alpha - physiology | Antigens, CD - physiology | Tumor Necrosis Factor-alpha - biosynthesis | Bone Marrow Cells - metabolism | Physiological aspects | Research | T cells | Tumor necrosis factor | Cells | Estrogen | Index Medicus | ovariectomy‖osteoporosis‖mouse‖pQCT
Journal Article
Journal of Clinical Investigation, ISSN 0021-9738, 04/2014, Volume 124, Issue 4, pp. 1537 - 1551
Development of host protective immunity against Mycobacterium tuberculosis infection is critically dependent on the inflammatory cytokine TNF. TNF signals... 
MEDICINE, RESEARCH & EXPERIMENTAL | TUMOR-NECROSIS-FACTOR | BACILLUS-CALMETTE-GUERIN | TRANSMEMBRANE TNF | DEFICIENT MICE | BACTERIAL-INFECTION | HUMAN ALVEOLAR MACROPHAGES | DENDRITIC CELL MATURATION | FACTOR RECEPTOR | INDUCED GRANULOMA-FORMATION | FACTOR-ALPHA | Tumor Necrosis Factor-alpha - metabolism | Tuberculosis, Pulmonary - microbiology | Tumor Necrosis Factor Decoy Receptors - genetics | Receptors, Tumor Necrosis Factor, Type I - immunology | Tumor Necrosis Factor Decoy Receptors - immunology | Dendritic Cells - immunology | Mycobacterium tuberculosis - immunology | Male | Receptors, Tumor Necrosis Factor, Type II - immunology | Host-Pathogen Interactions - immunology | CD4-Positive T-Lymphocytes - immunology | Mycobacterium tuberculosis - pathogenicity | Receptors, Tumor Necrosis Factor, Type I - deficiency | Signal Transduction - immunology | Female | Interleukin-12 Subunit p40 - metabolism | Tumor Necrosis Factor Decoy Receptors - deficiency | Lymphocyte Activation | Mice, Inbred C57BL | Solubility | Tuberculosis, Pulmonary - pathology | Receptors, Tumor Necrosis Factor, Type I - genetics | Receptors, Tumor Necrosis Factor, Type II - genetics | Mice, Knockout | Receptors, Tumor Necrosis Factor, Type II - deficiency | Granuloma - pathology | Tuberculosis, Pulmonary - immunology | Animals | Mice | Prevention | Care and treatment | Immune response | Mycobacterium tuberculosis | Tumor necrosis factor | Physiological aspects | Drug targeting | Regulation | Research | Studies | Tuberculosis | Cytokines | Rodents | Mortality | Ligands | Infections | Index Medicus | Abridged Index Medicus
Journal Article