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Cancer Prevention Research, ISSN 1940-6207, 06/2010, Volume 3, Issue 6, pp. 696 - 706
The selective estrogen-receptor modulator (SERM) tamoxifen became the first U. S. Food and Drug Administration (FDA)-approved agent for reducing breast cancer... 
WOMEN | RISK-FACTORS | ONCOLOGY | ESTROGEN | FOLLOW-UP | OUTCOMES | MUTATIONS | NSABP | Uterus - pathology | Raloxifene Hydrochloride - adverse effects | Raloxifene Hydrochloride - pharmacology | Adenocarcinoma - pathology | Raloxifene Hydrochloride - therapeutic use | Cataract - chemically induced | Follow-Up Studies | Thromboembolism - chemically induced | Fractures, Spontaneous - epidemiology | Estrogens | Humans | Middle Aged | Osteoporosis, Postmenopausal - complications | Endometrial Neoplasms - chemically induced | Selective Estrogen Receptor Modulators - adverse effects | Risk | Cataract - epidemiology | Thromboembolism - epidemiology | Drug Utilization | Incidence | Selective Estrogen Receptor Modulators - therapeutic use | Aged, 80 and over | Adult | Female | Osteoporosis, Postmenopausal - prevention & control | Breast Neoplasms - epidemiology | Myocardial Ischemia - epidemiology | Neoplasms, Hormone-Dependent - prevention & control | Double-Blind Method | Neoplasm Invasiveness | Myocardial Ischemia - prevention & control | Fractures, Spontaneous - etiology | Endometrial Neoplasms - epidemiology | Neoplasms, Hormone-Dependent - epidemiology | Breast Neoplasms - prevention & control | Adenocarcinoma - prevention & control | Fractures, Spontaneous - prevention & control | Breast Neoplasms - pathology | Neoplasms, Hormone-Dependent - pathology | Tamoxifen - therapeutic use | Tamoxifen - pharmacology | Aged | Tamoxifen - adverse effects | Adenocarcinoma - epidemiology | Selective Estrogen Receptor Modulators - pharmacology | Index Medicus
Journal Article
Journal of Clinical Oncology, ISSN 0732-183X, 11/2011, Volume 29, Issue 31, pp. 4160 - 4167
Journal Article
British Journal of Surgery, ISSN 0007-1323, 11/2018, Volume 105, Issue 12, pp. 1650 - 1657
Background Obesity increases the risk of several types of cancer. Whether bariatric surgery influences the risk of obesity‐related cancer is not clear. This... 
MORTALITY | TRIAL | SURGERY | GASTRIC BYPASS-SURGERY | METAANALYSIS | REFLUX | BARIATRIC SURGERY | ESOPHAGEAL ADENOCARCINOMA | Esophageal Neoplasms - etiology | Neoplasms, Hormone-Dependent - mortality | Postoperative Complications - etiology | Neoplasms - etiology | Endometrial Neoplasms - mortality | Bariatric Surgery - statistics & numerical data | Humans | Middle Aged | Breast Neoplasms - etiology | Postoperative Complications - prevention & control | Prostatic Neoplasms - etiology | Esophageal Neoplasms - prevention & control | Male | Neoplasms - prevention & control | Postoperative Complications - mortality | Endometrial Neoplasms - prevention & control | Young Adult | Colorectal Neoplasms - etiology | Aged, 80 and over | Esophageal Neoplasms - mortality | Adult | Female | Obesity - mortality | Prostatic Neoplasms - prevention & control | Neoplasms, Hormone-Dependent - prevention & control | Colorectal Neoplasms - mortality | Obesity - surgery | Obesity - complications | Risk Factors | Endometrial Neoplasms - etiology | Kaplan-Meier Estimate | Colorectal Neoplasms - prevention & control | Neoplasms - mortality | Prostatic Neoplasms - mortality | Breast Neoplasms - prevention & control | Propensity Score | Neoplasms, Hormone-Dependent - etiology | Adolescent | Bariatric Surgery - mortality | Breast Neoplasms - mortality | Aged | Obesity | Surgery | Oncology, Experimental | Colorectal cancer | Hormones | Research | Health aspects | Risk factors | Cancer
Journal Article
Journal Article
Breast Cancer Research and Treatment, ISSN 0167-6806, 5/2012, Volume 133, Issue 1, pp. 367 - 373
Journal Article
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 11/2014, Volume 111, Issue 46, pp. 16514 - 16519
It recently has been recognized that men develop colonic adenomas and carcinomas at an earlier age and at a higher rate than women. In the ApcPirc/+ (Pirc) rat... 
Testosterone | Animal models | Estrogens | Colorectal cancer | Rats | Adenoma | Hormones | Colon | Small intestine | Tumors | ANDROGEN RECEPTOR | animal models | androgens | TESTOSTERONE | colon cancer | MULTIDISCIPLINARY SCIENCES | estrogens | INTESTINAL TUMORIGENESIS | BETA | CARCINOGENESIS | COLORECTAL-CANCER | MOUSE MODEL | ESTROGEN | MICE | intestinal regionality | POSTMENOPAUSAL WOMEN | Species Specificity | Adenoma - epidemiology | Colonic Neoplasms - prevention & control | Humans | Rats, Inbred F344 | Adenoma - prevention & control | Adenomatous Polyposis Coli - epidemiology | Medroxyprogesterone Acetate - pharmacology | RNA, Messenger - analysis | Colonic Neoplasms - chemically induced | Male | Dihydrotestosterone - toxicity | Neoplasms, Hormone-Dependent - physiopathology | Azoxymethane - toxicity | Hormone Replacement Therapy | Carcinogens - toxicity | Genes, APC | Colonic Neoplasms - physiopathology | Gonadal Steroid Hormones - physiology | Rats, Mutant Strains | Receptors, Androgen - biosynthesis | Postmenopause | Female | Adenomatous Polyposis Coli - genetics | Animals, Congenic | Estradiol - pharmacology | Disease Models, Animal | Neoplasms, Hormone-Dependent - prevention & control | Ovariectomy | Mice, Inbred C57BL | Adenomatous Polyposis Coli - physiopathology | Estradiol - administration & dosage | Colonic Neoplasms - epidemiology | Random Allocation | Organ Specificity | Neoplasms, Hormone-Dependent - epidemiology | Orchiectomy | Adenoma - physiopathology | Animals | Receptors, Androgen - genetics | Sex Distribution | Medroxyprogesterone Acetate - administration & dosage | Mice | Mutation | Adenoma - chemically induced | Physiological aspects | Colon cancer | Health aspects | Biological Sciences
Journal Article
Journal Article
Cancer Research, ISSN 0008-5472, 10/2009, Volume 69, Issue 20, pp. 8017 - 8024
Heme oxygenase-1 (HO-1), a member of the heat shock protein family, plays a key role as a sensor and regulator of oxidative stress. Herein, we identify HO-1 as... 
OXIDATIVE STRESS | INVASION | METASTASIS | INHIBITION | ONCOLOGY | MICROTUBULE | OXYGENASE GENE | MECHANISMS | GROWTH-FACTOR | EXPRESSION | CELL CARCINOMA | Genetic Therapy | Heme Oxygenase-1 - metabolism | Prostatic Hyperplasia - pathology | Reactive Oxygen Species - metabolism | Oxidative Stress | Apoptosis - drug effects | Tissue Array Analysis | Humans | Neoplasms, Hormone-Dependent - secondary | Male | Immunoenzyme Techniques | Heme Oxygenase-1 - genetics | Lung Neoplasms - secondary | Neoplasms, Hormone-Dependent - enzymology | p38 Mitogen-Activated Protein Kinases - metabolism | Prostatic Neoplasms - prevention & control | Tumor Cells, Cultured | Neoplasms, Hormone-Dependent - prevention & control | Prostatic Neoplasms - pathology | Lung Neoplasms - enzymology | Signal Transduction | Neoplasm Invasiveness | RNA, Small Interfering - pharmacology | Lymphatic Metastasis | Cell Adhesion - drug effects | Mice, SCID | Heme Oxygenase-1 - antagonists & inhibitors | Blotting, Western | Disease Progression | Gene Expression Regulation, Enzymologic | Cell Movement - drug effects | Animals | Lung Neoplasms - prevention & control | Mitogen-Activated Protein Kinase 3 - metabolism | Prostatic Neoplasms - enzymology | Cell Proliferation - drug effects | Mice | Prostatic Hyperplasia - prevention & control | Prostatic Hyperplasia - enzymology | Mitogen-Activated Protein Kinase 1 - metabolism
Journal Article