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The American Journal of Human Genetics, ISSN 0002-9297, 09/2016, Volume 99, Issue 3, pp. 607 - 623
Journal Article
Neurodegenerative Diseases, ISSN 1660-2854, 09/2012, Volume 11, Issue 1, pp. 33 - 41
Background: Alzheimer’s disease (AD) is characterized by progressive neuronal loss and cognitive decline. Epidemiological studies suggest that the risk of AD... 
Original Paper | Neuroinflammation | β-Secretase-1 | Alzheimer's disease | Insulin-degrading enzyme | Spatial memory | APP | NEPRILYSIN | PROTEIN | ALZHEIMERS-DISEASE | PATHOLOGY | NEUROSCIENCES | PEPTIDE | CLINICAL NEUROLOGY | AMYLOID-BETA | GENDER | TRANSGENIC MOUSE MODEL | beta-Secretase-1 | BRAIN | Amyloid Precursor Protein Secretases - genetics | Glial Fibrillary Acidic Protein - genetics | Microglia - metabolism | Humans | Male | Aspartic Acid Endopeptidases - genetics | Interleukin-1beta - genetics | Glial Fibrillary Acidic Protein - metabolism | RNA, Messenger - metabolism | Antigens, CD - genetics | Antigens, CD - metabolism | Learning Disorders - pathology | CD11b Antigen - genetics | Interleukin-1beta - metabolism | Amyloid beta-Peptides - metabolism | Female | Neprilysin - genetics | Insulysin - metabolism | Enzyme-Linked Immunosorbent Assay | Maze Learning - physiology | Gene Expression Regulation - genetics | Presenilin-1 - genetics | Mice, Transgenic | Neprilysin - metabolism | Reaction Time - genetics | Sex Characteristics | Antigens, Differentiation, Myelomonocytic - genetics | Amyloid Precursor Protein Secretases - metabolism | Amyloid beta-Protein Precursor - genetics | Animals | Analysis of Variance | Antigens, Differentiation, Myelomonocytic - metabolism | Aspartic Acid Endopeptidases - metabolism | Insulysin - genetics | Learning Disorders - genetics | Mice | CD11b Antigen - metabolism
Journal Article
Journal of Endodontics, ISSN 0099-2399, 2009, Volume 35, Issue 10, pp. 1368 - 1376
Abstract Introduction During development and regeneration, odontogenesis and osteogenesis are initiated by a cascade of signals driven by several master... 
Endocrinology & Metabolism | Dentistry | odontoblast/osteoblast differentiation | periodontal ligament cells | Dental pulp cells | gene expression | SIGNALING PATHWAYS | ACTIVATION | NOTCH | CROSS-TALK | IN-VITRO | DENTISTRY, ORAL SURGERY & MEDICINE | MINERALIZATION | GROWTH | WNT | Cyclin D2 - genetics | Humans | Odontogenesis - genetics | Periodontal Ligament - injuries | Receptors, Notch - genetics | Insulin-Like Growth Factor I - genetics | Dentin - pathology | Receptor Cross-Talk - physiology | Cell Differentiation - genetics | Periodontal Ligament - cytology | Wnt Proteins - genetics | Regeneration - genetics | Dental Cementum - physiopathology | Adult | Neprilysin - genetics | Cadherins - genetics | Osteogenesis - genetics | Disease Models, Animal | Bone Morphogenetic Protein 2 - genetics | Dentin - physiopathology | Dental Pulp - injuries | Gene Expression Regulation - genetics | Membrane Proteins - genetics | Alveolar Process - pathology | Cells, Cultured | Intercellular Signaling Peptides and Proteins - genetics | Dental Pulp - cytology | Rats | Dental Cementum - pathology | Periodontal Ligament - physiology | Genes, APC - physiology | Animals | Transforming Growth Factor beta - genetics | Dental Pulp - physiology | Stem Cells - physiology | Keratin-15 - genetics | Alveolar Process - physiopathology | Gene expression | Stem cells | Genes
Journal Article
The American Journal of Human Genetics, ISSN 0002-9297, 03/2012, Volume 90, Issue 3, pp. 524 - 532
Journal Article
Journal of Neurochemistry, ISSN 0022-3042, 2010, Volume 115, Issue 1, pp. 47 - 57
P>The brain steady state level of beta-amyloid (A beta) is determined by the balance between its production and removal, the latter through egress across blood... 
insulin-degrading enzyme | Alzheimer's disease brain | neprilysin | endothelin-converting enzyme | degradation | Alzheimer's disease | amyloid-degrading enzymes | β-amyloid | MOUSE-BRAIN | PROTEIN A-BETA | BIOCHEMISTRY & MOLECULAR BIOLOGY | DISEASE PATHOGENESIS | MILD COGNITIVE IMPAIRMENT | NEUROSCIENCES | SENILE PLAQUES | beta-amyloid | AMYLOID-BETA PEPTIDE | FLUOROMETRIC ASSAY | HUMAN CEREBRAL-CORTEX | PLAQUE-FORMATION | TRANSGENIC MICE | Metalloendopeptidases - genetics | Tissue Banks | Prospective Studies | Aspartic Acid Endopeptidases - biosynthesis | Humans | Brain - enzymology | Insulysin - biosynthesis | Male | Aspartic Acid Endopeptidases - genetics | Gene Expression Regulation, Enzymologic - physiology | Neprilysin - biosynthesis | Endothelin-Converting Enzymes | RNA - genetics | Synaptophysin - genetics | Amyloid beta-Peptides - genetics | Aged, 80 and over | Female | Neprilysin - genetics | Alzheimer Disease - psychology | Brain Chemistry - genetics | Cognition Disorders - enzymology | Amyloid beta-Peptides - biosynthesis | Electrophoresis, Polyacrylamide Gel | Cognition Disorders - genetics | DNA Primers | Reverse Transcriptase Polymerase Chain Reaction | Blotting, Western | Metalloendopeptidases - biosynthesis | RNA - biosynthesis | Gene Expression Regulation, Enzymologic - genetics | Synaptophysin - biosynthesis | Alzheimer Disease - metabolism | Indicators and Reagents | Insulysin - genetics | Aged | Alzheimer Disease - genetics | Medical colleges | Brain | Messenger RNA | Proteases | Pharmacy | Fluorescence | Public health | Target marketing | Neurochemistry | Proteins | Enzymes | Alzheimers disease | Ribonucleic acid--RNA
Journal Article
The Journal of Cell Biology, ISSN 0021-9525, 8/2007, Volume 178, Issue 5, pp. 829 - 841
The tumor necrosis factor type 1 death receptor (TNFR1) contributes to apoptosis. TNFR1, a subgroup of the TNFR superfamily, contains a cytoplasmic death... 
Pathology | Neurons | Transgenic animals | Entorhinal cortex | Memory | Antibodies | Amyloids | Alzheimers disease | Hippocampus | Microglia | TNF RECEPTOR | SECRETASE EXPRESSION | KAPPA-B ACTIVATION | COGNITIVE DECLINE | CEREBRAL-HEMORRHAGE | IN-VIVO | PRECURSOR PROTEIN | TRANSGENIC MOUSE MODEL | NEURONAL LOSS | NEOCORTICAL NEUROFIBRILLARY TANGLES | CELL BIOLOGY | Tumor Necrosis Factor-alpha - metabolism | Amyloid Precursor Protein Secretases - genetics | Memory Disorders - physiopathology | Microglia - metabolism | Memory Disorders - genetics | Humans | Memory Disorders - metabolism | NF-kappa B - metabolism | Neurons - cytology | Aspartic Acid Endopeptidases - genetics | Receptors, Tumor Necrosis Factor, Type I - metabolism | Alzheimer Disease - pathology | Amyloid beta-Peptides - genetics | Gene Deletion | Amyloid beta-Peptides - metabolism | Neprilysin - genetics | Neurons - metabolism | Cerebral Amyloid Angiopathy - genetics | Disease Models, Animal | Microglia - cytology | Insulysin - metabolism | Promoter Regions, Genetic | Alzheimer Disease - physiopathology | Cerebral Amyloid Angiopathy - physiopathology | Gene Expression Regulation | Behavior, Animal - physiology | Mice, Transgenic | Neprilysin - metabolism | Receptors, Tumor Necrosis Factor, Type I - genetics | Mice, Knockout | Learning - physiology | Amyloid Precursor Protein Secretases - metabolism | Cerebral Amyloid Angiopathy - pathology | Animals | Aspartic Acid Endopeptidases - metabolism | Alzheimer Disease - metabolism | Insulysin - genetics | Mice | Alzheimer Disease - genetics
Journal Article
Oncology Reports, ISSN 1021-335X, 5/2015, Volume 33, Issue 5, pp. 2583 - 2591
Journal Article
Molecular Medicine Reports, ISSN 1791-2997, 02/2017, Volume 16, Issue 2, pp. 1635 - 1642
Solid pseudopapillary neoplasm (SPN) of the pancreas is a low-grade malignant neoplasm that accounts for ~5% of cystic pancreatic tumors and ~0.9-2.7% of... 
solid pseudopapillary neoplasm | tumor associated genes | functional enrichment analysis | differentially expressed genes | protein-protein interaction | Differentially expressed genes | Solid pseudopapillary neoplasm | Functional enrichment analysis | Tumor associated genes | Protein-protein interaction | MEDICINE, RESEARCH & EXPERIMENTAL | C-MYC | PROLIFERATION | LEVEL | BETA-CATENIN | CANCER | INVASION | ONCOLOGY | GROWTH | GENE-EXPRESSION | TUMOR-SUPPRESSOR | STROMAL CELLS | Receptor, Epidermal Growth Factor - genetics | Up-Regulation | Pancreatic Neoplasms - metabolism | Genes, Neoplasm | Genes, jun - genetics | Humans | Gene Expression Regulation, Neoplastic | Glucagon - genetics | Neoplasms, Glandular and Epithelial - metabolism | Gene Expression Profiling | Gene Regulatory Networks | Protein Interaction Maps | Proto-Oncogene Proteins c-fyn - genetics | Neoplasms, Glandular and Epithelial - genetics | Matrix Metalloproteinase 9 - metabolism | Proto-Oncogenes - genetics | Receptor, Epidermal Growth Factor - metabolism | Matrix Metalloproteinase 9 - genetics | Hyaluronan Receptors - metabolism | Neprilysin - genetics | Gonadotropin-Releasing Hormone - genetics | Matrix Metalloproteinase 2 - metabolism | Down-Regulation | Computational Biology | Pancreatic Neoplasms - genetics | Neprilysin - metabolism | Pancreas - metabolism | Transcription Factors - genetics | Hyaluronan Receptors - genetics | Proto-Oncogene Proteins c-myc - metabolism | Transcription Factors - metabolism | Matrix Metalloproteinase 2 - genetics | Gonadotropin-Releasing Hormone - metabolism | Glucagon - metabolism | Proto-Oncogene Proteins c-myc - genetics | Transcription factors | Pancreatic tumors | Genetic aspects | Diagnosis | Health aspects | Identification and classification | Computational biology | Ontology | Glucagon | Pathogenesis | c-Myc protein | Encyclopedias | Nervous system | Genomes | Myc protein | Matrix metalloproteinase | Data bases | Cell adhesion & migration | Proteins | Signal transduction | Epidermal growth factor | Annotations | CD44 antigen | Cell cycle | Metalloproteinase | Pancreas | Bioinformatics | Systems development | Fyn protein | Epidermal growth factor receptors | ErbB protein | Diabetes mellitus | c-Jun protein | Pituitary (anterior) | Gonadotropins | T cell receptors | Gene expression | Gelatinase B | Gelatinase A | Studies | Protein kinase | Endopeptidase | Software | Gonadotropin-releasing hormone | Mutation | Cancer | Tumors
Journal Article
Journal of Neurochemistry, ISSN 0022-3042, 07/2016, Volume 138, Issue 1, pp. 163 - 173
Journal Article
PLoS ONE, ISSN 1932-6203, 08/2012, Volume 7, Issue 8, p. e44583
Tissue-specific transcription factors are thought to cooperate with signaling pathways to promote patterned tissue specification, in part by co-regulating... 
HOMEOBOX-CONTAINING GENE | HUMAN PAX6 GENE | SINE-OCULIS | MORPHOGENETIC FURROW | MULTIDISCIPLINARY SCIENCES | EMBRYONIC STEM-CELLS | ECTOPIC EYE DEVELOPMENT | TWIN-OF-EYELESS | WING IMAGINAL DISC | PRONEURAL GENE | RETINAL DETERMINATION GENES | Wings, Animal - cytology | Receptors, Notch - metabolism | Hedgehog Proteins - metabolism | Molecular Sequence Data | Gene Expression Profiling | Drosophila Proteins - metabolism | RNA, Messenger - metabolism | Drosophila melanogaster - genetics | DNA-Binding Proteins - metabolism | Organ Specificity - genetics | Arthropod Antennae - metabolism | RNA Interference | Gene Expression Regulation, Developmental | Transcription, Genetic | Eye - metabolism | Amino Acid Sequence | RNA, Messenger - genetics | Drosophila melanogaster - cytology | Genome, Insect - genetics | Eye - cytology | Up-Regulation - genetics | Neprilysin - chemistry | Signal Transduction - genetics | Transcriptome - genetics | Drosophila Proteins - chemistry | Neprilysin - metabolism | Down-Regulation - genetics | Wings, Animal - metabolism | Animals | Drosophila melanogaster - growth & development | Drosophila Proteins - genetics | Photoreceptor Cells, Invertebrate - metabolism | DNA microarrays | DNA binding proteins | Comparative analysis | Gene expression | Genes | Drosophila | Cluster analysis | Transcription factors | Target recognition | Homology | Identification | Biology | Genomes | Kinases | Eye | Signal transduction | Pathways | Cell cycle | Metalloproteinase | Bioinformatics | RNA-mediated interference | Principal components analysis | Clustering | Ribonucleic acid--RNA | Pax6 protein | Signaling | Insects | Hedgehog protein | Stem cells | Regulation | Neprilysin | Notch protein | Alzheimers disease | Combinatorial analysis | RNA | Ribonucleic acid
Journal Article