Articles

Search Articles

Advanced search
Help

Catalogue

Articles

Databases

X
Search Filters
Format Format
Format Format
X
Sort by Item Count (A-Z)
Filter by Count
Journal Article (20332) 20332
Book Review (5349) 5349
Publication (1731) 1731
Book / eBook (318) 318
Book Chapter (144) 144
Conference Proceeding (130) 130
Dissertation (13) 13
Web Resource (13) 13
Magazine Article (5) 5
Data Set (1) 1
Journal / eJournal (1) 1
Paper (1) 1
Reference (1) 1
more...
Subjects Subjects
Subjects Subjects
X
Sort by Item Count (A-Z)
Filter by Count
index medicus (19720) 19720
humans (15758) 15758
animals (9833) 9833
female (8004) 8004
male (6672) 6672
cancer (5621) 5621
mice (4599) 4599
oncology (4573) 4573
middle aged (4028) 4028
adult (3261) 3261
neoplasms - physiopathology (3160) 3160
aged (3109) 3109
rats (2594) 2594
research (2585) 2585
medicine, research & experimental (2137) 2137
tumors (2067) 2067
cell line, tumor (2011) 2011
expression (1776) 1776
analysis (1718) 1718
cell biology (1672) 1672
care and treatment (1624) 1624
metastasis (1603) 1603
surgery (1469) 1469
abridged index medicus (1446) 1446
apoptosis (1420) 1420
neoplasms, experimental - physiopathology (1405) 1405
medicine & public health (1397) 1397
oncology, experimental (1352) 1352
breast cancer (1272) 1272
breast neoplasms - physiopathology (1264) 1264
health aspects (1248) 1248
risk factors (1226) 1226
models, biological (1223) 1223
biochemistry & molecular biology (1222) 1222
neoplasm transplantation (1215) 1215
article (1141) 1141
chemotherapy (1139) 1139
breast-cancer (1112) 1112
disease models, animal (1110) 1110
time factors (1105) 1105
treatment outcome (1081) 1081
prognosis (1070) 1070
neurosciences (1032) 1032
angiogenesis (1025) 1025
signal transduction (987) 987
cells (984) 984
tumor cells, cultured (954) 954
aged, 80 and over (949) 949
growth (943) 943
disease progression (940) 940
carcinoma (916) 916
brain neoplasms - physiopathology (909) 909
physiological aspects (909) 909
medicine (892) 892
radiology, nuclear medicine & medical imaging (887) 887
medical research (883) 883
neoplasms - pathology (877) 877
pathology (872) 872
development and progression (859) 859
neoplasms - drug therapy (845) 845
immunohistochemistry (830) 830
cell proliferation (827) 827
lung neoplasms - physiopathology (826) 826
proteins (826) 826
therapy (821) 821
neoplasm metastasis - physiopathology (817) 817
pharmacology & pharmacy (799) 799
clinical neurology (795) 795
gene expression (793) 793
research article (778) 778
radiotherapy (769) 769
cell line (768) 768
in-vivo (736) 736
endocrinology & metabolism (735) 735
mice, nude (733) 733
gastroenterology & hepatology (714) 714
adolescent (712) 712
neoplasm metastasis (711) 711
biology (710) 710
neovascularization, pathologic - physiopathology (706) 706
retrospective studies (695) 695
antineoplastic agents - therapeutic use (684) 684
mammary neoplasms, experimental - physiopathology (682) 682
gene expression regulation, neoplastic (681) 681
genetic aspects (674) 674
neoplasms (666) 666
mice, inbred c57bl (639) 639
liver neoplasms - physiopathology (638) 638
survival (631) 631
signal transduction - physiology (620) 620
antineoplastic agents - pharmacology (619) 619
physiology (616) 616
inflammation (614) 614
diagnosis (608) 608
quality of life (606) 606
multidisciplinary sciences (600) 600
gene-expression (599) 599
prostatic neoplasms - physiopathology (597) 597
cells, cultured (592) 592
activation (584) 584
more...
Library Location Library Location
Library Location Library Location
X
Sort by Item Count (A-Z)
Filter by Count
Gerstein Science - Stacks (259) 259
Online Resources - Online (17) 17
Collection Dvlpm't (Acquisitions) - Closed Orders (16) 16
Collection Dvlpm't (Acquisitions) - Vendor file (6) 6
St. Michael's Hospital - Stacks (4) 4
Mt Sinai Hospital - Pathology (3) 3
Sunnybrook Health Sciences Centre - Sunnybrook Stacks (3) 3
UofT at Scarborough - Stacks (3) 3
Credit Valley Hospital - Stacks (2) 2
Lakeridge Health Sciences - Oshawa (2) 2
Markham Stouffville Hospital - Stacks (2) 2
UofT at Mississauga - Stacks (2) 2
Chemistry (A D Allen) - Stacks (1) 1
Credit Valley Hospital - Reference (1) 1
Faculty of Information - Subject Analysis Systems (1) 1
Scarborough Hospital - General (1) 1
Toronto East General Hospital - Online (1) 1
West Park Healthcare Centre - Stacks (1) 1
more...
Language Language
Language Language
X
Sort by Item Count (A-Z)
Filter by Count
English (19957) 19957
Russian (269) 269
French (162) 162
German (105) 105
Japanese (70) 70
Chinese (48) 48
Polish (41) 41
Italian (16) 16
Spanish (14) 14
Czech (7) 7
Hungarian (3) 3
Portuguese (3) 3
Croatian (2) 2
Dutch (2) 2
Bulgarian (1) 1
Hebrew (1) 1
Korean (1) 1
Lithuanian (1) 1
Norwegian (1) 1
Romanian (1) 1
Serbian (1) 1
Slovak (1) 1
Swedish (1) 1
Ukrainian (1) 1
more...
Publication Date Publication Date
Click on a bar to filter by decade
Slide to change publication date range


1. Full Text Normalization of tumour blood vessels improves the delivery of nanomedicines in a size-dependent manner
by Chauhan, Vikash P and Stylianopoulos, Triantafyllos and Martin, John D and PopoviÄ, Zoran and Chen, Ou and Kamoun, Walid S and Bawendi, Moungi G and Fukumura, Dai and Jain, Rakesh K
Nature Nanotechnology, ISSN 1748-3387, 2012, Volume 7, Issue 6, pp. 383 - 388
The blood vessels of cancerous tumours are leaky(1-3) and poorly organized(4-7). This can increase the interstitial fluid pressure inside tumours and reduce... 
VASCULAR NORMALIZATION | ANTIANGIOGENIC THERAPY | TRANSPORT | SOLID TUMORS | FLUID-FLOW | ANGIOGENESIS | MATERIALS SCIENCE, MULTIDISCIPLINARY | GROWTH | PENETRATION | NANOSCIENCE & NANOTECHNOLOGY | CANCER | GLIOBLASTOMA PATIENTS | Angiogenesis Inhibitors - pharmacology | Mammary Neoplasms, Experimental - physiopathology | Mammary Neoplasms, Experimental - metabolism | Mice, SCID | Nanoparticles | Animals | Receptors, Vascular Endothelial Growth Factor - metabolism | Nanomedicine - methods | Mammary Neoplasms, Experimental - pathology | Receptors, Vascular Endothelial Growth Factor - antagonists & inhibitors | Female | Mammary Neoplasms, Experimental - blood supply | Mice | Drug Delivery Systems - methods | Mammary Neoplasms, Experimental - drug therapy | Index Medicus
Journal Article
Details down arrow
Digital rights down arrow
loading Loading Rights
2. Full Text The α2β1 integrin is a metastasis suppressor in mouse models and human cancer
by Ramirez, Norma E and Zhang, Zhonghua and Madamanchi, Aasakiran and Boyd, Kelli L and O'Rear, Lynda D and Nashabi, Abudi and Li, Zhengzi and Dupont, William D and Zijlstra, Andries and Zutter, Mary M
Journal of Clinical Investigation, ISSN 0021-9738, 01/2011, Volume 121, Issue 1, pp. 226 - 237
Integrins regulate cell-cell and cell-matrix adhesion and thereby play critical roles in tumor progression and metastasis. Although work in preclinical models... 
Prognosis | Humans | Male | Mammary Neoplasms, Experimental - physiopathology | DNA Primers - genetics | Prostatic Neoplasms - physiopathology | Breast Neoplasms - physiopathology | Mammary Neoplasms, Experimental - genetics | Prostatic Neoplasms - genetics | Neoplasm Metastasis - prevention & control | Base Sequence | Tumor Suppressor Proteins - genetics | Mammary Neoplasms, Experimental - pathology | Female | Prostatic Neoplasms - pathology | Tumor Stem Cell Assay | Neoplasm Invasiveness | Kaplan-Meier Estimate | Integrin alpha2beta1 - physiology | Mice, Transgenic | Biomarkers, Tumor - physiology | Mice, Knockout | Neoplasm Metastasis - genetics | Tumor Suppressor Proteins - physiology | Animals | Breast Neoplasms - genetics | Integrin alpha2beta1 - deficiency | Breast Neoplasms - pathology | Genes, erbB-2 | Neoplasm Metastasis - pathology | Neoplasm Metastasis - physiopathology | Biomarkers, Tumor - genetics | Mice | Integrin alpha2beta1 - genetics | In Vitro Techniques | Index Medicus | Abridged Index Medicus
Journal Article
Details down arrow
Digital rights down arrow
loading Loading Rights
3. Full Text The Six1 homeoprotein induces human mammary carcinoma cells to undergo epithelial-mesenchymal transition and metastasis in mice through increasing TGF-β signaling
by Micalizzi, Douglas S and Christensen, Kimberly L and Jedlicka, Paul and Coletta, Ricardo D and Barón, Anna E and Harrell, J. Chuck and Horwitz, Kathryn B and Billheimer, Dean and Heichman, Karen A and Welm, Alana L and Schiemann, William P and Ford, Heide L
Journal of Clinical Investigation, ISSN 0021-9738, 09/2009, Volume 119, Issue 9, pp. 2678 - 2690
Inappropriate activation of developmental pathways is a well-recognized tumor-promoting mechanism. Here we show that overexpression of the homeoprotein Six 1,... 
BREAST-CANCER | SURVIVAL | MEDICINE, RESEARCH & EXPERIMENTAL | CDNA ARRAY | OVEREXPRESSION | GROWTH-FACTOR-BETA | TUMOR PROGRESSION | MYOGENESIS | GENE-EXPRESSION PROFILES | TREATMENT RESPONSE | INSIGHTS | Neoplasm Transplantation | Prognosis | Humans | Mammary Neoplasms, Experimental - physiopathology | Transplantation, Heterologous | Smad3 Protein - metabolism | Breast Neoplasms - physiopathology | Mammary Neoplasms, Experimental - genetics | Mammary Neoplasms, Experimental - pathology | Female | Epithelium - pathology | Signal Transduction | Transforming Growth Factor beta - physiology | Mice, SCID | Homeodomain Proteins - genetics | Animals | Breast Neoplasms - genetics | Breast Neoplasms - pathology | Mice, Nude | Neoplasm Metastasis - pathology | Neoplasm Metastasis - physiopathology | Cell Line, Tumor | Mice, Inbred NOD | Mice | Homeodomain Proteins - physiology | Mesoderm - pathology | Index Medicus | Abridged Index Medicus
Journal Article
Details down arrow
Digital rights down arrow
loading Loading Rights
4. Full Text Endothelial podosome rosettes regulate vascular branching in tumour angiogenesis
by Seano, Giorgio and Seano, Giorgio and Chiaverina, Giulia and Gagliardi, Paolo Armando and Di Blasio, Laura and Puliafito, Alberto and Bouvard, Claire and Sessa, Roberto and Tarone, Guido and Sorokin, Lydia and Helley, Dominique and Jain, Rakesh K and Serini, Guido and Bussolino, Federico and Primo, Luca
Nature cell biology, ISSN 1465-7392, 10/2014, Volume 16, Issue 10, pp. 8 - 941
The mechanism by which angiogenic endothelial cells break the physical barrier of the vascular basement membrane and consequently sprout to form new vessels in... 
F-ACTIN | INVADOPODIA | TIP CELLS | INTEGRIN | BLOOD-VESSELS | EXTRACELLULAR-MATRIX | MICE | GROWTH-FACTOR | TRANSFORMED FIBROBLASTS | BASEMENT-MEMBRANE | CELL BIOLOGY | Neoplasms - metabolism | Cell Membrane Structures - physiology | Tetradecanoylphorbol Acetate - pharmacology | Humans | Lung Neoplasms - metabolism | Male | Melanoma, Experimental - metabolism | Green Fluorescent Proteins - genetics | Vascular Endothelial Growth Factor A - metabolism | Lung Neoplasms - physiopathology | Integrin alpha6beta1 - metabolism | Melanoma, Experimental - physiopathology | Cell Membrane Structures - metabolism | RNA Interference | HEK293 Cells | Integrin alpha6beta1 - genetics | Neovascularization, Pathologic - physiopathology | Neoplasms - physiopathology | Vascular Endothelial Growth Factor A - pharmacology | Endothelial Cells - physiology | Lung Neoplasms - blood supply | Green Fluorescent Proteins - metabolism | Cell Membrane Structures - drug effects | Melanoma, Experimental - blood supply | Endothelial Cells - metabolism | Mice, Inbred C57BL | Cells, Cultured | Neoplasms - blood supply | Matrix Metalloproteinase 14 - metabolism | Basement Membrane - metabolism | Mice, Knockout | Microscopy, Confocal | Animals | Cell Line, Tumor | Neovascularization, Pathologic - metabolism | HeLa Cells | Laminin - metabolism | Microscopy, Fluorescence | Endothelial Cells - drug effects | Blood vessels | Physiological aspects | Cell research | Neovascularization | Research | Index Medicus
Journal Article
Details down arrow
Digital rights down arrow
loading Loading Rights
5. Full Text S1PR1 on tumor-associated macrophages promotes lymphangiogenesis and metastasis via NLRP3/IL-1 beta
by Weichand, B and Popp, R and Dziumbla, S and Mora, J and Strack, E and Elwakeel, E and Frank, AC and Scholich, K and Pierre, S and Syed, SN and Olesch, C and Ringleb, J and Oren, B and Doring, C and Savai, R and Jung, M and von Knethen, A and Levkau, B and Fleming, I and Weigert, A and Brune, B
JOURNAL OF EXPERIMENTAL MEDICINE, ISSN 0022-1007, 09/2017, Volume 214, Issue 9, pp. 2695 - 2713
Metastasis is the primary cause of cancer death. The inflammatory tumor microenvironment contributes to metastasis, for instance, by recruiting blood and lymph... 
MEDICINE, RESEARCH & EXPERIMENTAL | CELLS | ACTIVATION | CARCINOGENESIS | ANGIOGENESIS | INFLAMMATION | SPHINGOSINE-1-PHOSPHATE | GROWTH | INFLAMMASOMES | IMMUNOLOGY | CANCER | EXPRESSION | Macrophages - physiology | Interleukin-1beta - physiology | Fibrosarcoma - physiopathology | Humans | Mice, Inbred C57BL | Lymphangiogenesis - physiology | Mammary Neoplasms, Experimental - physiopathology | Receptors, Lysosphingolipid - physiology | Lymphatic Metastasis | Breast Neoplasms - physiopathology | Animals | Breast Neoplasms - pathology | Neoplasm Metastasis - physiopathology | Female | Mice | NLR Family, Pyrin Domain-Containing 3 Protein - physiology | CD11b antigen | Carcinoma | Cell survival | Tumor-infiltrating lymphocytes | Blood vessels | Fibrosarcoma | Inflammation | Metastasis | Gene expression | Macrophages | IL-1β | Lymph nodes | Metastases | Immune systems | Angiogenesis | Signaling | Cell activation | Clonal deletion | Deletion | Mammary gland | Tumors | Cancer | Lymph | Index Medicus | 314 | 307 | 311
Journal Article
Details down arrow
Digital rights down arrow
loading Loading Rights
6. Full Text Perk-Dependent Translational Regulation Promotes Tumor Cell Adaptation and Angiogenesis in Response to Hypoxic Stress
by Jaime D. Blais and Christina L. Addison and Robert Edge and Theresa Falls and Huijun Zhao and Kishore Wary and Costas Koumenis and Heather P. Harding and David Ron and Martin Holcik and John C. Bell
Molecular and Cellular Biology, ISSN 0270-7306, 12/2006, Volume 26, Issue 24, pp. 9517 - 9532
Article Usage Stats Services MCB Citing Articles Google Scholar PubMed Related Content Social Bookmarking CiteULike Delicious Digg Facebook Google+ Mendeley... 
CANCER-CELLS | MESSENGER-RNA TRANSLATION | UNFOLDED-PROTEIN RESPONSE | ELEMENT-MEDIATED EXPRESSION | INDUCIBLE FACTOR-I | BIOCHEMISTRY & MOLECULAR BIOLOGY | GENE-EXPRESSION | TISSUE INHIBITOR | EXTRACELLULAR-MATRIX | UP-REGULATION | INTERNAL-RIBOSOME-ENTRY | CELL BIOLOGY | Fibroblasts - enzymology | Protein Biosynthesis | Oligonucleotide Array Sequence Analysis | Humans | Neoplasms, Experimental - enzymology | Gene Expression Profiling | eIF-2 Kinase - deficiency | Neovascularization, Pathologic - enzymology | Neoplasms, Experimental - genetics | Neovascularization, Pathologic - physiopathology | Neoplasms, Experimental - physiopathology | eIF-2 Kinase - genetics | Hypoxia - enzymology | Stress, Physiological - genetics | Stress, Physiological - physiopathology | Stress, Physiological - enzymology | HT29 Cells | Hypoxia - genetics | Animals | eIF-2 Kinase - physiology | Mice, Nude | Hypoxia - physiopathology | Neovascularization, Pathologic - genetics | Mice | Cell Line, Transformed | Index Medicus
Journal Article
Details down arrow
Digital rights down arrow
loading Loading Rights
7. Full Text Hepatitis B virus X (HBx) induces tumorigenicity of hepatic progenitor cells in 3,5‐diethoxycarbonyl‐1,4‐dihydrocollidine‐treated HBx transgenic mice
by Wang, Chao and Yang, Wen and Yan, He‐Xin and Luo, Tao and Zhang, Jian and Tang, Liang and Wu, Fu‐Quan and Zhang, Hui‐Lu and Yu, Le‐Xing and Zheng, Long‐Yi and Li, Yu‐Qiong and Dong, Wei and He, Ya‐Qin and Liu, Qiong and Zou, Shan‐Shan and Lin, Yan and Hu, Liang and Li, Zhong and Wu, Meng‐Chao and Wang, Hong‐Yang
Hepatology, ISSN 0270-9139, 01/2012, Volume 55, Issue 1, pp. 108 - 120
Hepatitis B virus X (HBx) protein is implicated in hepatitis B virus (HBV)‐associated liver carcinogenesis. However, it remains unclear whether HBx‐expressing... 
LIVER-CANCER | HEPATOCELLULAR-CARCINOMA CELLS | PROTEIN | RAT | DISEASE | CATENIN | STEM/PROGENITOR CELLS | MOUSE-LIVER | OVAL CELLS | GASTROENTEROLOGY & HEPATOLOGY | CANCER STEM-CELLS | Bile Duct Neoplasms - chemically induced | Carcinoma, Hepatocellular - chemically induced | Cholangiocarcinoma - chemically induced | Humans | Middle Aged | Liver Neoplasms, Experimental - chemically induced | Male | Cell Transformation, Neoplastic - chemically induced | Cholangiocarcinoma - virology | Bile Duct Neoplasms - physiopathology | Liver Neoplasms, Experimental - physiopathology | Trans-Activators - genetics | Bile Ducts, Intrahepatic | Female | Gene Expression Regulation, Neoplastic - drug effects | Gene Expression Regulation, Neoplastic - physiology | Disease Models, Animal | Stem Cells - virology | Bile Duct Neoplasms - virology | Carcinoma, Hepatocellular - physiopathology | Cholangiocarcinoma - physiopathology | Liver Neoplasms, Experimental - virology | Mice, Inbred C57BL | Mice, Transgenic | Carcinoma, Hepatocellular - virology | Mice, SCID | Animals | Pyridines - toxicity | Signal Transduction - drug effects | Stem Cells - drug effects | Mice, Inbred NOD | Signal Transduction - physiology | Stem Cells - physiology | Trans-Activators - metabolism | Mice | Hepatitis | Liver cancer | Transgenic animals | Rodents | Tumors | Index Medicus
Journal Article
Details down arrow
Digital rights down arrow
loading Loading Rights
8. Full Text In situ force mapping of mammary gland transformation
by Lopez, Jose I and Kang, Inkyung and You, Weon-Kyoo and McDonald, Donald M and Weaver, Valerie M
Integrative Biology, ISSN 1757-9694, 09/2011, Volume 3, Issue 9, pp. 910 - 921
Tumor progression is characterized by an incremental stiffening of the tissue. The importance of tissue rigidity to cancer is appreciated, yet the contribution... 
ELASTICITY | VISCOELASTIC PROPERTIES | BREAST-CANCER RISK | TISSUE | EXTRACELLULAR-MATRIX | SUBSTRATE STIFFNESS | ELASTOGRAPHY | MECHANICAL-PROPERTIES | CELL | TUMOR MICROENVIRONMENT | CELL BIOLOGY | Bioengineering | Tensile Strength | Biophysical Phenomena | Vitrification | Mammary Neoplasms, Experimental - physiopathology | Mice, Transgenic | Retroviridae Infections - etiology | Neoplasm Invasiveness - physiopathology | Retroviridae Infections - physiopathology | Biomechanical Phenomena | Animals | Cell Transformation, Neoplastic | Tumor Virus Infections - physiopathology | Extracellular Matrix - physiology | Mammary Tumor Virus, Mouse | Microscopy, Atomic Force | Female | Mice | Tumor Virus Infections - etiology | Mammary Neoplasms, Experimental - etiology | Mammary Glands, Animal - physiology | Index Medicus
Journal Article
Details down arrow
Digital rights down arrow
loading Loading Rights
9. Full Text Human CD133+ Progenitor Cells Promote the Healing of Diabetic Ischemic Ulcers by Paracrine Stimulation of Angiogenesis and Activation of Wnt Signaling
by Barcelos, Lucíola S and Duplaa, Cécile and Kränkel, Nicolle and Graiani, Gallia and Invernici, Gloria and Katare, Rajesh and Siragusa, Mauro and Meloni, Marco and Campesi, Ilaria and Monica, Manuela and Simm, Andreas and Campagnolo, Paola and Mangialardi, Giuseppe and Stevanato, Lara and Alessandri, Giulio and Emanueli, Costanza and Madeddu, Paolo
Circulation Research, ISSN 0009-7330, 05/2009, Volume 104, Issue 9, pp. 1095 - 1102
We evaluated the healing potential of human fetal aorta–derived CD133 progenitor cells and their conditioned medium (CD133 CCM) in a new model of ischemic... 
Angiogenesis | Wound healing | Diabetes | Ischemia | Stem cells | stem cells | wound healing | CARDIAC & CARDIOVASCULAR SYSTEMS | angiogenesis | ischemia | TRANSPLANTATION | IN-VITRO | PATHWAY | WOUNDS | EMBRYONIC STEM-CELLS | ENDOTHELIAL-CELLS | FOOT ULCERS | PERIPHERAL VASCULAR DISEASE | GROWTH-FACTOR | NEOVASCULARIZATION | HEMATOLOGY | diabetes | MOUSE SKIN | Cell Proliferation | Diabetes Mellitus, Experimental - surgery | Fetal Stem Cells - metabolism | Humans | Male | Fetal Stem Cells - immunology | Vascular Endothelial Growth Factor A - metabolism | Wound Healing | Wnt Proteins - metabolism | Antigens, CD - analysis | Stem Cell Transplantation | Intercellular Signaling Peptides and Proteins - metabolism | Time Factors | Ischemia - complications | Cell Differentiation | Diabetes Mellitus, Experimental - complications | Membrane Proteins - metabolism | Interleukin-8 - metabolism | Diabetes Mellitus, Experimental - metabolism | Diabetes Mellitus, Experimental - physiopathology | Diabetic Foot - physiopathology | Paracrine Communication | Signal Transduction | Cell Survival | Cells, Cultured | Diabetic Foot - surgery | Aorta - embryology | Diabetic Foot - etiology | Ischemia - metabolism | Ischemia - physiopathology | AC133 Antigen | Animals | Diabetic Foot - metabolism | Ischemia - surgery | Fetal Stem Cells - transplantation | Mice | Glycoproteins - analysis | Lower Extremity - blood supply | Neovascularization, Physiologic | Peptides - analysis | Cell Movement | Culture Media, Conditioned - metabolism | Index Medicus
Journal Article
Details down arrow
Digital rights down arrow
loading Loading Rights
10. Full Text Lymphatic Metastasis in the Absence of Functional Intratumor Lymphatics
by Timothy P. Padera and Ananth Kadambi and Emmanuelle di Tomaso and Carla Mouta Carreira and Edward B. Brown and Yves Boucher and Noah C. Choi and Douglas Mathisen and John Wain and Eugene J. Mark and Lance L. Munn and Rakesh K. Jain
Science, ISSN 0036-8075, 6/2002, Volume 296, Issue 5574, pp. 1883 - 1886
Lymphatic metastasis contributes to mortality from solid tumors. Whether metastasizing cancer cells reach lymph nodes via intratumor lymphatic vessels is... 
Lungs | Blood vessels | Lectins | Fibrosarcoma | Reports | Metastasis | Lymphatic vessels | Ferritins | Phatic acts | Tumors | Cancer | SYSTEM | RECRUITMENT | VESSELS | GROWTH-FACTOR-C | MULTIDISCIPLINARY SCIENCES | TUMOR LYMPHANGIOGENESIS | INDUCTION | CANCER | LYVE-1 | Immunohistochemistry | Microscopy - methods | Neoplasms - metabolism | Neoplasm Transplantation | Endothelial Growth Factors - metabolism | Adenocarcinoma - pathology | Fibrosarcoma - physiopathology | Vascular Endothelial Growth Factor C | Lymphatic System - pathology | Fibrosarcoma - secondary | Humans | Lung Neoplasms - pathology | Melanoma, Experimental - metabolism | Lung Neoplasms - physiopathology | Fibrosarcoma - metabolism | Melanoma, Experimental - physiopathology | Lymphography | Fibrosarcoma - pathology | Lung Neoplasms - secondary | Melanoma, Experimental - secondary | Neoplasms - physiopathology | Tumor Cells, Cultured | Adenocarcinoma - chemistry | Vesicular Transport Proteins | Extracellular Space - physiology | Lymphatic System - chemistry | Lymphatic System - physiology | Lung Neoplasms - chemistry | Melanoma, Experimental - pathology | Lymphatic Metastasis | Pressure | Mice, Inbred C3H | Animals | Mice, Nude | Mice | Glycoproteins - analysis | Neoplasms - pathology | Antigens, Surface - analysis | Physiological aspects | Lymphatics | Research | Analysis | Cancer cells | Index Medicus
Journal Article
Details down arrow
Digital rights down arrow
loading Loading Rights
11. Full Text Tumor-mediated liver X receptor-α activation inhibits CC chemokine receptor-7 expression on dendritic cells and dampens antitumor responses
by Valentinis, Barbara and Maggioni, Daniela and Sanvito, Francesca and Prinetti, Alessandro and Sonnino, Sandro and Bordignon, Claudio and Zhou, Dan and Raccosta, Laura and Fontana, Raffaella and Steffensen, Knut R and Negro, Aurora and Ponzoni, Maurilio and Russo, Vincenzo and Bregni, Marco and Villablanca, Eduardo J and Traversari, Catia and Gustafsson, Jan-Ake and Doglioni, Claudio
Nature Medicine, ISSN 1078-8956, 01/2010, Volume 16, Issue 1, pp. 98 - 105
Sterol metabolism has recently been linked to innate and adaptive immune responses through liver X receptor (LXR) signaling. Whether products of sterol... 
MIGRATION | MEDICINE, RESEARCH & EXPERIMENTAL | IMMUNE-RESPONSE | CHOLESTEROL | NUCLEAR RECEPTORS | BIOCHEMISTRY & MOLECULAR BIOLOGY | SULFOTRANSFERASE | CELL BIOLOGY | IMMUNOGENICITY | LXR-BETA | METABOLISM | IN-VIVO | REGULATORY T-CELLS | Antigens, CD - immunology | Immunoglobulins - immunology | Humans | Dendritic Cells - physiology | Gene Expression Regulation - physiology | Lymph Nodes - immunology | Receptors, CCR7 - biosynthesis | Tumor Escape - physiology | Cell Movement - physiology | Lymph Nodes - physiopathology | Animals | Melanoma - immunology | Liver X Receptors | Neoplasms, Experimental - immunology | Orphan Nuclear Receptors - physiology | Cell Line, Tumor | Signal Transduction - physiology | Mice | Membrane Glycoproteins - immunology | Neoplasms, Experimental - physiopathology | Melanoma - physiopathology | Tumor Escape - immunology | Index Medicus
Journal Article
Details down arrow
Digital rights down arrow
loading Loading Rights
12. Full Text Six1 expands the mouse mammary epithelial stem/progenitor cell pool and induces mammary tumors that undergo epithelial-mesenchymal transition
by McCoy, Erica L and Iwanaga, Ritsuko and Jedlicka, Paul and Abbey, Nee-Shamo and Chodosh, Lewis A and Heichman, Karen A and Welm, Alana L and Ford, Heide L
Journal of Clinical Investigation, ISSN 0021-9738, 09/2009, Volume 119, Issue 9, pp. 2663 - 2677
Six1 is a developmentally regulated homeoprotein with limited expression in most normal adult tissues and frequent misexpression in a variety of malignancies.... 
BREAST-CANCER | MEDICINE, RESEARCH & EXPERIMENTAL | OVEREXPRESSION | STEM-CELLS | HOMEOPROTEIN SIX1 | GENE-EXPRESSION PROFILES | BETA-CATENIN | ES CELLS | CARCINOMA | PROGRESSION | GLAND | Neoplasm Transplantation | Humans | Male | Mammary Neoplasms, Experimental - physiopathology | Transplantation, Heterologous | Breast Neoplasms - physiopathology | Mammary Neoplasms, Experimental - genetics | Mammary Neoplasms, Experimental - pathology | Neoplastic Stem Cells - pathology | Female | Epithelium - pathology | Gene Expression | Signal Transduction | Transforming Growth Factor beta - physiology | Mice, Transgenic | Mice, SCID | Homeodomain Proteins - genetics | Neoplasm Metastasis - genetics | Animals | Breast Neoplasms - genetics | Breast Neoplasms - pathology | Neoplasm Metastasis - physiopathology | Cell Line, Tumor | Mice, Inbred NOD | Mice | Homeodomain Proteins - physiology | Mesoderm - pathology | Care and treatment | Breast cancer | Cancer | Index Medicus | Abridged Index Medicus
Journal Article
Details down arrow
Digital rights down arrow
loading Loading Rights