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Neuron (Cambridge, Mass.), ISSN 0896-6273, 03/2010, Volume 65, Issue 5, pp. 597 - 611
To investigate the role of microRNAs in regulating oligodendrocyte (OL) differentiation and myelination, we utilized transgenic mice in which microRNA... 
DEVBIO | MOLNEURO | Neurosciences | Neurosciences & Neurology | Life Sciences & Biomedicine | Science & Technology | Central Nervous System - metabolism | MicroRNAs - metabolism | Green Fluorescent Proteins - genetics | S100 Proteins - genetics | Oligodendroglia - drug effects | Basic Helix-Loop-Helix Transcription Factors - metabolism | Animals, Newborn | Basic Helix-Loop-Helix Transcription Factors - genetics | Receptor, Platelet-Derived Growth Factor alpha - metabolism | S100 Calcium Binding Protein beta Subunit | Oligonucleotide Array Sequence Analysis - methods | Rats | Mice, Transgenic | Oligodendrocyte Transcription Factor 2 | Myelin Proteins - genetics | Rats, Sprague-Dawley | Nerve Growth Factors - genetics | Mice | MicroRNAs - genetics | Myelin Proteins - metabolism | SOXD Transcription Factors - genetics | Optic Nerve - metabolism | Age Factors | SOXD Transcription Factors - metabolism | Gene Expression Regulation, Developmental - genetics | Sciatic Nerve - growth & development | Myelin Sheath - metabolism | Central Nervous System - growth & development | DNA-Binding Proteins - metabolism | Sciatic Nerve - metabolism | Oligodendroglia - physiology | Transfection | DEAD-box RNA Helicases - metabolism | Cell Differentiation - physiology | Optic Nerve - growth & development | Brain - cytology | 2',3'-Cyclic-Nucleotide Phosphodiesterases - genetics | Ribonuclease III - genetics | Ribonuclease III - metabolism | Mice, Inbred C57BL | Cells, Cultured | Gene Expression Profiling - methods | Transcription Factors - genetics | 2',3'-Cyclic-Nucleotide Phosphodiesterases - metabolism | DNA-Binding Proteins - genetics | Nerve Tissue Proteins - genetics | Nerve Tissue Proteins - metabolism | Transcription Factors - metabolism | DEAD-box RNA Helicases - genetics | Animals | Cell Differentiation - drug effects | Receptor, Platelet-Derived Growth Factor alpha - genetics | Stem Cells - drug effects | Stem Cells - physiology | Proteins | Genotype & phenotype | Gene expression | Experiments | Rodents | Cell cycle | Index Medicus
Journal Article
Cancer immunology, immunotherapy, ISSN 1432-0851, 11/2008, Volume 58, Issue 7, pp. 1033 - 1045
Lenalidomide (Revlimid®; CC-5013) and pomalidomide (CC-4047) are IMiDs® proprietary drugs having immunomodulatory properties that have both shown activity in... 
Immunomodulatory drugs | Biomedicine | Immunology | Lenalidomide | T regulatory cells | Cancer Research | Oncology | Pomalidomide | IMiDs | Life Sciences & Biomedicine | Science & Technology | Forkhead Transcription Factors - immunology | Receptors, Nerve Growth Factor - immunology | Receptors, Transforming Growth Factor beta - immunology | Receptors, Nerve Growth Factor - drug effects | Humans | Receptors, Nerve Growth Factor - metabolism | Thalidomide - pharmacology | Colonic Neoplasms - metabolism | Receptors, OX40 - immunology | T-Lymphocytes, Regulatory - immunology | Thalidomide - analogs & derivatives | Receptors, OX40 - metabolism | Transforming Growth Factor beta - drug effects | Colonic Neoplasms - immunology | Forkhead Transcription Factors - metabolism | Interleukin-10 - metabolism | Female | Antineoplastic Agents - pharmacology | Forkhead Transcription Factors - antagonists & inhibitors | Immunosuppressive Agents - pharmacology | Receptors, Tumor Necrosis Factor - metabolism | Transforming Growth Factor beta - immunology | Glucocorticoid-Induced TNFR-Related Protein | T-Lymphocytes, Regulatory - drug effects | Animals | Receptors, Transforming Growth Factor beta - drug effects | Receptors, Transforming Growth Factor beta - metabolism | Cell Line, Tumor | Mice | Mice, Inbred BALB C | Receptors, Tumor Necrosis Factor - immunology | Interleukin-10 - immunology | Transforming Growth Factor beta - metabolism | Receptors, Tumor Necrosis Factor - drug effects | Receptors, OX40 - antagonists & inhibitors | Care and treatment | Dexamethasone | Multiple myeloma | Angiogenesis inhibitors | Transforming growth factors | T cells | Cancer | Index Medicus
Journal Article
Journal Article
Nature (London), ISSN 1476-4687, 07/2010, Volume 466, Issue 7302, pp. 133 - 137
...Cancers derive by clonal progression to appear as abnormal growths. At diagnosis, they can be at a stage ranging from low risk of metastasis and probable cure... 
Science & Technology - Other Topics | Multidisciplinary Sciences | Science & Technology | Bone and Bones - pathology | Neoplasm Transplantation | Neoplastic Stem Cells - cytology | Humans | Nerve Tissue Proteins - deficiency | Neural Crest - pathology | Receptors, Nerve Growth Factor - metabolism | Neoplasm Proteins - metabolism | DNA-Binding Proteins - deficiency | Neural Crest - metabolism | Neoplasm Metastasis | Lung Neoplasms - secondary | Neoplastic Stem Cells - metabolism | Antigens, Neoplasm - metabolism | Neoplastic Stem Cells - pathology | Skin Transplantation | Antigens, Neoplasm - analysis | Skin - pathology | Melanoma - metabolism | Melanoma-Specific Antigens | Neural Crest - cytology | Melanoma - pathology | DNA-Binding Proteins - genetics | Nerve Tissue Proteins - genetics | Neoplastic Stem Cells - transplantation | Receptors, Nerve Growth Factor - genetics | Mice, Knockout | Nerve Tissue Proteins - metabolism | Animals | Bone Transplantation | Neoplasm Proteins - analysis | Transplantation, Heterologous - pathology | Mice | Receptors, Nerve Growth Factor - deficiency | Care and treatment | Melanoma | Stem cells | Physiological aspects | Development and progression | Nerve growth factor | Genetic aspects | Growth factor receptors | Research | Risk factors | Transplants & implants | Skin & tissue grafts | Cancer | Tumors | Human | Enrichment | Antigens | Tumours | Bones | Transplantation | Index Medicus
Journal Article
PloS one, ISSN 1932-6203, 04/2014, Volume 9, Issue 4, pp. e94287 - e94287
.... Because much of early AD pathophysiology includes hippocampal abnormalities, a viable treatment strategy might be to use trophic factors that support hippocampal integrity and function... 
Science & Technology - Other Topics | Multidisciplinary Sciences | Science & Technology | Alzheimer Disease - complications | Growth Differentiation Factor 2 - metabolism | Microtubule-Associated Proteins - metabolism | Humans | Gliosis - physiopathology | Nerve Growth Factors - metabolism | Glial Fibrillary Acidic Protein - metabolism | Neurogenesis | Choline O-Acetyltransferase - metabolism | Alzheimer Disease - pathology | Gliosis - pathology | Amyloid beta-Peptides - metabolism | Hippocampus - enzymology | Amyloidosis - complications | Disease Models, Animal | Biomarkers - metabolism | Alzheimer Disease - physiopathology | Amyloidosis - pathology | Plaque, Amyloid - pathology | Cholinergic Neurons - metabolism | Activin Receptors, Type I - metabolism | Amyloidosis - physiopathology | Mice, Transgenic | Neuropeptides - metabolism | Hippocampus - pathology | Gliosis - complications | Insulin-Like Growth Factor II - metabolism | Plaque, Amyloid - complications | Hippocampus - metabolism | Animals | Activin Receptors, Type II | Plaque, Amyloid - physiopathology | Insulin-Like Growth Factor II - administration & dosage | Alzheimer Disease - metabolism | Mice | Cholinergic Neurons - pathology | Care and treatment | Physiological aspects | Fluorescence | Nerve growth factor | Acetylcholine | Amyloidosis | Diagnosis | Health aspects | Risk factors | Brain | Neuropathology | Insulin-like growth factor I | Memory | Insulin-like growth factors | Choline O-acetyltransferase | Neurotrophin 3 | Proteins | Infusion | Consolidation | Transgenic animals | Rodents | Doublecortin protein | Amyloid | Acetyltransferase | Insulin-like growth factor II | Trophic factors | Alzheimer's disease | Plaques | Neurodegenerative diseases | Neurons | Abnormalities | Gene expression | Insulin | Medicine | Pathology | Brain-derived neurotrophic factor | Presenilin 1 | Choline | Green fluorescent protein | Mutation | Laboratory animals | Bone morphogenetic protein 9 | Hippocampus | Index Medicus
Journal Article
Human molecular genetics, ISSN 0964-6906, 12/2014, Volume 23, Issue 23, pp. 6177 - 6190
.... We report on a transgenic mouse with impaired transforming growth factor beta (TGF beta)-signalling in forebrain-derived neural cells using a Foxg1-cre knock-in to drive the conditional knock-out of the... 
Biochemistry & Molecular Biology | Genetics & Heredity | Life Sciences & Biomedicine | Science & Technology | Cerebral Hemorrhage - metabolism | Neurons - pathology | Receptors, Transforming Growth Factor beta - genetics | Humans | Secretory Pathway | Vascular Endothelial Growth Factor A - metabolism | Vascular Endothelial Growth Factor A - secretion | Brain - metabolism | Brain - blood supply | Forkhead Transcription Factors - metabolism | Pericytes - pathology | Human Umbilical Vein Endothelial Cells - cytology | Telencephalon - pathology | Fibroblast Growth Factor 2 - metabolism | Transforming Growth Factor beta - secretion | Neurons - secretion | Neurons - metabolism | Protein-Serine-Threonine Kinases - metabolism | Telencephalon - metabolism | Pericytes - metabolism | Protein-Serine-Threonine Kinases - genetics | Mice, Transgenic | Forkhead Transcription Factors - genetics | Human Umbilical Vein Endothelial Cells - secretion | Nerve Tissue Proteins - genetics | Neural Stem Cells - pathology | Blood-Brain Barrier - metabolism | Nerve Tissue Proteins - metabolism | Telencephalon - blood supply | Culture Media, Conditioned | Animals | Receptors, Transforming Growth Factor beta - metabolism | Brain - pathology | Neural Stem Cells - secretion | Mice | Cerebral Hemorrhage - pathology | Neural Stem Cells - metabolism | Transforming Growth Factor beta - metabolism | Insulin-Like Growth Factor I - metabolism | Neovascularization, Physiologic | Cell Movement | Index Medicus
Journal Article