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PloS one, ISSN 1932-6203, 2012, Volume 7, Issue 11, p. e49023
Intrauterine infection and inflammation are major reasons for preterm birth. The switch from placenta-mediated to lung-mediated oxygen supply during birth is... 
INDUCED CELL-DEATH | ISCHEMIC TOLERANCE | MICROGLIAL TOXICITY | NITRIC-OXIDE SYNTHASE | MULTIDISCIPLINARY SCIENCES | PERIVENTRICULAR LEUKOMALACIA | DEVELOPING OLIGODENDROCYTES | NECROSIS-FACTOR-ALPHA | EXCITOTOXIC INJURY | INDUCED BRAIN-INJURY | BACTERIAL-ENDOTOXIN SENSITIZES | Inflammation - chemically induced | Inflammation - pathology | Microglia - metabolism | Oligodendroglia - metabolism | Rats, Wistar | Apoptosis - drug effects | Caspase 3 - metabolism | Brain - metabolism | Inflammation - metabolism | Oligodendroglia - drug effects | Nerve Fibers, Myelinated - pathology | Hyperoxia - metabolism | Microglia - pathology | Animals, Newborn | Nerve Fibers, Myelinated - metabolism | Leukoencephalopathies - pathology | Microglia - drug effects | Nerve Fibers, Myelinated - drug effects | Cells, Cultured | Rats | Leukoencephalopathies - metabolism | Rats, Sprague-Dawley | Brain - drug effects | Hyperoxia - pathology | Oligodendroglia - pathology | Animals | Lipopolysaccharides - pharmacology | Brain - pathology | Apoptosis - physiology | Immunohistochemistry | Brain | Cell death | Infants (Premature) | Inflammation | Research | Mitogens | Health aspects | Injuries | Neuroimaging | Neonates | Cell culture | Pediatrics | Transcription factors | Traumatic brain injury | Premature birth | Interleukin | Superoxide dismutase | Infants | Infections | Birth | Western blotting | Lipopolysaccharides | Head injuries | Newborn babies | Ischemia | Rodents | Oligodendrocytes | Oxygen | Pain perception | Cytokines | Maturation | Incubation | Mortality | Magnetic resonance | Gene expression | Substantia alba | Microglia | Hospitals | Placenta | Magnetic resonance imaging | Lungs | Magnetic permeability | Hyperoxia | Interleukin 10 | Brain damage | Hypoxia | Oxygen tension | Brain injury | Apoptosis | pathology | Leukoencephalopathies | Sprague-Dawley | pharmacology | Caspase 3 | Cultured | Basic Medicine | chemically induced | Myelinated | physiology | drug effects | Cells | Newborn | Wistar | Medicinska grundvetenskaper | Oligodendroglia | metabolism | Nerve Fibers
Journal Article
Nature (London), ISSN 1476-4687, 2017, Volume 541, Issue 7638, pp. 481 - 487
Journal Article
Clinical & Experimental Ophthalmology, ISSN 1442-6404, 03/2009, Volume 37, Issue 2, pp. 223 - 231
Purpose:  The aim of this study is to investigate markers of inflammation and oxidative stress in an early model of diabetic retinopathy, correlate retinal and... 
pericyte | diabetic retinopathy | inflammation | microglia | oxidative stress | Diabetic retinopathy | Oxidative stress | Pericyte | Inflammation | Microglia | CELLS | ANGIOGENESIS | ANTIOXIDANTS | DEATH | DAMAGE | PATHWAY | DISEASE | OPHTHALMOLOGY | RETINOPATHY | Retina - drug effects | Diabetic Retinopathy - drug therapy | Inflammation - pathology | Microglia - metabolism | Diabetes Mellitus, Experimental - drug therapy | Retina - metabolism | Oxidative Stress | Rats, Wistar | Tumor Necrosis Factor-alpha - blood | Superoxide Dismutase - blood | Male | Diabetic Retinopathy - pathology | Immunoenzyme Techniques | Inflammation - metabolism | Pericytes - pathology | Microglia - pathology | Free Radical Scavengers - therapeutic use | Acetylcysteine - therapeutic use | Diabetes Mellitus, Experimental - metabolism | Fluorescent Antibody Technique, Indirect | Enzyme-Linked Immunosorbent Assay | Macrophages - pathology | Pericytes - metabolism | Rats | Biomarkers - blood | Diabetic Retinopathy - metabolism | Macrophages - metabolism | Animals | Endothelium, Vascular - metabolism | Diabetes Mellitus, Experimental - pathology | Endothelium, Vascular - pathology | Isoprostanes - blood | Pericytes, metabolism | Endothelium, Vascular, metabolism | Microglia, metabolism | Superoxide Dismutase, blood | Diabetic Retinopathy, pathology | Isoprostanes, blood | Tumor Necrosis Factor-alpha, blood | Acetylcysteine, therapeutic use | Endothelium, Vascular, pathology | Inflammation, metabolism | Inflammation, pathology | Diabetic Retinopathy, metabolism | Diabetes Mellitus, Experimental, drug therapy | Macrophages, pathology | Retina, drug effects | Retina, metabolism | Diabetes Mellitus, Experimental, pathology | Pericytes, pathology | Diabetic Retinopathy, drug therapy | Diabetes Mellitus, Experimental, metabolism | Biological Markers, blood | Macrophages, metabolism | Microglia, pathology | Free Radical Scavengers, therapeutic use | Streptozocin | Heme | Acetylcysteine | Superoxide dismutase | Superoxide | Ophthalmology | Universities and colleges | Macrophages | Vascular endothelial growth factor | Endothelium
Journal Article
PloS one, ISSN 1932-6203, 2017, Volume 12, Issue 1, p. e0170814
Chemotherapy-induced peripheral neuropathy (CIPN) and associated neuropathic pain is a debilitating adverse effect of cancer treatment. Current understanding... 
MACROPHAGE INFILTRATION | PAIN | INDUCED MECHANICAL ALLODYNIA | MULTIDISCIPLINARY SCIENCES | REGULATORY T-CELLS | DORSAL-ROOT GANGLIA | SPINAL-CORD | NERVE INJURY | SENSORY NEURONS | SATELLITE GLIAL-CELLS | PACLITAXEL | Hyperalgesia - chemically induced | Neuralgia - genetics | Spleen - immunology | Chemokine CCL2 - immunology | Male | Spleen - drug effects | Activating Transcription Factor 3 - immunology | T-Lymphocytes, Regulatory - pathology | Hyperalgesia - pathology | T-Lymphocytes, Regulatory - immunology | Neurofilament Proteins - genetics | Neuralgia - immunology | Chemokine CCL3 - genetics | Microglia - pathology | Lymph Nodes - drug effects | Activating Transcription Factor 3 - genetics | Receptors, Purinergic P2Y12 - genetics | Gene Expression | Receptors, Purinergic P2Y12 - immunology | Mice, Transgenic | Lymph Nodes - immunology | Spinal Cord - immunology | Hyperalgesia - immunology | T-Lymphocytes, Regulatory - drug effects | Hyperalgesia - genetics | Ganglia, Spinal - pathology | Mice | CD8-Positive T-Lymphocytes - immunology | Ganglia, Spinal - drug effects | CD8-Positive T-Lymphocytes - pathology | Lymph Nodes - pathology | Spinal Cord - drug effects | Chemokine CCL3 - immunology | Sensory Receptor Cells - immunology | Neuralgia - pathology | Microglia - immunology | Antineoplastic Agents - adverse effects | Spinal Cord - pathology | Neuralgia - chemically induced | Neurofilament Proteins - immunology | Spleen - pathology | Ganglia, Spinal - immunology | Sensory Receptor Cells - pathology | Microglia - drug effects | Mice, Inbred C57BL | Paclitaxel - adverse effects | Chemokine CCL2 - genetics | Animals | Sensory Receptor Cells - drug effects | CD8-Positive T-Lymphocytes - drug effects | Organoplatinum Compounds - adverse effects | Usage | Chemotherapy | Care and treatment | Oxalic acid | Polyneuropathies | Research | Diagnosis | T cells | Cancer | Spinal cord | Neurosciences | CD8 antigen | Carbon tetrachloride | Interleukin | Central nervous system | Transgenic | Nervous system | Diabetic neuropathy | Lymphocytes T | Cerebrospinal fluid | Peripheral neuropathy | Macrophages | Lymph nodes | Proteins | Neurotoxicity | Interleukin 4 | Pain | Dorsal root ganglia | Lymphocytes | Rodents | Sensory neurons | Tumor necrosis factor-TNF | Horns | Dorsal horn | Peripheral nervous system | Pain sensitivity | Spleen | Pain perception | Immune response | Cytokines | Granulocyte-macrophage colony-stimulating factor | Hypersensitivity | CCL3 protein | Interleukin 12 | Inflammation | CCL4 protein | Microglia | CD4 antigen | Studies | White blood cells | TNF inhibitors | Infiltration | Chemokines | Monocyte chemoattractant protein 1
Journal Article
Journal of Dentistry, ISSN 0300-5712, 2016, Volume 56, pp. 19 - 32
Journal Article