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Nature Neuroscience, ISSN 1097-6256, 03/2007, Volume 10, Issue 3, pp. 321 - 330
Directed fusion of transmitter-laden vesicles enables rapid intercellular signaling in the central nervous system and occurs at synapses within gray matter.... 
ALPHA-LATROTOXIN | NMDA RECEPTORS | RAT CNS | GLIAL PROGENITOR CELLS | RHESUS-MONKEY | OLIGODENDROCYTE PRECURSOR CELLS | CORPUS-CALLOSUM | SYNAPTIC-TRANSMISSION | NG2 PROTEOGLYCAN | NEUROSCIENCES | ADULT | Excitatory Postsynaptic Potentials - radiation effects | Nerve Fibers, Unmyelinated - ultrastructure | Cerebral Cortex - cytology | Excitatory Postsynaptic Potentials - drug effects | Patch-Clamp Techniques - methods | Neuroglia - drug effects | Excitatory Postsynaptic Potentials - physiology | Neuroglia - cytology | Axons - ultrastructure | Macrolides - pharmacology | Microscopy, Immunoelectron - methods | Electric Stimulation - methods | Animals, Newborn | Receptor, Platelet-Derived Growth Factor alpha - metabolism | Vesicular Glutamate Transport Protein 1 - metabolism | Enzyme Inhibitors - pharmacology | Axons - metabolism | Proteoglycans - metabolism | Mice, Transgenic | Antigens - metabolism | Excitatory Amino Acid Antagonists - pharmacology | Neuroglia - radiation effects | Animals | Nerve Fibers, Unmyelinated - metabolism | Luminescent Proteins - genetics | Glutamic Acid - metabolism | Mice | In Vitro Techniques | Dose-Response Relationship, Radiation | Proteoglycans - genetics | Antigens - genetics | Luminescent Proteins - metabolism | Physiological aspects | Axons | Neural transmission | Research | Glutamate | Index Medicus
Journal Article
Pain, ISSN 0304-3959, 09/2017, Volume 158, Issue 9, pp. 1711 - 1722
Chronic pain and hyperalgesia, as well as pain resulting from episodes of vaso-occlusion, are characteristic features of sickle cell disease (SCD) and are... 
URB597 | Cannabinoid | Murine | FAAH inhibitor | Hyperalgesia | IN-SITU HYBRIDIZATION | ENDOGENOUS CANNABINOID ANANDAMIDE | BONE CANCER PAIN | NEUROBLASTOMA-CELLS | HIPPOCAMPAL-NEURONS | NEUROSCIENCES | CLINICAL NEUROLOGY | MURINE MODEL | CALCIUM-CHANNELS | PERIPHERAL MECHANISMS | MECHANICAL HYPERALGESIA | ANESTHESIOLOGY | PRIMARY SENSORY NEURONS | Anemia, Sickle Cell - pathology | Endocannabinoids - metabolism | Pain Threshold - physiology | Pyrazoles - therapeutic use | Hydrolysis - drug effects | Humans | Male | Receptor, Cannabinoid, CB2 - antagonists & inhibitors | Receptor, Cannabinoid, CB2 - genetics | Arachidonic Acids - metabolism | Benzamides - therapeutic use | Nerve Fibers, Unmyelinated - drug effects | Pain Threshold - drug effects | Morpholines - therapeutic use | Disease Models, Animal | Hemoglobins - metabolism | Hemoglobins - genetics | Mice, Transgenic | Polyunsaturated Alkamides - metabolism | Enzyme Inhibitors - therapeutic use | Hyperalgesia - physiopathology | Evoked Potentials - drug effects | Nerve Fibers, Unmyelinated - pathology | Receptor, Cannabinoid, CB2 - metabolism | Animals | Evoked Potentials - genetics | Hyperalgesia - drug therapy | Nerve Fibers, Unmyelinated - metabolism | Anemia, Sickle Cell - metabolism | Indoles - therapeutic use | Mice | Nociceptors - metabolism | Receptor, Cannabinoid, CB1 - antagonists & inhibitors | Carbamates - therapeutic use | Nociceptors - drug effects | Anemia, Sickle Cell - genetics | Index Medicus
Journal Article
BMC Neurology, ISSN 1471-2377, 09/2017, Volume 17, Issue 1, pp. 184 - 184
Journal Article