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PloS one, ISSN 1932-6203, 2017, Volume 12, Issue 7, p. e0177962
Adult neural crest stem-derived cells (NCSC) are of extraordinary high plasticity and promising candidates for use in regenerative medicine. Several locations... 
PROGENITOR CELLS | MESENCHYMAL STROMAL CELLS | STEM-CELLS | TRUNK | MULTIDISCIPLINARY SCIENCES | PERIODONTAL-LIGAMENT | ONTOGENY | GENERATION | SKIN-DERIVED PRECURSORS | DIFFERENTIATION | MELANOCYTES | RNA-Binding Proteins - genetics | Humans | Adipose Tissue - cytology | Melanocytes - metabolism | Neurons - cytology | Receptors, Nerve Growth Factor - metabolism | Neural Stem Cells - cytology | Schwann Cells - cytology | Neural Crest - metabolism | Adipose Tissue - metabolism | Mesenchymal Stromal Cells - cytology | Snail Family Transcription Factors - genetics | Melanocytes - cytology | Nestin - genetics | Adult | Female | Cell Differentiation | Neurons - metabolism | Dermis - cytology | Nuclear Proteins - genetics | Biomarkers - metabolism | SOX9 Transcription Factor - metabolism | Gene Expression | Neural Crest - cytology | Dermis - metabolism | Snail Family Transcription Factors - metabolism | Microinjections | Bone Marrow Cells - cytology | Neural Crest - growth & development | Mesenchymal Stromal Cells - metabolism | Nuclear Proteins - metabolism | Schwann Cells - metabolism | Transcription Factor Brn-3A - metabolism | Chick Embryo | Nerve Tissue Proteins - genetics | Receptors, Nerve Growth Factor - genetics | Nerve Tissue Proteins - metabolism | Nestin - metabolism | Animals | Transcription Factor Brn-3A - genetics | Twist-Related Protein 1 - genetics | Twist-Related Protein 1 - metabolism | Neural Stem Cells - metabolism | SOX9 Transcription Factor - genetics | Bone Marrow Cells - metabolism | Mesenchymal Stem Cell Transplantation | RNA-Binding Proteins - metabolism | Adipose tissues | Comparative analysis | Skin | Neurosciences | Nestin | Adipose tissue | Leukocyte migration | Laboratories | Sox9 protein | Dermis | Melanocytes | Nervous system | Human tissues | Regeneration (physiology) | Cell adhesion & migration | Msi1 protein | Plasticity (neural) | Ethics | Immunology | Pathways | Surgery | Bone marrow | Hair | Hematology | Neurons | Tissue engineering | Schwann cells | Brn-3 protein | Gene expression | Embryos | Neural crest | Medicine | Regeneration | Human performance | Stromal cells | Stem cells | Cell migration | Dental pulp
Journal Article
Neuron (Cambridge, Mass.), ISSN 0896-6273, 2010, Volume 65, Issue 5, pp. 597 - 611
To investigate the role of microRNAs in regulating oligodendrocyte (OL) differentiation and myelination, we utilized transgenic mice in which microRNA... 
DEVBIO | MOLNEURO | TRANSCRIPTION FACTORS | NERVOUS-SYSTEM | GLIAL PROGENITOR-CELL | PROTEIN | CYCLE EXIT | NEURONS | GROWTH-FACTOR | IDENTIFICATION | MICRORNA EXPRESSION | LINEAGE | NEUROSCIENCES | Central Nervous System - metabolism | MicroRNAs - metabolism | Green Fluorescent Proteins - genetics | S100 Proteins - genetics | Oligodendroglia - drug effects | Basic Helix-Loop-Helix Transcription Factors - metabolism | Animals, Newborn | Basic Helix-Loop-Helix Transcription Factors - genetics | Receptor, Platelet-Derived Growth Factor alpha - metabolism | S100 Calcium Binding Protein beta Subunit | Oligonucleotide Array Sequence Analysis - methods | Rats | Mice, Transgenic | Oligodendrocyte Transcription Factor 2 | Myelin Proteins - genetics | Rats, Sprague-Dawley | Nerve Growth Factors - genetics | Mice | MicroRNAs - genetics | Myelin Proteins - metabolism | SOXD Transcription Factors - genetics | Optic Nerve - metabolism | Age Factors | SOXD Transcription Factors - metabolism | Gene Expression Regulation, Developmental - genetics | Sciatic Nerve - growth & development | Myelin Sheath - metabolism | Central Nervous System - growth & development | DNA-Binding Proteins - metabolism | Sciatic Nerve - metabolism | Oligodendroglia - physiology | Transfection | DEAD-box RNA Helicases - metabolism | Cell Differentiation - physiology | Optic Nerve - growth & development | Brain - cytology | 2',3'-Cyclic-Nucleotide Phosphodiesterases - genetics | Ribonuclease III - genetics | Ribonuclease III - metabolism | Mice, Inbred C57BL | Cells, Cultured | Gene Expression Profiling - methods | Transcription Factors - genetics | 2',3'-Cyclic-Nucleotide Phosphodiesterases - metabolism | DNA-Binding Proteins - genetics | Nerve Tissue Proteins - genetics | Nerve Tissue Proteins - metabolism | Transcription Factors - metabolism | DEAD-box RNA Helicases - genetics | Animals | Cell Differentiation - drug effects | Receptor, Platelet-Derived Growth Factor alpha - genetics | Stem Cells - drug effects | Stem Cells - physiology | Proteins | Genotype & phenotype | Gene expression | Experiments | Rodents | Cell cycle
Journal Article
The Journal of clinical investigation, ISSN 0021-9738, 2009, Volume 119, Issue 5, pp. 1298 - 1311
.... Using a rat model of IPF, we explored the role of the proangiogenic and antiapoptotic growth factor VEGF in the vascular remodeling that underlies PH... 
MEDICINE, RESEARCH & EXPERIMENTAL | EPITHELIUM-DERIVED FACTOR | GROWTH-FACTOR-BETA | RECEPTOR | PROLIFERATION | GENE-TRANSFER | CELL APOPTOSIS | EXPRESSION | BINDING | PIGMENT-EPITHELIUM | Genetic Therapy | Idiopathic Pulmonary Fibrosis - therapy | Apoptosis - drug effects | Gene Expression - genetics | Humans | Transforming Growth Factor beta1 - metabolism | Caspase 3 - metabolism | Hypertension, Pulmonary - physiopathology | Microvessels - pathology | Idiopathic Pulmonary Fibrosis - chemically induced | Nerve Growth Factors - metabolism | Vascular Endothelial Growth Factor A - metabolism | Hypertension, Pulmonary - therapy | Idiopathic Pulmonary Fibrosis - metabolism | Vascular Endothelial Growth Factor Receptor-2 - genetics | Pulmonary Artery - metabolism | Idiopathic Pulmonary Fibrosis - complications | Vascular Endothelial Growth Factor A - pharmacology | Transforming Growth Factor beta1 - pharmacology | Disease Models, Animal | Fibroblasts - metabolism | Serpins - genetics | Endothelial Cells - metabolism | Smad2 Protein - metabolism | Rats | Lung - physiopathology | Nitric Oxide Synthase Type III - genetics | Rats, Sprague-Dawley | Eye Proteins - metabolism | Models, Biological | Nerve Growth Factors - genetics | Fibroblasts - drug effects | Idiopathic Pulmonary Fibrosis - pathology | Bronchoalveolar Lavage Fluid - chemistry | Hypertension, Pulmonary - etiology | Serum Albumin - metabolism | Phosphorylation | Female | Lung - metabolism | Eye Proteins - genetics | Lung - pathology | Serpins - metabolism | Vascular Endothelial Growth Factor A - therapeutic use | Microvessels - drug effects | Vascular Endothelial Growth Factor Receptor-2 - metabolism | Transforming Growth Factor beta1 - genetics | Animals | Endothelial Cells - cytology | Pulmonary Artery - pathology | Endothelial Cells - drug effects | Immunohistochemistry | Usage | Care and treatment | Genetic aspects | Research | Health aspects | Vascular endothelial growth factor | Pulmonary hypertension | Risk factors | Apoptosis | Index Medicus | Abridged Index Medicus
Journal Article
The Journal of clinical investigation, ISSN 0021-9738, 2012, Volume 122, Issue 7, pp. 2454 - 2468
In inflammatory CNS conditions such as multiple sclerosis (MS), current options to treat clinical relapse are limited, and more selective agents are needed.... 
MEDICINE, RESEARCH & EXPERIMENTAL | EXPERIMENTAL AUTOIMMUNE ENCEPHALOMYELITIS | MULTIPLE-SCLEROSIS | NEURITE OUTGROWTH | NITRIC-OXIDE SYNTHASE | EXPERIMENTAL ALLERGIC ENCEPHALOMYELITIS | IN-VIVO | CENTRAL-NERVOUS-SYSTEM | VASCULAR-PERMEABILITY | ENDOTHELIAL GROWTH-FACTOR | HUMAN GLIOBLASTOMA-MULTIFORME | Membrane Glycoproteins - metabolism | Humans | Male | Vascular Endothelial Growth Factor A - metabolism | Vascular Endothelial Growth Factor A - genetics | Occludin | Brain - metabolism | Inflammation - metabolism | Nitric Oxide Synthase Type III - metabolism | Demyelinating Diseases | Multiple Sclerosis - metabolism | Interleukin-1beta - physiology | Cytokines - metabolism | Mice, Inbred C57BL | Cells, Cultured | Gene Expression Regulation | Vascular Endothelial Growth Factor Receptor-2 - metabolism | Mice, Transgenic | Nuclear Proteins - metabolism | Permeability | Blood-Brain Barrier - metabolism | Membrane Glycoproteins - genetics | Membrane Proteins | Nerve Tissue Proteins - metabolism | Blood-Brain Barrier - pathology | Lymphocytes - pathology | Animals | Brain - pathology | Multiple Sclerosis - pathology | Mice | Primary Cell Culture | Vascular Endothelial Growth Factor A - physiology | Astrocytes - metabolism | Physiological aspects | Central nervous system diseases | Research | Blood-brain barrier | Vascular endothelial growth factor
Journal Article
Cancer immunology, immunotherapy, ISSN 1432-0851, 2008, Volume 58, Issue 7, pp. 1033 - 1045
Lenalidomide (Revlimid®; CC-5013) and pomalidomide (CC-4047) are IMiDs® proprietary drugs having immunomodulatory properties that have both shown activity in... 
Immunomodulatory drugs | Biomedicine | Immunology | Lenalidomide | T regulatory cells | Cancer Research | Oncology | Pomalidomide | IMiDs | MULTIPLE-MYELOMA | DENILEUKIN DIFTITOX | THERAPEUTIC IMPLICATIONS | IMiDs (R) | PERIPHERAL-BLOOD | IMMUNOLOGY | TUMOR-INFILTRATING LYMPHOCYTES | CANCER-PATIENTS | ONCOLOGY | IN-VIVO | THALIDOMIDE ANALOG | ANTITUMOR IMMUNITY | Forkhead Transcription Factors - immunology | Receptors, Nerve Growth Factor - immunology | Receptors, Transforming Growth Factor beta - immunology | Receptors, Nerve Growth Factor - drug effects | Humans | Receptors, Nerve Growth Factor - metabolism | Thalidomide - pharmacology | Colonic Neoplasms - metabolism | Receptors, OX40 - immunology | T-Lymphocytes, Regulatory - immunology | Thalidomide - analogs & derivatives | Receptors, OX40 - metabolism | Transforming Growth Factor beta - drug effects | Colonic Neoplasms - immunology | Forkhead Transcription Factors - metabolism | Interleukin-10 - metabolism | Female | Antineoplastic Agents - pharmacology | Forkhead Transcription Factors - antagonists & inhibitors | Immunosuppressive Agents - pharmacology | Receptors, Tumor Necrosis Factor - metabolism | Transforming Growth Factor beta - immunology | Glucocorticoid-Induced TNFR-Related Protein | T-Lymphocytes, Regulatory - drug effects | Animals | Receptors, Transforming Growth Factor beta - drug effects | Receptors, Transforming Growth Factor beta - metabolism | Cell Line, Tumor | Mice | Mice, Inbred BALB C | Receptors, Tumor Necrosis Factor - immunology | Interleukin-10 - immunology | Transforming Growth Factor beta - metabolism | Receptors, Tumor Necrosis Factor - drug effects | Receptors, OX40 - antagonists & inhibitors | Care and treatment | Dexamethasone | Multiple myeloma | Angiogenesis inhibitors | Transforming growth factors | T cells | Cancer
Journal Article
Neuron, ISSN 0896-6273, 02/2013, Volume 77, Issue 3, pp. 472 - 484
... have focused on the mechanisms governing its development, particularly on the role of transcription factors in CTA and CSA specification and on the function of subpall... 
MUTANT MICE | CORTICAL AXONS | THALAMOCORTICAL AXONS | GROWTH | SEMAPHORIN 3E | CENTRAL-NERVOUS-SYSTEM | GUIDANCE | CHICK HINDLIMB | CAJAL-RETZIUS CELLS | NEUROSCIENCES | CEREBRAL-CORTEX | Thyroid Nuclear Factor 1 | Age Factors | Embryo, Mammalian | Leukocyte L1 Antigen Complex - metabolism | Gene Expression Regulation, Developmental - genetics | Axons - physiology | Cerebral Cortex - cytology | Neural Pathways - physiology | DNA-Binding Proteins - metabolism | POU Domain Factors - genetics | tau Proteins - genetics | Thalamus - physiology | Contactin 2 - metabolism | Repressor Proteins - metabolism | Glycoproteins - genetics | Tumor Suppressor Proteins - metabolism | Wnt3A Protein - genetics | Membrane Proteins - genetics | Mice, Inbred C57BL | Mice, Transgenic | Nuclear Proteins - metabolism | Transcription Factors - genetics | Nerve Tissue Proteins - genetics | Homeodomain Proteins - genetics | Membrane Glycoproteins - genetics | Nerve Tissue Proteins - metabolism | S100 Calcium Binding Protein G - metabolism | Transcription Factors - metabolism | Animals | Calbindin 2 | Thalamus - cytology | Cerebral Cortex - physiology | Luminescent Proteins - genetics | Mice | Body Patterning - genetics | Luminescent Proteins - metabolism | Developmental biology | Neurons | Studies | Laboratories | Experiments | Repressor Proteins | Cerebral Cortex | Cellular Biology | Neural Pathways | tau Proteins | Life Sciences | Contactin 2 | Gene Expression Regulation, Developmental | Body Patterning | Thalamus | Membrane Glycoproteins | Luminescent Proteins | DNA-Binding Proteins | POU Domain Factors | Calcium-Binding Protein, Vitamin D-Dependent | Glycoproteins | Nerve Tissue Proteins | Nuclear Proteins | Membrane Proteins | Axons | Homeodomain Proteins | Leukocyte L1 Antigen Complex | Transcription Factors | Wnt3A Protein | Tumor Suppressor Proteins | reciprocal connections | handshake | waiting period | PlexinD1 | axon guidance | Sema3E | thalamocortical | corticothalamic
Journal Article
PLoS ONE, ISSN 1932-6203, 04/2014, Volume 9, Issue 4, p. e94287
.... Because much of early AD pathophysiology includes hippocampal abnormalities, a viable treatment strategy might be to use trophic factors that support hippocampal integrity and function... 
APP TRANSGENIC MICE | BASAL FOREBRAIN NEURONS | MEMORY DEFICITS | MULTIDISCIPLINARY SCIENCES | ACETYLCHOLINE-RELEASE | COGNITIVE IMPAIRMENT | APPSWE/PS1DE9 MOUSE MODEL | KINASE 1 ALK1 | RAT SEPTAL NEURONS | GROWTH-FACTOR-I | IMPAIRED NEUROGENESIS | Alzheimer Disease - complications | Growth Differentiation Factor 2 - metabolism | Microtubule-Associated Proteins - metabolism | Humans | Gliosis - physiopathology | Nerve Growth Factors - metabolism | Glial Fibrillary Acidic Protein - metabolism | Neurogenesis | Choline O-Acetyltransferase - metabolism | Alzheimer Disease - pathology | Gliosis - pathology | Amyloid beta-Peptides - metabolism | Hippocampus - enzymology | Amyloidosis - complications | Disease Models, Animal | Biomarkers - metabolism | Alzheimer Disease - physiopathology | Amyloidosis - pathology | Plaque, Amyloid - pathology | Cholinergic Neurons - metabolism | Activin Receptors, Type I - metabolism | Amyloidosis - physiopathology | Mice, Transgenic | Neuropeptides - metabolism | Hippocampus - pathology | Gliosis - complications | Insulin-Like Growth Factor II - metabolism | Plaque, Amyloid - complications | Hippocampus - metabolism | Animals | Activin Receptors, Type II | Plaque, Amyloid - physiopathology | Insulin-Like Growth Factor II - administration & dosage | Alzheimer Disease - metabolism | Mice | Cholinergic Neurons - pathology | Care and treatment | Physiological aspects | Fluorescence | Nerve growth factor | Acetylcholine | Amyloidosis | Diagnosis | Health aspects | Risk factors | Brain | Neuropathology | Insulin-like growth factor I | Memory | Reversing | Insulin-like growth factors | Choline O-acetyltransferase | Neurotrophin 3 | Proteins | Infusion | Consolidation | Transgenic animals | Rodents | Doublecortin protein | Amyloid | Acetyltransferase | Insulin-like growth factor II | Trophic factors | Alzheimer's disease | Plaques | Neurodegenerative diseases | Neurons | Abnormalities | Gene expression | Insulin | Medicine | Pathology | Brain-derived neurotrophic factor | Presenilin 1 | Choline | Green fluorescent protein | Mutation | Laboratory animals | Bone morphogenetic protein 9 | Hippocampus
Journal Article
American Journal of Physiology: Cell Physiology, ISSN 1522-1563, 2009, Volume 296, Issue 6, pp. C1248 - C1257
.... Moreover, recent results confirm that other transforming growth factor-β (TGF-β) members control muscle mass... 
Myostatin | Muscle atrophy | Akt | Hypertrophy | MYOBLAST DIFFERENTIATION | PHYSIOLOGY | hypertrophy | UBIQUITIN LIGASES | myostatin | CELL BIOLOGY | SKELETAL-MUSCLE | PATHWAY | GROWTH | ATROPHY INVOLVE | MICE | muscle atrophy | EXPRESSION | Phosphorylation | Receptors, Transforming Growth Factor beta - genetics | Age Factors | Muscle Denervation | Male | Muscle, Skeletal - innervation | Muscle, Skeletal - metabolism | Smad3 Protein - metabolism | Proto-Oncogene Proteins c-akt - genetics | Tripartite Motif Proteins | Muscle Development | Muscular Atrophy - physiopathology | Transfection | RNA Interference | Muscle Proteins - metabolism | Cell Differentiation | Muscular Atrophy - prevention & control | Myostatin - metabolism | Proto-Oncogene Proteins c-akt - metabolism | Protein-Serine-Threonine Kinases - metabolism | Disease Models, Animal | Muscular Atrophy - metabolism | Signal Transduction | Muscular Atrophy - pathology | Cells, Cultured | Protein-Serine-Threonine Kinases - genetics | Smad2 Protein - metabolism | Ubiquitin-Protein Ligases - metabolism | Mice, Transgenic | Phosphotransferases (Alcohol Group Acceptor) - metabolism | Animals | Carrier Proteins - metabolism | Receptors, Transforming Growth Factor beta - metabolism | Sciatic Nerve - surgery | Muscle, Skeletal - physiopathology | Mice | TOR Serine-Threonine Kinases | Muscle, Skeletal - pathology | Mutation | Transforming Growth Factor beta - metabolism | RNA, Small Interfering - metabolism
Journal Article
Nature (London), ISSN 1476-4687, 2010, Volume 466, Issue 7302, pp. 133 - 137
...Cancers derive by clonal progression to appear as abnormal growths. At diagnosis, they can be at a stage ranging from low risk of metastasis and probable cure... 
PROGENITORS | PROSPECTIVE IDENTIFICATION | MULTIDISCIPLINARY SCIENCES | CANCER STEM-CELLS | Bone and Bones - pathology | Neoplasm Transplantation | Neoplastic Stem Cells - cytology | Humans | Nerve Tissue Proteins - deficiency | Neural Crest - pathology | Receptors, Nerve Growth Factor - metabolism | Neoplasm Proteins - metabolism | DNA-Binding Proteins - deficiency | Neural Crest - metabolism | Neoplasm Metastasis | Lung Neoplasms - secondary | Neoplastic Stem Cells - metabolism | Antigens, Neoplasm - metabolism | Neoplastic Stem Cells - pathology | Skin Transplantation | Antigens, Neoplasm - analysis | Skin - pathology | Melanoma - metabolism | Melanoma-Specific Antigens | Neural Crest - cytology | Melanoma - pathology | DNA-Binding Proteins - genetics | Nerve Tissue Proteins - genetics | Neoplastic Stem Cells - transplantation | Receptors, Nerve Growth Factor - genetics | Mice, Knockout | Nerve Tissue Proteins - metabolism | Animals | Bone Transplantation | Neoplasm Proteins - analysis | Transplantation, Heterologous - pathology | Mice | Receptors, Nerve Growth Factor - deficiency | Care and treatment | Melanoma | Stem cells | Physiological aspects | Development and progression | Nerve growth factor | Genetic aspects | Growth factor receptors | Research | Risk factors | Transplants & implants | Skin & tissue grafts | Cancer | Tumors | Human | Enrichment | Antigens | Tumours | Bones | Transplantation
Journal Article