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Molecular neurobiology, ISSN 0893-7648, 11/2016, Volume 53, Issue 9, pp. 6459 - 6475
...Oxidative Stress, Disrupted Energy Metabolism, and Altered Signaling Pathways in Glutaryl-CoA Dehydrogenase Knockout Mice: Potential Implications of Quinolinic... 
Signaling pathways | Neurology | Neurosciences | Energy metabolism | Biomedicine | Glutaric acidemia | Redox homeostasis | Neurobiology | Inflammatory response | Quinolinic acid | Cell Biology | Neurosciences & Neurology | Life Sciences & Biomedicine | Science & Technology | Brain Diseases, Metabolic - pathology | Glutaryl-CoA Dehydrogenase - metabolism | Glutathione - metabolism | Antioxidants - metabolism | Fluoresceins - metabolism | Nitrites - metabolism | NF-kappa B - metabolism | Extracellular Signal-Regulated MAP Kinases - metabolism | Electron Transport Complex IV - metabolism | Amino Acid Metabolism, Inborn Errors - metabolism | Sulfhydryl Compounds - metabolism | Neostriatum - drug effects | Lipid Peroxidation - drug effects | Neostriatum - metabolism | Quinolinic Acid - administration & dosage | Quinolinic Acid - toxicity | Amino Acid Metabolism, Inborn Errors - pathology | Brain Diseases, Metabolic - enzymology | Proto-Oncogene Proteins c-akt - metabolism | Superoxide Dismutase - metabolism | Malondialdehyde - metabolism | Glutathione Peroxidase - metabolism | Neostriatum - pathology | Oxidation-Reduction | Brain Diseases, Metabolic - metabolism | Mice, Knockout | Nitrates - metabolism | Animals | Lactates - metabolism | Diet | Signal Transduction - drug effects | Models, Biological | NF-E2-Related Factor 2 - metabolism | Oxidative Stress - drug effects | Amino Acid Metabolism, Inborn Errors - enzymology | Energy Metabolism - drug effects | Glutaryl-CoA Dehydrogenase - deficiency | Antioxidants | Lysine | Creatine kinase | Glutathione transferase | Physiological aspects | Superoxide | Inflammation | Creatine | Homeostasis | Signal transduction | Oxidative stress | Bioenergetics | Index Medicus
Journal Article
Free radical biology & medicine, ISSN 0891-5849, 11/2012, Volume 53, Issue 10, pp. 1857 - 1867
Huntington’s disease (HD) is a CAG repeat disorder affecting the HD gene, which encodes for huntingtin (Htt) and is characterized by prominent cell death in... 
Striatal cells | Oxidative stress | Reactive oxygen species | Huntingtin | Huntington disease | Biochemistry & Molecular Biology | Endocrinology & Metabolism | Life Sciences & Biomedicine | Science & Technology | Reactive Oxygen Species - metabolism | Oxidative Stress | Glutathione - metabolism | Glutathione Reductase - metabolism | Huntington Disease - pathology | Caspase 3 - metabolism | RNA, Messenger - metabolism | Corpus Striatum - cytology | Corpus Striatum - metabolism | Glutathione - pharmacology | Biological Transport | Multidrug Resistance-Associated Proteins - genetics | Neostriatum - metabolism | Glutamate-Cysteine Ligase - metabolism | Glutathione Synthase - metabolism | Nuclear Proteins - genetics | Glutathione Peroxidase - metabolism | Glutathione - analogs & derivatives | Oxidation-Reduction | RNA, Messenger - genetics | Cells, Cultured | Glutathione Transferase - metabolism | Nuclear Proteins - metabolism | gamma-Glutamyltransferase - metabolism | Huntington Disease - metabolism | Nerve Tissue Proteins - genetics | Gene Knock-In Techniques | Nerve Tissue Proteins - metabolism | Huntingtin Protein | Animals | Huntington Disease - genetics | Mice | Mutation | Multidrug Resistance-Associated Proteins - metabolism | Antioxidants | Drug resistance in microorganisms | Messenger RNA | Thiols | Huntington's chorea | Ligases | Glutathione transferase | Cell death | Analysis | Peroxidase | Cells | Index Medicus
Journal Article
Journal Article
Neuron (Cambridge, Mass.), ISSN 0896-6273, 01/2015, Volume 85, Issue 2, pp. 296 - 302
Journal Article
Molecular neurobiology, ISSN 1559-1182, 10/2015, Volume 53, Issue 8, pp. 5782 - 5795
Clinical evidences showing zinc (Zn) accumulation in the post-mortem brain of Parkinson’s disease (PD) patients and experimental studies on rodents chronically... 
Neurology | Oxidative stress | Neurosciences | Biomedicine | Neurodegeneration | Neurobiology | Inflammation | Zinc | Cell Biology | Apoptosis | Neurosciences & Neurology | Life Sciences & Biomedicine | Science & Technology | Tumor Necrosis Factor-alpha - metabolism | Inflammation - pathology | Tyrosine 3-Monooxygenase - metabolism | Rats, Wistar | Substantia Nigra - pathology | Antigens, Nuclear - metabolism | Apoptosis - drug effects | Dopaminergic Neurons - pathology | bcl-X Protein - genetics | Nerve Degeneration - genetics | Apoptosis - genetics | Male | NF-kappa B - metabolism | RNA, Messenger - metabolism | Behavior, Animal | Proto-Oncogene Proteins c-bcl-2 - metabolism | Inflammation - metabolism | Interleukin-1beta - metabolism | Dopaminergic Neurons - metabolism | Lipid Peroxidation - drug effects | Apoptosis Regulatory Proteins - genetics | Dopamine - metabolism | Neostriatum - pathology | Cytochromes c - metabolism | RNA, Messenger - genetics | bcl-2-Associated X Protein - metabolism | Metabolome | Mitochondria - metabolism | Mitochondria - drug effects | Nerve Degeneration - pathology | Apoptosis Regulatory Proteins - metabolism | Gene Expression Regulation - drug effects | Nerve Tissue Proteins - metabolism | Animals | Serotonin - metabolism | Oxidative Stress - drug effects | Zinc - pharmacology | bcl-X Protein - metabolism | Proto-Oncogene Proteins c-bcl-2 - genetics | Tyrosine | Parkinson's disease | Corticosteroids | Neurons | Mitochondrial DNA | Superoxide | Sulfates | Cytochrome c | Zinc compounds | Metabolites | Interleukins | Physiological aspects | Lymphomas | Tumor proteins | Index Medicus
Journal Article
Nature communications, ISSN 2041-1723, 12/2018, Volume 9, Issue 1, pp. 1422 - 14
We report that half striatal cholinergic interneurons are dual transmitter cholinergic and GABAergic interneurons (CGINs) expressing ChAT, GAD65, Lhx7, and... 
Science & Technology - Other Topics | Multidisciplinary Sciences | Science & Technology | LIM-Homeodomain Proteins - metabolism | Sodium Potassium Chloride Symporter Inhibitors - pharmacology | gamma-Aminobutyric Acid - metabolism | Cholinergic Agents - pharmacology | Humans | Solute Carrier Family 12, Member 2 - genetics | Neuronal Plasticity - drug effects | Acetylcholine - metabolism | Corpus Striatum - metabolism | Dopamine - deficiency | Parkinson Disease, Secondary - pathology | Chlorides - metabolism | Parkinson Disease, Secondary - metabolism | Ion Transport | Corpus Striatum - pathology | Action Potentials - drug effects | Solute Carrier Family 12, Member 2 - metabolism | Acetylcholine - pharmacology | Cholinergic Neurons - metabolism | Gene Expression Regulation | Interneurons - drug effects | gamma-Aminobutyric Acid - pharmacology | Glutamate Decarboxylase - genetics | Mice, Transgenic | Cholinergic Agents - metabolism | Cholinergic Neurons - drug effects | Transcription Factors - genetics | Nerve Tissue Proteins - genetics | Bumetanide - pharmacology | Interneurons - pathology | Action Potentials - physiology | Nerve Tissue Proteins - metabolism | Transcription Factors - metabolism | LIM-Homeodomain Proteins - genetics | Patch-Clamp Techniques | Animals | Interneurons - metabolism | Parkinson Disease, Secondary - genetics | Vesicular Inhibitory Amino Acid Transport Proteins - metabolism | Vesicular Inhibitory Amino Acid Transport Proteins - genetics | Corpus Striatum - drug effects | Glutamate Decarboxylase - metabolism | Mice | Cholinergic Neurons - pathology | Inhibitory Postsynaptic Potentials - drug effects | Deprivation | Dopamine | Parkinson's disease | Interneurons | Neurodegenerative diseases | Chloride transport | Glutamate decarboxylase | γ-Aminobutyric acid | Restoration | Neostriatum | Inhibition | Bumetanide | Movement disorders | Index Medicus | Life Sciences | Neurons and Cognition
Journal Article
Cell metabolism, ISSN 1550-4131, 02/2017, Volume 25, Issue 2, pp. 335 - 344
Journal Article
The Journal of neuroscience, ISSN 1529-2401, 03/2010, Volume 30, Issue 9, pp. 3398 - 3408
Striatal dopamine (DA) and acetylcholine (ACh) regulate motivated behaviors and striatal plasticity. Interactions between these neurotransmitters may be... 
Neurosciences | Neurosciences & Neurology | Life Sciences & Biomedicine | Science & Technology | Receptor, Muscarinic M2 - genetics | Synaptic Transmission - physiology | Muscarinic Agonists - pharmacology | Nucleus Accumbens - metabolism | Basal Ganglia - metabolism | Receptor, Muscarinic M2 - drug effects | Electrophysiology | Receptors, Muscarinic - metabolism | Acetylcholine - metabolism | Corpus Striatum - cytology | Corpus Striatum - metabolism | Basal Ganglia - cytology | Receptors, Muscarinic - drug effects | Neostriatum - drug effects | Nucleus Accumbens - cytology | Neostriatum - metabolism | Synaptic Transmission - drug effects | Interneurons - cytology | Receptor, Muscarinic M4 - genetics | Dopamine - metabolism | Organ Culture Techniques | Receptors, Muscarinic - genetics | Receptors, Nicotinic - drug effects | Receptors, Nicotinic - metabolism | Neostriatum - cytology | Presynaptic Terminals - drug effects | Receptor, Muscarinic M2 - metabolism | Mice, Inbred C57BL | Interneurons - drug effects | Receptor, Muscarinic M4 - metabolism | Mice, Knockout | Patch-Clamp Techniques | Animals | Interneurons - metabolism | Receptor, Muscarinic M4 - drug effects | Corpus Striatum - drug effects | Presynaptic Terminals - metabolism | Mice | Nucleus Accumbens - drug effects | Basal Ganglia - drug effects | Index Medicus
Journal Article