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1. Full Text Serine/Threonine Kinase MLK4 Determines Mesenchymal Identity in Glioma Stem Cells in an NF-κB-dependent Manner
by Kim, Sung-Hak and Ezhilarasan, Ravesanker and Phillips, Emma and Gallego-Perez, Daniel and Sparks, Amanda and Taylor, David and Ladner, Katherine and Furuta, Takuya and Sabit, Hemragul and Chhipa, Rishi and Cho, Ju Hwan and Mohyeldin, Ahmed and Beck, Samuel and Kurozumi, Kazuhiko and Kuroiwa, Toshihiko and Iwata, Ryoichi and Asai, Akio and Kim, Jonghwan and Sulman, Erik P and Cheng, Shi-Yuan and Lee, L. James and Nakada, Mitsutoshi and Guttridge, Denis and DasGupta, Biplab and Goidts, Violaine and Bhat, Krishna P and Nakano, Ichiro
Cancer cell, ISSN 1535-6108, 02/2016, Volume 29, Issue 2, pp. 201 - 213
glioblastoma | proneural-mesenchymal transition | cancer stem cell | epithelial-to-mesenchymal transition | Cancer stem cell | Proneural-mesenchymal transition | Epithelial-to-mesenchymal transition | Glioblastoma | Oncology | Life Sciences & Biomedicine | Science & Technology | Cell Biology | Mesenchymal Stromal Cells - enzymology | Brain Neoplasms - enzymology | Phosphorylation | Glioma - enzymology | Signal Transduction | MAP Kinase Kinase Kinases - genetics | Humans | Brain Neoplasms - pathology | Gene Silencing | NF-kappa B - metabolism | MAP Kinase Kinase Kinases - metabolism | Animals | Glioma - pathology | Neoplastic Stem Cells - pathology | Mice | Mesenchymal Stromal Cells - pathology | Neoplastic Stem Cells - enzymology | Apoptosis | Index Medicus | proneural-Mesenchymal transition
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2. Full Text Breast cancer stem cells are regulated by mesenchymal stem cells through cytokine networks
by Liu, Suling and Ginestier, Christophe and Ou, Sing J and Clouthier, Shawn G and Patel, Shivani H and Monville, Florence and Korkaya, Hasan and Heath, Amber and Dutcher, Julie and Kleer, Celina G and Jung, Younghun and Dontu, Gabriela and Taichman, Russell and Wicha, Max S
Cancer research (Chicago, Ill.), ISSN 0008-5472, 01/2011, Volume 71, Issue 2, pp. 614 - 624
Life Sciences & Biomedicine | Oncology | Science & Technology | Gynecology. Andrology. Obstetrics | Mammary gland diseases | Pharmacology. Drug treatments | Biological and medical sciences | Medical sciences | Antineoplastic agents | Tumors | Mesenchymal Stromal Cells - enzymology | Aldehyde Dehydrogenase - analysis | Cytokines - metabolism | Bone Marrow Cells - cytology | Humans | Aldehyde Dehydrogenase - biosynthesis | Mesenchymal Stromal Cells - metabolism | Cell Communication - physiology | Transplantation, Heterologous | Mice, SCID | Breast Neoplasms - metabolism | Breast Neoplasms - enzymology | Animals | Neoplastic Stem Cells - metabolism | Breast Neoplasms - pathology | Neoplastic Stem Cells - pathology | Female | Mice, Inbred NOD | Mice | Mesenchymal Stromal Cells - pathology | Neoplastic Stem Cells - enzymology | Index Medicus | Life Sciences | Cellular Biology | Cancer
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3. Full Text Aldehyde dehydrogenase-expressing colon stem cells contribute to tumorigenesis in the transition from colitis to cancer
by Carpentino, Joseph E and Hynes, Mark J and Appelman, Henry D and Tong, Zheng and Steindler, Dennis A and Scott, Edward W and Huang, Emina H
Cancer research (Chicago, Ill.), ISSN 0008-5472, 10/2009, Volume 69, Issue 20, pp. 8208 - 8215
Life Sciences & Biomedicine | Oncology | Science & Technology | Biological and medical sciences | Medical sciences | Antineoplastic agents | Pharmacology. Drug treatments | Tumors | Precancerous Conditions - etiology | Colon - enzymology | Adenocarcinoma - pathology | Stromal Cells - pathology | Adenocarcinoma - etiology | Humans | Spheroids, Cellular - pathology | Colitis - pathology | Spheroids, Cellular - enzymology | Immunoenzyme Techniques | Stromal Cells - enzymology | Flow Cytometry | Neoplastic Stem Cells - pathology | Precancerous Conditions - pathology | Aldehyde Dehydrogenase - metabolism | Colonic Neoplasms - etiology | Precancerous Conditions - enzymology | Cytokines - metabolism | Colon - pathology | Adenocarcinoma - enzymology | Mice, SCID | Xenograft Model Antitumor Assays | Animals | Colitis - enzymology | Colonic Neoplasms - pathology | Fluorescent Antibody Technique | Protein Array Analysis | Mice, Inbred NOD | Colonic Neoplasms - enzymology | Mice | Neoplastic Stem Cells - enzymology | Index Medicus | Xenograft | Colitis | tumor microenvironment | Aldehyde dehydrogenase | Colon Cancer | Colon sphere
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4. Full Text PTEN deficiency reprogrammes human neural stem cells towards a glioblastoma stem cell-like phenotype
by Duan, Shunlei and Yuan, Guohong and Liu, Xiaomeng and Ren, Ruotong and Li, Jingyi and Zhang, Weizhou and Wu, Jun and Xu, Xiuling and Fu, Lina and Li, Ying and Yang, Jiping and Zhang, Weiqi and Bai, Ruijun and Yi, Fei and Suzuki, Keiichiro and Gao, Hua and Esteban, Concepcion Rodriguez and Zhang, Chuanbao and Belmonte, Juan Carlos Izpisua and Chen, Zhiguo and Wang, Xiaomin and Jiang, Tao and Qu, Jing and Tang, Fuchou and Liu, Guang-Hui
Nature communications, ISSN 2041-1723, 12/2015, Volume 6, Issue 1, pp. 10068 - 10068
Science & Technology - Other Topics | Multidisciplinary Sciences | Science & Technology | PTEN Phosphohydrolase - genetics | Brain Neoplasms - enzymology | Glioblastoma - enzymology | Neoplastic Stem Cells - cytology | PTEN Phosphohydrolase - deficiency | Cell Proliferation | Humans | Brain Neoplasms - pathology | Brain Neoplasms - genetics | Male | Mice, SCID | Neural Stem Cells - cytology | Neural Stem Cells - enzymology | Gene Knockdown Techniques | Phenotype | Animals | Glioblastoma - genetics | Glioblastoma - pathology | Cell Line, Tumor | Cell Differentiation | Mice | Neoplastic Stem Cells - enzymology | Index Medicus
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5. Full Text Tumorigenic and metastatic activity of human thyroid cancer stem cells
by Todaro, Matilde and Iovino, Flora and Eterno, Vincenzo and Cammareri, Patrizia and Gambara, Guido and Espina, Virginia and Gulotta, Gaspare and Dieli, Francesco and Giordano, Silvia and De Maria, Ruggero and Stassi, Giorgio
Cancer research (Chicago, Ill.), ISSN 0008-5472, 11/2010, Volume 70, Issue 21, pp. 8874 - 8885
Life Sciences & Biomedicine | Oncology | Science & Technology | Non tumoral diseases. Target tissue resistance. Benign neoplasms | Malignant tumors | Pharmacology. Drug treatments | Thyroid. Thyroid axis (diseases) | Biological and medical sciences | Endocrinopathies | Medical sciences | Antineoplastic agents | Tumors | Proto-Oncogene Proteins c-met - metabolism | Thyroid Neoplasms - enzymology | Cell Proliferation | Adenocarcinoma, Follicular - pathology | Humans | Middle Aged | Male | Thyroid Gland - enzymology | Case-Control Studies | Immunoenzyme Techniques | Young Adult | Flow Cytometry | Lung Neoplasms - secondary | Neoplastic Stem Cells - metabolism | Carcinoma - enzymology | Neoplastic Stem Cells - pathology | Adult | Female | Carcinoma, Papillary - enzymology | Carcinoma - pathology | Proto-Oncogene Proteins c-akt - metabolism | Aldehyde Dehydrogenase - metabolism | Tumor Stem Cell Assay | Lung Neoplasms - enzymology | Neoplasm Invasiveness | RNA, Messenger - genetics | Aldehyde Dehydrogenase - genetics | Mice, SCID | Reverse Transcriptase Polymerase Chain Reaction | Carcinoma, Papillary - pathology | Cell Adhesion | Adenocarcinoma, Follicular - enzymology | Blotting, Western | Xenograft Model Antitumor Assays | Animals | Mice, Nude | Thyroid Gland - pathology | Fluorescent Antibody Technique | Mice, Inbred NOD | Aged | Mice | Cell Transformation, Neoplastic - pathology | Cell Movement | Thyroid Neoplasms - pathology | Index Medicus
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6. Full Text ALDH1 Is a Marker of Normal and Malignant Human Mammary Stem Cells and a Predictor of Poor Clinical Outcome
by Ginestier, Christophe and Hur, Min Hee and Charafe-Jauffret, Emmanuelle and Monville, Florence and Dutcher, Julie and Brown, Marty and Jacquemier, Jocelyne and Viens, Patrice and Kleer, Celina G and Liu, Suling and Schott, Anne and Hayes, Dan and Birnbaum, Daniel and Wicha, Max S and Dontu, Gabriela
Cell stem cell, ISSN 1934-5909, 11/2007, Volume 1, Issue 5, pp. 555 - 567
STEMCELL | CELLCYCLE | Cell & Tissue Engineering | Life Sciences & Biomedicine | Science & Technology | Cell Biology | Immunohistochemistry | Mammary Glands, Human - immunology | Up-Regulation | Cell Proliferation | Breast Neoplasms - immunology | Tissue Array Analysis | Humans | Neoplastic Stem Cells - immunology | Adult Stem Cells - immunology | Retinal Dehydrogenase | Breast Neoplasms - therapy | Stem Cell Transplantation | Breast Neoplasms - enzymology | Flow Cytometry | Isoenzymes - metabolism | Neoplastic Stem Cells - pathology | Female | Cell Differentiation | Aldehyde Dehydrogenase - metabolism | Adult Stem Cells - enzymology | Cell Separation | Biomarkers - analysis | Cells, Cultured | Treatment Outcome | Mice, SCID | Hyaluronan Receptors - analysis | Xenograft Model Antitumor Assays | Cell Lineage | Phenotype | Animals | Breast Neoplasms - pathology | CD24 Antigen - analysis | Survival Analysis | Mice, Inbred NOD | Mice | Mammary Glands, Human - enzymology | Neoplastic Stem Cells - enzymology | Index Medicus
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7. Full Text Hyaluronan synthase HAS2 promotes tumor progression in bone by stimulating the interaction of breast cancer stem-like cells with macrophages and stromal cells
by Okuda, Hiroshi and Kobayashi, Aya and Xia, Bo and Watabe, Misako and Pai, Sudha K and Hirota, Shigeru and Xing, Fei and Liu, Wen and Pandey, Puspa R and Fukuda, Koji and Modur, Vishnu and Ghosh, Arnab and Wilber, Andrew and Watabe, Kounosuke
Cancer research (Chicago, Ill.), ISSN 0008-5472, 01/2012, Volume 72, Issue 2, pp. 537 - 547
Life Sciences & Biomedicine | Oncology | Science & Technology | Gynecology. Andrology. Obstetrics | Mammary gland diseases | Pharmacology. Drug treatments | Biological and medical sciences | Medical sciences | Antineoplastic agents | Tumors | Up-Regulation | Stromal Cells - pathology | Humans | Gene Expression Regulation, Neoplastic | Bone Neoplasms - secondary | Gene Expression Profiling | Bone Neoplasms - pathology | Cell Movement - physiology | Hymecromone - analogs & derivatives | Breast Neoplasms - enzymology | Stromal Cells - enzymology | Neoplastic Stem Cells - pathology | Female | Bone Neoplasms - genetics | Hyaluronan Synthases | Hymecromone - pharmacology | Bone Neoplasms - enzymology | Macrophages - pathology | Neoplasm Invasiveness | Disease Progression | Macrophages - enzymology | Animals | Breast Neoplasms - genetics | Glucuronosyltransferase - metabolism | Breast Neoplasms - pathology | Cell Adhesion - physiology | Mice, Nude | Glucuronosyltransferase - antagonists & inhibitors | Mice | Neoplastic Stem Cells - enzymology | Index Medicus | Bone cancer | microenvironment | tumor associated macrophages | HAS2
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8. Full Text The MEK inhibitor PD184352 enhances BMS-214662-induced apoptosis in CD34+ CML stem progenitor cells
by Pellicano, F and Imara, P and Sinclair, A and Helgason, G.V and Copland, M and Grant, S and Holyoake, T.L
Leukemia, ISSN 0887-6924, 07/2011, Volume 25, Issue 7, pp. 1159 - 1167
BMS-214662 | HSC | MEK inhibitor | CML | TKI | PD184352 | Oncology | Life Sciences & Biomedicine | Hematology | Science & Technology | Hematologic and hematopoietic diseases | Leukemias. Malignant lymphomas. Malignant reticulosis. Myelofibrosis | Biological and medical sciences | Medical sciences | Antigens, CD34 - analysis | Apoptosis - drug effects | Neoplastic Stem Cells - drug effects | Humans | Leukemia, Myeloid, Chronic-Phase - pathology | Neoplasm Proteins - antagonists & inhibitors | K562 Cells - drug effects | Blast Crisis - enzymology | MAP Kinase Kinase 1 - genetics | Tumor Cells, Cultured - enzymology | Genes, Dominant | Leukemia, Myelogenous, Chronic, BCR-ABL Positive - pathology | Benzamides - pharmacology | Farnesyltranstransferase - antagonists & inhibitors | MAP Kinase Kinase Kinases - antagonists & inhibitors | Hematopoietic Stem Cells - drug effects | Leukemia, Myelogenous, Chronic, BCR-ABL Positive - enzymology | Benzodiazepines - pharmacology | Blast Crisis - pathology | Tumor Cells, Cultured - drug effects | Enzyme Inhibitors - pharmacology | Imidazoles - pharmacology | Leukemia, Myeloid, Chronic-Phase - enzymology | K562 Cells - enzymology | Drug Synergism | Recombinant Fusion Proteins - genetics | Genes, ras | Hematopoietic Stem Cells - enzymology | Neoplastic Stem Cells - enzymology | Drug Screening Assays, Antitumor | Oncogene Protein p21(ras) - genetics | Antimitotic agents | Physiological aspects | Chronic myeloid leukemia | Research | Drug therapy | Antineoplastic agents | Protein kinases | Health aspects | Apoptosis | Index Medicus | FTI | CD34
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9. Full Text Targeting carbonic anhydrase IX depletes breast cancer stem cells within the hypoxic niche
by Lock, F.E and McDonald, P.C and Lou, Y and Serrano, I and Chafe, S.C and Ostlund, C and Aparicio, S and Winum, J.-Y and Supuran, C.T and Dedhar, S
Oncogene, ISSN 0950-9232, 10/2013, Volume 32, Issue 44, pp. 5210 - 5219
mTOR | hypoxia | breast cancer | CAIX | cancer stem cell | EMT | Biochemistry & Molecular Biology | Oncology | Genetics & Heredity | Life Sciences & Biomedicine | Science & Technology | Cell Biology | Cell Proliferation | Paclitaxel - pharmacology | Neoplastic Stem Cells - drug effects | Humans | Tumor Microenvironment | Spheroids, Cellular - enzymology | Carbonic Anhydrases - metabolism | Cell Hypoxia | Breast Neoplasms - enzymology | Lung Neoplasms - secondary | Antigens, Neoplasm - metabolism | Female | Antineoplastic Agents - pharmacology | Carbonic Anhydrase IX | Lung Neoplasms - enzymology | Stem Cell Niche | Neoplasm Invasiveness | Carbonic Anhydrase Inhibitors - pharmacology | Mice, SCID | Sulfonamides - pharmacology | Breast Neoplasms - drug therapy | Drug Synergism | Xenograft Model Antitumor Assays | Animals | Lung Neoplasms - prevention & control | Breast Neoplasms - pathology | Cell Line, Tumor | Mice, Inbred NOD | Mice | Phenylurea Compounds - pharmacology | Neoplastic Stem Cells - enzymology | Care and treatment | Breast cancer | Genetic aspects | Research | Health aspects | Stem cells | Tumorigenesis | Metastasis | Tumors | Index Medicus
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