X
Search Filters
Format Format
Format Format
X
Sort by Item Count (A-Z)
Filter by Count
Journal Article (24915) 24915
Conference Proceeding (19) 19
Book Chapter (16) 16
Newsletter (14) 14
Publication (11) 11
Book / eBook (7) 7
Dissertation (6) 6
Magazine Article (5) 5
Book Review (1) 1
Web Resource (1) 1
more...
Subjects Subjects
Subjects Subjects
X
Sort by Item Count (A-Z)
Filter by Count
humans (20238) 20238
animals (15445) 15445
angiogenesis (11676) 11676
mice (9128) 9128
female (8449) 8449
neovascularization, pathologic - metabolism (7874) 7874
oncology (7082) 7082
male (7016) 7016
neovascularization, pathologic (6553) 6553
cancer (5527) 5527
neovascularization, pathologic - pathology (5465) 5465
expression (5339) 5339
cell line, tumor (4653) 4653
endothelial growth-factor (4559) 4559
vascular endothelial growth factor a - metabolism (4265) 4265
vascular endothelial growth factor (3938) 3938
neovascularization, pathologic - drug therapy (3669) 3669
middle aged (3568) 3568
immunohistochemistry (3411) 3411
cell biology (3241) 3241
neovascularization, pathologic - genetics (3195) 3195
metastasis (2987) 2987
neovascularization (2938) 2938
aged (2918) 2918
adult (2851) 2851
tumors (2836) 2836
apoptosis (2767) 2767
cell proliferation (2743) 2743
signal transduction (2713) 2713
mice, nude (2658) 2658
vegf (2592) 2592
biochemistry & molecular biology (2472) 2472
cells (2383) 2383
growth (2168) 2168
tumor angiogenesis (2138) 2138
cells, cultured (2135) 2135
disease models, animal (2115) 2115
rats (2107) 2107
research (2017) 2017
analysis (1995) 1995
mice, inbred c57bl (1972) 1972
in-vivo (1968) 1968
prognosis (1934) 1934
endothelial cells - metabolism (1897) 1897
angiogenesis inhibitors - pharmacology (1893) 1893
gene expression regulation, neoplastic (1884) 1884
neoplasms - blood supply (1860) 1860
gene expression (1845) 1845
hypoxia (1810) 1810
cell proliferation - drug effects (1798) 1798
neovascularization, pathologic - prevention & control (1771) 1771
mice, inbred balb c (1748) 1748
neoplasms - pathology (1729) 1729
medicine, research & experimental (1714) 1714
neoplasms - metabolism (1714) 1714
pathology (1638) 1638
neovascularization, pathologic - physiopathology (1618) 1618
xenograft model antitumor assays (1579) 1579
endothelium (1559) 1559
proteins (1520) 1520
endothelial-cells (1492) 1492
endothelial cells (1478) 1478
vascular endothelial growth factor a - genetics (1475) 1475
activation (1472) 1472
in-vitro (1444) 1444
angiogenesis inhibitors - therapeutic use (1440) 1440
inflammation (1440) 1440
endothelium, vascular - metabolism (1439) 1439
carcinoma (1432) 1432
vascular endothelial growth factor a (1402) 1402
disease progression (1400) 1400
health aspects (1395) 1395
tumor cells, cultured (1369) 1369
inhibition (1352) 1352
pharmacology & pharmacy (1341) 1341
cell movement (1338) 1338
research article (1328) 1328
physiological aspects (1327) 1327
development and progression (1287) 1287
blotting, western (1283) 1283
vascular endothelial growth factors (1280) 1280
neoplasm transplantation (1270) 1270
up-regulation (1257) 1257
antineoplastic agents - pharmacology (1256) 1256
proliferation (1252) 1252
growth-factor (1243) 1243
neoplasm invasiveness (1240) 1240
care and treatment (1238) 1238
breast-cancer (1222) 1222
reverse transcriptase polymerase chain reaction (1205) 1205
aged, 80 and over (1204) 1204
multidisciplinary sciences (1198) 1198
neoplasm metastasis (1188) 1188
therapy (1184) 1184
neoplasms - drug therapy (1172) 1172
signal transduction - drug effects (1167) 1167
cell movement - drug effects (1149) 1149
apoptosis - drug effects (1148) 1148
time factors (1145) 1145
hematology (1104) 1104
more...
Library Location Library Location
Language Language
Language Language
X
Sort by Item Count (A-Z)
Filter by Count
English (24369) 24369
Chinese (485) 485
Japanese (174) 174
Polish (89) 89
French (85) 85
Russian (75) 75
German (26) 26
Spanish (21) 21
Hungarian (16) 16
Italian (9) 9
Korean (6) 6
Dutch (5) 5
Swedish (4) 4
Portuguese (3) 3
Danish (2) 2
Romanian (2) 2
Serbian (2) 2
Ukrainian (2) 2
Bulgarian (1) 1
Czech (1) 1
Finnish (1) 1
Turkish (1) 1
more...
Publication Date Publication Date
Click on a bar to filter by decade
Slide to change publication date range


Journal Article
Proceedings of the National Academy of Sciences - PNAS, ISSN 1091-6490, 2015, Volume 112, Issue 39, pp. 12163 - 12168
Pathologic ocular neovascularization commonly causes blindness. It is critical to identify the factors altered in pathologically proliferating versus normally quiescent vessels to develop effective targeted therapeutics... 
MiR-150 | MicroRNA | Retinopathy | Neovascularization | Endothelial cells | B-CELL DEVELOPMENT | PROLIFERATIVE RETINOPATHY | endothelial cells | ANGIOGENESIS | OXYGEN-INDUCED RETINOPATHY | MULTIDISCIPLINARY SCIENCES | neovascularization | MOUSE | C-MYB | MODEL | HYPOXIA | retinopathy | microRNA | miR-150 | INDUCED CHOROIDAL NEOVASCULARIZATION | Intravitreal Injections | Humans | 3' Untranslated Regions - genetics | Laser Capture Microdissection | Molecular Sequence Data | MicroRNAs - metabolism | Intracellular Signaling Peptides and Proteins - metabolism | Retinal Neovascularization - metabolism | MicroRNAs - pharmacology | Luciferases | Base Sequence | HEK293 Cells | Retinal Vessels - cytology | Receptors, CXCR4 - genetics | Retinal Neovascularization - genetics | Membrane Proteins - metabolism | Intracellular Signaling Peptides and Proteins - genetics | Retinal Vessels - metabolism | Endothelial Cells - metabolism | Membrane Proteins - genetics | MicroRNAs - administration & dosage | Frizzled Receptors - metabolism | Mice, Knockout | Receptors, CXCR4 - metabolism | Gene Expression Regulation - drug effects | Cell Movement - drug effects | Animals | Choroidal Neovascularization - metabolism | Frizzled Receptors - genetics | Cell Proliferation - drug effects | Mice | MicroRNAs - genetics | Choroidal Neovascularization - genetics | Physiological aspects | Genetic aspects | Blindness | Biological Sciences
Journal Article
Investigative ophthalmology & visual science, ISSN 1552-5783, 2012, Volume 53, Issue 6, pp. 2968 - 2977
PURPOSE. Retinopathy of prematurity (ROP) is a leading cause of blindness in children and is, in its most severe form, characterized by uncontrolled growth of vision-threatening pathologic vessels... 
RETINAL NEOVASCULARIZATION | BLOCKERS | MOUSE MODEL | RAT RETINA | OPHTHALMOLOGY | INFANTILE HEMANGIOMAS | FACTOR EXPRESSION | PREMATURITY | INFANCY | ENDOTHELIAL GROWTH-FACTOR | Up-Regulation | Cell Proliferation | Angiopoietin-2 - genetics | Humans | Gene Expression Profiling | Retinal Neovascularization - metabolism | Vascular Endothelial Growth Factor A - metabolism | Angiopoietin-1 - metabolism | Vascular Endothelial Growth Factor A - genetics | Angiopoietin-2 - metabolism | Dose-Response Relationship, Drug | Receptors, Vascular Endothelial Growth Factor - metabolism | Retina - cytology | Propranolol - administration & dosage | Injections, Subcutaneous | Retinal Neovascularization - prevention & control | Erythropoietin - metabolism | Receptors, Vascular Endothelial Growth Factor - genetics | Drug Evaluation, Preclinical | Receptors, Adrenergic, beta - metabolism | Adrenergic beta-Antagonists - administration & dosage | Infant, Newborn | Disease Models, Animal | RNA - metabolism | Animals, Newborn | Oxygen - toxicity | Enzyme-Linked Immunosorbent Assay | Administration, Oral | Injections, Intraperitoneal | Reverse Transcriptase Polymerase Chain Reaction | Angiopoietin-1 - genetics | Erythropoietin - genetics | Animals | Retinopathy of Prematurity - prevention & control | Mice | Retinopathy of Prematurity - metabolism | In Vitro Techniques | Endothelial Cells - drug effects | Disease Models | Other Medical Sciences | Endothelial Cells/drug effects | Oxygen/toxicity | Drug Evaluation | Vascular Endothelial Growth Factor A/genetics/metabolism | Infant | Propranolol/administration & dosage | Adrenergic | Oral | Intraperitoneal | RNA/metabolism | Receptors | Retinopathy of Prematurity/metabolism/prevention & control | beta/metabolism | Preclinical | Adrenergic beta-Antagonists/administration & dosage | Drug | Administration | Retinal Neovascularization/metabolism/prevention & control | Subcutaneous | Angiopoietin-2/genetics/metabolism | Erythropoietin/genetics/metabolism | Retina/cytology | Angiopoietin-1/genetics/metabolism | Injections | Newborn | Annan medicin och hälsovetenskap | Animal | Dose-Response Relationship | Vascular Endothelial Growth Factor/genetics/metabolism
Journal Article
Arteriosclerosis, Thrombosis, and Vascular Biology, ISSN 1079-5642, 03/2014, Volume 34, Issue 3, pp. 581 - 586
Journal Article
Journal Article
Investigative Ophthalmology and Visual Science, ISSN 0146-0404, 04/2011, Volume 52, Issue 5, pp. 2809 - 2816
PURPOSE. Macular telangiectasia (MacTel) is a vision-threatening retinal disease with unknown pathogenesis and no approved treatment. Very low-density... 
IDIOPATHIC MACULAR TELANGIECTASIA | OXIDATIVE STRESS | SUBRETINAL NEOVASCULARIZATION | MOUSE MODEL | OPHTHALMOLOGY | ANGIOMATOUS PROLIFERATION | PARAFOVEAL TELANGIECTASIS | INTRAVITREAL BEVACIZUMAB | DENSITY LIPOPROTEIN RECEPTOR | TUMOR-GROWTH | ENDOTHELIAL GROWTH-FACTOR
Journal Article
Gastroenterology, ISSN 0016-5085, 2016, Volume 150, Issue 4, pp. 982 - 997.e30
.... In a search for ways to inhibit pathologic production or activities of VEGF without affecting its normal production or functions, we investigated the post-transcriptional regulation of VEGF... 
Gastroenterology and Hepatology | Alternative RNA Processing | Portal Hypertension | Cytoplasmic Polyadenylation | CPEB | CELLS | RECRUITMENT | VEGF | P110-ALPHA ISOFORM | TRANSLATIONAL CONTROL | MESSENGER-RNA | SIGNALING PATHWAY | GENE-EXPRESSION | MICE | GASTROENTEROLOGY & HEPATOLOGY | PORTAL-HYPERTENSION | Hypertension, Portal - genetics | RNA-Binding Proteins - genetics | mRNA Cleavage and Polyadenylation Factors - metabolism | Humans | Middle Aged | Transcription Factors - deficiency | Neovascularization, Pathologic | Male | Vascular Endothelial Growth Factor A - metabolism | Mice, 129 Strain | RNA, Messenger - metabolism | mRNA Cleavage and Polyadenylation Factors - deficiency | Case-Control Studies | Transfection | RNA Interference | Time Factors | Liver Cirrhosis - metabolism | Adult | Female | 3' Untranslated Regions | mRNA Cleavage and Polyadenylation Factors - genetics | Disease Models, Animal | Cell Line | Signal Transduction | Hypertension, Portal - pathology | Mice, Inbred C57BL | RNA, Messenger - genetics | Gene Expression Regulation | Liver Cirrhosis, Biliary - pathology | Transcription Factors - genetics | Liver Cirrhosis, Biliary - genetics | Rats, Sprague-Dawley | Transcription Factors - metabolism | Animals | Liver Cirrhosis - virology | Hypertension, Portal - metabolism | Polyadenylation | Liver Cirrhosis - pathology | Chronic Disease | Liver Cirrhosis, Biliary - metabolism | RNA-Binding Proteins - metabolism | Hypertension | Liver diseases | Genetic research | Research institutes | Binding proteins | Vascular endothelial growth factor | Protein binding
Journal Article
Journal Article
Journal Article
Nature (London), ISSN 1476-4687, 2015, Volume 520, Issue 7546, pp. 192 - 197
The metabolism of endothelial cells during vessel sprouting remains poorly studied... 
OXIDATION | DISTRIBUTIONS | GLYCOLYSIS | REDUCTIVE GLUTAMINE-METABOLISM | ANGIOGENESIS | MULTIDISCIPLINARY SCIENCES | TUMOR | PROLIFERATION | GROWTH-FACTOR | CANCER | CULTURE | Carnitine O-Palmitoyltransferase - genetics | Human Umbilical Vein Endothelial Cells - metabolism | Blood Vessels - metabolism | Blood Vessels - pathology | Humans | Neovascularization, Pathologic - pathology | Nucleotides - chemistry | Carnitine O-Palmitoyltransferase - antagonists & inhibitors | Adenosine Triphosphate - metabolism | Carnitine O-Palmitoyltransferase - metabolism | Human Umbilical Vein Endothelial Cells - cytology | Nucleotides - biosynthesis | Nucleotides - pharmacology | Oxidation-Reduction - drug effects | DNA - biosynthesis | Carnitine O-Palmitoyltransferase - deficiency | Fatty Acids - metabolism | Disease Models, Animal | Carbon - metabolism | Fatty Acids - chemistry | Human Umbilical Vein Endothelial Cells - drug effects | Endothelial Cells - metabolism | Gene Silencing | Acetic Acid - pharmacology | Blood Vessels - cytology | Citric Acid Cycle | Animals | Retinopathy of Prematurity - drug therapy | Endothelial Cells - cytology | Neovascularization, Pathologic - drug therapy | Blood Vessels - drug effects | Cell Line, Tumor | Glucose - metabolism | Human Umbilical Vein Endothelial Cells - pathology | Cell Proliferation - drug effects | Mice | Neovascularization, Pathologic - metabolism | Retinopathy of Prematurity - metabolism | Endothelial Cells - enzymology | Endothelial Cells - drug effects | Retinopathy of Prematurity - pathology | Physiological aspects | Deoxyribonucleotides | Fatty acids | Carbon | Endothelium | Proteins | Angiogenesis | Protein synthesis | Glucose | Kinases | Deoxyribonucleic acid--DNA | Cells
Journal Article
Scientific Reports, ISSN 2045-2322, 01/2016, Volume 6, Issue 1, p. 18828
.... Netrin-4 displays pathologic roles in tumorigenesis and neovascularization. To answer the question whether netrin-4 acts either pro-or anti-angiogenic, angiogenesis in the retina was assessed in Ntn-4... 
VASCULAR DEVELOPMENT | RETINAL VASCULATURE | ANGIOGENESIS | OXYGEN-INDUCED RETINOPATHY | MULTIDISCIPLINARY SCIENCES | MOUSE | MICE | CHOROIDAL NEOVASCULARIZATION | IDENTIFICATION | EXPRESSION | ENDOTHELIAL GROWTH-FACTOR | Nerve Growth Factors - deficiency | Retina - metabolism | Nerve Growth Factors - metabolism | Retinal Neovascularization - metabolism | Vascular Endothelial Growth Factor A - metabolism | RNA, Messenger - metabolism | Vascular Endothelial Growth Factor A - genetics | Retinal Diseases - etiology | Retinal Diseases - pathology | Retinal Neovascularization - genetics | Eye Diseases - pathology | Netrin Receptors | Disease Models, Animal | Retinal Diseases - diagnosis | RNA, Messenger - genetics | Eye Diseases - etiology | Gene Expression Regulation | Receptors, Cell Surface - metabolism | Netrins | Eye Diseases - diagnosis | Mice, Knockout | Animals | Choroidal Neovascularization - metabolism | Nerve Growth Factors - genetics | Neovascularization, Pathologic - genetics | Neuroglia - metabolism | Mice | Choroidal Neovascularization - genetics | Retina - pathology | Receptors, Cell Surface - genetics | Retinopathy | Astrocytes | Netrin-1 | Colorectal carcinoma | Blood vessels | Homeostasis | Retina | Inflammation | Gene expression | Mimicry | Vascularization | Membrane proteins | Eye | Angiogenesis | Rodents | Hypoxia | Tumorigenesis | Receptor mechanisms | DCC protein
Journal Article
Blood, ISSN 1528-0020, 2012, Volume 120, Issue 14, pp. 2925 - 2929
Inflammatory cytokines and growth factors drive angiogenesis independently; however, their integrated role in pathologic and physiologic angiogenesis is not fully understood... 
RETINAL NEOVASCULARIZATION | SUPPRESSOR | OXYGEN-INDUCED RETINOPATHY | ENDOTHELIAL-CELLS | GROWTH | RESISTANCE | HEMATOLOGY | EXPRESSION | BRAIN | Endothelium, Vascular - cytology | Tumor Necrosis Factor-alpha - metabolism | Cell Proliferation | TOR Serine-Threonine Kinases - metabolism | Tumor Necrosis Factor-alpha - genetics | Male | Insulin-Like Growth Factor I - genetics | Carcinoma, Lewis Lung - blood supply | Neovascularization, Pathologic - etiology | Melanoma, Experimental - prevention & control | TOR Serine-Threonine Kinases - genetics | Integrases - metabolism | Neovascularization, Pathologic - prevention & control | Real-Time Polymerase Chain Reaction | Paraneoplastic Syndromes, Ocular - prevention & control | STAT3 Transcription Factor - genetics | STAT3 Transcription Factor - metabolism | Paraneoplastic Syndromes, Ocular - pathology | Melanoma, Experimental - blood supply | Signal Transduction | Mice, Inbred C57BL | RNA, Messenger - genetics | Melanoma, Experimental - pathology | Reverse Transcriptase Polymerase Chain Reaction | Blotting, Western | Carcinoma, Lewis Lung - prevention & control | Suppressor of Cytokine Signaling 3 Protein | Animals | Endothelium, Vascular - metabolism | Hypoxia - pathology | Carcinoma, Lewis Lung - pathology | Suppressor of Cytokine Signaling Proteins - physiology | Mice | Insulin-Like Growth Factor I - metabolism | Brief Report | Vascular Biology
Journal Article