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Neuron, ISSN 0896-6273, 2010, Volume 65, Issue 5, pp. 597 - 611
To investigate the role of microRNAs in regulating oligodendrocyte (OL) differentiation and myelination, we utilized transgenic mice in which microRNA... 
DEVBIO | MOLNEURO | TRANSCRIPTION FACTORS | NERVOUS-SYSTEM | GLIAL PROGENITOR-CELL | PROTEIN | CYCLE EXIT | NEURONS | GROWTH-FACTOR | IDENTIFICATION | MICRORNA EXPRESSION | LINEAGE | NEUROSCIENCES | Central Nervous System - metabolism | MicroRNAs - metabolism | Green Fluorescent Proteins - genetics | S100 Proteins - genetics | Oligodendroglia - drug effects | Basic Helix-Loop-Helix Transcription Factors - metabolism | Animals, Newborn | Basic Helix-Loop-Helix Transcription Factors - genetics | Receptor, Platelet-Derived Growth Factor alpha - metabolism | S100 Calcium Binding Protein beta Subunit | Oligonucleotide Array Sequence Analysis - methods | Rats | Mice, Transgenic | Oligodendrocyte Transcription Factor 2 | Myelin Proteins - genetics | Rats, Sprague-Dawley | Nerve Growth Factors - genetics | Mice | MicroRNAs - genetics | Myelin Proteins - metabolism | SOXD Transcription Factors - genetics | Optic Nerve - metabolism | Age Factors | SOXD Transcription Factors - metabolism | Gene Expression Regulation, Developmental - genetics | Sciatic Nerve - growth & development | Myelin Sheath - metabolism | Central Nervous System - growth & development | DNA-Binding Proteins - metabolism | Sciatic Nerve - metabolism | Oligodendroglia - physiology | Transfection | DEAD-box RNA Helicases - metabolism | Cell Differentiation - physiology | Optic Nerve - growth & development | Brain - cytology | 2',3'-Cyclic-Nucleotide Phosphodiesterases - genetics | Ribonuclease III - genetics | Ribonuclease III - metabolism | Mice, Inbred C57BL | Cells, Cultured | Gene Expression Profiling - methods | Transcription Factors - genetics | 2',3'-Cyclic-Nucleotide Phosphodiesterases - metabolism | DNA-Binding Proteins - genetics | Nerve Tissue Proteins - genetics | Nerve Tissue Proteins - metabolism | Transcription Factors - metabolism | DEAD-box RNA Helicases - genetics | Animals | Cell Differentiation - drug effects | Receptor, Platelet-Derived Growth Factor alpha - genetics | Stem Cells - drug effects | Stem Cells - physiology | Proteins | Genotype & phenotype | Gene expression | Experiments | Rodents | Cell cycle
Journal Article
Nature, ISSN 0028-0836, 07/2010, Volume 466, Issue 7302, pp. 133 - 137
The question of whether tumorigenic cancer stem cells exist in human melanomas has arisen in the last few years(1). Here we show that in melanomas, tumour stem... 
PROGENITORS | PROSPECTIVE IDENTIFICATION | MULTIDISCIPLINARY SCIENCES | CANCER STEM-CELLS | Bone and Bones - pathology | Neoplasm Transplantation | Neoplastic Stem Cells - cytology | Humans | Nerve Tissue Proteins - deficiency | Neural Crest - pathology | Receptors, Nerve Growth Factor - metabolism | Neoplasm Proteins - metabolism | DNA-Binding Proteins - deficiency | Neural Crest - metabolism | Neoplasm Metastasis | Lung Neoplasms - secondary | Neoplastic Stem Cells - metabolism | Antigens, Neoplasm - metabolism | Neoplastic Stem Cells - pathology | Skin Transplantation | Antigens, Neoplasm - analysis | Skin - pathology | Melanoma - metabolism | Melanoma-Specific Antigens | Neural Crest - cytology | Melanoma - pathology | DNA-Binding Proteins - genetics | Nerve Tissue Proteins - genetics | Neoplastic Stem Cells - transplantation | Receptors, Nerve Growth Factor - genetics | Mice, Knockout | Nerve Tissue Proteins - metabolism | Animals | Bone Transplantation | Neoplasm Proteins - analysis | Transplantation, Heterologous - pathology | Mice | Receptors, Nerve Growth Factor - deficiency | Care and treatment | Melanoma | Stem cells | Physiological aspects | Development and progression | Nerve growth factor | Genetic aspects | Growth factor receptors | Research | Risk factors | Transplants & implants | Skin & tissue grafts | Cancer | Tumors | Human | Enrichment | Antigens | Tumours | Bones | Transplantation
Journal Article
ONCOGENE, ISSN 0950-9232, 06/2012, Volume 31, Issue 26, pp. 3190 - 3201
Epithelial-mesenchymal transition (EMT) is a crucial event in wound healing, tissue repair and cancer progression in adult tissues. We have recently shown that... 
delta EF1 | METASTASIS | alternative splicing | BIOCHEMISTRY & MOLECULAR BIOLOGY | breast cancer | NETWORKS | INDUCTION | ESRP | CELL-LINES | EMT | CELL BIOLOGY | BREAST-CANCER | TGF-beta | GROWTH-FACTOR-BETA | ONCOLOGY | GENETICS & HEREDITY | RECEPTORS | EXPRESSION | PROGRESSION | INSIGHTS | RNA-Binding Proteins - genetics | Homeodomain Proteins - metabolism | Humans | Epithelial-Mesenchymal Transition - drug effects | Epithelial-Mesenchymal Transition - genetics | Receptors, Fibroblast Growth Factor - genetics | Gene Expression Regulation, Neoplastic - drug effects | Cadherins - genetics | Gene Expression Regulation, Neoplastic - genetics | Transcription Factors - genetics | Down-Regulation - drug effects | Nerve Tissue Proteins - genetics | Disease Progression | Down-Regulation - genetics | Homeodomain Proteins - genetics | Transcription Factors - metabolism | Transforming Growth Factor beta - pharmacology | Phenotype | Alternative Splicing - drug effects | Animals | Breast Neoplasms - pathology | Cell Line, Tumor | Mice | RNA-Binding Proteins - metabolism | Protein Isoforms - genetics | Zinc Finger E-box-Binding Homeobox 1 | Alternative splicing | Wound healing | Transcription | Exons | Transforming growth factor | Breast cancer | regulatory proteins | Transforming growth factor- beta | Tumor cell lines | Promoters | Fibroblast growth factor receptors | Tumors | δEF1 | TGF-β | Original
Journal Article
Journal of Clinical Investigation, ISSN 0021-9738, 05/2009, Volume 119, Issue 5, pp. 1298 - 1311
Idiopathic pulmonary fibrosis (IPF) can lead to the development of secondary pulmonary hypertension (PH) and ultimately death. Despite this known association,... 
MEDICINE, RESEARCH & EXPERIMENTAL | EPITHELIUM-DERIVED FACTOR | GROWTH-FACTOR-BETA | ANGIOGENESIS | ARTERIAL-HYPERTENSION | DISEASE | SMOOTH-MUSCLE-CELLS | PROLIFERATION | GENE-TRANSFER | EXPRESSION | PIGMENT-EPITHELIUM | Genetic Therapy | Idiopathic Pulmonary Fibrosis - therapy | Apoptosis - drug effects | Gene Expression - genetics | Humans | Transforming Growth Factor beta1 - metabolism | Caspase 3 - metabolism | Hypertension, Pulmonary - physiopathology | Microvessels - pathology | Idiopathic Pulmonary Fibrosis - chemically induced | Nerve Growth Factors - metabolism | Vascular Endothelial Growth Factor A - metabolism | Hypertension, Pulmonary - therapy | Idiopathic Pulmonary Fibrosis - metabolism | Vascular Endothelial Growth Factor Receptor-2 - genetics | Pulmonary Artery - metabolism | Idiopathic Pulmonary Fibrosis - complications | Vascular Endothelial Growth Factor A - pharmacology | Transforming Growth Factor beta1 - pharmacology | Disease Models, Animal | Fibroblasts - metabolism | Serpins - genetics | Endothelial Cells - metabolism | Smad2 Protein - metabolism | Rats | Lung - physiopathology | Nitric Oxide Synthase Type III - genetics | Rats, Sprague-Dawley | Eye Proteins - metabolism | Models, Biological | Nerve Growth Factors - genetics | Fibroblasts - drug effects | Idiopathic Pulmonary Fibrosis - pathology | Bronchoalveolar Lavage Fluid - chemistry | Hypertension, Pulmonary - etiology | Serum Albumin - metabolism | Phosphorylation | Female | Lung - metabolism | Eye Proteins - genetics | Lung - pathology | Serpins - metabolism | Vascular Endothelial Growth Factor A - therapeutic use | Microvessels - drug effects | Vascular Endothelial Growth Factor Receptor-2 - metabolism | Transforming Growth Factor beta1 - genetics | Animals | Endothelial Cells - cytology | Pulmonary Artery - pathology | Endothelial Cells - drug effects | Immunohistochemistry | Usage | Care and treatment | Genetic aspects | Research | Health aspects | Vascular endothelial growth factor | Pulmonary hypertension | Risk factors | Apoptosis | Index Medicus | Abridged Index Medicus
Journal Article
PLoS ONE, ISSN 1932-6203, 05/2014, Volume 9, Issue 5, p. e92596
Background: Large-scale genomic analyses of patient cohorts have revealed extensive heterogeneity between individual tumors, contributing to treatment failure... 
CANCER-CELLS | METASTATIC MELANOMA | FACTOR-BETA | MULTIDISCIPLINARY SCIENCES | TRANSCRIPTION | EPIGENETIC REGULATION | NEURAL CREST | SOX10 | PROMOTES | TUMOR MICROENVIRONMENT | CD133 | Meta-Analysis as Topic | Antigens, CD - biosynthesis | Humans | Peptides - genetics | Male | Antigens, CD - genetics | Gene Knockdown Techniques | Neoplastic Stem Cells - metabolism | Melanoma - genetics | Nerve Tissue Proteins - biosynthesis | Neoplastic Stem Cells - pathology | Female | Neoplasm Proteins - genetics | SOXE Transcription Factors - biosynthesis | Glycoproteins - genetics | Melanoma - metabolism | Receptors, Nerve Growth Factor - biosynthesis | Neoplasm Proteins - biosynthesis | Melanoma - pathology | AC133 Antigen | Nerve Tissue Proteins - genetics | Receptors, Nerve Growth Factor - genetics | Glycoproteins - biosynthesis | Animals | Cell Line, Tumor | Mice, Inbred NOD | Biomarkers, Tumor - genetics | Mice | Biomarkers, Tumor - biosynthesis | SOXE Transcription Factors - genetics | Nerve growth factor | Drug resistance | Analysis | Genomics | Stem cells | Transcription factors | Laboratories | Genes | Genomes | Metastasis | Cell surface | Metastases | Genotype & phenotype | Heterogeneity | Rodents | Fibroblasts | Growth factors | Antigens | Colonies | Markers | Melanoma | Cell division | Tumorigenicity | Gene expression | Neural crest | Sox10 protein | Pathology | Properties (attributes) | Cell lines | Epigenetics | Neural stem cells | Surface markers | Cancer | Tumors
Journal Article
Journal Article