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smooth-muscle-cells (282) 282
myocytes, smooth muscle - metabolism (264) 264
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time factors (224) 224
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cell biology (185) 185
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carotid arteries - pathology (178) 178
cell movement (176) 176
neointima - metabolism (170) 170
transfection (170) 170
article (168) 168
cell proliferation - drug effects (168) 168
phenotype (168) 168
gene expression (164) 164
biochemistry & molecular biology (163) 163
intimal hyperplasia (162) 162
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vascular injury (152) 152
migration (151) 151
apoptosis (150) 150
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medicine, research & experimental (137) 137
phosphorylation (136) 136
gene expression regulation (135) 135
activation (133) 133
injury (130) 130
analysis (128) 128
rna interference (125) 125
carotid artery injuries - metabolism (122) 122
cardiovascular (116) 116
endothelium (116) 116
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immunohistochemistry (114) 114
carotid arteries - metabolism (112) 112
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angioplasty (107) 107
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gene therapy (103) 103
atherosclerosis - pathology (102) 102
rodents (102) 102
balloon injury (101) 101
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Journal of Molecular and Cellular Cardiology, ISSN 0022-2828, 2008, Volume 46, Issue 3, pp. 292 - 299
Abstract Macrophage-triggered chronic inflammation and smooth muscle cell-initiated vascular remodeling are two major pathophysiologic events during... 
Cardiovascular | Smooth muscle cell | Atherogenesis | CIITA | Protein degradation | MHC II | Collagen type I | HDAC2-mediated deacetylation | RFX5 | Transcriptional regulation | Macrophage | INTERFERON-GAMMA REPRESSION | CARDIAC & CARDIOVASCULAR SYSTEMS | DENDRITIC CELLS | IFN-GAMMA | NEOINTIMA FORMATION | TRICHOSTATIN-A | CELL BIOLOGY | GENE-EXPRESSION | COLLAGEN COL1A2 TRANSCRIPTION | ATHEROSCLEROTIC PLAQUE | HISTONE DEACETYLASE | Transcription, Genetic - drug effects | Atherosclerosis - genetics | Humans | Interferon-gamma - metabolism | Promoter Regions, Genetic - genetics | Repressor Proteins - antagonists & inhibitors | DNA-Binding Proteins - metabolism | Inflammation - metabolism | Collagen Type I - genetics | Inflammation - drug therapy | Trans-Activators - genetics | Interferon-gamma - genetics | Nuclear Proteins - genetics | Hydroxamic Acids - pharmacology | Myocytes, Smooth Muscle - metabolism | Repressor Proteins - metabolism | Cell Line | Atherosclerosis - drug therapy | Collagen Type I - metabolism | Histone Deacetylases - genetics | Histocompatibility Antigens Class II - biosynthesis | Repressor Proteins - genetics | Histone Deacetylase 2 | Histone Deacetylases - metabolism | Nuclear Proteins - metabolism | Transcription Factors - genetics | DNA-Binding Proteins - genetics | Regulatory Factor X Transcription Factors | Atherosclerosis - metabolism | Transcription Factors - metabolism | Leupeptins - pharmacology | Acetylation - drug effects | Macrophages - metabolism | Animals | Cysteine Proteinase Inhibitors - pharmacology | Inflammation - genetics | Trans-Activators - metabolism | Histone Deacetylase Inhibitors | Mice | Protein Processing, Post-Translational | Histocompatibility Antigens Class II - genetics | Transcription, Genetic - genetics | Medical colleges | Proteolysis | Collagen | Atherosclerosis | Macrophages | Biological response modifiers | Interferon gamma | Index Medicus
Journal Article
Arteriosclerosis, Thrombosis, and Vascular Biology, ISSN 1079-5642, 09/2016, Volume 36, Issue 9, pp. 1947 - 1961
OBJECTIVE—Key augmented processes in atherosclerosis have been identified, whereas less is known about downregulated pathways. Here, we applied a systems... 
Actin cytoskeleton | Downregulation | Smooth muscle cells | Hyperplasia | Atherosclerosis | atherosclerosis | VASCULAR DEVELOPMENT | ALPHA-ACTIN | PROTEIN | downregulation | NETWORKS | PLAQUE | actin cytoskeleton | smooth muscle cells | RAT CAROTID-ARTERY | DISEASE | GENE-EXPRESSION | SYNAPTOPODIN | hyperplasia | PERIPHERAL VASCULAR DISEASE | DIFFERENTIATION | HEMATOLOGY | Carotid Arteries - metabolism | Apolipoproteins E - deficiency | Cytoskeletal Proteins - genetics | Atherosclerosis - genetics | Humans | Middle Aged | Carotid Artery Diseases - physiopathology | Cell Dedifferentiation | Male | LIM Domain Proteins - metabolism | Autoantigens - genetics | Neointima | RNA Interference | Time Factors | Carotid Artery Diseases - pathology | Carotid Artery Injuries - genetics | Disease Models, Animal | Atherosclerosis - pathology | Actin Cytoskeleton - metabolism | Signal Transduction | Down-Regulation | Plaque, Atherosclerotic | Rats, Sprague-Dawley | Atherosclerosis - metabolism | Mice, Knockout | Phenotype | Carotid Artery Diseases - genetics | Calcium-Binding Proteins - genetics | Autoantigens - metabolism | Muscle, Smooth, Vascular - metabolism | Myocytes, Smooth Muscle - pathology | Muscle, Smooth, Vascular - physiopathology | Case-Control Studies | Transfection | Intermediate Filament Proteins - genetics | Cytoskeletal Proteins - metabolism | Microfilament Proteins - metabolism | Carotid Artery Diseases - metabolism | Microfilament Proteins - genetics | Myocytes, Smooth Muscle - metabolism | Carotid Artery Injuries - metabolism | Calcium-Binding Proteins - metabolism | Genetic Association Studies | Cells, Cultured | Vasoconstriction | Muscle, Smooth, Vascular - pathology | Animals | Carotid Arteries - physiopathology | Apolipoproteins E - genetics | Adaptor Proteins, Signal Transducing - genetics | LIM Domain Proteins - genetics | Carotid Arteries - pathology | Adaptor Proteins, Signal Transducing - metabolism | Intermediate Filament Proteins - metabolism | Index Medicus
Journal Article
Atherosclerosis, ISSN 0021-9150, 10/2017, Volume 265, pp. 133 - 139
The factor VII activating protease (FSAP) knockout mice have a bigger neointima after vascular injury and a larger infarct volume after stroke. The Marburg I... 
Atherosclerosis | Protease activated receptors | Inflammation | Gene expression | FSAP | Thrombosis | HABP2 | CARDIAC & CARDIOVASCULAR SYSTEMS | RECEPTOR | RISK | NEOINTIMA FORMATION | PROTEOLYSIS | INDUCTION | IL-6 | INTERLEUKIN-6 EXPRESSION | PERIPHERAL VASCULAR DISEASE | MARBURG-I POLYMORPHISM | PROMOTES | Inflammation - pathology | Interleukin-8 - genetics | Receptor, PAR-1 - metabolism | Humans | Apoptosis - genetics | Gene Expression Profiling | Time Factors | Cyclooxygenase 2 - genetics | E-Selectin - genetics | Inflammation Mediators - metabolism | Serine Endopeptidases - genetics | Amphiregulin - metabolism | Vascular Cell Adhesion Molecule-1 - genetics | Interleukin-8 - metabolism | Interleukin-6 - metabolism | Amphiregulin - genetics | Cell Proliferation - genetics | Interleukin-6 - genetics | Signal Transduction | Myocytes, Smooth Muscle - enzymology | Cells, Cultured | Gene Expression Regulation | E-Selectin - metabolism | Receptor, PAR-1 - genetics | Muscle, Smooth, Vascular - pathology | Human Umbilical Vein Endothelial Cells - enzymology | Cyclooxygenase 2 - metabolism | Inflammation - genetics | Serine Endopeptidases - metabolism | Vascular Cell Adhesion Molecule-1 - metabolism | Inflammation - enzymology | Muscle, Smooth, Vascular - enzymology | Single nucleotide polymorphisms | Proteases | Genes | Endothelium | Anopheles | Platelet-derived growth factor | Analysis | Genetic research | Smooth muscle | Fibroblast growth factors | Index Medicus
Journal Article
Gene Therapy, ISSN 0969-7128, 03/2013, Volume 20, Issue 3, pp. 248 - 254
Targeting diseased cells is a challenging issue in both pharmacological and biological therapeutics. Gene therapy is emerging as a novel approach for treating... 
MEDICINE, RESEARCH & EXPERIMENTAL | calcium | proliferation | BIOCHEMISTRY & MOLECULAR BIOLOGY | HEART-FAILURE | TRANSCRIPTION | MUSCLE-CELL PROLIFERATION | NEOINTIMA FORMATION | SERCA2a | vascular | NFAT | SMOOTH-MUSCLE | BIOTECHNOLOGY & APPLIED MICROBIOLOGY | CARDIAC-HYPERTROPHY | RETICULUM CA2+-ATPASE | GENETICS & HEREDITY | CALCINEURIN | regulated promoter | EXPRESSION | Luciferases - metabolism | Rats, Wistar | Calcium - metabolism | Humans | Endoplasmic Reticulum - metabolism | Male | Promoter Regions, Genetic - genetics | Luciferases - genetics | Cattle | Adenoviridae - genetics | Serum | Carotid Artery Injuries - genetics | Myocytes, Smooth Muscle - metabolism | Cytomegalovirus - genetics | Sarcoplasmic Reticulum Calcium-Transporting ATPases - metabolism | Cells, Cultured | Rats | Muscle, Smooth, Vascular - cytology | Genetic Vectors - genetics | Carotid Artery Injuries - therapy | Gene Expression Regulation - drug effects | Microscopy, Confocal | Animals | Culture Media - pharmacology | Cell Proliferation - drug effects | Sarcoplasmic Reticulum Calcium-Transporting ATPases - genetics | Genetic Therapy - methods | NFATC Transcription Factors - genetics | Promoters (Genetics) | Calcium, Dietary | Cytomegaloviruses | Viral genetics | Physiological aspects | Genetic aspects | Research | Gene therapy | T cells | Health aspects | Proteins | Cell growth | Calcium | Gene expression | Veins & arteries | Index Medicus
Journal Article
Free Radical Biology and Medicine, ISSN 0891-5849, 08/2016, Volume 97, pp. 212 - 222
Journal Article
Circulation, ISSN 0009-7322, 10/2014, Volume 130, Issue 17, pp. 1452 - 1465
Journal Article
Arteriosclerosis, Thrombosis, and Vascular Biology, ISSN 1079-5642, 03/2015, Volume 35, Issue 3, pp. 547 - 557
OBJECTIVE—Atherosclerosis, an inflammatory disease of arterial vessel walls, requires migration and matrix metalloproteinase (MMP)-9–dependent invasion of... 
Inflammation | Migration and invasion protein | Atherosclerosis | Serum-glucocorticoid regulated kinase | atherosclerosis | migration and invasion protein | APOE-NULL MICE | ATHEROSCLEROTIC PLAQUES | INCREASED EXPRESSION | PATHOGENIC T(H)17 CELLS | MONOCYTE CHEMOATTRACTANT PROTEIN-1 | NEOINTIMA FORMATION | MATRIX METALLOPROTEINASE-3 | inflammation | MUSCLE-CELL MIGRATION | PERIPHERAL VASCULAR DISEASE | serum-glucocorticoid regulated kinase | HEMATOLOGY | NF-KAPPA-B | E-DEFICIENT MICE | Inflammation - pathology | Protein-Serine-Threonine Kinases - deficiency | Apolipoproteins E - deficiency | Atherosclerosis - genetics | Humans | Male | Immediate-Early Proteins - metabolism | Matrix Metalloproteinase 9 - metabolism | Carotid Artery Diseases - pathology | Matrix Metalloproteinase 9 - genetics | Peritonitis - genetics | Carotid Arteries - enzymology | Immediate-Early Proteins - deficiency | Transcription, Genetic | Protein-Serine-Threonine Kinases - metabolism | Disease Models, Animal | Atherosclerosis - pathology | Macrophages - pathology | NF-kappa B p50 Subunit - metabolism | Signal Transduction | Matrix Metalloproteinase 2 - metabolism | Plaque, Atherosclerotic | Macrophages - enzymology | Mice, Knockout | Aorta - pathology | Aortic Diseases - genetics | Peritonitis - enzymology | Immediate-Early Proteins - genetics | Carotid Artery Diseases - enzymology | Carotid Artery Diseases - genetics | Mutation | Inflammation - enzymology | Vascular Remodeling | I-kappa B Proteins - metabolism | Atherosclerosis - enzymology | Transfection | I-kappa B Kinase - metabolism | Thioglycolates | Aortic Diseases - enzymology | Active Transport, Cell Nucleus | Aorta - enzymology | Aortic Diseases - pathology | Cell Line | Mice, Inbred C57BL | Protein-Serine-Threonine Kinases - genetics | Peritonitis - chemically induced | Chemotaxis | Gene Expression Regulation, Enzymologic | Animals | Apolipoproteins E - genetics | Inflammation - genetics | Carotid Arteries - pathology | Index Medicus
Journal Article
Journal of Biological Chemistry, ISSN 0021-9258, 08/2015, Volume 290, Issue 32, pp. 19641 - 19652
The JAK-STAT3 signaling pathway is one of the critical pathways regulating cell proliferation, differentiation, and apoptosis. Myocardin is regarded as a key... 
SIGNALING PATHWAYS | DEPENDENT TRANSCRIPTION | INDUCED APOPTOSIS | BIOCHEMISTRY & MOLECULAR BIOLOGY | SERUM RESPONSE FACTOR | GENE-EXPRESSION | NEOINTIMA FORMATION | DIFFERENTIATION | VEGF-A | TUBE FORMATION | ENDOTHELIAL GROWTH-FACTOR | Serum Response Factor - genetics | Cell Proliferation | Muscle, Smooth, Vascular - metabolism | Muscle Contraction - genetics | Humans | Janus Kinases - metabolism | Vascular Endothelial Growth Factor A - metabolism | Vascular Endothelial Growth Factor A - genetics | Mitogen-Activated Protein Kinase 1 - genetics | Trans-Activators - genetics | Cell Differentiation | Myocytes, Smooth Muscle - drug effects | Nuclear Proteins - genetics | Myocytes, Smooth Muscle - cytology | Myocytes, Smooth Muscle - metabolism | Vascular Endothelial Growth Factor A - pharmacology | STAT3 Transcription Factor - genetics | STAT3 Transcription Factor - metabolism | Muscle, Smooth, Vascular - drug effects | Cell Line | Janus Kinases - genetics | Mitogen-Activated Protein Kinase 3 - genetics | Signal Transduction | Serum Response Factor - metabolism | Gene Expression Regulation | Nuclear Proteins - metabolism | Muscle, Smooth, Vascular - cytology | Feedback, Physiological | Phenotype | Mitogen-Activated Protein Kinase 3 - metabolism | Trans-Activators - metabolism | Mitogen-Activated Protein Kinase 1 - metabolism | Index Medicus | cardiovascular disease | transcription factor | transcription regulation | cardiovascular | cell signaling | Cell Biology
Journal Article
Arteriosclerosis, Thrombosis, and Vascular Biology, ISSN 1079-5642, 01/2015, Volume 35, Issue 1, pp. 175 - 183
OBJECTIVE—Vascular calcification is a characteristic feature of atherosclerosis, diabetes mellitus, and end-stage renal disease. We have demonstrated that... 
PTEN | AKT | FOXO1/3 | ubiquitination | vascular calcification | Runx2 | TRANSCRIPTION FACTORS | OSTEOBLAST DIFFERENTIATION | TUMOR-SUPPRESSOR GENE | NEOINTIMA FORMATION | INACTIVATION | PROSTATE-CANCER | PERIPHERAL VASCULAR DISEASE | SMOOTH-MUSCLE-CELLS | HEMATOLOGY | Phosphorylation | Apolipoproteins E - deficiency | Muscle, Smooth, Vascular - metabolism | Atherosclerosis - genetics | Vascular Calcification - genetics | Myocytes, Smooth Muscle - pathology | Aorta - metabolism | Core Binding Factor Alpha 1 Subunit - metabolism | Aortic Diseases - metabolism | Vascular Calcification - metabolism | Ubiquitination | Transfection | RNA Interference | Time Factors | Forkhead Transcription Factors - metabolism | Proto-Oncogene Proteins c-akt - metabolism | Microfilament Proteins - genetics | Myocytes, Smooth Muscle - metabolism | Aortic Diseases - pathology | Disease Models, Animal | PTEN Phosphohydrolase - genetics | Atherosclerosis - pathology | PTEN Phosphohydrolase - deficiency | Signal Transduction | Cells, Cultured | Gene Expression Regulation | Genotype | Forkhead Transcription Factors - genetics | Vascular Calcification - pathology | Atherosclerosis - metabolism | Mice, Knockout | Protein Transport | Aorta - pathology | Muscle Proteins - genetics | Aortic Diseases - genetics | Muscle, Smooth, Vascular - pathology | Phenotype | Animals | Apolipoproteins E - genetics | Forkhead Box Protein O1 | Enzyme Activation | Forkhead Box Protein O3 | Integrases - genetics | Index Medicus | Vascular calcification | FOXO1
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Journal Article