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Journal Article
Nature (London), ISSN 1476-4687, 2019, Volume 571, Issue 7766, pp. 565 - 569
Parkinson's disease is a neurodegenerative disorder with motor symptoms linked to the loss of dopaminergic neurons in the substantia nigra compacta. Although... 
RECOGNIZE | MODELS | MULTIDISCIPLINARY SCIENCES | OCCURS | ASSOCIATION | BRAIN | CLONE | LYMPHOCYTES | Protein Kinases - genetics | CD8-Positive T-Lymphocytes - pathology | Dopaminergic Neurons - pathology | Neostriatum - physiopathology | Male | Mitochondria - immunology | Parkinson Disease - drug therapy | Citrobacter rodentium - pathogenicity | Intestines - immunology | Enterobacteriaceae Infections - immunology | Ubiquitin-Protein Ligases - immunology | Female | Protein Kinases - deficiency | Disease Models, Animal | Neostriatum - pathology | Citrobacter rodentium - immunology | Dopaminergic Neurons - immunology | Intestines - pathology | Neostriatum - microbiology | Levodopa - therapeutic use | Enterobacteriaceae Infections - microbiology | Enterobacteriaceae Infections - pathology | Mitochondria - pathology | Parkinson Disease - genetics | Antigen Presentation - immunology | Parkinson Disease - physiopathology | Enterobacteriaceae Infections - physiopathology | Animals | Intestines - microbiology | Parkinson Disease - microbiology | Protein Kinases - immunology | Autoantigens - immunology | Axons - pathology | Ubiquitin-Protein Ligases - deficiency | Mice | CD8-Positive T-Lymphocytes - immunology | Ubiquitin-Protein Ligases - genetics | Neostriatum - immunology | Ubiquitin | Brain | Animal models | Parkinson's disease | Peptides | CD8 antigen | Substantia nigra | Immune clearance | Parkinsons disease | Cytotoxicity | Lymphocytes T | Infections | Kinases | Immunity | Proteins | Mitochondria | Lymphocytes | Intestine | Neostriatum | Bacteria | Levodopa | Gram-negative bacteria | Ubiquitin-protein ligase | Age | Movement disorders | Immune system | Dopamine receptors | Spleen | Dihydroxyphenylalanine | Antigen presentation | Antigens | Repressing | Signs and symptoms | Dopamine | Bacterial infections | Cytokines | Neurodegenerative diseases | Neurons | Cloning | Inflammation | PTEN-induced putative kinase | Parkin protein | Mutation | Dopa
Journal Article
Nature neuroscience, ISSN 1546-1726, 2000, Volume 3, Issue 12, pp. 1301 - 1306
The cause of Parkinson's disease (PD) is unknown, but epidemiological studies suggest an association with pesticides and other environmental toxins, and... 
ELECTRON-TRANSPORT CHAIN | SKELETAL-MUSCLE | OXIDATIVE-PHOSPHORYLATION | BRAIN MITOCHONDRIA | TOXIN 3-NITROPROPIONIC ACID | COMPLEX-I | MITOCHONDRIAL-FUNCTION | ALPHA-SYNUCLEIN | SUBSTANTIA-NIGRA | NEUROSCIENCES | LEWY BODIES | Substantia Nigra - physiopathology | Neurons - pathology | Neural Pathways - drug effects | Presynaptic Terminals - pathology | Electron Transport Complex I | Substantia Nigra - pathology | Lewy Bodies - drug effects | Neostriatum - physiopathology | Nerve Degeneration - physiopathology | Rats, Inbred Lew | Male | Neural Pathways - pathology | Nerve Degeneration - chemically induced | Neostriatum - drug effects | Rotenone - toxicity | Neurons - metabolism | Lewy Bodies - metabolism | Neural Pathways - physiopathology | Parkinson Disease, Secondary - chemically induced | NADH, NADPH Oxidoreductases - metabolism | Neurons - drug effects | Lewy Bodies - pathology | NADH, NADPH Oxidoreductases - drug effects | Dopamine - metabolism | Neostriatum - pathology | Parkinson Disease - pathology | Presynaptic Terminals - drug effects | Dyskinesias - pathology | Dyskinesias - physiopathology | Rats | Dyskinesias - etiology | Parkinson Disease - physiopathology | Rats, Sprague-Dawley | Nerve Degeneration - pathology | Environmental Exposure - adverse effects | Substantia Nigra - drug effects | Animals | Presynaptic Terminals - metabolism | Oxidative stress | Control | Parkinson's disease | Gene mutations | Neurons | Physiological aspects | Genetic aspects | Research | Risk factors
Journal Article
PloS one, ISSN 1932-6203, 07/2013, Volume 8, Issue 7, p. e69190
Journal Article
Biological Psychiatry, ISSN 0006-3223, 2011, Volume 69, Issue 5, pp. 407 - 414
Journal Article
PloS one, ISSN 1932-6203, 07/2014, Volume 9, Issue 7, p. e100869
Synucleinopathies, characterized by intracellular aggregation of alpha-synuclein protein, share a number of features in pathology and disease progression... 
ADENOASSOCIATED VIRAL VECTORS | DOPAMINE RELEASE | HUMAN ALPHA-SYNUCLEIN | ALZHEIMERS-DISEASE | MULTIDISCIPLINARY SCIENCES | MOUSE MODEL | NEURONS | LEWY BODY DISEASE | SUBSTANTIA-NIGRA | NEURODEGENERATIVE DISEASES | PARKINSONS-DISEASE | Dependovirus - genetics | Humans | Motor Activity - drug effects | Lewy Body Disease - genetics | Cell Nucleus - metabolism | Synapses - metabolism | Neostriatum - drug effects | Exploratory Behavior - drug effects | Microglia - pathology | Female | alpha-Synuclein - genetics | Prosencephalon - metabolism | Disease Models, Animal | Prosencephalon - pathology | Animals, Newborn | Dopamine Agonists - pharmacology | Neostriatum - pathology | Synapses - drug effects | Microglia - drug effects | Interneurons - drug effects | Lewy Body Disease - pathology | Axons - drug effects | Axons - metabolism | Rats | Cholinergic Neurons - drug effects | Rats, Sprague-Dawley | Disease Progression | Genetic Vectors - genetics | Interneurons - pathology | Protein Transport | Lewy Body Disease - metabolism | Pregnancy | Phenotype | Animals | Cell Nucleus - drug effects | Cholinergic Neurons - pathology | alpha-Synuclein - metabolism | Lewy Body Disease - physiopathology | Neonates | Neurosciences | Animal models | Parkinson's disease | Interneurons | Substantia nigra | Parkinsons disease | Forebrain | Synuclein | Autophagy | Neurodegeneration | Cortex (frontal) | Rodents | Surgery | Inclusion bodies | Neostriatum | Dementia disorders | Degeneration | Movement disorders | Dopamine receptors | Dopamine | Gene transfer | Neurodegenerative diseases | Neurons | Vectors (Biology) | Apomorphine | Microglia | Lewy bodies | Pathology | Locomotion | Overexpression | Biomarkers | Laboratory animals | Alzheimers disease | Dementia | Basic Medicine | Medical and Health Sciences | Medicin och hälsovetenskap | Medicinska och farmaceutiska grundvetenskaper | Neurovetenskaper
Journal Article
The European journal of neuroscience, ISSN 0953-816X, 2018, Volume 48, Issue 4, pp. 2050 - 2070
Functional recovery from central nervous system injury is likely to be partly due to a rearrangement of neural circuits. In this context, the corticobulbar... 
spinal cord injury | motor cortex | anterograde tracing | brainstem | nonhuman primate | cortical lesion | Parkinson | FUNCTIONAL RECOVERY | PONTOMEDULLARY RETICULAR-FORMATION | NOGO-A | NEUROSCIENCES | BRAIN-CELL AUTOTRANSPLANTATION | PRIMATE RETICULOSPINAL TRACT | MPTP-TREATED MONKEYS | A ANTIBODY TREATMENT | CORTICOSPINAL TRACT | SPINAL-CORD-INJURY | UNILATERAL SECTION | Parkinson Disease - therapy | Parkinson Disease - pathology | Nogo Proteins - immunology | Neuroanatomical Tract-Tracing Techniques | Motor Cortex - injuries | Macaca fascicularis | Cell Transplantation | Male | Transplantation, Autologous | Brain Injuries - therapy | Reticular Formation - pathology | Hand - pathology | Motor Cortex - pathology | Antibodies, Blocking - administration & dosage | Spinal Cord Injuries - pathology | Animals | Female | Spinal Cord Injuries - therapy | Brain Injuries - pathology | Pyramidal Tracts - pathology | Disease Models, Animal | Rhombencephalon - pathology | Viral antibodies | Primates | Antibodies | Neural circuitry | Tracers (Biology) | Central nervous system | Cortex (premotor) | Presynapse | Dopamine | Parkinson's disease | Neurodegenerative diseases | Parkinsons disease | Recovery of function | Reticular formation | Nervous system | Cortex (motor) | Spinal cord injury | Nogo protein | Neural networks | Neostriatum | Lesions | Movement disorders | Dopamine receptors | Research Report | Systems Neuroscience
Journal Article