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Neuron, ISSN 0896-6273, 02/2012, Volume 73, Issue 4, pp. 729 - 742
Following damage to peripheral nerves, a remarkable process of clearance and regeneration takes place. Axons downstream of the injury degenerate, while the... 
SCIATIC-NERVE | DEMYELINATION | MYELINATION | HUMAN CONNEXIN32 | MOUSE MODEL | MONOCYTE CHEMOATTRACTANT PROTEIN-1 | INJURY | ENSHEATHMENT | NEUREGULIN-1 | NEUROSCIENCES | NF1 LOSS | Cyclin D1 - metabolism | Leukocytes - pathology | MAP Kinase Signaling System - physiology | Diphenylamine - pharmacology | Receptor, Nerve Growth Factor - genetics | Recovery of Function - drug effects | Motor Activity - drug effects | Estrogen Antagonists - pharmacology | Male | Microscopy, Immunoelectron | Myelin Sheath - metabolism | Diphenylamine - analogs & derivatives | MAP Kinase Signaling System - genetics | Peripheral Nerve Injuries - physiopathology | T-Lymphocytes - metabolism | Time Factors | Reaction Time - drug effects | Schwann Cells - physiology | Benzamides - pharmacology | T-Lymphocytes - pathology | Neutrophils - metabolism | Receptor, Nerve Growth Factor - metabolism | Neutrophils - pathology | Animals, Newborn | Nerve Regeneration - genetics | Microscopy, Electron, Transmission | Recovery of Function - genetics | Receptors, Estrogen - genetics | Cytokines - metabolism | Proto-Oncogene Proteins c-raf - genetics | Gene Expression Regulation - genetics | Mice, Inbred C57BL | Peripheral Nerve Injuries - pathology | Mice, Transgenic | Reaction Time - genetics | Proto-Oncogene Proteins c-raf - metabolism | Gene Expression Regulation - drug effects | Cell Movement - drug effects | Microscopy, Confocal | Motor Activity - genetics | Animals | MAP Kinase Signaling System - drug effects | Schwann Cells - ultrastructure | Mast Cells - pathology | Myelin Sheath - genetics | Nerve Regeneration - drug effects | Tamoxifen - pharmacology | Mice | Research | Analysis | Oncology, Experimental | Cells | Cancer | Proteins | Dehydrogenases | Rodents | Nervous system | Ligands | Kinases | Phosphatase | Vascular endothelial growth factor | Chemokines | Cell adhesion & migration | Tumors | Ataxia
Journal Article
Neuron, ISSN 0896-6273, 02/2014, Volume 81, Issue 3, pp. 561 - 573
The ability of injured axons to regenerate declines with age, yet the mechanisms that regulate axon regeneration in response to age are not known. Here we show... 
LIFE-SPAN | NERVOUS-SYSTEM | CYCLIC-AMP | NEURONAL REGENERATION | TISSUE-SPECIFIC ACTIVITIES | MAP KINASE PATHWAY | TUMOR-SUPPRESSOR | SPINAL-CORD-INJURY | C-ELEGANS | NEMATODE CAENORHABDITIS-ELEGANS | NEUROSCIENCES | Humans | Nerve Regeneration - physiology | Nerve Degeneration - physiopathology | Caenorhabditis elegans Proteins - metabolism | Nerve Degeneration - genetics | Green Fluorescent Proteins - genetics | Insulin-Like Growth Factor I - genetics | Time Factors | Immunosuppressive Agents - pharmacology | Disease Models, Animal | Nerve Regeneration - genetics | PTEN Phosphohydrolase - genetics | Animals, Genetically Modified | Gene Expression Regulation - genetics | PTEN Phosphohydrolase - metabolism | Phosphotransferases (Alcohol Group Acceptor) - genetics | Transcription Factors - genetics | Sirolimus - pharmacology | Nerve Degeneration - pathology | Phosphotransferases (Alcohol Group Acceptor) - metabolism | Transcription Factors - metabolism | Caenorhabditis elegans | Insulin - metabolism | Animals | Aging - physiology | Signal Transduction - drug effects | Signal Transduction - physiology | Caenorhabditis elegans Proteins - genetics | Forkhead Transcription Factors | Insulin-Like Growth Factor I - metabolism | Neurosciences | Medical colleges | Aged | Neurons | Proteins | Transcription factors | Young adults | Aging | Nematodes | Genomes | Genetic testing | Insulin | Deoxyribonucleic acid--DNA
Journal Article
Nature Medicine, ISSN 1078-8956, 03/2018, Volume 24, Issue 3, pp. 338 - 351
Deficits in Schwann cell-mediated remyelination impair functional restoration after nerve damage, contributing to peripheral neuropathies. The mechanisms... 
MEDICINE, RESEARCH & EXPERIMENTAL | NERVOUS-SYSTEM | ACTIVATION | BIOCHEMISTRY & MOLECULAR BIOLOGY | AXON REGENERATION | INJURY | CELL BIOLOGY | REPAIR | DISEASE | DIFFERENTIATION | SCHWANN-CELL DEVELOPMENT | INHERITED DEMYELINATING NEUROPATHIES | MOLECULAR-MECHANISMS | Sciatic Nerve - injuries | Humans | Peripheral Nerve Injuries - rehabilitation | E1A-Associated p300 Protein - genetics | Nerve Degeneration - physiopathology | Nerve Degeneration - genetics | Sciatic Nerve - growth & development | Myelin Sheath - metabolism | Sciatic Nerve - metabolism | Peripheral Nerve Injuries - physiopathology | Nerve Regeneration - genetics | Recovery of Function - genetics | Signal Transduction | Histone Deacetylases | Mice, Transgenic | Schwann Cells - pathology | Schwann Cells - metabolism | Transcription Factors - genetics | DNA-Binding Proteins - genetics | Muscle Proteins - genetics | Animals | Myelin Sheath - genetics | Genome | Peripheral Nerve Injuries - genetics | Remyelination - genetics | Regeneration | Nervous system | Genetic aspects | Enzyme inhibitors | Health aspects | Myelin proteins | Histone deacetylase | Chromatin | Myelin | Recovery of function | Homeostasis | Event-related potentials | AKT protein | Genomes | Histone acetyltransferase | Machinery | Neuromodulation | Modulators | 1-Phosphatidylinositol 3-kinase | Signaling | Myelination | Clonal deletion | Restoration | Nerves | Epigenetics | Deletion | Neuregulin | Mice | Inhibition
Journal Article
Proceedings of the National Academy of Sciences of the United States of America, ISSN 0027-8424, 5/2012, Volume 109, Issue 19, pp. 7517 - 7522
Axon regeneration in the central nervous system normally fails, in part because of a developmental decline in the intrinsic ability of CNS projection neurons... 
Axons | Regeneration | Spinal cord | Neurites | Neuroscience | Transcription factors | Growth promotion | Neurons | Central nervous system | Adults | Laser capture microdissection | Gene therapy | Spinal cord injury | Adeno associated virus | spinal cord injury | FUNCTIONAL RECOVERY | NERVE REGENERATION | CELLS | PROTEIN | adeno associated virus | MULTIDISCIPLINARY SCIENCES | gene therapy | laser capture microdissection | SENSORY NEURONS | IN-VIVO | GROWTH | GENE-EXPRESSION | FACTOR KLF7 | SPINAL-CORD-INJURY | Herpes Simplex Virus Protein Vmw65 - genetics | Immunohistochemistry | Neurites - physiology | Humans | Nerve Regeneration - physiology | Transcriptional Activation | Neurons - cytology | Axons - physiology | Green Fluorescent Proteins - genetics | Spinal Cord Injuries - genetics | Kruppel-Like Transcription Factors - metabolism | HEK293 Cells | Neurons - physiology | Pyramidal Tracts - metabolism | Female | Neurons - metabolism | Pyramidal Tracts - physiology | Nerve Regeneration - genetics | Green Fluorescent Proteins - metabolism | Gene Expression | Spinal Cord Injuries - metabolism | Mice, Inbred C57BL | Cells, Cultured | Axons - metabolism | Etoposide | Rats | Neurites - metabolism | Reverse Transcriptase Polymerase Chain Reaction | Luminescent Measurements - methods | Rats, Sprague-Dawley | Animals | Genetic Engineering | Mice | Spinal Cord Injuries - physiopathology | Mutation | Pyramidal Tracts - cytology | Kruppel-Like Transcription Factors - genetics | Physiological aspects | Genetic transcription | Research | Biological Sciences
Journal Article
Neuron, ISSN 0896-6273, 06/2010, Volume 66, Issue 5, pp. 663 - 670
Journal Article
Nature Neuroscience, ISSN 1097-6256, 05/2013, Volume 16, Issue 5, pp. 571 - 579
Journal Article
Neuron, ISSN 0896-6273, 2009, Volume 64, Issue 5, pp. 617 - 623
Axon regeneration failure accounts for permanent functional deficits following CNS injury in adult mammals. However, the underlying mechanisms remain elusive.... 
MOLNEURO | SIGNALING | SURVIVAL | RETINAL GANGLION-CELLS | SIGNALING PATHWAYS | STIMULATES AXON REGENERATION | GROWTH | LEUKEMIA INHIBITORY FACTOR | SPINAL-CORD-INJURY | NEUROSCIENCES | SENSORY NEURONS | ADULT | TRANSGENIC MICE | Nerve Regeneration - physiology | Green Fluorescent Proteins - genetics | Suppressor of Cytokine Signaling Proteins - deficiency | Retinal Ganglion Cells - pathology | Dose-Response Relationship, Drug | Tubulin - metabolism | Time Factors | Ciliary Neurotrophic Factor - genetics | Ciliary Neurotrophic Factor - pharmacology | Organ Culture Techniques | Disease Models, Animal | Optic Nerve Injuries - drug therapy | Animals, Newborn | Nerve Regeneration - genetics | Retinal Ganglion Cells - physiology | Cholera Toxin - metabolism | Gene Expression Regulation - genetics | Mice, Inbred C57BL | Optic Nerve Injuries - physiopathology | Axons - drug effects | Axons - metabolism | Mice, Transgenic | Cytokine Receptor gp130 - deficiency | Phosphotransferases (Alcohol Group Acceptor) - metabolism | Optic Nerve Injuries - genetics | Suppressor of Cytokine Signaling 3 Protein | Animals | Carrier Proteins - metabolism | Analysis of Variance | Axons - pathology | Nerve Regeneration - drug effects | Suppressor of Cytokine Signaling Proteins - physiology | Mice | TOR Serine-Threonine Kinases | Injections, Intraventricular - methods | Neurosciences | Ciliary neurotrophic factor | Peptide hormones | Neurons | Analysis | Proteins | Studies | Medical research | Optic nerve | Rodents | Mammals | Index Medicus
Journal Article
Journal Article