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Journal Article
Journal of Translational Medicine, ISSN 1479-5876, 11/2012, Volume 10, Issue 1, pp. 230 - 230
Background: The mechanism regarding rapid progression of residual hepatocellular carcinoma (HCC) after insufficient radiofrequency ablation (RFA) has been... 
Radiofrequency ablation | Hepatocellular carcinoma | Metastasis | Invasiveness | Tumor-associated endothelial cells | MEDICINE, RESEARCH & EXPERIMENTAL | SELECTIN | COLORECTAL LIVER METASTASES | CANCER | ADHESION | INVASION | SERUM-LEVELS | GROWTH | COMPLICATIONS | RAPID PROGRESSION | EXPRESSION | Neoplasm, Residual - pathology | Up-Regulation | Cell Proliferation | Humans | Catheter Ablation | NF-kappa B - metabolism | Extracellular Signal-Regulated MAP Kinases - metabolism | Neovascularization, Pathologic - pathology | Cell Shape | Hyperthermia, Induced | Carcinoma, Hepatocellular - blood supply | Liver Neoplasms - pathology | Liver Neoplasms - enzymology | Proto-Oncogene Proteins c-akt - metabolism | STAT3 Transcription Factor - metabolism | Green Fluorescent Proteins - metabolism | Reproducibility of Results | Signal Transduction | Cell Separation | Neoplasm Invasiveness | Cell Communication | Carcinoma, Hepatocellular - surgery | Liver Neoplasms - surgery | Carcinoma, Hepatocellular - enzymology | Neoplasm, Residual - enzymology | Cell Adhesion | Liver Neoplasms - blood supply | Carcinoma, Hepatocellular - pathology | Cell Line, Tumor | Endothelial Cells - pathology | Endothelial Cells - enzymology | Development and progression | Ablation (Surgery) | Hepatoma | Low density lipoproteins | Analysis | Endothelium | Proteins | Liver cancer | Tumors | Cancer
Journal Article
European Journal of Cancer, ISSN 0959-8049, 2017, Volume 79, pp. 214 - 225
Abstract Background High-grade serous ovarian cancer (HGSOC) causes 80% of all ovarian cancer (OC) deaths. In this setting, the role of cancer stem-like cells... 
Hematology, Oncology and Palliative Medicine | BRCA | Prognosis | ALDH1 | Cancer stem-like cell | Survival | Aldehyde dehydrogenase-1 | Ovarian cancer | CD133 | BRCA2 MUTATIONS | METAANALYSIS | MARKER | ALDEHYDE DEHYDROGENASE 1 | CHEMOTHERAPY | ONCOLOGY | POOR-PROGNOSIS | CLINICAL-TRIALS | ALDH1 EXPRESSION | CARCINOMA | Neoplasm, Residual - pathology | Humans | Middle Aged | Retinal Dehydrogenase - metabolism | Ovarian Neoplasms - pathology | Antineoplastic Agents - therapeutic use | Ovarian Neoplasms - mortality | Neoplasm Recurrence, Local - mortality | Neoplasm Recurrence, Local - enzymology | Neoplasm Recurrence, Local - pathology | Platinum Compounds - therapeutic use | Neoplasms, Glandular and Epithelial - mortality | Isoenzymes - metabolism | Biomarkers, Tumor - metabolism | Neoplastic Stem Cells - pathology | Adult | Female | Neoplasm, Residual - mortality | Neoplasms, Glandular and Epithelial - pathology | Ubiquitin-Protein Ligases - metabolism | Neoplasm, Residual - enzymology | Ovarian Neoplasms - enzymology | Neoplasms, Glandular and Epithelial - enzymology | BRCA2 Protein - metabolism | AC133 Antigen - metabolism | Survival Analysis | Carcinoma, Ovarian Epithelial | Aged | Neoplastic Stem Cells - enzymology | Clonal Evolution | Antimitotic agents | Care and treatment | Relapse | Oncology, Experimental | Analysis | Research | Antineoplastic agents | Aldehydes | Health aspects | Cancer | Diseases
Journal Article
Nature Medicine, ISSN 1078-8956, 07/2012, Volume 18, Issue 7, pp. 1052 - 1059
Neoadjuvant chemotherapy (NAC) induces a pathological complete response (pCR) in similar to 30% of patients with breast cancer. However, many patients have... 
MEDICINE, RESEARCH & EXPERIMENTAL | STEM-CELLS | METASTASIS | PREDICTOR | BIOCHEMISTRY & MOLECULAR BIOLOGY | KINASE | CELL BIOLOGY | EPITHELIAL-MESENCHYMAL TRANSITION | MEK | INHIBITION | GENE-EXPRESSION | RECURRENCE | KI67 | Tissue Banks | Humans | Gene Expression Regulation, Neoplastic | ras Proteins - metabolism | Ki-67 Antigen - metabolism | Gene Expression Profiling | Extracellular Signal-Regulated MAP Kinases - metabolism | RNA, Messenger - metabolism | Gene Knockdown Techniques | Neoplasm, Residual | Paraffin Embedding | Mitogen-Activated Protein Kinase Kinases - metabolism | Breast Neoplasms - enzymology | MAP Kinase Signaling System - genetics | Dual-Specificity Phosphatases - genetics | Female | Neoadjuvant Therapy | Tissue Fixation | Genes, Neoplasm - genetics | Breast Neoplasms - classification | Cell Survival | Dual-Specificity Phosphatases - metabolism | RNA, Messenger - genetics | Mitogen-Activated Protein Kinase Kinases - antagonists & inhibitors | Mitogen-Activated Protein Kinase Phosphatases - deficiency | Treatment Outcome | Breast Neoplasms - drug therapy | Mitogen-Activated Protein Kinase Phosphatases - genetics | Dual-Specificity Phosphatases - deficiency | Drug Resistance, Neoplasm - genetics | Animals | Breast Neoplasms - genetics | Mice | Enzyme Activation | Apoptosis | Mitogen-Activated Protein Kinase Phosphatases - metabolism | Adjuvant treatment | Breast cancer | Research | Drug resistance | Drug therapy | Health aspects | Cancer | Chemotherapy
Journal Article
Blood, ISSN 0006-4971, 01/2013, Volume 121, Issue 3, pp. 485 - 488
Journal Article
Journal Article
Pediatric Blood & Cancer, ISSN 1545-5009, 06/2019, Volume 66, Issue 6, pp. e27681 - n/a
Journal Article
Journal Article
Histochemistry and Cell Biology, ISSN 0948-6143, 11/2010, Volume 134, Issue 5, pp. 469 - 481
Journal Article
Journal of Clinical Oncology, ISSN 0732-183X, 01/2017, Volume 35, Issue 3, pp. 298 - 305
Journal Article
BMC Cancer, ISSN 1471-2407, 05/2014, Volume 14, Issue 1, p. 317
Journal Article