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Journal Article
Journal Article
PloS one, ISSN 1932-6203, 2012, Volume 7, Issue 11, p. e49023
Intrauterine infection and inflammation are major reasons for preterm birth. The switch from placenta-mediated to lung-mediated oxygen supply during birth is... 
INDUCED CELL-DEATH | ISCHEMIC TOLERANCE | MICROGLIAL TOXICITY | NITRIC-OXIDE SYNTHASE | MULTIDISCIPLINARY SCIENCES | PERIVENTRICULAR LEUKOMALACIA | DEVELOPING OLIGODENDROCYTES | NECROSIS-FACTOR-ALPHA | EXCITOTOXIC INJURY | INDUCED BRAIN-INJURY | BACTERIAL-ENDOTOXIN SENSITIZES | Inflammation - chemically induced | Inflammation - pathology | Microglia - metabolism | Oligodendroglia - metabolism | Rats, Wistar | Apoptosis - drug effects | Caspase 3 - metabolism | Brain - metabolism | Inflammation - metabolism | Oligodendroglia - drug effects | Nerve Fibers, Myelinated - pathology | Hyperoxia - metabolism | Microglia - pathology | Animals, Newborn | Nerve Fibers, Myelinated - metabolism | Leukoencephalopathies - pathology | Microglia - drug effects | Nerve Fibers, Myelinated - drug effects | Cells, Cultured | Rats | Leukoencephalopathies - metabolism | Rats, Sprague-Dawley | Brain - drug effects | Hyperoxia - pathology | Oligodendroglia - pathology | Animals | Lipopolysaccharides - pharmacology | Brain - pathology | Apoptosis - physiology | Immunohistochemistry | Brain | Cell death | Infants (Premature) | Inflammation | Research | Mitogens | Health aspects | Injuries | Neuroimaging | Neonates | Cell culture | Pediatrics | Transcription factors | Traumatic brain injury | Premature birth | Interleukin | Superoxide dismutase | Infants | Infections | Birth | Western blotting | Lipopolysaccharides | Head injuries | Newborn babies | Ischemia | Rodents | Oligodendrocytes | Oxygen | Pain perception | Cytokines | Maturation | Incubation | Mortality | Superoxide | Gene expression | Substantia alba | Microglia | Hospitals | Placenta | Magnetic resonance imaging | Lungs | Magnetic permeability | Hyperoxia | Interleukin 10 | Brain damage | Hypoxia | Oxygen tension | Brain injury | Apoptosis | pathology | Leukoencephalopathies | Sprague-Dawley | pharmacology | Caspase 3 | Cultured | Basic Medicine | chemically induced | Myelinated | physiology | drug effects | Cells | Newborn | Wistar | Medicinska grundvetenskaper | Oligodendroglia | metabolism | Nerve Fibers
Journal Article
Journal of Neuroscience Research, ISSN 0360-4012, 07/2017, Volume 95, Issue 7, pp. 1373 - 1390
Bidirectional interactions between neurons and myelinating glial cells result in formation of axonal domains along myelinated fibers... 
axonal degeneration | muscle atrophy | myelinated fibers | neuromuscular junction | nerve conduction | axonal domains | NODES | RANVIER | MARIE-TOOTH-DISEASE | MECHANISMS | INHERITED NEUROPATHIES | NEUROSCIENCES | ORGANIZATION | MAINTENANCE | NERVE CRUSH INJURY | CYTOSKELETAL SCAFFOLDING PROTEIN | GLIA INTERACTIONS | Membrane Proteins - genetics | Mice, Inbred C57BL | Muscular Atrophy - pathology | Male | Muscular Atrophy - genetics | Neural Conduction - physiology | Nerve Fibers, Myelinated - ultrastructure | Mice, 129 Strain | Mutation - genetics | Nerve Tissue Proteins - genetics | Neuromuscular Junction - ultrastructure | Cell Adhesion Molecules, Neuronal - genetics | Mice, Knockout | Animals | Nerve Fibers, Myelinated - pathology | Axons - pathology | Axons - ultrastructure | Female | Mice | Neuromuscular Junction - pathology | Medical colleges | Nervous system diseases | Electric properties | Conduction | Synaptogenesis | Disintegration | Disorders | Glial cells | Neuronal-glial interactions | Defects | Fibers | Atrophy | Nerve conduction | Reduction | Mitochondria | Demyelination | Contactin | Neurodegeneration | Degeneration | Neurodegenerative diseases | Demyelinating diseases | Mutants | Pathology | Axons | Properties (attributes) | Neuromuscular junctions | Axon guidance | Synapses | Denervation | Structure-function relationships | Integrity | Axonal Degeneration | Muscle Atrophy | Myelinated fibers | Neuromuscular Junction | Axonal Domains | Nerve Conduction
Journal Article
PloS one, ISSN 1932-6203, 2016, Volume 11, Issue 11, p. e0165637
Journal Article