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Immunological Reviews, ISSN 0105-2896, 07/2013, Volume 254, Issue 1, pp. 78 - 101
Summary Systemic chronic immune activation is considered today as the driving force of CD4+ T‐cell depletion and acquired immunodeficiency syndrome (AIDS). A... 
SIV | pathogenesis | HIV | inflammation | immune activation | natural hosts | Immune activation | Inflammation | Pathogenesis | Natural hosts | LONG-TERM NONPROGRESSORS | RECEIVING ANTIRETROVIRAL THERAPY | AFRICAN-GREEN MONKEYS | CD4(+) T-CELLS | NONPATHOGENIC SIV INFECTION | NECROSIS-FACTOR-ALPHA | IMMUNOLOGY | PLASMACYTOID DENDRITIC CELLS | SIMIAN IMMUNODEFICIENCY VIRUS | LYMPHOID-TISSUE | INFECTED SOOTY MANGABEYS | HIV-1 - metabolism | Lymphoid Tissue - virology | HIV Infections - virology | Humans | Viral Proteins - immunology | Lymphocyte Depletion | Lymphoid Tissue - immunology | CD4-Positive T-Lymphocytes - immunology | HIV Infections - immunology | HIV-1 - immunology | Lymphocyte Activation - immunology | Simian Acquired Immunodeficiency Syndrome - virology | Animals | Simian Acquired Immunodeficiency Syndrome - immunology | Virus Replication | CD4-Positive T-Lymphocytes - virology | HIV (Viruses) | T cells | HIV infection | Lymphoid Tissue/immunology | HIV-1/immunology | HIV Infections/immunology | HIV-1/metabolism | Lymphoid Tissue/virology | Virology | Life Sciences | Simian Acquired Immunodeficiency Syndrome/virology | Microbiology and Parasitology | Immunology | CD4-Positive T-Lymphocytes/immunology | Lymphocyte Activation/immunology | HIV Infections/virology | CD4-Positive T-Lymphocytes/virology | Simian Acquired Immunodeficiency Syndrome/immunology | Viral Proteins/immunology
Journal Article
PLoS Pathogens, ISSN 1553-7366, 2015, Volume 11, Issue 9, p. e1005153
Two of the crucial aspects of human immunodeficiency virus (HIV) infection are (i) viral persistence in reservoirs (precluding viral eradication) and (ii)... 
LONG-TERM NONPROGRESSORS | MESSENGER-RNA EXPRESSION | MICROBIOLOGY | NECROSIS-FACTOR-ALPHA | HUMAN-IMMUNODEFICIENCY-VIRUS | HUMAN ADIPOCYTES | VIROLOGY | ACTIVE ANTIRETROVIRAL THERAPY | SIVMAC251 INFECTION | IMMUNE ACTIVATION | METABOLIC-DISORDERS | MEMORY T-CELLS | PARASITOLOGY | Simian Acquired Immunodeficiency Syndrome - metabolism | HIV - physiology | Stromal Cells - pathology | Coculture Techniques | Humans | Middle Aged | Macaca fascicularis | Male | HIV - immunology | Adipose Tissue - immunology | CD4-Positive T-Lymphocytes - pathology | CD4-Positive T-Lymphocytes - immunology | Simian Immunodeficiency Virus - isolation & purification | Adipose Tissue - metabolism | HIV Infections - immunology | Simian Acquired Immunodeficiency Syndrome - virology | Macrophages - virology | HIV Infections - pathology | Stromal Cells - virology | Adult | Female | Panniculitis - immunology | Disease Reservoirs | CD4-Positive T-Lymphocytes - virology | Simian Acquired Immunodeficiency Syndrome - pathology | Macrophages - immunology | Simian Immunodeficiency Virus - immunology | Macrophages - pathology | Adipose Tissue - virology | HIV Infections - virology | Stromal Cells - metabolism | Adipose Tissue - pathology | CD4-Positive T-Lymphocytes - metabolism | Cells, Cultured | Stromal Cells - immunology | HIV - isolation & purification | Immunity, Innate | Panniculitis - virology | Simian Immunodeficiency Virus - physiology | Host-Pathogen Interactions | Macrophages - metabolism | Animals | Simian Acquired Immunodeficiency Syndrome - immunology | Aged | Panniculitis - pathology | Panniculitis - metabolism | HIV Infections - metabolism | Adipose tissues | Host-virus relationships | HIV (Viruses) | Simian immunodeficiency virus | Observations | Health aspects | Life Sciences | Human health and pathology | Antiretroviral drugs | Lymphatic system | Mortality | Infections | Inflammation | Adipocytes | Metabolism | Experiments | HIV | Lymphocytes | Rodents | Deoxyribonucleic acid | DNA | Human immunodeficiency virus | Drug therapy
Journal Article
Journal Article
PLoS ONE, ISSN 1932-6203, 07/2014, Volume 9, Issue 7, p. e102598
Characterizing the mechanisms by which West Nile virus (WNV) causes blood-brain barrier (BBB) disruption, leukocyte infiltration into the brain and... 
MIGRATION | MATRIX METALLOPROTEINASES | TRANSMIGRATION | ENCEPHALITIS | MULTIDISCIPLINARY SCIENCES | CENTRAL-NERVOUS-SYSTEM | NECROSIS-FACTOR-ALPHA | INFECTION | ICAM-1 | EXPRESSION | T-CELLS | Leukocytes - pathology | Humans | Brain - virology | Monocytes - metabolism | Brain - metabolism | Lymphocytes - virology | Monocytes - pathology | Blood-Brain Barrier - virology | Transendothelial and Transepithelial Migration - physiology | Endothelial Cells - virology | West Nile Fever - pathology | Lymphocytes - metabolism | West Nile Fever - virology | West Nile virus - physiology | Endothelial Cells - metabolism | Cells, Cultured | West Nile Fever - metabolism | E-Selectin - metabolism | Permeability | Blood-Brain Barrier - metabolism | Monocytes - virology | Intercellular Adhesion Molecule-1 - metabolism | Lymphocytes - pathology | Cell Adhesion - physiology | Endothelium, Vascular - metabolism | Endothelium, Vascular - virology | Leukocytes - virology | Endothelium, Vascular - pathology | Endothelial Cells - pathology | In Vitro Techniques | Vascular Cell Adhesion Molecule-1 - metabolism | Leukocytes - metabolism | Brain | Encephalitis | Analysis | West Nile fever | Models | Spyware | Endothelium | Animal models | Vascular cell adhesion molecule 1 | Leukocyte migration | Neuropathology | Pathogenesis | Central nervous system | Blocking antibodies | Membrane permeability | Antibodies | Viruses | Infections | Leukocytes | VLA-4 antigen | Cell adhesion & migration | Cell adhesion molecules | Blood-brain barrier | Mac1 protein | Lymphocytes | Cell adhesion | Intercellular adhesion molecule 1 | Invasiveness | Blocking | Pharmacology | Inflammation | Adhesion | Endothelial cells | Medicine | White blood cells | E-selectin | Monocytes | Brain research | Infectious diseases | West Nile virus | Infiltration | Microvasculature | Cams | Disruption | Cell migration | Chemokines
Journal Article
Journal Article
PLoS ONE, ISSN 1932-6203, 2009, Volume 4, Issue 12, pp. e8072 - e8072
Human disease caused by highly pathogenic avian influenza (HPAI) H5N1 can lead to a rapidly progressive viral pneumonia leading to acute respiratory distress... 
A H5N1 | APOPTOSIS | INTERFERON | ACTIVATION | MULTIDISCIPLINARY SCIENCES | CYTOKINE SIGNALING-1 | INNATE IMMUNE-RESPONSE | PROLIFERATION | INFECTION | KAPPA-B | FAMILY | Influenza A Virus, H1N1 Subtype - immunology | Reproducibility of Results | Oligonucleotide Array Sequence Analysis | Monocytes - cytology | Humans | Cells, Cultured | Birds - virology | Up-Regulation - genetics | Gene Expression Profiling | Influenza in Birds - immunology | Interferon Type I - immunology | Reverse Transcriptase Polymerase Chain Reaction | Down-Regulation - genetics | Host-Pathogen Interactions - immunology | Animals | Macrophages - virology | Host-Pathogen Interactions - genetics | Time Factors | Influenza A Virus, H5N1 Subtype - immunology | Influenza in Birds - virology | Influenza, Human - virology | Seasons | Macrophages - immunology | Influenza, Human - immunology | Avian influenza | Acute respiratory distress syndrome | Genes | Swine influenza | Avian influenza viruses | Interferon | Biological response modifiers | Comparative analysis | Macrophages | Gene expression | Animal models | Pandemics | Pathogenesis | Viruses | Inflammatory response | Activation | Infections | Kinases | Avian flu | Pathways | Alveoli | Cytokines | Inflammation | Tumor necrosis factor-α | Epithelium | Biological activity | DNA microarrays | Respiratory distress syndrome | Lungs | Influenza | Apoptosis | Up-Regulation | Influenza in Birds | Down-Regulation | Birds | Influenza A Virus, H5N1 Subtype | Interferon Type I | Virology | Host-Pathogen Interactions | Life Sciences | Microbiology and Parasitology | Monocytes | Influenza, Human | Influenza A Virus, H1N1 Subtype
Journal Article