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2006, ISBN 9780470012949, xvii, 350 p., [8] p. of plates
Book
Journal of biochemistry (Tokyo), ISSN 0021-924X, 2012, Volume 153, Issue 1, pp. 13 - 19
Vascular endothelial growth factors (VEGFs) belong to the platelet-derived growth factor supergene family, and they play central roles in the regulation of... 
VEGFR | hypoxia | preeclampsia | VEGF | tumor angiogenesis | SUPPRESSES TUMOR-GROWTH | CELLS | TYROSINE KINASE | BIOCHEMISTRY & MOLECULAR BIOLOGY | BONE-MARROW | VEGF RECEPTOR-1 | BEVACIZUMAB PLUS IRINOTECAN | NUTRIENT STARVATION | DNA-SYNTHESIS | FLT-1 | IN-VIVO | Neoplasms - metabolism | Vascular Endothelial Growth Factor Receptor-1 - antagonists & inhibitors | Humans | Male | Neoplasm Proteins - antagonists & inhibitors | Vascular Endothelial Growth Factor A - metabolism | Vascular Endothelial Growth Factor A - antagonists & inhibitors | Molecular Targeted Therapy | Neoplasm Proteins - metabolism | Vascular Endothelial Growth Factor A - chemistry | Vascular Endothelial Growth Factor Receptor-2 - antagonists & inhibitors | Angiogenesis Inhibitors - therapeutic use | Female | Pre-Eclampsia - metabolism | Lymphangiogenesis | Angiogenesis Inhibitors - pharmacology | Vascular Endothelial Growth Factor Receptor-2 - metabolism | Neoplasm Proteins - chemistry | Vascular Endothelial Growth Factor Receptor-1 - metabolism | Vascular Endothelial Growth Factor Receptor-1 - chemistry | Neoplasms - drug therapy | Pregnancy | Vascular Endothelial Growth Factor Receptor-2 - chemistry | Animals | Pre-Eclampsia - drug therapy | Signal Transduction - drug effects | Neovascularization, Pathologic - drug therapy | Neovascularization, Pathologic - metabolism | Neovascularization, Physiologic | JB Reviews
Journal Article
Journal Article
Journal Article
Glia, ISSN 0894-1491, 2019, Volume 67, Issue 2, pp. 332 - 344
Ischemia‐induced angiogenesis contributes to various neuronal and retinal diseases, and often results in neurodegeneration and visual impairment. Current... 
CCL2 | miR‐30a‐5p | microglia | angiogenesis | ischemia | miR-30a-5p | CHEMOKINE LIGAND 2 | ACTIVATION | OXYGEN-INDUCED RETINOPATHY | MACROPHAGES | MOUSE | NEUROSCIENCES | DEFICIENCY | DEGENERATION | INFLAMMATION | CENTRAL-NERVOUS-SYSTEM | BRAIN | Microglia - metabolism | Neovascularization, Physiologic - drug effects | Apoptosis - drug effects | Humans | Apoptosis - genetics | MicroRNAs - metabolism | RNA, Messenger - metabolism | fas Receptor - metabolism | Lectins - metabolism | Chemokine CCL2 - metabolism | RNA Interference - physiology | Disease Models, Animal | Animals, Newborn | Cell Proliferation - genetics | Endothelial Cells - metabolism | Gene Expression Regulation - genetics | Mice, Inbred C57BL | Chemokine CCL2 - genetics | Mice, Transgenic | Gene Expression Regulation - drug effects | Neovascularization, Physiologic - physiology | Animals | Neovascularization, Pathologic - drug therapy | Cell Proliferation - drug effects | Mice | MicroRNAs - genetics | Neovascularization, Pathologic - metabolism | Cell Line, Transformed | Endothelial Cells - drug effects | Diabetic retinopathy | Medical colleges | Medical research | Analysis | Physiological aspects | Medicine, Experimental | Ophthalmology | Neovascularization | Vascular endothelial growth factor | Endothelium | Disabled people | Retinopathy | Cell survival | Therapeutic applications | Pathogenesis | Crosstalk | Diabetes mellitus | Retina | Tissues | Endothelial cells | Microglia | Vascularization | Angiogenesis | Phagocytes | FasL protein | Ischemia | Neurodegeneration | Inhibition | Repair | Monocyte chemoattractant protein 1 | Cell migration | Apoptosis
Journal Article
Cell stem cell, ISSN 1934-5909, 2015, Volume 16, Issue 1, pp. 51 - 66
Mesenchymal stem cells (MSCs) reside in the perivascular niche of many organs, including kidney, lung, liver, and heart, although their roles in these tissues... 
REGULATOR | ORIGIN | SONIC HEDGEHOG | PATHWAY | BONE-MARROW NICHE | MYOFIBROBLASTS | NG2 PROTEOGLYCAN | EXPRESSION | MESENCHYMAL STEM-CELLS | GROWTH FACTOR-AA | CELL & TISSUE ENGINEERING | CELL BIOLOGY | Organ Specificity - drug effects | Diphtheria Toxin - pharmacology | Neovascularization, Physiologic - drug effects | Pericytes - drug effects | Blood Vessels - metabolism | Blood Vessels - pathology | Humans | Fibrosis - metabolism | Cell Lineage - drug effects | Myofibroblasts - metabolism | Mesenchymal Stromal Cells - cytology | Mesenchymal Stromal Cells - ultrastructure | Kruppel-Like Transcription Factors - metabolism | Pericytes - pathology | Bone Marrow Cells - drug effects | Colony-Forming Units Assay | Aorta - physiopathology | Homeostasis - drug effects | Heart Ventricles - pathology | Receptor, Platelet-Derived Growth Factor beta - metabolism | Mesenchymal Stromal Cells - drug effects | Endothelial Cells - metabolism | Aorta - drug effects | Pericytes - metabolism | Cells, Cultured | Proteoglycans - metabolism | Antigens - metabolism | Aorta - pathology | Myofibroblasts - cytology | Animals | Heart Ventricles - physiopathology | Cell Differentiation - drug effects | Endothelial Cells - cytology | Blood Vessels - drug effects | Mice | Stem Cell Niche - drug effects | Zinc Finger Protein GLI1 | Fibrosis - pathology | Bone Marrow Cells - metabolism | Endothelial Cells - drug effects | Heart Ventricles - drug effects
Journal Article
Journal Article
Molecular Endocrinology, ISSN 1944-9917, 2016, Volume 30, Issue 6, pp. 660 - 676
Journal Article