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JACC (Journal of the American College of Cardiology), ISSN 0735-1097, 2009, Volume 53, Issue 5, pp. S35 - S42
Hyperglycemia can promote vascular complications by multiple mechanisms, with formation of advanced glycation end products and increased oxidative stress... 
Cardiovascular | Internal Medicine | atherosclerosis | glucose | inflammation | advanced glycation end products | oxidative stress | PROTEIN-KINASE-C | CARDIAC & CARDIOVASCULAR SYSTEMS | INCREASED EXPRESSION | SUPEROXIDE-PRODUCTION | INSULIN-RESISTANCE | HIGH GLUCOSE | ADHESION MOLECULE-1 ICAM-1 | PHENOTYPIC HETEROGENEITY | CORONARY-ARTERY-DISEASE | ENDOTHELIAL GROWTH-FACTOR | GLYCATION END-PRODUCTS | Cardiovascular Diseases - etiology | Inflammation - pathology | Reactive Oxygen Species | Oxidative Stress | Humans | Microvessels - pathology | Podocytes - pathology | Cardiovascular Diseases - pathology | Diabetes Complications - blood | Diabetes Complications - etiology | Neovascularization, Pathologic - complications | Inflammation - complications | Diabetes Mellitus, Type 2 - blood | Pericytes - pathology | Cardiovascular Diseases - blood | Endothelium, Vascular - pathology | Glycation End Products, Advanced - blood | Diabetes Complications - pathology | Diabetes Mellitus, Type 2 - pathology | Diabetes Mellitus, Type 2 - complications | Type 2 diabetes | Blood sugar | Lipids | Protein C | Endothelium | Animal pigments | Hyperglycemia | Synthesis | Atherosclerosis | Armored personnel carriers | Neovascularization | Kidney diseases | Cardiovascular diseases | Vascular endothelial growth factor | Blood circulation disorders | Wound healing | Muscular system | Kinases | Nitric oxide | Rodents
Journal Article
Nature, ISSN 0028-0836, 05/2012, Volume 485, Issue 7398, pp. 333 - 338
Peripartum cardiomyopathy (PPCM) is an often fatal disease that affects pregnant women who are near delivery, and it occurs more frequently in women with... 
UNITED-STATES | SKELETAL-MUSCLE | TRANSCRIPTIONAL COACTIVATOR PGC-1-ALPHA | MITOCHONDRIAL BIOGENESIS | EJECTION FRACTION | MULTIDISCIPLINARY SCIENCES | HEART-FAILURE | ANGIOTENSIN-II | PREECLAMPSIA | TYROSINE KINASE 1 | MICE LACKING | Pregnancy Complications, Cardiovascular - physiopathology | Neovascularization, Physiologic - drug effects | Humans | Male | Cardiomyopathies - etiology | Pre-Eclampsia - physiopathology | Bromocriptine - therapeutic use | Neovascularization, Pathologic - complications | Cardiomyopathies - physiopathology | Peroxisome Proliferator-Activated Receptor Gamma Coactivator 1-alpha | Trans-Activators - genetics | Female | Neovascularization, Pathologic - physiopathology | Vascular Endothelial Growth Factor A - pharmacology | Disease Models, Animal | Vascular Endothelial Growth Factor Receptor-1 - genetics | Cardiomyopathies - drug therapy | Heart - physiopathology | Cardiomyopathies - blood | Vascular Endothelial Growth Factor A - therapeutic use | Kaplan-Meier Estimate | Vascular Endothelial Growth Factor Receptor-1 - metabolism | Bromocriptine - pharmacology | Mice, Knockout | Pregnancy | Neovascularization, Physiologic - physiology | Vascular Endothelial Growth Factor Receptor-1 - pharmacology | Animals | Myocytes, Cardiac - drug effects | Trans-Activators - deficiency | Pregnancy Complications, Cardiovascular - drug therapy | Neovascularization, Pathologic - drug therapy | Heart - drug effects | Myocytes, Cardiac - metabolism | Trans-Activators - metabolism | Mice | Transcription Factors | Vascular Endothelial Growth Factor Receptor-1 - blood | Pregnancy Complications, Cardiovascular - etiology | Pregnancy Complications, Cardiovascular - blood | Cardiomyopathy | Physiological aspects | Research | Heart diseases | Vascular endothelial growth factor | Risk factors | Proteins | Angiogenesis | Musculoskeletal system | Womens health | Rodents | Preeclampsia | Tomography | Cardiomyocytes | Infections
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 8/2009, Volume 106, Issue 32, pp. 13505 - 13510
Diabetes is associated with poor outcomes following acute vascular occlusive events. This results in part from a failure to form adequate compensatory... 
Diabetic angiopathies | Wound healing | Diabetes complications | Diabetes mellitus | Fibroblasts | Hypoxia | Mice | Diabetes | Type 2 diabetes mellitus | Transactivation | HIF-1 | Hyperglycemia | Ischemia | Deferoxamine | MOBILIZATION | STABILIZATION | MULTIDISCIPLINARY SCIENCES | TRANSCRIPTION | VESSEL FORMATION | MELLITUS | ischemia | HYDROXYLASES | hyperglycemia | deferoxamine | METABOLISM | COMPLICATIONS | INDUCIBLE FACTOR-1 | ENDOTHELIAL GROWTH-FACTOR | Diabetes Mellitus - pathology | Reactive Oxygen Species - metabolism | Humans | Transcriptional Activation - drug effects | Hyperglycemia - complications | Vascular Endothelial Growth Factor A - metabolism | Neovascularization, Pathologic - complications | Neovascularization, Pathologic - pathology | Hypoxia-Inducible Factor 1, alpha Subunit - metabolism | Protein Binding - drug effects | Hyperglycemia - pathology | Diabetes Mellitus, Experimental - complications | Diabetes Complications - pathology | Diabetes Mellitus, Experimental - metabolism | Wound Healing - drug effects | Disease Models, Animal | Deferoxamine - pharmacology | Pyruvaldehyde - pharmacology | Hypoxia-Inducible Factor 1, alpha Subunit - genetics | Diabetes Complications - metabolism | Cells, Cultured | Diabetes Mellitus - metabolism | Glucose - pharmacology | Hypoxia - complications | Up-Regulation - drug effects | p300-CBP Transcription Factors - metabolism | Animals | Diabetes Mellitus, Experimental - pathology | Research | Neovascularization | Vascular endothelial growth factor | Biological Sciences
Journal Article
Stroke, ISSN 0039-2499, 01/2015, Volume 46, Issue 1, pp. 182 - 189
Journal Article
Journal Article
Human Molecular Genetics, ISSN 0964-6906, 08/2010, Volume 19, Issue 15, pp. 3011 - 3020
Journal Article
Blood, ISSN 0006-4971, 08/2006, Volume 108, Issue 4, pp. 1158 - 1164
We present results of 2 similarly designed but separate phase 2 studies involving single-agent lenalidomide (CC-5013, Revlimid) in a total of 68 patients with... 
MULTIPLE-MYELOMA | GROWTH-FACTOR-BETA | LOW-DOSE THALIDOMIDE | CC-5013 | MYELOPROLIFERATIVE-DISORDERS | ANGIOGENESIS | ANALOGS | MUTATION | CELL TRANSPLANTATION | MYELODYSPLASTIC-SYNDROMES | HEMATOLOGY | Humans | Male | Primary Myelofibrosis - pathology | Neovascularization, Pathologic - complications | Primary Myelofibrosis - blood | Thrombocytopenia - genetics | Thalidomide - analogs & derivatives | Anemia - genetics | Anemia - drug therapy | Anemia, Myelophthisic - genetics | Time Factors | Splenomegaly - blood | Neutropenia - blood | Thalidomide - adverse effects | Primary Myelofibrosis - complications | Splenomegaly - genetics | Administration, Oral | Anemia, Myelophthisic - pathology | Thrombocytopenia - chemically induced | Thalidomide - administration & dosage | Thrombocytopenia - pathology | Remission Induction | Neovascularization, Pathologic - drug therapy | Anemia, Myelophthisic - complications | Sequence Deletion | Splenomegaly - drug therapy | Anemia - blood | Anemia - pathology | Splenomegaly - pathology | Anemia, Myelophthisic - blood | Neovascularization, Pathologic - blood | Neovascularization, Pathologic - pathology | Janus Kinase 2 | Splenomegaly - complications | Protein-Tyrosine Kinases - genetics | Thrombocytopenia - drug therapy | Neutropenia - pathology | Adult | Female | Neutropenia - chemically induced | Neutropenia - genetics | Primary Myelofibrosis - drug therapy | Proto-Oncogene Proteins - genetics | Thrombocytopenia - blood | Anemia, Myelophthisic - drug therapy | Point Mutation | Anemia - complications | Primary Myelofibrosis - genetics | Platelet Count | Neovascularization, Pathologic - genetics | Aged | Hemoglobins - analysis
Journal Article
Journal of Molecular Medicine, ISSN 0946-2716, 11/2013, Volume 91, Issue 11, pp. 1315 - 1327
Journal Article