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The Journal of neuroscience, ISSN 1529-2401, 10/2012, Volume 32, Issue 40, pp. 13718 - 13728
The symptoms of Parkinson's disease (PD) are related to changes in the frequency and pattern of activity in the reciprocally connected GABAergic external... 
Neurosciences | Neurosciences & Neurology | Life Sciences & Biomedicine | Science & Technology | Dopaminergic Neurons - pathology | Nerve Tissue Proteins - analysis | Male | Synaptic Transmission | Isonicotinic Acids - pharmacology | Exploratory Behavior - physiology | RNA, Messenger - biosynthesis | Dopamine - physiology | Exploratory Behavior - drug effects | Nerve Tissue Proteins - biosynthesis | Dopaminergic Neurons - physiology | Globus Pallidus - physiopathology | Vesicular Inhibitory Amino Acid Transport Proteins - biosynthesis | Globus Pallidus - pathology | Receptors, GABA-A - biosynthesis | Parkinsonian Disorders - physiopathology | Subthalamic Nucleus - physiopathology | Membrane Proteins - genetics | Carrier Proteins - biosynthesis | Mice, Inbred C57BL | RNA, Messenger - genetics | Inhibitory Postsynaptic Potentials - physiology | Rats | Nerve Tissue Proteins - genetics | Rats, Sprague-Dawley | Carrier Proteins - genetics | Subthalamic Nucleus - pathology | Membrane Proteins - biosynthesis | Oxidopamine - toxicity | Patch-Clamp Techniques | Animals | Parkinsonian Disorders - pathology | Receptors, GABA-A - genetics | Vesicular Inhibitory Amino Acid Transport Proteins - genetics | Mice | Nerve Degeneration | Inhibitory Postsynaptic Potentials - drug effects | Index Medicus | Parkinsonian Disorders | Subthalamic Nucleus | Neurons and Cognition | Neurobiology | Receptors, GABA-A | Oxidopamine | Life Sciences | Inhibitory Postsynaptic Potentials | Vesicular Inhibitory Amino Acid Transport Proteins | Carrier Proteins | Exploratory Behavior | Dopamine | Globus Pallidus | Nerve Tissue Proteins | Membrane Proteins | Dopaminergic Neurons | RNA, Messenger | Isonicotinic Acids
Journal Article
Blood, ISSN 0006-4971, 01/2015, Volume 125, Issue 4, pp. 710 - 719
The precise mechanism for reduced thrombosis in prekallikrein null mice (Klkb1(-/-)) is unknown. Klkb1(-/-) mice have delayed carotid artery occlusion times on... 
Life Sciences & Biomedicine | Hematology | Science & Technology | Epoprostenol - genetics | Receptor, Bradykinin B2 - genetics | Receptors, G-Protein-Coupled - metabolism | Epoprostenol - biosynthesis | Peptide Fragments - pharmacology | Carotid Artery Thrombosis - metabolism | Pyrones - pharmacology | Sirtuin 1 - genetics | Thromboplastin - antagonists & inhibitors | Nerve Tissue Proteins - biosynthesis | Synaptotagmins - biosynthesis | Carotid Artery Thrombosis - chemically induced | Prekallikrein | Thromboplastin - biosynthesis | Proto-Oncogene Proteins - metabolism | Kruppel-Like Transcription Factors - biosynthesis | Proto-Oncogene Proteins - antagonists & inhibitors | Angiotensin II - pharmacology | Receptor, Bradykinin B2 - biosynthesis | Sirtuin 1 - biosynthesis | Proto-Oncogene Proteins - genetics | Imidazoles - pharmacology | Angiotensin II - analogs & derivatives | Sulfonamides - pharmacology | Nerve Tissue Proteins - genetics | Sirtuin 1 - antagonists & inhibitors | Mice, Knockout | Naphthalenes - pharmacology | Animals | Partial Thromboplastin Time | Thromboplastin - genetics | Receptors, G-Protein-Coupled - antagonists & inhibitors | Mice | Receptors, G-Protein-Coupled - genetics | Synaptotagmins - genetics | Carotid Artery Thrombosis - genetics | Carotid Artery Thrombosis - pathology | Kruppel-Like Transcription Factors - antagonists & inhibitors | Kruppel-Like Transcription Factors - genetics | RNA, Messenger | Index Medicus | Abridged Index Medicus | Thrombosis and Hemostasis
Journal Article
PLoS genetics, ISSN 1553-7390, 2015, Volume 11, Issue 12, pp. e1005634 - e1005634
Journal Article
The Journal of neuroscience, ISSN 1529-2401, 03/2006, Volume 26, Issue 11, pp. 3066 - 3076
The deep cerebellar nuclei (DCN) are the main output centers of the cerebellum, but little is known about their development. Using transcription factors as... 
reeler | Tbr2 | Pax6 | Tbr1 | Fastigial nucleus | Brn2 | Neurosciences | Neurosciences & Neurology | Life Sciences & Biomedicine | Science & Technology | Paired Box Transcription Factors - biosynthesis | Paired Box Transcription Factors - genetics | Cerebellar Nuclei - embryology | Axonal Transport | Cerebellar Nuclei - cytology | Extracellular Matrix Proteins - biosynthesis | Efferent Pathways - physiology | Red Nucleus - embryology | T-Box Domain Proteins - biosynthesis | Efferent Pathways - embryology | Neurons - cytology | Mice, Neurologic Mutants | Morphogenesis | Rhombencephalon - cytology | Cerebellar Nuclei - abnormalities | Nerve Tissue Proteins - biosynthesis | Serine Endopeptidases - genetics | Neurons - metabolism | Eye Proteins - genetics | Rhombencephalon - embryology | Homeodomain Proteins - biosynthesis | Serine Endopeptidases - biosynthesis | Extracellular Matrix Proteins - genetics | Mice, Inbred C57BL | Glutamic Acid - physiology | Repressor Proteins - genetics | Transcription Factors - biosynthesis | PAX6 Transcription Factor | Transcription Factors - genetics | DNA-Binding Proteins - genetics | Gestational Age | Nerve Tissue Proteins - genetics | T-Box Domain Proteins - genetics | Cell Adhesion Molecules, Neuronal - genetics | Homeodomain Proteins - genetics | Red Nucleus - cytology | Cell Lineage | Animals | Repressor Proteins - biosynthesis | Cell Adhesion Molecules, Neuronal - biosynthesis | Biomarkers | Rhombencephalon - metabolism | Mice | DNA-Binding Proteins - biosynthesis | Microscopy, Fluorescence | Cell Movement | Eye Proteins - biosynthesis | Index Medicus | fastigial nucleus
Journal Article
PloS one, ISSN 1932-6203, 03/2013, Volume 8, Issue 3, pp. e57208 - e57208
... disruption of tissue (primary injury), as well as through delayed and potentially reversible molecular and cellular pathophysiological mechanisms, which cause... 
Science & Technology - Other Topics | Multidisciplinary Sciences | Science & Technology | Nitric Oxide Synthase Type II - biosynthesis | I-kappa B Proteins - immunology | Cerebral Cortex - pathology | MAP Kinase Kinase 4 - immunology | NF-kappa B - metabolism | Brain Injuries - metabolism | Interleukin-1beta - genetics | Tyrosine - analogs & derivatives | Inflammation - metabolism | Tryptases - immunology | NF-KappaB Inhibitor alpha | Toll-Like Receptor 4 - genetics | Toll-Like Receptor 4 - immunology | Brain Injuries - genetics | Poly(ADP-ribose) Polymerases - biosynthesis | Tyrosine - immunology | Mice, Knockout | Tyrosine - metabolism | Cerebral Cortex - immunology | Tryptases - biosynthesis | MAP Kinase Kinase 4 - genetics | Mice | Poly (ADP-Ribose) Polymerase-1 | Poly(ADP-ribose) Polymerases - immunology | Toll-Like Receptor 4 - biosynthesis | NF-kappa B - immunology | Brain Infarction - metabolism | Brain Infarction - pathology | I-kappa B Proteins - metabolism | I-kappa B Proteins - genetics | Cerebral Cortex - metabolism | Glial Fibrillary Acidic Protein | MAP Kinase Kinase 4 - metabolism | Brain Injuries - immunology | Nitric Oxide Synthase Type II - immunology | Nerve Tissue Proteins - biosynthesis | Interleukin-1beta - biosynthesis | Tryptases - genetics | Nerve Tissue Proteins - immunology | Gene Expression Regulation - genetics | Gene Expression Regulation - immunology | Interleukin-1beta - immunology | Inflammation - immunology | Nerve Tissue Proteins - genetics | Nerve Tissue Proteins - metabolism | Animals | Poly(ADP-ribose) Polymerases - genetics | NF-kappa B - genetics | Nitric Oxide Synthase Type II - genetics | Brain Infarction - immunology | Inflammation - genetics | Brain Infarction - genetics | Brain Injuries - pathology | Tyrosine - genetics | Brain | Brain damage | Brain research | Neurons | Injuries | Neuroprotection | Animal models | Traumatic brain injury | Bax protein | Bcl-2 protein | Control methods | Kinases | Proteins | Signal transduction | Receptors | Head injuries | Animal tissues | Toll-like receptors | Immune system | Translocation | NF-κB protein | Glial fibrillary acidic protein | Cortex | Poly(ADP-ribose) polymerase | JNK protein | Inflammation | TLR4 protein | IL-1β | Trauma | Nitric-oxide synthase | Chymase | Augmentation | Signaling | Injury prevention | Tryptase | Nitrotyrosine | Apoptosis | Index Medicus
Journal Article
Cerebral cortex (New York, N.Y. 1991), ISSN 1460-2199, 01/2014, Volume 24, Issue 1, pp. 186 - 198
Journal Article
Nature (London), ISSN 1476-4687, 07/2010, Volume 466, Issue 7307, pp. 765 - 768
Chronic myelogenous leukaemia (CML) can progress from a slow growing chronic phase to an aggressive blast crisis phase, but the molecular basis of this... 
Science & Technology - Other Topics | Multidisciplinary Sciences | Science & Technology | RNA-Binding Proteins - genetics | Up-Regulation | Oncogene Proteins, Fusion - metabolism | Prognosis | Homeodomain Proteins - metabolism | Humans | Gene Expression Regulation, Neoplastic | Leukemia, Myelogenous, Chronic, BCR-ABL Positive - genetics | Nuclear Pore Complex Proteins - genetics | Cell Differentiation - genetics | RNA-Binding Proteins - biosynthesis | Leukemia, Myelogenous, Chronic, BCR-ABL Positive - pathology | Nerve Tissue Proteins - biosynthesis | Membrane Proteins - metabolism | Blast Crisis - pathology | Signal Transduction | Membrane Proteins - genetics | Nuclear Pore Complex Proteins - metabolism | Mice, Inbred C57BL | Tumor Suppressor Protein p53 - metabolism | Receptor, Notch1 - metabolism | Nerve Tissue Proteins - genetics | Disease Progression | Homeodomain Proteins - genetics | Nerve Tissue Proteins - metabolism | Fusion Proteins, bcr-abl - genetics | Membrane Proteins - biosynthesis | Animals | Oncogene Proteins, Fusion - genetics | Leukemia, Myelogenous, Chronic, BCR-ABL Positive - metabolism | Blast Crisis - genetics | Mice | Blast Crisis - metabolism | Fusion Proteins, bcr-abl - metabolism | RNA-Binding Proteins - metabolism | Myelocytic leukemia | Molecular genetics | Physiological aspects | Development and progression | Nonlymphoid leukemia | Cellular signal transduction | Genetic aspects | Research | Genetic regulation | Gene expression | Medical research | Stem cells | Chronic illnesses | Index Medicus
Journal Article
Nature neuroscience, ISSN 1546-1726, 03/2015, Volume 18, Issue 5, pp. 690 - 697
The developing mammalian brain is destined for a female phenotype unless exposed to gonadal hormones during a perinatal sensitive period. It has been assumed... 
Neurosciences | Neurosciences & Neurology | Life Sciences & Biomedicine | Science & Technology | DNA (Cytosine-5-)-Methyltransferases - physiology | Nerve Tissue Proteins - deficiency | Nerve Tissue Proteins - analysis | Testosterone - physiology | Cytidine - pharmacology | Male | Copulation - drug effects | Brain - growth & development | Cytidine - analogs & derivatives | DNA (Cytosine-5-)-Methyltransferases - antagonists & inhibitors | Preoptic Area - physiopathology | Testosterone - pharmacology | Phthalimides - pharmacology | DNA Methylation | Gene Expression Regulation, Developmental | Tryptophan - analogs & derivatives | Nerve Tissue Proteins - biosynthesis | Female | Disorders of Sex Development - physiopathology | Estradiol - physiology | Nerve Tissue Proteins - antagonists & inhibitors | Nerve Tissue Proteins - physiology | DNA (Cytosine-5-)-Methyltransferases - deficiency | Microfilament Proteins - analysis | Rats | Sex Characteristics | DNA, Intergenic - genetics | Disorders of Sex Development - genetics | Nerve Tissue Proteins - genetics | Rats, Sprague-Dawley | DNA (Cytosine-5-)-Methyltransferases - genetics | Sex Differentiation - physiology | Copulation - physiology | Animals | Protein Isoforms - biosynthesis | CpG Islands | Mice | Tryptophan - pharmacology | Preoptic Area - enzymology | Protein Isoforms - genetics | Enzymes | Hormones, Sex | Regulation | Gene expression | Properties | Health aspects | Index Medicus
Journal Article