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Journal Article
Glia, ISSN 0894-1491, 05/2007, Volume 55, Issue 7, pp. 746 - 757
Paranodal axo‐glial junctions are important for ion channel clustering and rapid action potential propagation in myelinated nerve fibers. Paranode formation... 
lipid raft | axo‐glial junction | autoimmune neuropathy | node of Ranvier | ganglioside | Node of Ranvier | Lipid raft | Autoimmune neuropathy | Axo-glial junction | Ganglioside | SYSTEM | NODES | RANVIER | axo-glial junction | GUILLAIN-BARRE-SYNDROME | GLIAL INTERACTIONS | NEUROSCIENCES | LACKING COMPLEX GANGLIOSIDES | PERIPHERAL-NERVE | AXON INITIAL SEGMENTS | BETA-IV-SPECTRIN | MICE | Membrane Microdomains - metabolism | Polyradiculoneuropathy - metabolism | Spinal Nerve Roots - metabolism | Ranvier's Nodes - metabolism | Membrane Microdomains - ultrastructure | Gangliosides - genetics | Potassium Channels - metabolism | Nerve Fibers, Myelinated - pathology | Gangliosides - metabolism | Sodium Channels - metabolism | Peripheral Nerves - metabolism | CASP8 and FADD-Like Apoptosis Regulating Protein - metabolism | Nerve Fibers, Myelinated - metabolism | Microscopy, Electron, Transmission | G(M1) Ganglioside - genetics | Neural Conduction - genetics | Axons - metabolism | Polyradiculoneuropathy - genetics | Schwann Cells - metabolism | Peripheral Nerves - pathology | Aging - pathology | Mice, Knockout | Peripheral Nerves - physiopathology | Animals | Ion Channels - metabolism | Polyradiculoneuropathy - physiopathology | G(M1) Ganglioside - analogs & derivatives | Mice | Ranvier's Nodes - pathology | Spinal Nerve Roots - pathology | Neurofilament Proteins - metabolism | G(M1) Ganglioside - metabolism | Aging - metabolism | Index Medicus
Journal Article
Nature Medicine, ISSN 1078-8956, 11/2015, Volume 21, Issue 11, pp. 1326 - 1331
Mechanical allodynia, induced by normally innocuous low-threshold mechanical stimulation, represents a cardinal feature of neuropathic pain. Blockade or... 
MEDICINE, RESEARCH & EXPERIMENTAL | FLAGELLIN | NOCICEPTORS | BIOCHEMISTRY & MOLECULAR BIOLOGY | TACTILE ALLODYNIA | INNATE | RATS | SPINAL-CORD | NERVE INJURY | CELL BIOLOGY | TOLL-LIKE RECEPTOR-5 | ITCH | PRIMARY SENSORY NEURONS | Lidocaine - pharmacology | Neuralgia - genetics | Capsaicin - pharmacology | Toll-Like Receptor 5 - genetics | Humans | Middle Aged | Neuralgia - metabolism | Male | Neurons - cytology | Ganglia, Spinal - cytology | Anesthetics, Local - pharmacology | Antineoplastic Agents - toxicity | Sensory System Agents - pharmacology | Flagellin - pharmacology | Paclitaxel - toxicity | Neuralgia - etiology | Nerve Fibers, Unmyelinated - drug effects | Adult | Female | Neurons - metabolism | Neurons - drug effects | Nerve Fibers, Myelinated - metabolism | Peripheral Nerve Injuries - complications | Hyperalgesia - metabolism | Nerve Fibers, Myelinated - drug effects | Mice, Knockout | Lidocaine - analogs & derivatives | Animals | Hyperalgesia - genetics | Hyperalgesia - etiology | Aged | Mice | Ganglia, Spinal - drug effects | Neurofilament Proteins - metabolism | Diabetic Neuropathies - complications | Ganglia, Spinal - metabolism | Genetically modified mice | Care and treatment | Pain | Dorsal root ganglia | Management | Research | TLR5 | Chronic pain | Pharmacology | Neurological disorders | Pain management | Immune system | Index Medicus
Journal Article
Neuron, ISSN 0896-6273, 01/2017, Volume 93, Issue 1, pp. 179 - 193
Journal Article
Journal Article