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American Journal of Physiology - Heart and Circulatory Physiology, ISSN 0363-6135, 05/2012, Volume 302, Issue 10, pp. H1953 - H1964
Journal Article
Journal Article
Free radical biology & medicine, ISSN 0891-5849, 2016, Volume 97, pp. 212 - 222
Journal Article
Journal of atherosclerosis and thrombosis, ISSN 1340-3478, 2019, Volume 26, Issue 2, pp. 183 - 197
Journal Article
The Journal of clinical investigation, ISSN 0021-9738, 2013, Volume 123, Issue 8, pp. 3600 - 3613
Dysfunctional bone morphogenetic protein receptor-2 (BMPR2) signaling is implicated in the pathogenesis of pulmonary arterial hypertension (PAH). We used a... 
MEDICINE, RESEARCH & EXPERIMENTAL | GENE | BONE MORPHOGENETIC PROTEIN | ARTERIAL-HYPERTENSION | HEART-FAILURE | IN-VIVO | APELIN | SMOOTH-MUSCLE-CELLS | TGF-BETA RECEPTOR | EXPRESSION | T-CELLS | Cell Proliferation | Humans | Microvessels - pathology | Male | Cell Hypoxia | Tacrolimus - pharmacology | Hypertension, Pulmonary - drug therapy | Endothelial Cells - physiology | Bone Morphogenetic Protein Receptors, Type II - genetics | Signal Transduction | Bone Morphogenetic Protein 4 - physiology | Endothelium, Vascular - physiopathology | Rats | Hypertension, Pulmonary - metabolism | Inhibitor of Differentiation Protein 1 - metabolism | Neointima - metabolism | Rats, Sprague-Dawley | Bone Morphogenetic Protein Receptors, Type II - metabolism | Mice, Knockout | Animals | High-Throughput Screening Assays | Neointima - drug therapy | Neointima - pathology | Tacrolimus Binding Protein 1A - metabolism | Cell Line, Tumor | Inhibitor of Differentiation Protein 1 - genetics | Endothelium, Vascular - pathology | Mice | Hypertension, Pulmonary - pathology | Smad Proteins - metabolism | Pulmonary Artery - pathology | Apoptosis | Endothelial Cells - drug effects | Hypertension | Cell receptors | Physiological aspects | Cellular signal transduction | Research | Properties | Endothelium | Proteins | Medical research | Scholarships & fellowships | Research & development--R&D | Pulmonary arteries | Rodents | Hypoxia | Ligands | Mutation | Drug dosages
Journal Article
Journal Article
Journal of cellular and molecular medicine, ISSN 1582-1838, 2017, Volume 21, Issue 8, pp. 1523 - 1531
Platelets play an important role in the pathogenesis of vascular remodelling after injury. Junctional adhesion molecule A (JAM‐A) was recently described to... 
ARTERIAL INJURY | junctional adhesion molecule | neointima | leucocyte | ATHEROSCLEROSIS | LEUKOCYTE INTEGRIN MAC-1 | COUNTERRECEPTOR | GLYCOPROTEIN IB-ALPHA | IN-VIVO | VASCULAR INFLAMMATION | MONOCYTE RECRUITMENT | platelet | JAM-A | MONONUCLEAR-CELL RECRUITMENT | MEDICINE, RESEARCH & EXPERIMENTAL | CELL BIOLOGY | Blood Platelets - pathology | Carotid Arteries - metabolism | Cell Adhesion Molecules - genetics | Hyperlipidemias - genetics | Apolipoproteins E - deficiency | Atherosclerosis - genetics | Myocytes, Smooth Muscle - pathology | Male | Monocytes - metabolism | Neointima - genetics | Carotid Artery Injuries - complications | Monocytes - pathology | Female | Carotid Artery Injuries - genetics | Myocytes, Smooth Muscle - metabolism | Carotid Artery Injuries - metabolism | Hyperlipidemias - complications | Carotid Artery Injuries - pathology | Atherosclerosis - pathology | Hyperlipidemias - metabolism | Signal Transduction | Mice, Inbred C57BL | Cell Adhesion Molecules - deficiency | Gene Expression Regulation | Atherosclerosis - complications | Cell Adhesion | Neointima - metabolism | Atherosclerosis - metabolism | Mice, Knockout | Animals | Vascular Remodeling - genetics | Blood Platelets - metabolism | Apolipoproteins E - genetics | Receptors, Cell Surface - deficiency | Neointima - pathology | Hyperlipidemias - pathology | Carotid Arteries - pathology | Mice | Neointima - complications | Receptors, Cell Surface - genetics | Apolipoproteins | Atherosclerosis | Pathogenesis | Smooth muscle | Leukocytes (mononuclear) | Wire | Macrophages | Adhesion | Recruitment | Monocytes | Clonal deletion | Blood platelets | Microscopy | Apolipoprotein E | Rodents | Arteriosclerosis | Carotid artery | Lesions | Platelets | Injuries | Injury analysis | Original
Journal Article
Journal of the Royal Society interface, ISSN 1742-5689, 2017, Volume 14, Issue 136, pp. 20170327 - 20170327
The embryonic lineage of intramural cells, microstructural organization of the extracellular matrix, local luminal and wall geometry, and haemodynamic loads... 
Smooth muscle | Inflammatory cells | Angiotensin II | Stress | Aortic stiffness | stress | MULTIDISCIPLINARY SCIENCES | INVOLVEMENT | smooth muscle | ATHEROSCLEROSIS | ANGIOTENSIN-II | MODEL | II-INDUCED HYPERTENSION | aortic stiffness | angiotensin II | inflammatory cells | GROWTH | SMOOTH-MUSCLE-CELLS | EXTRACELLULAR-MATRIX | MICE | MECHANOTRANSDUCTION | Aneurysm, Dissecting - genetics | Aneurysm, Dissecting - metabolism | Aortic Aneurysm - metabolism | Muscle, Smooth, Vascular - metabolism | Models, Cardiovascular | Aortic Aneurysm - physiopathology | Extracellular Matrix - metabolism | Male | Aneurysm, Dissecting - pathology | Muscle, Smooth, Vascular - physiopathology | Neointima - metabolism | Mice, Knockout | Extracellular Matrix - genetics | Neointima - physiopathology | Muscle, Smooth, Vascular - pathology | Neointima - genetics | Animals | Aneurysm, Dissecting - physiopathology | Fibrosis | Neointima - pathology | Mice | Aortic Aneurysm - pathology | Aortic Aneurysm - genetics | Extracellular Matrix - pathology | Hypertension | Animal models | Cyclic loads | Aneurysm | Muscles | Mechanical properties | Aneurysms | Stiffening | Muscle contraction | Biomechanics | Arteriosclerosis | Coronary vessels | Collagen | Atherosclerosis | Aorta | Extracellular matrix | Stiffness | Blood pressure | Dissection | Stretching | 1004 | Life Sciences–Engineering interface
Journal Article
Proceedings of the National Academy of Sciences - PNAS, ISSN 1091-6490, 2018, Volume 115, Issue 37, pp. E8660 - E8667
Journal Article